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1 ement of the Cra regulator characteristic of Enterobacteriales.
2  or TTSS-secreted proteins, are required for enterobacterial aggregative multicellular behavior.
3 developed genetics of E. coli by integrating enterobacterial ampRC genes into the E. coli chromosome.
4 (IL-1beta) level; the relative abundances of Enterobacteriales and Enterobacteriaceae and the interfe
5 relations between the relative abundances of Enterobacteriales and Enterobacteriaceae and the sCD14 l
6  relative abundances of Gammaproteobacteria, Enterobacteriales, and Enterobacteriaceae and the interl
7                                              Enterobacterial animal pathogens exhibit aggregative mul
8 igh homology to previously reported ViI-like enterobacterial bacteriophage genomes.
9 sly constructed an assay system for studying enterobacterial beta-lactam resistance mutations using t
10 rial amyloids, are an important component of enterobacterial biofilms.
11  responses in the gut can generate transient enterobacterial blooms in which conjugative transfer occ
12 tics to inhibit enteric pathogens and reduce enterobacterial blooms.
13             The mechanism of sugar uptake by enterobacterial channels, such as Escherichia coli LamB
14 igosaccharides preferred by LamB and related enterobacterial channels.
15 tification of a water-soluble cyclic form of enterobacterial common antigen (ECA(CYC)) from Escherich
16                      Phosphoglyceride-linked enterobacterial common antigen (ECA(PG)) is a cell surfa
17 embly of the phosphoglyceride-linked form of enterobacterial common antigen (ECA(PG)) occurs by a mec
18                 The polysaccharide chains of enterobacterial common antigen (ECA) are comprised of th
19 tools revealed that rffH, a gene involved in enterobacterial common antigen (ECA) biosynthesis, is pa
20                                              Enterobacterial common antigen (ECA) is expressed by Gra
21 ese loci is responsible for synthesis of the enterobacterial common antigen (ECA), a glycolipid situa
22 otransferase involved in the biosynthesis of enterobacterial common antigen (ECA), a non-essential ou
23        Here, we characterize the role of the enterobacterial common antigen (ECA), a surface glycolip
24 ogues of genes required for the synthesis of enterobacterial common antigen (ECA), suggesting that H.
25 cteriaceae express a polysaccharide known as enterobacterial common antigen (ECA), which is an attrac
26 accharide repeat unit in the biosynthesis of enterobacterial common antigen (ECA).
27 d A and complete LOS core (galU), as well as enterobacterial common antigen (wecB and wecC), is impor
28 en with the use of MAB-T88 in the bacteremic enterobacterial common antigen group (p <.05).
29 tive sepsis or in those patients with proven enterobacterial common antigen infections.
30 in wzyE, encoding an enzyme that polymerizes enterobacterial common antigen, a surface polysaccharide
31 ibits growth in bile only in the presence of enterobacterial common antigen, an outer-membrane glycol
32 occurrence of a water-soluble cyclic form of enterobacterial common antigen, ECA(CYC), purified from
33 n the biosynthesis of lipopolysaccharide and enterobacterial common antigen.
34 n insertion in wecE was unable to synthesize enterobacterial common antigen.
35 luding group I capsule, group II capsule and enterobacterial common antigen; (iii) genes involved in
36 he type 5 capsule biosynthetic locus restore enterobacterial common-antigen expression to Escherichia
37 -1 fimbrial subunit, FimH, was the necessary enterobacterial component for mast-cell activation and n
38 egion differs substantially from the typical enterobacterial cores.
39 uences occurs at the attachment site of each enterobacterial element, apparently serving as a transcr
40 ance of Proteobacteria, Gammaproteobacteria, Enterobacteriales, Enterobacteriaceae, Erysipelotrichi,
41 a that differences in structural features in enterobacterial FlhD are responsible for differential su
42 n DNA condensation and is a key regulator of enterobacterial gene expression in response to changes i
43         Mauve has been applied to align nine enterobacterial genomes and to determine global rearrang
44 ion as well as those of additional published enterobacterial genomes is underway and will be publicly
45     The EnteriX suite currently includes >15 enterobacterial genomes, generates views centered on fou
46 er 300 vertically inherited prophages within enterobacterial genomes.
47  A backbone, usually present in highly toxic enterobacterial Gram-negative lipid A.
48 haperones, facilitating incorporation of the enterobacterial hook-associated axial proteins (HAPs) Fl
49 NF-alpha) to recruit neutrophils to sites of enterobacterial infection.
50                          Chlamydial and many enterobacterial infections can trigger reactive arthriti
51 idely distributed among Salmonella and other enterobacterial isolates from agricultural sources and h
52  glucosamine disaccharide characteristic for enterobacterial lipid A was replaced by a 2,3-diamino-2,
53  --> 6)-glucosamine disaccharide, typical of enterobacterial lipid A.
54        Exposure of mononuclear phagocytes to enterobacterial LPS induces a state of transient hypores
55                                              Enterobacterial LPS is recognized by the TLR4 signaling
56  activity was established: IL-6 induction by enterobacterial LPS was inhibited by cylindrically shape
57 demonstrate that in contrast to protein-free enterobacterial LPS, a similarly purified preparation of
58 ta support the conclusion that TLR4 mediates enterobacterial LPS-induced HIV transcription via IL-1 s
59 surface receptor and molecular mechanisms of enterobacterial LPS-induced HIV transcription.
60 to differ structurally and functionally from enterobacterial LPS.
61                                              Enterobacterial MlaA proteins form stable complexes with
62 he genes are designated mntH because the two enterobacterial NRAMPs encode H+-stimulated, highly sele
63 4 signaling complex, whereas LPS of some non-enterobacterial organisms is capable of signaling indepe
64  in contrast to the 2 position seen with the enterobacterial PagP.
65 d TCSTs in Erwinia amylovora, a severe plant enterobacterial pathogen, at genome-wide level.
66 ugment and curate annotations for genomes of enterobacterial pathogens and for additional genome sequ
67                                  Most of the enterobacterial pathogens encode at least one T3SS, a ma
68                                      In some enterobacterial pathogens, but not in Escherichia coli,
69                                          The enterobacterial phytopathogen Erwinia amylovora causes f
70                         The ardA gene of the enterobacterial plasmid CollbP-9 acts to alleviate restr
71 which is closely related to the genes in the enterobacterial plasmid R64.
72 atory ability of this procedure with that of enterobacterial repeat intergenic consensus (ERIC2) PCR,
73 wo Polymerase Chain Reaction (PCR) analyses: Enterobacterial Repetitive Intergenic Consensus (ERIC) a
74 ined by polymerase chain reaction (PCR) with enterobacterial repetitive intergenic consensus (ERIC) p
75  phylogenetic groups was further assessed by enterobacterial repetitive intergenic consensus (ERIC) t
76  larvae, which largely confirms the previous enterobacterial repetitive intergenic consensus (ERIC)-p
77 ection of Escherichia coli isolates typed by enterobacterial repetitive intergenic consensus (ERIC)-P
78  by pulsed-field gel electrophoresis (PFGE), enterobacterial repetitive intergenic consensus (ERIC)-P
79 tis were compared by DNA fingerprinting with enterobacterial repetitive intergenic consensus primers.
80 lsed-field gel electrophoresis (PFGE) and/or enterobacterial repetitive intergenic consensus sequence
81 ss discriminating than MLST, ribotyping, and enterobacterial repetitive intergenic consensus sequence
82                Genetic profiling was done by enterobacterial repetitive intergenic consensus sequence
83 al and clinical isolates were genotyped with enterobacterial repetitive intergenic consensus sequence
84 isruptor whose omission helped stabilize the Enterobacteriales root.
85  to modulate the tissue tropism of different enterobacterial species represents a novel function for
86 ison of rsmB sequences from several of these enterobacterial species revealed a highly conserved 34-m
87   The phylogenetic relationships of multiple enterobacterial species were reconstructed based on 16S
88 xed and user-supplied sequences from related enterobacterial species, anchored on a reference genome.
89 a collection of 17 SPI-7 related ICEs within enterobacterial species, of which six are novel.
90 enomic analysis of TCSTs in 53 genomes of 16 enterobacterial species.
91 n of fliA and motility varies depending upon enterobacterial species.
92  important step in the evolution of virulent enterobacterial strains.
93 a and their phages, and (3) dCTP/dUTPases in enterobacterial T4-like phages.
94  distinct binding specificities of different enterobacterial type 1 fimbriae cannot be ascribed solel
95  in the scale (from 1 to 2 target operons in Enterobacteriales up to 20 operons in Aeromonadales) and
96 Instability was observed for the root of the Enterobacteriales, with nearly equal subsets of the prot

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