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1 icate goblet cells, whereas others implicate enterochromaffin cells).
2 e adult stage in a subset of enteroendocrine/enterochromaffin cells.
3 by 5-hydroxytryptamine (5-HT) released from enterochromaffin cells.
4 g cells are identified as a subpopulation of enterochromaffin cells.
5 in neuroendocrine, including chromaffin and enterochromaffin, cells.
6 onstrated that 5-HT released from intestinal enterochromaffin cells activates 5-HT3 receptors on vaga
8 5-Hydroxytryptamine (5-HT) is released from enterochromaffin cells and activates neural reflex progr
9 e of 5-hydroxytryptamine (5-HT) from mucosal enterochromaffin cells and activation of 5-HT(3) recepto
10 e transcription factor Lmx1a is expressed in enterochromaffin cells and functions downstream of Nkx2.
12 sis, consistent with localization of TGR5 to enterochromaffin cells and intrinsic primary afferent ne
15 release serotonin (5-hydroxytryptamine) from enterochromaffin cells, and stretch reflexes that determ
16 vious results showing guanylin expression in enterochromaffin cells appear to be a consequence of ant
20 biota promote 5-HT biosynthesis from colonic enterochromaffin cells (ECs), which supply 5-HT to the m
21 tion to neurons, a subset of 5-HT-containing enterochromaffin cells expressed 5-HT1A immunoreactivity
23 onsistent with the hypothesis that 5-HT from enterochromaffin cells in response to mucosal stimuli in
28 g kg-1) indicating that endogenous 5-HT from enterochromaffin cells is not essential for transduction
29 erotonin (5-HT), which abounds in intestinal enterochromaffin cells, is released in response to vario
30 e populations, and identify Lmx1a as a novel enterochromaffin cell marker that is also essential for
31 athematical modeling, we show that, in human enterochromaffin cells, Myrip (1) inhibits a class of SG
34 , it is unclear whether they act directly on enterochromaffin cells or indirectly through an intermed
36 er RNA, encoding the rate-limiting enzyme in enterochromaffin cell serotonin (5-hydroxytryptamine [5H
37 kx2.2 mutant conditions, serotonin-producing enterochromaffin cells were the most severely reduced en
39 release of endogenous 5-HT from the mucosal enterochromaffin cells, which acts on the 5-HT3 receptor
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