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1 f traits contributing to the pathogenesis of enterococcal infection.
2 detected more episodes of streptococcal and enterococcal infection.
3 d inflammation contributes to the control of enterococcal infection.
4 options for the treatment and prevention of enterococcal infections.
5 y be useful for prophylaxis and treatment of enterococcal infections.
6 tive for some strains of multidrug-resistant enterococcal infections.
7 l antimicrobial targets for the treatment of enterococcal infections.
8 a serious threat to the treatment of serious enterococcal infections.
9 ibiotic resistances, making the treatment of enterococcal infections an increasingly difficult proble
10 ion was only minimally altered by subsequent enterococcal infection and was not suppressed by inhibit
11 been an increase in the number of nosocomial enterococcal infections caused by strains resistant to v
12 ly assayed sera collected from patients with enterococcal infections for the presence of anti-Ace A a
14 idly, to the point where over one-quarter of enterococcal infections in intensive care units are now
16 gainst VRE, but its clinical use for serious enterococcal infections is unclear due to low serum leve
17 nosocomial pathogen, yet the pathogenesis of enterococcal infections, particularly of urinary tract i
18 ling, this monoclonal identified the site of enterococcal infection, providing a rare example of mole
20 of vancomycin resistance and association of enterococcal infections with significant mortality warra
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