ライフサイエンスコーパス: enterocolitis

1 ia, severe cerebral lesions, and necrotizing enterocolitis).

2 icular leukomalacia >grade 1, or necrotizing enterocolitis).

3 mon to all infants who developed necrotizing enterocolitis.

4 e, norovirus, cytomegalovirus, and bacterial enterocolitis.

5 s that may lead to diseases like necrotizing enterocolitis.

6 is restricted to APCs, in the development of enterocolitis.

7 tract, or cerebrospinal fluid or necrotizing enterocolitis.

8 et inflammatory bowel disease with apoptotic enterocolitis.

9 oundly depleted in newborns with necrotizing enterocolitis.

10 es and T cells that lead to severe apoptotic enterocolitis.

11 ouse homologs of CXCL8 in the early phase of enterocolitis.

12 actice reduces the prevalence of necrotizing enterocolitis.

13 owards a diminished incidence of necrotizing enterocolitis.

14 she was diagnosed with allergic eosinophilic enterocolitis.

15 and host immune elements such as necrotizing enterocolitis.

16 regulation with life-threatening early-onset enterocolitis.

17 ntributes to the pathogenesis of necrotizing enterocolitis.

18 ch as the transfusion-associated necrotizing enterocolitis.

19 testinal atresias, volvulus, and necrotizing enterocolitis.

20 inflammatory bowel disease, and necrotizing enterocolitis.

21 cular leukomalacia; and death or necrotizing enterocolitis.

22 rious respiratory conditions and necrotizing enterocolitis.

23 en in developing intestine as in necrotizing enterocolitis.

24 , rapid clinical improvement, and healing of enterocolitis.

25 enuated Salmonella-induced and noninfectious enterocolitis.

26 osal inflammation in a murine model of acute enterocolitis.

27 of neonatal bacterial sepsis and necrotizing enterocolitis.

28 ich likely contributes to the development of enterocolitis.

29 weeks of age with intestinal distension and enterocolitis.

30 enterocytes during experimental necrotizing enterocolitis.

31 IL-10-deficient mice spontaneously develop enterocolitis.

32 ute to the pathogenesis of C. jejuni-induced enterocolitis.

33 preterm infants with perforated necrotizing enterocolitis.

34 s during chronic, experimental granulomatous enterocolitis.

35 and management of patients with neutropenic enterocolitis.

36 l die at 3 to 5 days of age with evidence of enterocolitis.

37 lammatory bowel diseases such as necrotizing enterocolitis.

38 gest that only toxin A mediates diarrhea and enterocolitis.

39 ifier, on the risk of developing necrotising enterocolitis.

40 ficantly higher rate of neonatal necrotising enterocolitis.

41 ce and invasion assay, and the calf model of enterocolitis.

42 ng-related antecedents of severe necrotising enterocolitis.

43 eriventricular leukomalacia, and necrotising enterocolitis.

44 intestinal pathologies including necrotizing enterocolitis.

45 criteria for Bell's stage 2 or 3 necrotising enterocolitis.

46 t typhoid fever, there are none that prevent enterocolitis.

47 ricular hemorrhage, and death or necrotizing enterocolitis.

48 ants who did and did not develop necrotizing enterocolitis.

49 rate from the best quartile for necrotizing enterocolitis.

50 se or colitis (6.3% and 2.7%), and bacterial enterocolitis (0.9% and 0%) (P = .03).

51 =0.045) and an increased rate of necrotizing enterocolitis (10.4% vs. 8.0%; relative risk, 1.31; 95%

52 to treat to prevent one case of necrotising enterocolitis 114, 95% CI 87 to 136).

53 CI, 16.5%-18.6%]), and death or necrotizing enterocolitis (19.3% [95% CI, 18.1%-20.4%]).

54 auses of the abdominal pain were neutropenic enterocolitis (28%) and small bowel obstruction (12%); t

55 ncreases in deaths attributed to necrotizing enterocolitis (30 [95% CI, 27 to 34] vs. 23 [95% CI, 20

56 .3%, 19.1%, and 11.7%), death or necrotizing enterocolitis (48.1%, 37.1%, and 32.5%), and death or br

57 and developed a CD8(+) T cell-mediated acute enterocolitis 5 days after oral L. monocytogenes-encodin

58 significantly less was death or necrotizing enterocolitis (73.5% with exposure to antenatal corticos

59 ary cohort, of whom 28 developed necrotising enterocolitis; 94 infants were used as controls.

60 ht predispose to or protect from necrotising enterocolitis, a severe illness linked to prematurity.

61 nchiolitis, arthralgia, ocular inflammation, enterocolitis, absence of autoantibodies, and mild immun

62 f 30 566 VLBW infants, 1879 with necrotizing enterocolitis, according to the level of care and VLBW c

63 , diarrhea, Helicobacter pylori, necrotizing enterocolitis, allergy, and inflammatory bowel disease.

64 Neutropenic enterocolitis, also referred to as typhlitis, is a life-

65 ifferential diagnosis in outbreaks of severe enterocolitis among puppies between 4 days and 21 weeks

66 protected mice from T-cell transfer-induced enterocolitis and death.

67 These data indicate that chronic enterocolitis and diarrhea are associated, in part, with

68 tissues collected from animals with chronic enterocolitis and diarrhea in comparison with clinically

69 The fear of necrotizing enterocolitis and feeding intolerance are major factors

70 sequencing of DNA from 1 infant with severe enterocolitis and her parents.

71 everity in mouse models of acute and chronic enterocolitis and improved, in synergy with glucocortico

72 icated as a pathogenic factor in necrotizing enterocolitis and inflammatory bowel disease.

73 ylobacter jejuni is a leading cause of human enterocolitis and is associated with postinfectious comp

74 breve BBG-001 for prevention of necrotising enterocolitis and late-onset sepis in very preterm infan

75 Probiotics may reduce necrotising enterocolitis and late-onset sepsis after preterm birth.

76 species cause zoonotic infections, including enterocolitis and plague.

77 wborns has been shown to prevent necrotizing enterocolitis and reduce all-cause mortality.

78 We enrolled 40 patients with early-onset enterocolitis and screened for mutations in IL10/IL10R u

79 psy findings showed features of eosinophilic enterocolitis and she was diagnosed with allergic eosino

80 ibrosis associated with chronic experimental enterocolitis and stimulates expression of antifibrogeni

81 or and in protection against immune mediated enterocolitis and these phenomena are significantly asso

82 ses a spectrum of human infections including enterocolitis and typhoid fever.

83 cal courses were consistent with necrotising enterocolitis and whose radiographs fulfilled criteria f

84 e PDA closure, including sepsis, necrotizing enterocolitis, and a dependence on mechanical ventilatio

85 sease, irritable bowel syndrome, necrotizing enterocolitis, and a variety of other disorders.

86 e development of auto-inflammatory diseases, enterocolitis, and cancer.

87 ere intraventricular hemorrhage, necrotizing enterocolitis, and chronic lung disease among infants le

88 ensive loss of intestinal villi, obstructive enterocolitis, and lethality within 10 days.

89 sia, retinopathy of prematurity, necrotizing enterocolitis, and periventricular leukomalacia.

90 mmatory diseases such as asthma, necrotizing enterocolitis, and sepsis.

91 oss, bronchopulmonary dysplasia, necrotizing enterocolitis, and severe retinopathy of prematurity.

92 emature infants with and without necrotizing enterocolitis, and successfully provided a total number

93 c meningitis, neutropenic fever, neutropenic enterocolitis, and transfussion-associated GVHD.

94 1%), and Hirschsprung's disease, necrotising enterocolitis, and volvulus neonatorum in 23 (45.1%) eac

95 ical phenotype of Crohn disease, necrotizing enterocolitis, and, perhaps, intestinal manifestations o

96 g enterocolitis, the severity of necrotizing enterocolitis, and/or bacterial sepsis.

97 (AOR, 0.98; 95% CI, 0.70-1.37), necrotizing enterocolitis (AOR, 0.88; 95% CI, 0.65-1.20), severe neu

98 Four other patients with steroid-refractory enterocolitis appeared to respond promptly to tumor necr

99 or intraventricular hemorrhage, necrotizing enterocolitis, aspiration, retinopathy of prematurity, a

100 Pseudomembranous enterocolitis associated with Clostridium difficile infe

101 butor to the enhanced severity of Salmonella enterocolitis associated with helminth coinfection.

102 actors, including development of necrotising enterocolitis, associated with gut bacterial populations

103 less than 1500 g and perforated necrotizing enterocolitis at 15 pediatric centers to undergo primary

104 ets for interventions to prevent necrotising enterocolitis, at least among infants born at less than

105 We determined the severity of enterocolitis based on disease activity index, histology

106 that could predispose infants to necrotising enterocolitis before we can develop new strategies for p

107 The primary outcomes were necrotising enterocolitis (Bell stage 2 or 3), blood culture positiv

108 Infants with necrotizing enterocolitis born into midlevel hospitals (low-volume l

109 morbidities: late-onset sepsis, necrotizing enterocolitis, bronchopulmonary dysplasia, periventricul

110 roducts might reduce the risk of necrotising enterocolitis, but the absolute reduction is small.

111 roducts might reduce the risk of necrotising enterocolitis, but the absolute reduction is small.

112 ased the histopathologic severity of chronic enterocolitis by repairing crypt epithelium and simultan

113 18 infants developed necrotising enterocolitis (cases) and 26 were controls.

114 infants who subsequently develop necrotising enterocolitis (cases) and those who do not (controls).

115 nt complement-mediated killing compared with enterocolitis-causing strains of S Typhimurium.

116 (9%) in the probiotic group had necrotising enterocolitis compared with 66 (10%) in the placebo grou

117 n infants who went on to develop necrotising enterocolitis compared with controls.

118 fy the burden of severe or fatal necrotising enterocolitis confirmed by laparotomy, leading to death,

119 In mice with S typhimurium-induced enterocolitis, CoPP reduced the numbers of live S typhim

120 development was most impaired by necrotizing enterocolitis (d = -0.40; P < .001) and meningitis (d =

121 development was most impaired by necrotizing enterocolitis (d = -0.66; P < .001).

122 Necrotizing enterocolitis, defined as Bell stage 2 or greater by pre

123 Enterocolitis, defined by grade 3/4 clinical presentatio

124 the national incidence of severe necrotising enterocolitis, describe variation across neonatal networ

125 eration performed for perforated necrotizing enterocolitis does not influence survival or other clini

126 Experimental enterocolitis does not occur in a sterile (germ-free) en

127 mall-bowel resection (SBR) after necrotizing enterocolitis expands absorptive surface areas and promo

128 for more than one decade with chronic active enterocolitis, fistula disease as well as previous oral

129 Food protein-induced enterocolitis (FPIES) is a non-IgE cell- mediated food a

130 tity that can help differentiate neutropenic enterocolitis from other gastrointestinal complications.

131 g diagnoses: intestinal atresia, necrotizing enterocolitis, gastroschisis, and midgut volvulus.

132 tients with clinical features of necrotising enterocolitis had raised faecal calprotectin concentrati

133 eatments for biliary atresia and necrotising enterocolitis have been achieved through specialisation,

134 such as reduction in sepsis and necrotizing enterocolitis, have been reported for premature infants

135 in six patients (43%), including dermatitis, enterocolitis, hepatitis, and hypophysitis, and mediated

136 ne mediated toxicities including dermatitis, enterocolitis, hypophysitis, uveitis, hepatitis, and nep

137 onic intestinal inflammation and necrotizing enterocolitis in animal models.

138 on between certain Campylobacter species and enterocolitis in humans and nonhuman primates is well es

139 spontaneous colitis in mice and early onset enterocolitis in humans.

140 hogen that causes meningitis and necrotizing enterocolitis in infants.

141 lammatory bowel diseases (IBD) in humans and enterocolitis in mice.

142 sence of IL-10 signaling causes inflammatory enterocolitis in neonates.

143 pathogens frequently associated with chronic enterocolitis in normal, immunocompetent rhesus monkeys

144 may be associated with neonatal necrotizing enterocolitis in premature infants.

145 ally may reduce the incidence of necrotizing enterocolitis in preterm infants.

146 can be used to aid diagnosis of necrotising enterocolitis in preterm infants.

147 y that inhibited Slamf1 reduced the level of enterocolitis in Rag(-/-) mice.

148 onalcoholic fatty liver disease, necrotizing enterocolitis in very low birth weight infants, and hepa

149 specially Negativicutes) precede necrotising enterocolitis in very low birthweight infants.

150 nd find that specific fatty acids exacerbate enterocolitis in zebrafish.

151 more than 12 hours, were severe necrotizing enterocolitis, infection, bronchopulmonary dysplasia, se

152 events were reported in five patients (6%): enterocolitis, infusion-related reaction, aminotransfera

153 In mixed models, the time-by-necrotising-enterocolitis interaction was positively associated with

154 Owing to the rarity of severe necrotising enterocolitis, international collaborations are needed f

155 had significantly lower rates of necrotizing enterocolitis, intraventricular hemorrhage, and need for

156 Necrotizing enterocolitis is a devastating inflammatory condition of

157 Necrotizing enterocolitis is a major cause of death and complication

158 Perforated necrotizing enterocolitis is a major cause of morbidity and mortalit

159 Necrotising enterocolitis is a neonatal gastrointestinal inflammator

160 Neutropenic enterocolitis is a serious, potentially lethal complicat

161 Necrotizing enterocolitis is an overwhelming gastrointestinal emerge

162 We have reported this case because allergic enterocolitis is becoming a topic of concern.

163 Necrotising enterocolitis is one of the most common gastrointestinal

164 Necrotising enterocolitis is one of the most devastating and unpredi

165 Necrotizing enterocolitis is the leading cause of death from gastroi

166 Chronic enterocolitis is the leading cause of morbidity in colon

167 acterium breve BBG-001 to reduce necrotising enterocolitis, late-onset sepsis, and death in preterm i

168 suggested, as TLR4 activation in necrotizing enterocolitis led to reduced proliferation and increased

169 Infections and necrotizing enterocolitis, major causes of mortality and morbidity i

170 composite of late-onset sepsis, necrotising enterocolitis (modified Bell stage >/=2), or death in in

171 bronchopulmonary dysplasia, and necrotizing enterocolitis, most therapeutic approaches have failed t

172 ), sepsis (n = 1922), sepsis and necrotizing enterocolitis (n = 279), or meningitis with or without s

173 ), intestinal atresia (n=5), and necrotizing enterocolitis (n=4).

174 Necrotizing enterocolitis (NEC) affects up to 10% of premature infan

175 wn to protect neonatal rats from necrotizing enterocolitis (NEC) and are good therapeutic candidates

176 though the incidence of neonatal necrotizing enterocolitis (NEC) and the mortality stemming from this

177 (RBC) transfusion and anemia to necrotizing enterocolitis (NEC) are conflicting.

178 een associated with outbreaks of necrotizing enterocolitis (NEC) as well as infant sepsis and meningi

179 Necrotizing enterocolitis (NEC) continues to be a major cause of mor

180 Necrotizing enterocolitis (NEC) develops in response to elevated TLR

181 Necrotizing enterocolitis (NEC) has long remained a significant caus

182 ation of severe diseases such as necrotizing enterocolitis (NEC) in neonates or bowel wall rupture in

183 Necrotizing enterocolitis (NEC) is a common and often fatal inflamma

184 Necrotizing enterocolitis (NEC) is a devastating disease of prematur

185 Necrotizing enterocolitis (NEC) is a devastating inflammatory bowel

186 Necrotizing enterocolitis (NEC) is a devastating intestinal disease

187 Neutropenic enterocolitis (NEC) is a life-threatening disease with s

188 Necrotizing enterocolitis (NEC) is a major cause of morbidity and mo

189 Necrotizing enterocolitis (NEC) is a major cause of neonatal morbidi

190 Necrotizing enterocolitis (NEC) is a severe disease of the gastroint

191 Necrotizing enterocolitis (NEC) is a severe disease that affects the

192 Necrotizing enterocolitis (NEC) is an inflammatory disease of the in

193 Necrotizing enterocolitis (NEC) is an inflammatory intestinal disord

194 Experimental necrotizing enterocolitis (NEC) is characterized by circulating endo

195 Necrotizing enterocolitis (NEC) is characterized by interferon-gamma

196 Development of necrotising enterocolitis (NEC) is considered to be dependent on the

197 Necrotizing enterocolitis (NEC) is one of the most serious disorders

198 Necrotizing enterocolitis (NEC) is the leading cause of death from g

199 Necrotizing enterocolitis (NEC) is the leading cause of death from g

200 Necrotizing enterocolitis (NEC) is the leading cause of gastrointest

201 Necrotizing enterocolitis (NEC) is the most common and serious gastr

202 Necrotizing enterocolitis (NEC) is the most common gastrointestinal

203 Necrotizing enterocolitis (NEC) is the most common gastrointestinal

204 Necrotizing enterocolitis (NEC) is the most common gastrointestinal

205 nd is involved in development of necrotizing enterocolitis (NEC) of the immature intestine.

206 itial laparotomy or drainage for necrotizing enterocolitis (NEC) or isolated intestinal perforation (

207 regulate enterocyte apoptosis in necrotizing enterocolitis (NEC) remain incompletely understood, alth

208 The pathophysiology of necrotizing enterocolitis (NEC) remains poorly understood.We assesse

209 sequence of intestinal injury in necrotizing enterocolitis (NEC) remains unknown.

210 ut inflammation such as neonatal necrotizing enterocolitis (NEC) result after an injury to the mucosa

211 ave proposed using outcomes from necrotising enterocolitis (NEC) surgery for revalidation of neonatal

212 gen associated with the cases of necrotizing enterocolitis (NEC) that result from formula contaminati

213 mmatory cascade, is activated in necrotizing enterocolitis (NEC), a devastating condition of intestin

214 sease in the setting of neonatal necrotizing enterocolitis (NEC), a life-threatening gastrointestinal

215 of the intestinal leukocytes in necrotizing enterocolitis (NEC), a severe disease affecting prematur

216 Necrotizing enterocolitis (NEC), a severe intestinal inflammation in

217 ture neonates are predisposed to necrotizing enterocolitis (NEC), an idiopathic, inflammatory bowel n

218 antibiotic resistance, fungemia, necrotizing enterocolitis (NEC), and mortality.

219 , otitis media, gastroenteritis, necrotizing enterocolitis (NEC), and sudden infant death syndrome (S

220 ontribute to the pathogenesis of necrotizing enterocolitis (NEC), but it is unknown whether their int

221 ead use of plain films to detect necrotizing enterocolitis (NEC), it is considered a time-consuming m

222 nal diseases, including neonatal necrotizing enterocolitis (NEC), the leading cause of death from gas

223 Necrotizing enterocolitis (NEC), the leading cause of gastrointestin

224 HBM) attenuates the incidence of necrotizing enterocolitis (NEC), which remains a leading and intract

225 h can lead to the development of necrotizing enterocolitis (NEC)--a devastating inflammatory disease

226 egnancy-related risk factors for necrotizing enterocolitis (NEC)-associated deaths during infancy.

227 racterized by ISC loss including necrotizing enterocolitis (NEC).

228 in prophylaxis and treatment of necrotizing enterocolitis (NEC).

229 order to reduce the incidence of necrotizing enterocolitis (NEC).

230 feeding interventions to prevent necrotizing enterocolitis (NEC).

231 y associated with development of necrotizing enterocolitis (NEC).

232 is, SL mortality predictors were necrotizing enterocolitis (NEC; surgical odds ratio, 5.95; medical o

233 ated in the mutant animals, with less severe enterocolitis observed in vivo and reduced macrophage TN

234 Late-onset invasive infection or necrotizing enterocolitis occurred in 32% of infants (19 of 60) in g

235 Necrotizing enterocolitis occurred less frequently in the liberal ox

236 Necrotizing enterocolitis occurred more frequently in infants on res

237 age of 32 weeks developed severe necrotising enterocolitis, of whom 222 (48.1%) died.

238 1 (0.4%) babies developed severe necrotising enterocolitis, of whom 247 (46.5%) died (139 after lapar

239 78) and the combined outcomes of necrotizing enterocolitis or death and severe intraventricular hemor

240 y bypass, as well as in neonatal necrotizing enterocolitis or persistent ductus arteriosus.

241 y of prematurity and surgery for necrotizing enterocolitis or spontaneous intestinal perforation were

242 vere retinopathy of prematurity, necrotizing enterocolitis, or late-onset sepsis) by 36 weeks of post

243 nfection (sepsis or meningitis), necrotizing enterocolitis, or mortality during the first 60 days of

244 lar leukomalacia, proven sepsis, necrotizing enterocolitis, or perinatal death).

245 ntricular leukomalacia, surgical necrotizing enterocolitis, or stage 3 or greater retinopathy of prem

246 Interestingly, we find that necrotizing enterocolitis patients also exhibit decreased expression

247 10/IL-10R-deficient patients had intractable enterocolitis, perianal disease, and fistula formation.

248 re thrombocytopenia (eg, sepsis, necrotizing enterocolitis, perinatal asphyxia, and the immune thromb

249 unreported syndrome featuring neonatal-onset enterocolitis, periodic fever, and fatal or near-fatal e

250 diographic evidence of extensive necrotizing enterocolitis (pneumatosis intestinalis), gestational ag

251 he adjusted network incidence of necrotising enterocolitis ranged from 2.51% (95% CI 1.13-3.60) to 3.

252 s system injury decreased, while necrotizing enterocolitis-related deaths increased.

253 Infants with necrotizing enterocolitis represent a high-risk subgroup of the very

254 Cases presented with severe apoptotic enterocolitis resembling acute intestinal graft-versus-h

255 compared with 11% and 2% in patients without enterocolitis, respectively (P = .0065 for MM and P = .0

256 Most patients who developed enterocolitis responded to high-dose systemic corticoste

257 h or bronchopulmonary dysplasia, necrotizing enterocolitis, retinopathy of prematurity, and severe in

258 neonatal morbidities, including necrotizing enterocolitis, retinopathy of prematurity, bronchopulmon

259 ome, and significance of the immune-mediated enterocolitis seen with ipilimumab is presented.

260 also decreases the prevalence of necrotizing enterocolitis, sepsis, and intraventricular hemorrhage (

261 antly affects risk of mortality, necrotizing enterocolitis, sepsis, chronic lung disease, intraventri

262 No differences in rates of necrotizing enterocolitis, severe bronchopulmonary dysplasia, or sev

263 hage but not with differences in necrotizing enterocolitis, severe bronchopulmonary dysplasia, or sev

264 f prematurity requiring surgery, necrotizing enterocolitis, spontaneous intestinal perforation, and n

265 maturity (stage 3 or higher), or necrotizing enterocolitis (stages 2-3).

266 The estimated numbers of admissions for enterocolitis suggest an increasing trend from 466 to 40

267 Food protein-induced enterocolitis syndrome (FPIES) is a non-IgE-mediated foo

268 Food protein-induced enterocolitis syndrome (FPIES) is a non-IgE-mediated foo

269 Food protein-induced enterocolitis syndrome (FPIES) is a non-IgE-mediated foo

270 Food protein-induced enterocolitis syndrome (FPIES) is a non-IgE-mediated gas

271 Food protein-induced enterocolitis syndrome (FPIES) is a non-IgE-mediated gas

272 oderate to severe acute food protein-induced enterocolitis syndrome (FPIES) typically consists of int

273 and pathophysiology of food protein-induced enterocolitis syndrome (FPIES), an under-recognized and

274 d allergies and include food protein-induced enterocolitis syndrome (FPIES), food protein-induced all

275 l food allergy known as food protein-induced enterocolitis syndrome (FPIES), with several recent publ

276 nique entities, such as food protein-induced enterocolitis syndrome with acute presentation.

277 , pollen food syndrome, food-protein-induced enterocolitis syndrome, food-induced proctocolitis, eosi

278 ssociated with susceptibility to spontaneous enterocolitis that is microbiome dependent.

279 The frequencies of air leaks and necrotizing enterocolitis, the duration of respiratory support, and

280 bacteria may reduce the rate of necrotizing enterocolitis, the severity of necrotizing enterocolitis

281 , reports regarding the risk for necrotizing enterocolitis, the utility of lactate as an index of sys

282 The former effect occurs in necrotizing enterocolitis; the latter influence is seen in septic pa

283 nfants, 3586 stools, 46 cases of necrotising enterocolitis), there were increased proportions of Gamm

284 erleukin (IL)10-null mice are susceptible to enterocolitis, they maintained the same body weight as t

285 range of disease manifestations ranging from enterocolitis to typhoid fever.

286 y (the UK Neonatal Collaborative Necrotising Enterocolitis [UKNC-NEC] Study) of babies born in Englan

287 sms against intestinal pathogens, Salmonella enterocolitis (using Salmonella enterica serovar Typhimu

288 its absence, mice develop aggravated chronic enterocolitis via an imbalance of colitogenic Th1 cells

289 Chronic enterocolitis was induced by the transfer of wild-type o

290 tricular hemorrhage and death or necrotizing enterocolitis was lowest among infants born in hospitals

291 The occurrence of necrotizing enterocolitis was significantly higher in the control gr

292 us parents with severe exfoliative apoptotic enterocolitis; we also detected TTC7A mutations in 2 unr

293 ective tumor response rates in patients with enterocolitis were 36% for MM and 35% for RCC, compared

294 , just 28.6% of the infants with necrotizing enterocolitis were born into high-level, high-volume hos

295 If vaccines preventing enterocolitis were to be developed, they would likely pr

296 d colitis and Salmonella typhimurium-induced enterocolitis were used as animal models to study coliti

297 pidemic of poultry-associated, self-limiting enterocolitis, whereas in sub-Saharan Africa it is a maj

298 Chorioamnionitis, necrotizing enterocolitis, white matter injury on cranial ultrasound

299 ted rabbits included erosive and necrotizing enterocolitis with adherent bacterial rods, proliferativ

300 a 5-month-old female infant who had allergic enterocolitis with protein-losing enteropathy and had lo