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1 osure of calpastatin-overexpressing cells to enteropathogenic Escherichia coli.
2 and were reported to contain the eae gene of enteropathogenic Escherichia coli.
3 and one is similar to the AIDA-I adhesin of enteropathogenic Escherichia coli.
4 o gastric epithelial cells resembles that of enteropathogenic Escherichia coli.
5 causes disease similar to the human pathogen enteropathogenic Escherichia coli.
6 rmation of attaching and effacing lesions by enteropathogenic Escherichia coli.
7 uptake as well as Cdc42-dependent uptake of enteropathogenic Escherichia coli.
8 shigella, salmonella, Yersinia species, and enteropathogenic Escherichia coli.
9 he attachment and effacement associated with enteropathogenic Escherichia coli.
10 iated with the type IV pilus gene cluster of enteropathogenic Escherichia coli, a transposase from Vi
12 nding cis-complemented derivatives of rabbit enteropathogenic Escherichia coli and compared their abi
14 s homologous to those of the human pathogens enteropathogenic Escherichia coli and enterohemorrhagic
15 derstanding of the molecular pathogenesis of enteropathogenic Escherichia coli and enterohemorrhagic
17 related clinically important human pathogens enteropathogenic Escherichia coli and enterohemorrhagic
18 n secretion and translocation from wild-type enteropathogenic Escherichia coli and hypersecretion fro
20 homology to type III secreted proteins from enteropathogenic Escherichia coli and Yersinia and, base
21 perone-delivered to the translocase, EscV in enteropathogenic Escherichia coli, and cross it in stric
22 bacter rodentium is the rodent equivalent of enteropathogenic Escherichia coli, and it causes colitis
26 We purified the PulE homologue BfpD of the enteropathogenic Escherichia coli bundle-forming pilus (
29 ity island' from the prototype AE bacterium, enteropathogenic Escherichia coli, containing all previo
30 gens such as Yersinia pseudotuberculosis and enteropathogenic Escherichia coli disarm host cells by i
32 intimate attachment of enterohemorrhagic and enteropathogenic Escherichia coli (EHEC and EPEC) to mam
33 The human pathogens enterohemorrhagic and enteropathogenic Escherichia coli (EHEC and EPEC), as we
34 ith SadA from Salmonella enterica, EhaG from enteropathogenic Escherichia coli (EHEC), and UpaG from
36 cellular (S. Typhimurium) and extracellular (enteropathogenic Escherichia coli) enteric pathogens, vi
41 eotide sequence was determined for pMAR7, an enteropathogenic Escherichia coli (EPEC) adherence facto
43 l invasion by pathogenic bacteria, including enteropathogenic Escherichia coli (EPEC) and Citrobacter
44 of attaching and effacing pathogens such as enteropathogenic Escherichia coli (EPEC) and Citrobacter
50 Regulation of virulence gene expression in enteropathogenic Escherichia coli (EPEC) and enterohemor
52 It had been suggested that the flagella of enteropathogenic Escherichia coli (EPEC) and enterohemor
53 on of several important virulence factors in enteropathogenic Escherichia coli (EPEC) and reduced EPE
54 ues and fluids in response to infection with enteropathogenic Escherichia coli (EPEC) and Shiga-toxig
60 The type IV bundle-forming pili (BFP) of enteropathogenic Escherichia coli (EPEC) are required fo
61 ude two hydrophobic proteins, represented in enteropathogenic Escherichia coli (EPEC) by EspB and Esp
71 lifA, for lymphocyte inhibitory factor A) in enteropathogenic Escherichia coli (EPEC) encoding a prot
72 The attaching and effacing (A/E) pathogen enteropathogenic Escherichia coli (EPEC) forms character
75 yte effacement (LEE) pathogenicity island of enteropathogenic Escherichia coli (EPEC) has not been de
79 rs were performed to determine whether prior enteropathogenic Escherichia coli (EPEC) infection confe
106 The plasmid-encoded Per regulatory locus of enteropathogenic Escherichia coli (EPEC) is generally co
108 diarrhea induced by the food-borne pathogen enteropathogenic Escherichia coli (EPEC) is not known.
109 ression of the bundle-forming pilus (BFP) of enteropathogenic Escherichia coli (EPEC) is regulated at
114 rial pathogens like Salmonella, Shigella and enteropathogenic Escherichia coli (EPEC) is the transloc
115 initial steps in biofilm development, and in enteropathogenic Escherichia coli (EPEC) it is mediated
118 Although the bundle-forming pilus (BFP) of enteropathogenic Escherichia coli (EPEC) mediates microc
119 ttens that were presumptively diagnosed with enteropathogenic Escherichia coli (EPEC) on the basis of
126 Production of type IV bundle-forming pili by enteropathogenic Escherichia coli (EPEC) requires BfpB,
127 tion of type IV bundle-forming pili (BFP) by enteropathogenic Escherichia coli (EPEC) requires the pr
131 on the enteroadherent factor plasmid of the enteropathogenic Escherichia coli (EPEC) strain B171-8 (
132 and virulence-associated -components in the enteropathogenic Escherichia coli (EPEC) strain E2348/69
133 gative transfer system identified in O119:H2 enteropathogenic Escherichia coli (EPEC) strain MB80 by
140 n, CesT, serves a chaperone function for the enteropathogenic Escherichia coli (EPEC) translocated in
145 Here, we report that the bacterial pathogen enteropathogenic Escherichia coli (EPEC) uses the type I
146 Outer membrane intimin directs attachment of enteropathogenic Escherichia coli (EPEC) via its Tir rec
148 to inhibit attachment of microcolony-forming enteropathogenic Escherichia coli (EPEC) was investigate
151 ive proteins are secreted extracellularly by enteropathogenic Escherichia coli (EPEC), a leading caus
158 ns and actin polymerization, the hallmark of enteropathogenic Escherichia coli (EPEC), enterohemorrha
159 es, collected semimonthly, were screened for enteropathogenic Escherichia coli (EPEC), enterotoxigeni
160 ded type IV bundle-forming pilus produced by enteropathogenic Escherichia coli (EPEC), has recently b
161 creen outer membrane proteins from 50 rabbit enteropathogenic Escherichia coli (EPEC), human EPEC, an
165 processes as well as actin-based motility of enteropathogenic Escherichia coli (EPEC), vaccinia, and
166 e factor in two groups of enteric pathogens: enteropathogenic Escherichia coli (EPEC), which is a maj
167 E) lesions is central to the pathogenesis of enteropathogenic Escherichia coli (EPEC)-mediated diseas
177 mmon organisms detected by the GI panel were enteropathogenic Escherichia coli (EPEC, n = 21), norovi
178 zyme in the interaction between the host and enteropathogenic Escherichia coli(EPEC) and Shiga-toxige
181 rt the 1.9 A resolution crystal structure of enteropathogenic Escherichia coli GfcC, a periplasmic pr
183 and Campylobacter coli, Cryptosporidium spp, enteropathogenic Escherichia coli, heat-stable enterotox
184 cted with Citrobacter rodentium, a model for enteropathogenic Escherichia coli infection in humans, t
185 acter rodentium infection, a mouse model for enteropathogenic Escherichia coli infection, Hvem-/- mic
186 secretion system effector protein NleE from enteropathogenic Escherichia coli plays a key role in th
187 rodentium, a murine model pathogen for human enteropathogenic Escherichia coli, predominantly coloniz
188 s an attaching and effacing strain of rabbit enteropathogenic Escherichia coli (REPEC) that causes di
189 how that important other pathogens including enteropathogenic Escherichia coli, Shigella flexneri, an
190 orum sensing to TTS in enterohemorrhagic and enteropathogenic Escherichia coli show that quorum sensi
192 enterocyte effacement (LEE) is necessary for enteropathogenic Escherichia coli to cause characteristi
193 entium uses virulence factors similar to the enteropathogenic Escherichia coli to produce attaching a
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