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1 olate cotransporting polypeptide, within the enterohepatic circulation.
2 ossing biological membranes, and clearing by enterohepatic circulation.
3 ed rats and mice, and in mice with an intact enterohepatic circulation.
4 al CFTR(inh)-172 accumulation facilitated by enterohepatic circulation.
5 a measurement (one patient) is suggestive of enterohepatic circulation.
6 ll intestine and only in animals with intact enterohepatic circulation.
7 e quantity of bile salts fluxing through the enterohepatic circulation.
8 nal selective FXR reactivation normalized BA enterohepatic circulation along with up-regulation of in
9 transformation enzyme activities, changes in enterohepatic circulation, altered bioavailability of en
12 results suggest that systemic alterations in enterohepatic circulation, as well as host and microbiot
13 acid (BA) synthesis and transport within the enterohepatic circulation has revealed potential targets
15 bile salts, a critical determinant of their enterohepatic circulation, is mediated primarily by the
16 ects of bile acids on tissues outside of the enterohepatic circulation may be of major pathophysiolog
18 We analyzed expressions of factors mediating enterohepatic circulation of BA using ileal and colonic
19 r, they suggest a potential role for altered enterohepatic circulation of BAs in improving insulin se
20 tic BA uptake machinery maintains a (slower) enterohepatic circulation of BAs, although it is occasio
23 n of bile acids, a rate-limiting step in the enterohepatic circulation of bile acids and transactivat
26 l diarrhea, steatorrhea, interruption of the enterohepatic circulation of bile acids, and reduced pla
27 hASBT, SLC10A2) plays a critical role in the enterohepatic circulation of bile acids, as well as in c
34 oduct is likely to play an essential role in enterohepatic circulation of bile acids; further charact
35 transporter (ASBT, SLC10A2) facilitates the enterohepatic circulation of bile salts and plays a key
37 nse of the transport process involved in the enterohepatic circulation of bile salts to obstructive c
43 ted charcoal, to interrupt enterovascular or enterohepatic circulations, offers benefit compared with
45 gation of bile acids entering liver from the enterohepatic circulation rather than in de novo bile ac
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