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1 rates intestinal radiation injury (radiation enteropathy).
2 due to severe noninflammatory protein-losing enteropathy.
3 cluding HAART-related adverse events and HIV enteropathy.
4 c disease is a diet-induced, T cell-mediated enteropathy.
5 spected to have an associated protein-losing enteropathy.
6 and histologic evidence of gluten-sensitive enteropathy.
7 ly includes a minority of patients with mild enteropathy.
8 itis, motility, malabsorption, and radiation enteropathy.
9 y drug users develop subclinical small bowel enteropathy.
10 ations of nonsteroidal antiinflammatory drug enteropathy.
11 the key factor in staphylococcal enterotoxin enteropathy.
12 to be correlated with the degree (extent) of enteropathy.
13 ntrol mice or those exhibiting NSAID-induced enteropathy.
14 c event, and 1 (2%) developed protein-losing enteropathy.
15 ular NSAID users may be prone to small bowel enteropathy.
16 or hypoalbuminemia are useful clues to NSAID enteropathy.
17 ovesicular rash and is often associated with enteropathy.
18 t the disease arises secondary to hereditary enteropathy.
19 esents an in vitro model of gluten-sensitive enteropathy.
20 d Bob activation is a plausible cause of HIV enteropathy.
21 l small intestine in a murine model of NSAID enteropathy.
22 contributes substantially to the associated enteropathy.
23 rotein is feasible and ameliorates radiation enteropathy.
24 role for drug adduct formation in diclofenac enteropathy.
25 some infection was primarily associated with enteropathy.
26 ths and no patients developed protein losing enteropathy.
27 hunting, thromboembolism, and protein-losing enteropathy.
28 been additionally diagnosed with celiac-like enteropathy.
29 CeD) is a common gluten-sensitive autoimmune enteropathy.
30 as revealed as an AIE-75-positive autoimmune enteropathy.
31 with HIV status, and also with environmental enteropathy.
32 of, and, ideally, assess interventions for, enteropathy.
33 viously carried out studies on environmental enteropathy.
34 ong-term effects and avoid chronic allograft enteropathy.
35 rization of the pathogenesis of HIV-mediated enteropathy.
36 ymphocytes that may also play a role in some enteropathies.
37 syndrome (SBS) (59%), PNDD (14%), congenital enteropathies (10%), chronic intestinal pseudo-obstructi
41 unction, such as environmental (or tropical) enteropathy, affects zinc absorption, losses, and homeos
44 is factor (TNF) in graft-versus-host disease enteropathy, an adenoviral vector encoding a TNF inhibit
45 cations for the pathogenesis of TNF-mediated enteropathies and chronic inflammatory diseases of the i
47 microbial component, including environmental enteropathy and chronic colitis-associated colorectal ca
48 c has the potential to resolve environmental enteropathy and clear bacterial pathogens, we aimed to a
51 r human X-linked neonatal diabetes mellitus, enteropathy and endocrinopathy syndrome (IPEX; MIM 30493
52 developments in the area of acute radiation enteropathy and examine the current state of knowledge r
54 s to be the predominant isoform in radiation enteropathy and may be more important in the mechanisms
58 despite reducing biomarkers of environmental enteropathy and the prevalence of pathogenic intestinal
61 s herpetiformis (DH) have a gluten-sensitive enteropathy and while on gluten-containing diets have el
64 correlated with all parameters of radiation enteropathy and, after adjusting for radiation dose and
65 sive means of detecting and monitoring NSAID enteropathy (and possibly other gastrointestinal mucosal
66 entified 5 children with polyendocrinopathy, enteropathy, and dermatitis reminiscent of IPEX syndrome
68 t presented with lymphedema, protein loosing enteropathy, and sclerosing cholangitis and was diagnose
69 Immune dysregulation, polyendocrinopathy, enteropathy, and X-linked inheritance (IPEX) is one of a
73 alabsorption caused by chronic environmental enteropathy are associated with growth faltering seen in
74 inal translocation of microbial products and enteropathy as well as alterations in gut bacterial comm
75 trast enhanced ultrasound (CEUS) findings in enteropathy associated T-cell lymphoma (EATL) complicati
78 he intestinal microbiota might contribute to enteropathy associated with use of nonsteroidal anti-inf
85 ac disease) appears to be a manifestation of enteropathy-associated T-cell lymphoma in most cases.
86 tion-to-treat analysis in 252 nodal PTCL and enteropathy-associated T-cell lymphoma patients (excludi
87 negative anaplastic large cell lymphoma, and enteropathy-associated T-cell lymphoma were enrolled.
92 fections and their associated complications, enteropathy, autoimmunity, and lymphoproliferative disor
94 data supporting the notion of protein-losing enteropathy being a consequence of severe iron deficienc
95 the nutritional components of environmental enteropathy by analyzing the specific metabolic and gut-
97 n reduced faecal biomarkers of environmental enteropathy (calprotectin, myeloperoxidase, alpha1-antit
98 t interactions, anorexia, and protein-losing enteropathy can all contribute to the protein-calorie ma
107 mmunodeficiency-associated and environmental enteropathies, celiac disease, inflammatory bowel diseas
109 disease is a gluten-induced immune-mediated enteropathy characterized by a specific genetic genotype
111 eliac disease is a gluten-induced autoimmune enteropathy characterized by the presence of tissue tran
112 l lymphangiectasia (PIL) is a protein-losing enteropathy characterized by tortuous and dilated lymph
113 me, rejection particularly chronic allograft enteropathy continues to be a major barrier to long-term
114 a for prevention and treatment of radiation, enteropathy could become a reality and would be of subst
120 e from a patient with attaching-and-effacing enteropathy displayed the localized adherence attaching-
125 is model, we found that Salmonella-inflicted enteropathy elicits parallel blooms of the pathogen and
126 genital syndrome characterized by autoimmune enteropathy, endocrinopathy, dermatitis, and other autoi
127 a useful drug in the treatment of autoimmune enteropathy, even in patients who have not responded to
128 tinal permeability with diarrhea, called HIV enteropathy, even without enteric opportunistic infectio
129 and allergic manifestations including severe enteropathy, food allergies, atopic dermatitis, hyper-Ig
131 pecimens from patients with gluten-sensitive enteropathy (GSE) (obtained during gluten challenge) as
133 tinal pathogens and associated environmental enteropathy has been proposed to explain this problem.
135 s of heart failure death were protein-losing enteropathy (HR, 7.1; P=0.0043), single morphologically
136 oody diarrhea, malabsorption, protein-losing enteropathy, hypoalbuminemia, and failure to thrive.
137 idosis presenting with severe protein-losing enteropathy, hypoalbuminemia, and proximal myopathy who
140 el syndrome in 12 patients (27%), intestinal enteropathy in 20 patients (44%), and motility disorder
141 NO may therefore be an important mediator of enteropathy in both Th1- and Th2-inducing conditions.
145 wasting syndrome characterized by severe SIV enteropathy in the absence of opportunistic infections.
146 TNF inhibition decreases the severity of enteropathy in the DBA/2 --> B6D2F1 murine model of colo
147 marker and supports a role for environmental enteropathy in the pathogenesis of growth shortfall.
148 a) model of diarrhea, causing characteristic enteropathies, including inflammation, necrosis, and col
149 rated structural manifestations of radiation enteropathy, including radiation injury score (6.5 +/- 0
151 l administration of anti-CD3 ameliorated the enteropathy induced by intraperitoneal injection of the
152 present controlled study, the effect of the enteropathy induced by this organism on the retinal vasc
159 ng the pathophysiology and diagnosis of this enteropathy is the difficulty of obtaining information a
163 Celiac disease (CD), or gluten-sensitive enteropathy, is a common multifactorial disorder resulti
166 sociated autoimmune disease gluten-sensitive enteropathy, leaving little room for a differential envi
169 us, suggesting that EE with superimposed HIV enteropathy may be a distinct pathophysiological conditi
171 Myosin-5B malfunction causes the congenital enteropathy microvillus inclusion disease, underlining i
172 endothelial cells and in vivo in a radiation enteropathy mouse model confirm that genes involved in N
173 s trigger celiac disease (CD), an autoimmune enteropathy occurring in genetically susceptible persons
174 ole in the pathogenesis of acute and chronic enteropathies of dogs, including idiopathic inflammatory
175 wed characteristics typical of proliferative enteropathies of other animals, i.e., intracellular colo
179 findings (eg, heart failure, protein-losing enteropathy, or new arrhythmias), and somatic growth.
183 Heart Association III/IV, or protein-losing enteropathy/plastic bronchitis) 20 years after Fontan wa
184 chronic effusions (n = 4) or protein-losing enteropathy (PLE) (n = 5) after lateral tunnel-type Font
193 ac disease (CD) is an increasingly diagnosed enteropathy (prevalence, 1:200-1:300) that is induced by
195 crolimus (FK506) in patients with autoimmune enteropathy refractory to steroids and cyclosporine.
197 year-old boy presented with a protein-losing enteropathy secondary to a hypertrophic gastropathy.
198 howed that in those patients with ataxia and enteropathy, separate antibody populations react with th
199 ercise capacity, arrhythmias, protein-losing enteropathy, somatic growth retardation, neo-aortic valv
201 Celiac disease (CD) is a gluten-sensitive enteropathy that develops in genetically susceptible ind
202 medications in the world, yet they induce an enteropathy that is associated with high morbidity and m
203 , angiopathic thrombosis, and protein-losing enteropathy (the CHAPLE syndrome) is caused by abnormal
204 sal gene expression contribute to intestinal enteropathy, the role of small noncoding RNAs, specifica
205 entricular failure, cyanosis, protein-losing enteropathy, thromboembolism, and dysrhythmias often lea
206 g-term follow-up of patients with autoimmune enteropathy treated with tacrolimus is currently availab
209 Celiac disease (CD) is an immune-mediated enteropathy triggered by gluten in genetically susceptib
210 Celiac disease (CD) is an immune-mediated enteropathy triggered by the ingestion of gluten-contain
212 treatment of 31 patients with a diagnosis of enteropathy-type intestinal T-cell lymphoma treated at t
214 s, showed that in addition to protein-losing enteropathy, ventricular indexed end-diastolic volume >1
218 ith clinical parameters only, protein-losing enteropathy was associated with transplantation-free sur
221 ium and the causative agent of proliferative enteropathy, was developed using an Original Space Bag i
222 In contrast the patient with an autoimmune enteropathy, was HLA-DQ9/DQ6-positive, also arguing agai
223 To identify candidate etiologies for AIDS enteropathy, we used next-generation sequencing to defin
224 ss, and physiological changes resembling HIV enteropathy were previously found in the HT-29 intestina
226 ncy virus (SIV) infection is associated with enteropathy, which likely contributes to AIDS progressio
227 Three patients with diagnosed autoimmune enteropathy who continued to have intractable diarrhea d
228 DDs) are a collection of rare, heterogeneous enteropathies with early onset and often severe outcomes
229 nclusion disease (MVID) is a rare intestinal enteropathy with an onset within a few days to months af
230 iarrhea caused by early-onset protein-losing enteropathy with primary intestinal lymphangiectasia, ed
231 The clinical effects of gluten-sensitive enteropathy with villous atrophy limited to the duodenal
234 of immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) and IPEX-related disorders.
235 Immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome is a rare, fatal a
236 th immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome, associated with a
237 ith immunodysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome, the human disease
238 de immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome, which is caused b
241 Immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX; OMIM 304930) syndrome is a
242 he immune dysregulation, polyendocrinopathy, enteropathy, X-linked syndrome (IPEX) and found them to
243 of immune dysregulation, polyendocrinopathy, enteropathy, X-linked syndrome (IPEX) with or without FO
246 the immune dysregulation polyendocrinopathy, enteropathy, X-linked syndrome, or X-linked autoimmunity
248 me; immunodysregulation, polyendocrinopathy, enteropathy, X-linked syndrome; Crohn's disease; and fam
251 X (immune dysregulation, polyendocrinopathy, enteropathy, X-linked), caused by a lack of regulatory T
252 d an immune dysregulation-polyendocrinopathy-enteropathy-X-linked (IPEX)-like phenotype for STAT1 mut
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