ライフサイエンスコーパス: enteritis

1 ases of CMV disease (one pneumonitis and one enteritis).

2 Clostridium difficile toxin A (TxA)-induced enteritis.

3 ctivators are proinflammatory in TxA-induced enteritis.

4 e are markedly resistant to lethal radiation enteritis.

5 and one because of rejection after rotavirus enteritis.

6 inclusion body hepatitis, splenomegaly, and enteritis.

7 the pathogenesis of Clostridium perfringens enteritis.

8 with antibiotic treatment of E coli O157:H7 enteritis.

9 reas one dog was euthanized on day 17 due to enteritis.

10 elopment of S. flexneri-induced inflammatory enteritis.

11 vidence of inflammation in a rabbit model of enteritis.

12 n shown to be necessary for the induction of enteritis.

13 veloped bile duct and liver disease, but not enteritis.

14 of cattle results in a chronic granulomatous enteritis.

15 n and the pathogenesis of C. jejuni-mediated enteritis.

16 infiltration associated with toxin A-induced enteritis.

17 nflammatory cell influx, fluid secretion and enteritis.

18 to neutrophil mucosal influx during toxin A enteritis.

19 ulence feature underlying Salmonella-induced enteritis.

20 ought to be essential in the pathogenesis of enteritis.

21 nce of IBS 3 months or more after infectious enteritis.

22 isolated enterectomy due to cytomegalovirus enteritis.

23 1 and SN2/SNAT5 by mast cells during chronic enteritis.

24 distress and users of antibiotics during the enteritis.

25 ith a well-established diagnosis of regional enteritis.

26 ress, Paneth cell impairment and spontaneous enteritis.

27 ell into two distinct groups, bacteremia and enteritis.

28 enic fever with symptoms of mucositis and/or enteritis.

29 worldwide leading cause of bacterial-induced enteritis.

30 rs for, and outcomes of IBS after infectious enteritis.

31 Two patients developed cytomegalovirus enteritis.

32 reptomycin to result in gut-restricted acute enteritis.

33 ammation and enterocyte apoptosis in toxin A enteritis.

34 among random Campylobacter isolates causing enteritis, 275 enteritis-associated isolates, randomly c

35 among random Campylobacter isolates causing enteritis, 275 random enteritis-associated isolates of C

36 neumonia (69.2%), meningoencephalitis (50%), enteritis (46.2%), colitis (38.5%), syndrome (42.3%), vi

37 ree patients (FCL: 125) coincided with viral enteritis, 51 samples from 21 patients (FCL: 207) coinci

38 n (13.3% and 11.8%, respectively), norovirus enteritis (8.2% and 3%), cytomegalovirus disease or coli

39 flammation without rejection (group D: acute enteritis, 9; Helicobacter pylori, 1; Streptococcal phar

40 r role in the pathogenesis of avian necrotic enteritis, a disease that has emerged due to the removal

41 moderate, and severe rejections, nonspecific enteritis), although there was sufficient overlap to pro

42 n in individuals who did not have infectious enteritis, although there was heterogeneity among studie

43 reen 325 patients for inflammatory bacterial enteritis and a negative predictive value of 99.4% when

44 ARs can ameliorate C. difficile TcdA-induced enteritis and alter the outcome of C. difficile infectio

45 the duodenum was associated with nonspecific enteritis and CD8(+) T-cell activation.

46 A(2B)ARs mediate C. difficile toxin-induced enteritis and disease.

47 ch cause enteritis necroticans in humans and enteritis and enterotoxaemias of domestic animals, typic

48 Clostridium perfringens type B causes enteritis and enterotoxemia in domestic animals.

49 teritis necroticans in humans or necrotizing enteritis and enterotoxemia in domestic animals.

50 mportant toxins for C. perfringens diseases (enteritis and enterotoxemia) originating in the gastroin

51 uxiliary virulence factor for C. perfringens enteritis and enterotoxemia.

52 C isolates cause both haemorrhagic necrotic enteritis and fatal enterotoxemias (where toxins produce

53 cells in the lamina propria and eosinophilic enteritis and fibrosis in the small intestine.

54 ous complications included a cytomegalovirus enteritis and four fungal infections (related to central

55 ed isolates from patients with uncomplicated enteritis and GBS, as well as isolates from animal sourc

56 obally distributed cause of human food-borne enteritis and has been linked to chronic joint and neuro

57 bacter jejuni is an important cause of human enteritis and has been linked to the development of auto

58 Escherichia coli O157:H7 causes severe enteritis and hemolytic-uremic syndrome, mostly in young

59 cells on a BALB/c background succumbed with enteritis and hepatitis.

60 ithelial cells (IECs) results in spontaneous enteritis and increased susceptibility to induced coliti

61 Campylobacter fetus is a cause of enteritis and invasive extraintestinal disease in humans

62 two graft losses: one because of adenoviral enteritis and one because of rejection after rotavirus e

63 s to have only short-term effects, bacterial enteritis and protozoan and helminth infections are foll

64 effective control measures against necrotic enteritis and providing potential new tools to the field

65 aninum has been associated with eosinophilic enteritis and suggested as a possible cause of diffuse u

66 ry but not sufficient for the development of enteritis and that C57BL/6 IL-10(-/-) mice can serve as

67 Escherichia coli O157 causes severe enteritis and the extraintestinal complication hemolytic

68 compared with individuals without infectious enteritis) and host- and enteritis-related risk factors.

69 GI toxicity (mucositis, enteritis, and diarrhea) appears to be the major combine

70 for CN3685 to cause haemorrhagic necrotizing enteritis, apparently because the Agr-like system regula

71 Although the mechanism of C.jejuni-mediated enteritis appears to be multifactorial, flagella play co

72 Women-particularly those with severe enteritis-are at increased risk for developing IBS, as a

73 mucosal adenovirus infection associated with enteritis as well as parvovirus viremia in animals with

74 ary point prevalence of IBS after infectious enteritis, as well as relative risk (compared with indiv

75 acter isolates causing enteritis, 275 random enteritis-associated isolates of Campylobacter jejuni we

76 ampylobacter isolates causing enteritis, 275 enteritis-associated isolates, randomly collected in the

77 After giardiasis enteritis at least 5% developed clinical characteristics

78 or inflammation, especially after infectious enteritis, but this has not yet resulted in changes in t

79 dii inhibits C. difficile toxin A-associated enteritis by blocking the activation of Erk1/2 MAP kinas

80 However, cytomegalovirus (CMV) enteritis can cause complications.

81 s, 41.9% developed IBS, and of patients with enteritis caused by bacterial infection, 13.8% developed

82 Enteritis caused by Clostridium difficile toxin (Tx) is

83 erizing the virulence mechanisms influencing enteritis caused by non-typhoid Salmonella spp.

84 Of patients with enteritis caused by protozoa or parasites, 41.9% develop

85 this period, two patients had granulomatous enteritis characteristic of Crohn's disease in multiple

86 y which this bacterium invades its host, the enteritis characteristically associated with salmonellos

87 In a Caco-2 cell model of enteritis, culture supernatants of the type B agrB null

88 s, we found >10% of patients with infectious enteritis develop IBS later; risk of IBS was 4-fold high

89 Ten patients experienced 21 episodes of CMV enteritis, diagnosed by histopathology, virology, or bot

90 Rates of colonization and enteritis did not differ between male and female mice.

91 f the Salmonella virulence factors affecting enteritis do not appear to be required for infection of

92 that causes millions of cases of food-borne enteritis each year.

93 , in the absence of both, severe spontaneous enteritis emerges.

94 studies, comprising 21,421 individuals with enteritis, followed for 3 months to 10 years for develop

95 incidence, timing, and outcome of infectious enteritis (IE) after intestinal transplantation (ITx).

96 on included hypothyroidism in 5 subjects and enteritis in 1 subject.

97 specimens was used for the diagnosis of CMV enteritis in 21 patients.

98 pe C isolate CN3685 to cause bloody necrotic enteritis in a rabbit ileal loop model and also showed t

99 rombotic microangiopathy (iTMA) and ischemic enteritis in approximately 50% of infected human gut xen

100 phimurium mutants for their ability to cause enteritis in calves.

101 acteria that can cause chronic granulomatous enteritis in cattle, are difficult to distinguish on the

102 tion is the most commonly notified bacterial enteritis in Germany.

103 ause attaching and effacing (AE) lesions and enteritis in humans and animals.

104 ens type C isolates cause enterotoxemias and enteritis in humans and livestock.

105 n serve as models for the study of C. jejuni enteritis in humans.

106 continues to be a leading cause of bacterial enteritis in humans.

107 ts a mechanism involved in Shigella-elicited enteritis in humans.

108 ngens type D strains cause enterotoxemia and enteritis in livestock via epsilon toxin production.

109 e role of beta2-toxin in the pathogenesis of enteritis in neonatal pigs.

110 whether this protein plays the same role in enteritis in other animal species.

111 ases in domestic animals, including necrotic enteritis in piglets and typhlocolitis in horses.

112 ssist with the rapid laboratory diagnosis of enteritis in puppies and highlight the need for continue

113 -type CN3685 caused haemorrhagic necrotizing enteritis in rabbit ileal loops.

114 ic agrB or luxS mutants to cause necrotizing enteritis in rabbit small intestinal loops or enterotoxe

115 protease in preventing C. difficile toxin A enteritis in rat ileum and determine whether it protects

116 t S. boulardii inhibits C. difficile toxin A enteritis in rats by releasing a 54-kDa protease which d

117 ter a large community outbreak of giardiasis enteritis in the city of Bergen, Norway were evaluated w

118 2-fold higher in patients who had infectious enteritis in the past 12 months than in those who had no

119 ether antibiotic treatment of E coli O157:H7 enteritis increases the risk of HUS.

120 with natural Treg, and in vivo they suppress enteritis induced by mouse effector T cells.

121 Enteritis induced by non-typhoid pathogenic Salmonella i

122 al inflammatory diseases, such as infectious enteritis, inflammatory bowel disease, and necrotizing e

123 Toxin A enteritis involves release of PGE(2), which activates th

124 Campylobacter enteritis is a food-borne or waterborne illness caused a

125 to the laboratory diagnosis of Campylobacter enteritis is based on the recovery of the organism from

126 The type E animal enteritis isolates carrying these silent cpe sequences d

127 paratuberculosis results in a granulomatous enteritis (Johne's disease) that is often fatal.

128 at endothelial infection, iTMA, and ischemic enteritis might be central mechanisms underlying severe

129 In the enteritis model, TLR5KO mice had more severe gut patholo

130 tibiotic regimens commonly applied to murine enteritis models are used to examine the impact of antib

131 old higher in individuals who had infectious enteritis more than 12 months ago than in individuals wh

132 rodentium, a model pathogen for severe human enteritis, more rapidly than did WT mice.

133 s with the emerging infectious disease poult enteritis mortality syndrome.

134 evidence of acute rejection (n = 12), viral enteritis (n = 5), and nonspecific inflammation (n = 16)

135 e most common cause of bleeding, followed by enteritis (n=24), portal hypertensive lesions (n=15), Ro

136 he etiology of the poultry diseases necrotic enteritis (NE) and poultry gangrene (PG).

137 Necrotic enteritis (NE) caused by Clostridium perfringens is one

138 lostridium perfringens type C to cause human enteritis necroticans (EN) is attributed to beta toxin (

139 Enteritis necroticans (pigbel), an often fatal illness c

140 In December 1998, enteritis necroticans developed in a 12-year-old boy wit

141 ium perfringens type C isolates, which cause enteritis necroticans in humans and enteritis and entero

142 lostridium perfringens type C isolates cause enteritis necroticans in humans or necrotizing enteritis

143 pidly fatal diseases in domestic animals and enteritis necroticans in humans, contain the genes for a

144 The causative organism of enteritis necroticans is Clostridium perfringens type C,

145 ing, gas gangrene (clostridial myonecrosis), enteritis necroticans, and non-foodborne gastrointestina

146 ding gas gangrene (clostridial myonecrosis), enteritis necroticans, antibiotic-associated diarrhea, a

147 athogen, is one of the most common causes of enteritis necroticans, gas gangrene and food poisoning.

148 e severe diseases, including myonecrosis and enteritis necroticans, in humans and animals.

149 They are responsible for enteritis necroticans, which was termed Darmbrand when o

150 nts--findings consistent with a diagnosis of enteritis necroticans.

151 he acute pathogenesis of type A FP or type C enteritis necroticans.

152 ), occurs worldwide as chronic granulomatous enteritis of domestic and wild ruminants.

153 E isolates, all associated with hemorrhagic enteritis of neonatal calves, were identified by multipl

154 Johne's disease is a chronic enteritis of ruminants associated with enormous worldwid

155 n in conditions ranging from infective acute enteritis or colitis to inflammatory bowel disease is ac

156 of each branch of the UPR causes spontaneous enteritis or creates higher susceptibility for intestina

157 Since the lack of overt signs of enteritis or enterocolitis due to Salmonella infections

158 y higher than those from patients with viral enteritis or normal biopsies [198 mg/kg compared with 7

159 the IFN-gamma knockouts either succumbed to enteritis or survived to develop marked triaditis, porta

160 amma knockout donors either developed severe enteritis or survived to develop triaditis, cholangitis,

161 ired for the development of lethal radiation enteritis or the microbiota-associated enhancement of en

162 the reliable detection of invasive bacterial enteritis or the reliable selection of specimens for cul

163 sumptive diagnosis of inflammatory bacterial enteritis or which can be used to determine the suitabil

164 patients suspected of acute gastroenteritis, enteritis, or colitis.

165 a, F. nucleatum does not exacerbate colitis, enteritis, or inflammation-associated intestinal carcino

166 porcine sapelovirus (PSV) is known to cause enteritis, pneumonia, polioencephalomyelitis, and reprod

167 without infectious enteritis) and host- and enteritis-related risk factors.

168 95% CI, 1.6-6.5), and clinical indicators of enteritis severity.

169 s of patients with documented E coli O157:H7 enteritis, some of whom developed HUS; had clear definit

170 ence genes tested, but 66% of nonbacteremic, enteritis strains also contained all the tested virulenc

171 an outbreak of inclusion body hepatitis and enteritis that affected neonatal Northern aplomado (Falc

172 hat activated LPMs secrete SP during toxin A enteritis that can lead to secretion of cytokines, sugge

173 Campylobacter jejuni can cause an enteritis that is associated with an acute inflammatory

174 rt of patients presenting with Campylobacter enteritis to be 1.17/1000 person-years, a rate 77 times

175 onal neuropathy (AMAN), Campylobacter jejuni enteritis triggers the production of anti-ganglioside Ab

176 Hemorrhagic enteritis virus (HEV), a type II avian adenovirus, cause

177 Mink enteritis virus (MEV) is associated with rapid, high-lev

178 valence of IBS at 12 months after infectious enteritis was 10.1% (95% confidence interval [CI], 7.2-1

179 and at more than 12 months after infectious enteritis was 14.5% (95% CI, 7.7-25.5).

180 Toxin A enteritis was accompanied by increased cellular infiltra

181 (2)-dependent Fas/FasL activation in toxin A enteritis was further assessed in either scid or FasL an

182 In contrast, CMV enteritis was identified in only one patient, who subseq

183 ting, diarrhea, and histologic gastritis and enteritis were commonly observed in dogs treated with th

184 ic neoplasm, and ulcerative colitis/regional enteritis were included.

185 rm of ocular adnexal involvement in regional enteritis, which affects the orbit far more frequently t

186 might reflect enhanced host defense against enteritis, which is more severe in those with acquired o

187 hat an individual who develops Campylobacter enteritis will also develop GBS during the subsequent 2-

188 arasite Cryptosporidium parvum causes severe enteritis with substantial morbidity and mortality among

189 liary tract infection, abdominal abscess, or enteritis) with those who did not to identify clinical f

190 IDS patients can have episodes of Salmonella enteritis without dissemination.

191 uni is a major cause of bacterial food-borne enteritis worldwide, and invasion into intestinal epithe

192 ticus, a leading cause of seafood-associated enteritis worldwide, is dependent upon a type III secret