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1 icate goblet cells, whereas others implicate enterochromaffin cells).
2 e adult stage in a subset of enteroendocrine/enterochromaffin cells.
3  by 5-hydroxytryptamine (5-HT) released from enterochromaffin cells.
4 g cells are identified as a subpopulation of enterochromaffin cells.
5  in neuroendocrine, including chromaffin and enterochromaffin, cells.
6 onstrated that 5-HT released from intestinal enterochromaffin cells activates 5-HT3 receptors on vaga
7     5-Hydroxytryptamine (5-HT) released from enterochromaffin cells activates secretory and peristalt
8  5-Hydroxytryptamine (5-HT) is released from enterochromaffin cells and activates neural reflex progr
9 e of 5-hydroxytryptamine (5-HT) from mucosal enterochromaffin cells and activation of 5-HT(3) recepto
10 e transcription factor Lmx1a is expressed in enterochromaffin cells and functions downstream of Nkx2.
11              Serotonin is also released from enterochromaffin cells and inflammatory/immune cells to
12 sis, consistent with localization of TGR5 to enterochromaffin cells and intrinsic primary afferent ne
13 med FFA2 and FFA3, are expressed in duodenal enterochromaffin cells and L cells, respectively.
14 ivity was abundant in both enteric plexuses, enterochromaffin cells, and pancreatic ganglia.
15 release serotonin (5-hydroxytryptamine) from enterochromaffin cells, and stretch reflexes that determ
16 vious results showing guanylin expression in enterochromaffin cells appear to be a consequence of ant
17              These findings imply a role for enterochromaffin cells as "glucose sensors" during inges
18           We show that the number of mucosal enterochromaffin cells containing 5-HT changes with the
19                       Paracrine signaling by enterochromaffin cells (EC), which release 5-HT, has not
20 biota promote 5-HT biosynthesis from colonic enterochromaffin cells (ECs), which supply 5-HT to the m
21 tion to neurons, a subset of 5-HT-containing enterochromaffin cells expressed 5-HT1A immunoreactivity
22                                      Mucosal enterochromaffin cells have been postulated to be pressu
23 onsistent with the hypothesis that 5-HT from enterochromaffin cells in response to mucosal stimuli in
24  neurons were activated by 5-HT release from enterochromaffin cells in the mucosa.
25                           5-HT released from enterochromaffin cells initiates peristaltic, secretory,
26                 Expression of uroguanylin in enterochromaffin cells is consistent with the hypothesis
27                              5HT produced by enterochromaffin cells is critical in motility and secre
28 g kg-1) indicating that endogenous 5-HT from enterochromaffin cells is not essential for transduction
29 erotonin (5-HT), which abounds in intestinal enterochromaffin cells, is released in response to vario
30 e populations, and identify Lmx1a as a novel enterochromaffin cell marker that is also essential for
31 athematical modeling, we show that, in human enterochromaffin cells, Myrip (1) inhibits a class of SG
32 imiting biosynthetic enzyme for serotonin in enterochromaffin cells of the duodenum.
33  a decrease in the synthesis of serotonin by enterochromaffin cells of the gut.
34 , it is unclear whether they act directly on enterochromaffin cells or indirectly through an intermed
35                                          The enterochromaffin cell population was decreased in severe
36 er RNA, encoding the rate-limiting enzyme in enterochromaffin cell serotonin (5-hydroxytryptamine [5H
37 kx2.2 mutant conditions, serotonin-producing enterochromaffin cells were the most severely reduced en
38                Human BON cells, derived from enterochromaffin cells, were treated with D-glucose, gal
39  release of endogenous 5-HT from the mucosal enterochromaffin cells, which acts on the 5-HT3 receptor

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