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2 hown to be manipulated during infection with enteropathogenic and enterohaemorrhagic Escherichia coli
3 n infections with the attaching and effacing enteropathogenic and enterohaemorrhagic Escherichia coli
7 uman and animal enteric pathogens, including enteropathogenic and enterohaemorrhagic Escherichia coli
9 ane localization domains of intimin (Int) of enteropathogenic and enterohaemorrhagic Escherichia coli
10 molecule involved in intimate attachment of enteropathogenic and enterohaemorrhagic Escherichia coli
11 secreted by the type III secretion system of enteropathogenic and enterohemorrhagic Escherichia coli
14 lasia and colitis and is used as a model for enteropathogenic and enterohemorrhagic Escherichia coli
16 lence of the closely related human pathogens enteropathogenic and enterohemorrhagic Escherichia coli
17 oC SboC and SeoC are homologues of EspJ from enteropathogenic and enterohemorrhagic Escherichia coli
18 hing and effacing mouse pathogen that models enteropathogenic and enterohemorrhagic Escherichia coli
19 rocyte effacement-encoded regulator (Ler) of enteropathogenic and enterohemorrhagic Escherichia coli
21 cus of enterocyte effacement (LEE), a PAI of enteropathogenic and enterohemorrhagic Escherichia coli.
22 e occasions within the different lineages of enteropathogenic and enterohemorrhagic Escherichia coli.
23 ntium is a natural mouse pathogen related to enteropathogenic and enterohemorrhagic Escherichia coli.
24 ses resembling those of humans infected with enteropathogenic and enterohemorrhagic Escherichia coli.
26 e investigated the effects of nonpathogenic, enteropathogenic, and probiotic bacteria on the dynamics
27 sing chaperone HdeA promotes the survival of enteropathogenic bacteria during transit through the har
29 P are less prevalent and are mainly found in enteropathogenic bacteria, where they play key roles in
32 t successful establishment in the gut by the enteropathogenic bacterium Salmonella enterica serovar T
36 and histological data characterizing a novel enteropathogenic BEC strain, NB, detected in fecal speci
38 ater secretion, antibiotic prescription, and enteropathogenic colonization, each of which involves an
39 Porcine epidemic diarrhea virus (PEDV) is an enteropathogenic coronavirus causing lethal watery diarr
41 hazard ratio [HR] 1.9; 0.99-3.5) and typical enteropathogenic E coli (HR 2.6; 1.6-4.1) in infants age
43 Shigella, ST-ETEC, Cryptosporidium, typical enteropathogenic E coli) can substantially reduce the bu
44 t hybridization, strains were categorized as enteropathogenic E. coli (1.8%), enterotoxigenic E. coli
45 nterohemorrhagic Escherichia coli (EHEC) and enteropathogenic E. coli (EPEC) adhesion to cultured hum
46 a coli genome were assessed in 21 strains of enteropathogenic E. coli (EPEC) and enterohemorrhagic E.
47 that cause significant human disease are the enteropathogenic E. coli (EPEC) and enterotoxigenic E. c
48 compared these TIR interactions in EHEC and enteropathogenic E. coli (EPEC) and found that five inte
49 Detailed analysis of the H-NST proteins from enteropathogenic E. coli (EPEC) and uropathogenic E. col
50 Enterotoxigenic Escherichia coli (ETEC) and enteropathogenic E. coli (EPEC) are common causes of dia
51 nterohemorrhagic Escherichia coli (EHEC) and enteropathogenic E. coli (EPEC) are enteric bacterial pa
52 beled Shiga toxin-producing E. coli; whereas enteropathogenic E. coli (EPEC) are LEE+ and often carry
57 EC O157:H7 serotype arose from its ancestor, enteropathogenic E. coli (EPEC) O55:H7 (sorbitol ferment
58 menal bacteria, (3) prevent the adherence of enteropathogenic E. coli (EPEC) to epithelial monolayers
59 detect the role of phenotypic variability in enteropathogenic E. coli (EPEC), an important human path
60 s enterohemorrhagic Escherichia coli (EHEC), enteropathogenic E. coli (EPEC), and Citrobacter rodenti
62 er enterohemorrhagic E. coli (EHEC), typical enteropathogenic E. coli (EPEC), or atypical EPEC, depen
63 coli (EAEC), enterotoxigenic E. coli (ETEC), enteropathogenic E. coli (EPEC), Shigella spp., Campylob
66 15:H-) and compared it with those from human enteropathogenic E. coli (EPEC, O127:H6) and enterohemor
68 on was found between norovirus GII + typical enteropathogenic E. coli (OR = 0.09, 95% CI: 0.01, 0.95)
69 osubstrate, also significantly decreased the enteropathogenic E. coli -associated decrease in transep
70 a diverse array of clonal groups, including enteropathogenic E. coli 2 (EPEC 2), enterohemorrhagic E
71 coli [EAEC], enterotoxigenic E. coli [ETEC], enteropathogenic E. coli [EPEC], and Shiga-toxigenic E.
72 ntial regulation of protein kinase C-zeta by enteropathogenic E. coli and enterohemorrhagic E. coli m
73 tion of tight junctions after infection with enteropathogenic E. coli and enterohemorrhagic E. coli.
74 and lt for enterotoxigenic E. coli, eaeA for enteropathogenic E. coli and Shiga toxin-producing E. co
76 ithelial tight junctions, but the effects of enteropathogenic E. coli are more profound than those of
77 and probably contributes to the survival of enteropathogenic E. coli during the intestinal inflammat
78 activated luxS in three E. coli backgrounds: enteropathogenic E. coli E2348-69, and enterohaemorrhagi
79 found that uropathogenic E. coli CFT073 and enteropathogenic E. coli E2348/69 occupy intestinal nich
82 fall into any of the four classical EHEC and enteropathogenic E. coli groups but instead was closely
88 We constructed a tightly inducible clone of enteropathogenic E. coli O127:H6 lifA for affinity purif
90 g the O157:H7 serotype and are found in some enteropathogenic E. coli O55:H7 strains but are absent f
91 intestinal epithelial monolayers infected by enteropathogenic E. coli or enterohemorrhagic E. coli we
92 stal structure of the intimin-Tir complex of enteropathogenic E. coli predicts that each of these fou
94 esolution crystal structures of Gmm from the enteropathogenic E. coli strain O128: the structure of t
95 lian cells that had been preinfected with an enteropathogenic E. coli strain that expresses Tir but n
97 employed two proteins, intimin and tir, from enteropathogenic E. coli that are critical to the bacter
98 equenced pathovars, enterotoxigenic E. coli, enteropathogenic E. coli, and enteroaggregative E. coli.
99 fficile, enteroaggregative Escherichia coli, enteropathogenic E. coli, and enterotoxigenic E. coli),
100 O1, enterotoxigenic Escherichia coli (ETEC), enteropathogenic E. coli, Campylobacter jejuni, and Giar
101 ral pathogenic strains of E. coli, including enteropathogenic E. coli, enterohemorrhagic E. coli, ent
102 a enterocolitica, enteroaggregative E. coli, enteropathogenic E. coli, enterotoxigenic E. coli, Shiga
103 c from EAEC and Shigella flexneri, EspC from enteropathogenic E. coli, EspP from enterohemorrhagic E.
104 aggregative Escherichia coli, Campylobacter, enteropathogenic E. coli, rotavirus, and Entamoeba histo
105 ichia coli, particularly enterotoxigenic and enteropathogenic E. coli, rotavirus, Giardia lamblia, an
113 ded enterotoxigenic Escherichia coli (ETEC), enteropathogenic E.coli (EPEC), Listeria monocytogenes,
114 three CPE-positive type C EN strains caused enteropathogenic effects in rabbit small intestinal loop
115 ing that both CPB and CPE are needed for the enteropathogenic effects of CN3758 MDS lysate supernatan
117 nsequently, CPE and CPB contributions to the enteropathogenic effects of MDS lysate supernatants of C
118 t or reversing the cpb mutation restored the enteropathogenic effects of MDS lysate supernatants.
120 ell as lower proportions of enterotoxigenic, enteropathogenic, enterohemorrhagic, and commensal E. co
124 The human pathogens enterohemorrhagic and enteropathogenic Escherichia coli (EHEC and EPEC), as we
125 ith SadA from Salmonella enterica, EhaG from enteropathogenic Escherichia coli (EHEC), and UpaG from
126 eotide sequence was determined for pMAR7, an enteropathogenic Escherichia coli (EPEC) adherence facto
127 of attaching and effacing pathogens such as enteropathogenic Escherichia coli (EPEC) and Citrobacter
129 It had been suggested that the flagella of enteropathogenic Escherichia coli (EPEC) and enterohemor
134 Regulation of virulence gene expression in enteropathogenic Escherichia coli (EPEC) and enterohemor
136 on of several important virulence factors in enteropathogenic Escherichia coli (EPEC) and reduced EPE
137 ues and fluids in response to infection with enteropathogenic Escherichia coli (EPEC) and Shiga-toxig
143 The type IV bundle-forming pili (BFP) of enteropathogenic Escherichia coli (EPEC) are required fo
144 ude two hydrophobic proteins, represented in enteropathogenic Escherichia coli (EPEC) by EspB and Esp
152 lifA, for lymphocyte inhibitory factor A) in enteropathogenic Escherichia coli (EPEC) encoding a prot
153 The attaching and effacing (A/E) pathogen enteropathogenic Escherichia coli (EPEC) forms character
155 The type III secretion system (TTSS) of enteropathogenic Escherichia coli (EPEC) has been associ
156 yte effacement (LEE) pathogenicity island of enteropathogenic Escherichia coli (EPEC) has not been de
179 The plasmid-encoded Per regulatory locus of enteropathogenic Escherichia coli (EPEC) is generally co
181 diarrhea induced by the food-borne pathogen enteropathogenic Escherichia coli (EPEC) is not known.
185 rial pathogens like Salmonella, Shigella and enteropathogenic Escherichia coli (EPEC) is the transloc
186 initial steps in biofilm development, and in enteropathogenic Escherichia coli (EPEC) it is mediated
189 Although the bundle-forming pilus (BFP) of enteropathogenic Escherichia coli (EPEC) mediates microc
190 ttens that were presumptively diagnosed with enteropathogenic Escherichia coli (EPEC) on the basis of
196 Production of type IV bundle-forming pili by enteropathogenic Escherichia coli (EPEC) requires BfpB,
197 tion of type IV bundle-forming pili (BFP) by enteropathogenic Escherichia coli (EPEC) requires the pr
201 and virulence-associated -components in the enteropathogenic Escherichia coli (EPEC) strain E2348/69
202 gative transfer system identified in O119:H2 enteropathogenic Escherichia coli (EPEC) strain MB80 by
206 n, CesT, serves a chaperone function for the enteropathogenic Escherichia coli (EPEC) translocated in
211 Here, we report that the bacterial pathogen enteropathogenic Escherichia coli (EPEC) uses the type I
212 Outer membrane intimin directs attachment of enteropathogenic Escherichia coli (EPEC) via its Tir rec
214 to inhibit attachment of microcolony-forming enteropathogenic Escherichia coli (EPEC) was investigate
217 ive proteins are secreted extracellularly by enteropathogenic Escherichia coli (EPEC), a leading caus
224 ns and actin polymerization, the hallmark of enteropathogenic Escherichia coli (EPEC), enterohemorrha
225 es, collected semimonthly, were screened for enteropathogenic Escherichia coli (EPEC), enterotoxigeni
226 ded type IV bundle-forming pilus produced by enteropathogenic Escherichia coli (EPEC), has recently b
229 processes as well as actin-based motility of enteropathogenic Escherichia coli (EPEC), vaccinia, and
230 e factor in two groups of enteric pathogens: enteropathogenic Escherichia coli (EPEC), which is a maj
237 mmon organisms detected by the GI panel were enteropathogenic Escherichia coli (EPEC, n = 21), norovi
238 nding cis-complemented derivatives of rabbit enteropathogenic Escherichia coli and compared their abi
239 derstanding of the molecular pathogenesis of enteropathogenic Escherichia coli and enterohemorrhagic
241 related clinically important human pathogens enteropathogenic Escherichia coli and enterohemorrhagic
243 s homologous to those of the human pathogens enteropathogenic Escherichia coli and enterohemorrhagic
244 n secretion and translocation from wild-type enteropathogenic Escherichia coli and hypersecretion fro
246 homology to type III secreted proteins from enteropathogenic Escherichia coli and Yersinia and, base
247 Using the bundle-forming pilus (BFP) of enteropathogenic Escherichia coli as a model Tfp system,
248 We purified the PulE homologue BfpD of the enteropathogenic Escherichia coli bundle-forming pilus (
250 gens such as Yersinia pseudotuberculosis and enteropathogenic Escherichia coli disarm host cells by i
252 rt the 1.9 A resolution crystal structure of enteropathogenic Escherichia coli GfcC, a periplasmic pr
254 cted with Citrobacter rodentium, a model for enteropathogenic Escherichia coli infection in humans, t
255 acter rodentium infection, a mouse model for enteropathogenic Escherichia coli infection, Hvem-/- mic
256 secretion system effector protein NleE from enteropathogenic Escherichia coli plays a key role in th
257 orum sensing to TTS in enterohemorrhagic and enteropathogenic Escherichia coli show that quorum sensi
258 entium uses virulence factors similar to the enteropathogenic Escherichia coli to produce attaching a
261 zyme in the interaction between the host and enteropathogenic Escherichia coli(EPEC) and Shiga-toxige
263 cellular (S. Typhimurium) and extracellular (enteropathogenic Escherichia coli) enteric pathogens, vi
264 perone-delivered to the translocase, EscV in enteropathogenic Escherichia coli, and cross it in stric
265 bacter rodentium is the rodent equivalent of enteropathogenic Escherichia coli, and it causes colitis
269 and Campylobacter coli, Cryptosporidium spp, enteropathogenic Escherichia coli, heat-stable enterotox
270 rodentium, a murine model pathogen for human enteropathogenic Escherichia coli, predominantly coloniz
271 how that important other pathogens including enteropathogenic Escherichia coli, Shigella flexneri, an
279 d that extrinsic stress signals generated by enteropathogenic infection are epistatic to the hypoplas
281 lacks the major adhesins and invasins of its enteropathogenic relatives Yersinia enterocolitica and Y
283 in, the major proinflammatory determinant of enteropathogenic Salmonella, which was found to be glyco
285 the plague bacillus Yersinia pestis and two enteropathogenic species, Yersinia pseudotuberculosis an
287 he identification of a large gene present in enteropathogenic strains of Escherichia coli (EPEC) that
288 y cascade as the primary regulon controlling enteropathogenic virulence functions in S. typhimurium.
289 not clear why these avian coronaviruses are enteropathogenic, whereas other closely related avian co
290 ed to mount a robust immune response against enteropathogenic Y. pseudotuberculosis by promoting Th17
291 es) do not display altered susceptibility to enteropathogenic Yersinia compared to wild-type mice.
293 plasmids (pCD in Yersinia pestis and pYV in enteropathogenic Yersinia pseudotuberculosis and Yersini
295 es have demonstrated that TTSS expression in enteropathogenic Yersinia spp. also inhibits the respira
296 oonotic pathogen hepatitis E virus (HEV) and enteropathogenic Yersinia spp. were analyzed in parallel
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