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1 olate cotransporting polypeptide, within the enterohepatic circulation.
2 ossing biological membranes, and clearing by enterohepatic circulation.
3 ed rats and mice, and in mice with an intact enterohepatic circulation.
4 al CFTR(inh)-172 accumulation facilitated by enterohepatic circulation.
5 a measurement (one patient) is suggestive of enterohepatic circulation.
6 ll intestine and only in animals with intact enterohepatic circulation.
7 e quantity of bile salts fluxing through the enterohepatic circulation.
8 nal selective FXR reactivation normalized BA enterohepatic circulation along with up-regulation of in
9 transformation enzyme activities, changes in enterohepatic circulation, altered bioavailability of en
10           (11)C-cholylsarcosine underwent an enterohepatic circulation and reappeared in liver tissue
11 terocyte, along with the contribution of the enterohepatic circulation, are considered.
12 results suggest that systemic alterations in enterohepatic circulation, as well as host and microbiot
13 acid (BA) synthesis and transport within the enterohepatic circulation has revealed potential targets
14          Cholate feeding to rats with intact enterohepatic circulation increased mdr2 transcriptional
15  bile salts, a critical determinant of their enterohepatic circulation, is mediated primarily by the
16 ects of bile acids on tissues outside of the enterohepatic circulation may be of major pathophysiolog
17                                    Thus, the enterohepatic circulation of all conjugated bile acids w
18 We analyzed expressions of factors mediating enterohepatic circulation of BA using ileal and colonic
19 r, they suggest a potential role for altered enterohepatic circulation of BAs in improving insulin se
20 tic BA uptake machinery maintains a (slower) enterohepatic circulation of BAs, although it is occasio
21 defects in gallbladder emptying that disrupt enterohepatic circulation of BAs.
22  in the distal ileum plays a key role in the enterohepatic circulation of BAs.
23 n of bile acids, a rate-limiting step in the enterohepatic circulation of bile acids and transactivat
24                                          The enterohepatic circulation of bile acids is maintained by
25                          Interruption of the enterohepatic circulation of bile acids leads to increas
26 l diarrhea, steatorrhea, interruption of the enterohepatic circulation of bile acids, and reduced pla
27 hASBT, SLC10A2) plays a critical role in the enterohepatic circulation of bile acids, as well as in c
28 and intestine and controls the synthesis and enterohepatic circulation of bile acids.
29 ns (ILBPs) are involved in the transport and enterohepatic circulation of bile acids.
30  acid amidation, a critical component of the enterohepatic circulation of bile acids.
31  bile acid cotransporter participates in the enterohepatic circulation of bile acids.
32 y pump, a critical component involved in the enterohepatic circulation of bile acids.
33 otransporter superfamily and function in the enterohepatic circulation of bile acids.
34 oduct is likely to play an essential role in enterohepatic circulation of bile acids; further charact
35  transporter (ASBT, SLC10A2) facilitates the enterohepatic circulation of bile salts and plays a key
36                                          The enterohepatic circulation of bile salts is an important
37 nse of the transport process involved in the enterohepatic circulation of bile salts to obstructive c
38 g activity of FGF19 in organs engaged in the enterohepatic circulation of bile salts.
39 roteins and is thought to play a role in the enterohepatic circulation of bile salts.
40  function in the transcellular transport and enterohepatic circulation of bile salts.
41               We propose the existence of an enterohepatic circulation of lymphocytes, whereby some m
42 fecal excretion, suggests the possibility of enterohepatic circulation of this drug.
43 ted charcoal, to interrupt enterovascular or enterohepatic circulations, offers benefit compared with
44 to 57% +/- 5% of paired controls with intact enterohepatic circulation (P < 0.0001).
45 gation of bile acids entering liver from the enterohepatic circulation rather than in de novo bile ac

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