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1 ogenes, Staphylococcus aureus and Salmonella enteritidis.
2 ribute to successful egg contamination by S. Enteritidis.
3 87/91 is a recently evolved descendent of S. Enteritidis.
4 VA) method for subtyping Salmonella serotype Enteritidis.
5 reaks caused by Salmonella enterica serotype Enteritidis.
6 n of outbreaks caused by Salmonella serotype Enteritidis.
7 with progeny response to S. enterica serovar Enteritidis.
8 t in the transmission of S. enterica serovar Enteritidis.
9 st OMPs isolated from S. Choleraesuis and S. Enteritidis.
10 -transferase-LpfA fusion protein of serotype Enteritidis.
11 ge type (PT) reference strains of Salmonella enteritidis.
12 methods for the epidemiological typing of S. enteritidis.
13 ous than the flagella observed on Salmonella enteritidis.
14 he chicken ovaries by invasive strains of S. enteritidis.
15 ied nonpasteurized liquid eggs containing S. enteritidis.
16 rapid, and powerful subtyping method for S. enteritidis.
17 detection and characterization method for S. Enteritidis.
18 of the isolates, 3 of which were Salmonella Enteritidis.
19 Typhimurium and 1608 (15.8%) were Salmonella Enteritidis.
20 d ecological origins of S. enterica serotype Enteritidis.
21 e development of a SNP typing scheme with S. Enteritidis.
22 aradox surrounds Salmonella enterica serovar Enteritidis.
23 ia monocytogenes and Salmonella enterica sv. Enteritidis.
24 did bacterial lipopolysaccharides Salmonella enteritidis (0.24 nmol/L, 5-fold) greater than Helicobac
26 inst challenge with S. typhimurium F98 or S. enteritidis 27A PT 8 in birds from vaccinated hens and i
27 f cross-immunizing infection with Salmonella Enteritidis; (3) an increase in the duration of infectio
28 mologous (Typhimurium F98) and heterologous (Enteritidis 4973 and S. enterica O6,14,24: e,h-monophasi
30 proportion than Typhimurium (6%), including Enteritidis (7%), Heidelberg (13%), Choleraesuis (57%),
34 ubsequent challenge with virulent Salmonella enteritidis a selection against lpf phase-on variants wa
35 l showing the highest binding affinity to S. enteritidis, a DNA sequence of high affinity to the bact
36 e phenotypic response to S. enterica serovar Enteritidis, an F1 population of chickens was created by
37 lates serotyped, 160 (45.6%) were Salmonella Enteritidis and 152 (43.3%) were Salmonella Typhimurium.
38 ng NTS isolates, >/=80% (79.7% of Salmonella Enteritidis and 90.2% of Salmonella Typhimurium isolates
39 is of a patient with disseminated Salmonella enteritidis and a homozygous splice acceptor mutation in
40 mprises a positive selection step against S. enteritidis and a negative selection step against a mixt
48 ng Dice similarities for S. enterica serovar Enteritidis and S. enterica serovar Typhimurium strains
49 enzyme combined PFGE for S. enterica serovar Enteritidis and S. enterica serovar Typhimurium strains,
51 rs have the capacity to inhibit growth of S. enteritidis and S. typhimurium in bacterial cultures; th
52 amers, twelve rounds of selection to live S. enteritidis and S. typhimurium were performed, alternati
54 rain M5a1, Salmonella eastbourne, Salmonella enteritidis and Salmonella gelsenkirchen, respectively.
56 ing methods for Salmonella enterica serotype Enteritidis and survey the population structure of commo
58 Nontyphoidal Salmonella enterica serovars Enteritidis and Typhimurium are a common cause of gastro
60 Salmonella enterica serovar Enteritidis (S. enteritidis) and closely related serovars, suggesting th
61 Salmonella enterica serotype Enteritidis (S. enteritidis) and Escherichia coli O157:H7, has generated
62 m, 10% (10) were Salmonella enterica serovar Enteritidis, and 3% (3) were Salmonella enterica serovar
63 tions indicated that clinical isolates of S. enteritidis are highly heterogeneous in their ability to
68 dual strains of Salmonella enterica serotype Enteritidis beyond the phenotypic level; however, a cons
69 mbinant His-tagged flagellin from Salmonella enteritidis bound to TLR5 in detergent lysates from COS-
70 ears and case fatality was 20.3%; Salmonella Enteritidis case fatality (27.8%) was higher than for ot
72 ella (NTS), mainly serotypes Typhimurium and Enteritidis, cause invasive infections with high mortali
77 t IgY from multiple animals infected with S. Enteritidis compared to those infected with S. Hadar.
78 Stanleyville (91% vaccine efficacy), and S. Enteritidis CVD 1944 protected mice against the group D
79 The vaccines S. Typhimurium CVD 1931 and S. Enteritidis CVD 1944 were immunogenic and protected BALB
80 t international travel by linking Salmonella Enteritidis data from the National Antimicrobial Resista
81 Typhimurium and Salmonella enterica serovar Enteritidis DeltaguaBA DeltaclpX live oral vaccines to p
82 xtensive analysis of clinical isolates of S. enteritidis, demonstrate the complex nature of Salmonell
83 hly selective and can successfully detect S. enteritidis down to 600 CFU mL(-1) (equivalent to 18 CFU
84 Salmonella enterica subsp. enterica serovar Enteritidis, drugs that are not appropriate for therapy
85 ction against lpf phase ON cells of serotype Enteritidis during a subsequent challenge, suggesting th
90 ofiles of Listeria monocytogenes, Salmonella enteritidis, Escherichia coli, during growth in the pres
91 gainst some food-borne pathogens (Salmonella enteritidis, Escherichia coli, Listeria monocytogenes an
92 e of related pathogens, including Salmonella enteritidis, Escherichia coli, Staphylococcus aureus, Ps
93 Clinical isolates of S. enterica serovar Enteritidis exhibit a wide spectrum of virulence in mice
96 ave analyzed the pathway by which Salmonella enteritidis flagellin (FliC) activates murine and human
98 cs of Salmonella BSI in Blantyre; Salmonella Enteritidis from 1999 to 2002, Salmonella Typhimurium fr
99 nome sequence analysis of 675 isolates of S. Enteritidis from 45 countries, we show the existence of
102 of diversity within 104 isolates of serotype Enteritidis from eight unaffiliated poultry farms in Eng
103 on the molecular fingerprinting of serotype Enteritidis from poultry environments in the United King
104 tal of 192 NTS isolates (114 Typhimurium, 78 Enteritidis) from blood and stools from pediatric admiss
106 To investigate whether changes in serovar Enteritidis gene content contributed to this increased p
107 dentified in the Salmonella enterica serovar Enteritidis genome that is predicted to encode a protein
108 phism (SNP)-based cluster analysis of the S. Enteritidis genomes revealed well supported clades, with
110 Overall, MLVA typing of Salmonella serotype Enteritidis had enhanced resolution, good reproducibilit
115 The lipopolysaccharide (LPS) of Salmonella enteritidis has been implicated as a virulence factor of
117 injected intramuscularly 2 weeks later with Enteritidis, (ii) hens were contact infected with Kentuc
118 contact infected with Kentucky and then with Enteritidis, (iii) hens were injected with Enteritidis o
122 sociated food poisoning caused by Salmonella enteritidis in eggs because the avirulent S. typhimurium
123 a survival advantage to S. enterica serovar Enteritidis in eggs by repairing DNA damage caused by eg
125 environments, which may ultimately assist S. Enteritidis in persistent and silent colonization of chi
127 uding two rare strains classified as serovar Enteritidis in the Salmonella reference collection B, on
128 ence that SEF14 fimbriae are expressed by S. enteritidis in vivo, previous studies showed that SEF14
129 The isolation rate of Salmonella serotype Enteritidis increased until 1996, whereas declines were
130 cted to evaluate whether NO production in S. Enteritidis-infected HD11 cells can be used as a biomark
132 demonstrate that NO-based screening using S. Enteritidis-infected HD11 cells is a viable tool to iden
134 e highly discriminatory for IgY following S. Enteritidis infection (p < 0.05) compared to infections
135 implications for Salmonella enterica serovar Enteritidis infection and transmission to eggs, along wi
136 We defined an outbreak-associated case of S. enteritidis infection as one in which S. enteritidis was
137 ure of ice cream associated with cases of S. enteritidis infection were compared with those of produc
141 acid-resistant Salmonella enterica serotype Enteritidis infections in the United States and recent i
143 ization after pathogenic S. enterica serovar Enteritidis inoculation and for circulating antibody lev
144 Results of this study demonstrated that S. enteritidis interacts with granulosa cells in a specific
145 and transmission electron micrographs of S. enteritidis invasion of granulosa cells showed organisms
146 Salmonella enterica subsp. enterica serovar Enteritidis is a common food-borne pathogen, often assoc
153 ogical characteristic of S. enterica serovar Enteritidis is its association with chicken shell eggs,
154 Salmonella enterica serovar Typhimurium, S. Enteritidis is known to have pathobiology specific to ch
157 le each year and among pathogens, Salmonella Enteritidis is most widely found bacteria causing food b
158 y in recent decades, and S. enterica serovar Enteritidis is now one of the leading serovars of Salmon
161 Salmonella enterica serovar Enteritidis (S. Enteritidis) is a major etiologic agent of nontyphoid sa
162 Salmonella enterica serotype Enteritidis (S. enteritidis) is a major food-borne pathogen, and its inc
163 Salmonella enterica serovars Typhimurium and Enteritidis, is responsible for a major global burden of
164 type element with homology to the Salmonella enteritidis IS1351 element and Yersinia enterocolitica I
165 we conducted a retrospective analysis of S. Enteritidis isolates from seven epidemiologically confir
166 The resultant phylogeny allocated most S. Enteritidis isolates into two distinct clades (clades I
168 al and animal origins of Salmonella serotype Enteritidis isolates may have a considerable influence o
172 la Typhimurium and 30% (24/79) of Salmonella Enteritidis isolates tested were found to be multidrug r
177 d the effects of Salmonella enterica serovar Enteritidis lipopolysaccharide (LPS) and recombinant Sal
178 core polysaccharide-OPS (COPS) of Salmonella Enteritidis lipopolysaccharide (LPS) to flagellin protei
179 the published level of glucosylation for S. enteritidis LPS as well as for S. enteritidis LPS purcha
180 ion for S. enteritidis LPS as well as for S. enteritidis LPS purchased from Sigma Chemical Co., the 1
181 N-gamma +/+ mice following injection with S. enteritidis LPS, despite sustaining 11-fold reductions i
184 kers for the response to S. enterica serovar Enteritidis may result in the enhancement of the immune
185 fferent hosts, including S. enterica serovar Enteritidis (multiple hosts), S. Gallinarum (birds), and
187 riae on colonization of mice with a serotype Enteritidis mutant in which the lpf promoter region was
188 f the 687 NTS isolates, including Salmonella Enteritidis (n = 244 [35.5%]), Salmonella Typhimurium (n
190 0), Typhimurium (n = 163), Newport (n = 93), Enteritidis (n = 45), Dublin (n = 25), Pullorum (n = 9),
191 (two outbreaks caused by Salmonella serotype Enteritidis, one by Norwalk-like virus), and food items
192 h Enteritidis, (iii) hens were injected with Enteritidis only, and (iv) hens were contact infected wi
194 ontaminated with Salmonella enterica serovar Enteritidis or Salmonella enterica serovar Typhimurium a
195 ety of strains, which suggests that serotype Enteritidis organisms representing different genomic gro
196 a single culture-proven foodborne Salmonella enteritidis outbreak in 1994, Salmonella-induced gastroe
197 of commonly encountered S. enterica serotype Enteritidis outbreak isolates in the United States.
198 a single culture-proven foodborne Salmonella enteritidis outbreak that involved 1811 patients (mostly
199 erefore, more information is needed about S. Enteritidis pathobiology in comparison to that of S. Typ
201 monella enterica subspecies enterica serovar Enteritidis phage type (PT) 4, which peaked in 1993.
202 arge outbreak of Salmonella enterica serovar Enteritidis phage type 14b affecting more than 30 patien
203 ection B, only eleven regions of the serovar Enteritidis phage type 4 (PT4) chromosome (sequenced at
205 determine the relationships between some S. Enteritidis phage types (PTs) commonly associated with f
206 confer protection against challenge with S. enteritidis, presumably because lpf phase-off variants w
207 falling since 1997, and levels of Salmonella Enteritidis PT4 have fallen to preepidemic levels and ha
208 host-promiscuous Salmonella enterica serovar Enteritidis PT4 isolate P125109 and a chicken-restricted
209 roarray comparisons of the sequenced serovar Enteritidis PT4 to isolates of the closely related serov
210 Whereas CVD 1931 did not protect against S. Enteritidis R11, CVD 1944 did mediate protection against
211 wild-type strains S. Typhimurium I77 and S. Enteritidis R11, respectively, were constructed by delet
214 sruption of this gene in S. enterica serovar Enteritidis rendered the organism more susceptible to eg
218 re only found in Salmonella enterica serovar Enteritidis (S. enteritidis) and closely related serovar
219 hogens, such as Salmonella enterica serotype Enteritidis (S. enteritidis) and Escherichia coli O157:H
222 ent protein from Salmonella enterica serovar Enteritidis, S. enterica serovar Typhimurium, and Pseudo
223 genes of Salmonella typhimurium, Salmonella enteritidis, Salmonella arizonae, Shigella sonnei, and S
224 O157:H7, Salmonella typhimurium, Salmonella enteritidis, Salmonella arizonae, Shigella sonnei, Shige
225 uginosa, Pseudomonas fluorescens, Salmonella Enteritidis, Salmonella Typhimurium, Escherichia coli).
226 onsin, reported Salmonella enterica serotype enteritidis (SE) infections during 1997 more than double
230 mbria genes were replaced both in Salmonella enteritidis (sefA, agfA and fimC) and Escherichia coli (
231 ctivity against Escherichia coli, Salmonella enteritidis, Staphylococcus aureus, and Mycobacterium bo
232 chicken macrophage HD11 cells, while dead S. Enteritidis stimulates a high level of NO production, su
233 osomal tlpA gene rendered a virulent serovar Enteritidis strain defective in intracellular survival a
234 for strain differentiation of more clonal S. Enteritidis strains and provides core genotypic markers
236 -old and nineteen 10- to 20-year-old serovar Enteritidis strains from various hosts, using a Salmonel
239 Typhimurium and Salmonella enterica serovar Enteritidis strains that can serve as live oral vaccines
240 the subtyping of Salmonella enterica serovar Enteritidis strains to an epidemiologically relevant lev
243 obal epidemic clade and two new clades of S. Enteritidis that are geographically restricted to distin
244 sed subtyping scheme for S. enterica serovar Enteritidis that relies on a single combined cluster ana
245 n a whole-gene scale were needed for serovar Enteritidis to become more prevalent in domestic fowl.
251 ggest that a bivalent (S. Typhimurium and S. Enteritidis) vaccine would provide broad protection agai
253 e used transposon mutagenesis to identify S. Enteritidis virulence genes by assay of invasiveness in
255 S. enteritidis infection as one in which S. enteritidis was cultured from a person who became ill in
259 an previously known, as a second clade of S. Enteritidis was revealed that is distinct from those com
261 e characteristics of clinical isolates of S. enteritidis, we determined the 50% lethal doses (LD(50))
262 he RNI/ROI resistance of S. enterica serovar Enteritidis, we transformed a genomic DNA library of SE2
264 used to examine a panel of 29 isolates of S. enteritidis which had been previously characterized by o
265 serovar Typhimurium and S. enterica serovar Enteritidis, which are usually linked to localized gastr
266 I as most concordant for S. enterica serovar Enteritidis, while XbaI, BlnI, and SpeI were most concor
267 tified a gene, yafD from S. enterica serovar Enteritidis, whose overexpression conferred upon S. ente
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