ライフサイエンスコーパス: enterotoxin

1 ge plasmids, and express both beta-toxin and enterotoxin.

2 duce additional toxins, e.g., beta2 toxin or enterotoxin.

3 eron response, and the first described viral enterotoxin.

4 pendent of the expression of the heat-labile enterotoxin.

5 ween disease and the presence of B. fragilis enterotoxin.

6 ory therapy of diarrheas caused by bacterial enterotoxins.

7 etween asthma and exposure to staphylococcal enterotoxins.

8 ntestinal receptor for bacterial heat-stable enterotoxins.

9 viral virulence factor from other microbial enterotoxins.

10 rheas caused by cholera and Escherichia coli enterotoxins.

11 s are the major receptors for staphylococcal enterotoxins.

12 inhibitors, second messengers, and bacterial enterotoxins.

13 ificantly reduced in animals exposed to both enterotoxins.

14 of simultaneous exposure to these two potent enterotoxins.

15 jor virulence factor of the strain, Shigella enterotoxin 1, H4 flagellin, and O104 lipopolysaccharide

16 3 decades, while detection of staphylococcal enterotoxin A (SEA) and toxic shock syndrome toxin (TSST

17 ated the effect of intranasal staphylococcal enterotoxin A (SEA) exposure on murine lung.

18 bacterial isolates containing staphylococcal enterotoxin A (SEA) from the affected skin of CTCL patie

19 reference materials (RMs) for Staphylococcal enterotoxin A (SEA) in cheese.

20 l agents; and PCR analysis of staphylococcal enterotoxin A (SEA) to SEH, toxic shock syndrome toxin 1

21 SM cells pulsed with the SAg, staphylococcal enterotoxin A (SEA), elicited adherence and clustering o

22 Ovalbumin (OVA)-specific, staphylococcal enterotoxin A (SEA)-nonreactive naive CD4 Tcon cells wer

23 , we establish Clostridium difficile and its enterotoxin A (TcdA) as Pyrin-activating agents and show

24 response to the superantigen staphylococcal enterotoxin A on dendritic cells and a reduced number of

25 that after inhalation, Staphylococcus aureus enterotoxin A rapidly entered the bloodstream and induce

26 e model of SIRS that utilizes staphylococcal enterotoxin A specific for Vbeta3(+) T cells, we show th

27 ed innate immunity through the action of the enterotoxin A subunit.

28 terial toxins: cholera toxin, staphylococcal enterotoxin A, and toxic shock syndrome toxin.

29 re found to be 0.01 ng/mL for staphylococcal enterotoxin A, cholera toxin, botulinum toxin A, and ric

30 ying downstream of the TCR in staphylococcal enterotoxin A-specific CD8(+) T cells.

31 genic T cell stimulant Staphylococcus aureus enterotoxin A.

32 Food poisoning due to staphylococcal enterotoxins A and B (SEA and SEB) affects hundreds of t

33 abile toxin, and three S. aureus toxins (the enterotoxins A and B and the toxic shock syndrome toxin)

34 , the limits of detection for staphylococcal enterotoxins A and B, cholera toxin, botulinum toxin A,

35 cation of six protein toxins (staphylococcal enterotoxins A and B, cholera toxin, ricin, botulinum to

36 allenges including new data on the rotavirus enterotoxin, a potential antiviral target.

37 Staphylococcus aureus, called staphylococcal enterotoxins (abbreviated SEA to SEU).

38 human and animal pathogen, and the cytotoxic enterotoxin (Act) is a crucial virulence factor of this

39 Since enterotoxin and beta toxin are produced in the intestine

40 omoted the cytotoxic activity and binding of enterotoxin and beta toxin more strongly than did full-l

41 toxicity-enhancing effects on C. perfringens enterotoxin and beta toxin, which are also important tox

42 mid-encoded toxins, including C. perfringens enterotoxin and beta2 toxin, encoded by the cpe and cbp2

43 mpared to that of an established V. cholerae enterotoxin and Escherichia coli heat-labile enterotoxin

44 Our findings define a role for B. fragilis enterotoxin and its activating protease in the pathogene

45 hemolysin; (2) a group harboring the EAST-1 enterotoxin and the flagellar type H33 but no other prev

46 rhea, produce heat-stable and/or heat-labile enterotoxins and at least 25 different colonization fact

47 the first cells to respond to staphylococcal enterotoxins and contribute to the cytokine production a

48 s both heat-labile (LT) and heat-stable (ST) enterotoxins and is a major cause of diarrhea in infants

49 The genes encoding the enterotoxins and most of the colonization factors are lo

50 The role of enterotoxins and pathogenicity during repeat ETEC infect

51 with S. aureus led to decreased secretion of enterotoxins and phenotypic growth alterations consisten

52 lent strains that produce copious amounts of enterotoxins and spores.

53 ase C (GC-C), the receptor for diarrheagenic enterotoxins and the paracrine ligands guanylin and urog

54 with or without co-expression of heat-labile enterotoxin), and Shigella.

55 on of toxic shock syndrome toxin 1 (TSST-1), enterotoxin, and other superantigens by coagulase-negati

56 , GCC mediates diarrhea induced by bacterial enterotoxins, and an inverse relationship exists between

57 nd we establish that it is stable, expresses enterotoxins, and is not obviously transmissible by phag

58 e of absence of genes encoding for classical enterotoxins, and lack of plasmids encoding genes promot

59 Both types of enterotoxins are regulated at the level of transcription

60 the heat-stable (ST) and/or heat-labile (LT) enterotoxins, as well as surface structures, known as co

61 b had these effects, indicating that type II enterotoxins augment Ab responses by other mechanisms.

62 versus 0.007%; p = 0.04) and staphylococcal enterotoxin B (Fo = 0.49% versus 0.26%; p = 0.04).

63 and IgA antibodies to Staphylococcus aureus enterotoxin B (SAEB) (P = 0.003) in nasal secretions fro

64 Staphylococcal enterotoxin B (SEB) causes food poisoning in humans.

65 We observed that staphylococcal enterotoxin B (SEB) enhanced the IL-4 Der p 1-specific T

66 ere selected for detection of staphylococcal enterotoxin B (SEB) from 77 clinical Staphylococcus aure

67 Using anti-staphylococcal enterotoxin B (SEB) IgG as a "gate" and SEB as an "actua

68 e detection of a model toxin, staphylococcal enterotoxin B (SEB) in buffer, apple juice, and milk is

69 Intranasal exposure to staphylococcal enterotoxin B (SEB) in C57BL/6 wild-type mice caused acu

70 resent the x-ray structure of staphylococcal enterotoxin B (SEB) in complex with its receptors, the T

71 d to improve the detection of Staphylococcal enterotoxin B (SEB) in food.

72 Staphylococcal enterotoxin B (SEB) is a bacterial superantigen that bin

73 Staphylococcal enterotoxin B (SEB) is a potent superantigen that contri

74 Staphylococcal enterotoxin B (SEB) is a potent toxin that can cause tox

75 Staphylococcal enterotoxin B (SEB) is a superantigen known to be a modu

76 Staphylococcal enterotoxin B (SEB) is a superantigen that cross-links t

77 -cell stimulating activity of Staphylococcal enterotoxin B (SEB) is an important factor in the pathog

78 f two recombinantly expressed Staphylococcal Enterotoxin B (SEB) mutants, a single point mutant (Y89A

79 Systemic exposure to staphylococcal enterotoxin B (SEB) rapidly and selectively recruited CD

80 on (IFN) gamma in response to staphylococcal enterotoxin B (SEB) stimulation in 382 healthy infants a

81 When mice were injected with staphylococcal enterotoxin B (SEB) superantigen and H57-597 mAb, the ex

82 ogeneous detection method for staphylococcal enterotoxin B (SEB) utilizing core-shell-structured iron

83 or immunological detection of staphylococcal enterotoxin B (SEB) was designed, fabricated, and tested

84 id and sensitive detection of staphylococcal enterotoxin B (SEB) was developed using a novel acoustic

85 e (SPR) detection signal from staphylococcal enterotoxin B (SEB) was dramatically increased when the

86 Staphylococcal enterotoxin B (SEB), a potential biological warfare agen

87 Staphylococcal enterotoxin B (SEB), a shock-inducing exotoxin synthesiz

88 fringens epsilon toxin (ETX), staphylococcal enterotoxin B (SEB), shiga toxin (STX), and plant toxin

89 role played by superantigens, staphylococcal enterotoxin B (SEB), staphylococcal enterotoxin C (SEC),

90 erparts for responsiveness to staphylococcal enterotoxin B (SEB).

91 icin, cholera toxin (CT), and staphylococcal enterotoxin B (SEB).

92 o neutralize the potent toxin staphylococcal enterotoxin B (SEB).

93 of a common food-borne toxin, Staphylococcal enterotoxin B (SEB).

94 sponsive to SAgs, typified by staphylococcal enterotoxin B (SEB); ii) the human MAIT cell response to

95 e, heat-labile enterotoxin (LT), heat-stable enterotoxin b (STb), and enteroaggregative E. coli heat-

96 tinuously delivering the SAg, staphylococcal enterotoxin B (total of 10 mug/mouse), or PBS over 4 wk.

97 In this study, we show that staphylococcal enterotoxin B activates a Galphaq and PLCbeta2-dependent

98 +) T lymphocytes with soluble staphylococcal enterotoxin B and anti-CD28.

99 ton-Valentine leukocidin, and staphylococcal enterotoxin B and C negative, toxic shock syndrome toxin

100 r (PBS-BSA) and 0.1 ng/mL for staphylococcal enterotoxin B and LT.

101 cally important superantigens staphylococcal enterotoxin B and TSS toxin-1 with a single agent.

102 N-gamma(+) cells activated by staphylococcus enterotoxin B at 2 (P = .015) and 7 (P = .05) months of

103 e organic photodiode detected Staphylococcal enterotoxin B at concentrations as low as 0.5 ng/mL.

104 ity, and death in response to staphylococcal enterotoxin B challenge compared with wild-type mice.

105 a house dust mite extract and Staphylococcal enterotoxin B induced eczematous skin lesions.

106 MC were cultured for 7 d with staphylococcal enterotoxin B or IL-7 in the absence or presence of 100

107 on mitogenic stimulation with staphylococcal enterotoxin B or on antigenic stimulation with Candida a

108 Naive T cells primed by (staphylococcal enterotoxin B or tumor-associated protein-loaded) DC.Tbe

109 Choric exposure to staphylococcal enterotoxin B precipitated a lupus-like inflammatory dis

110 Chronic exposure to staphylococcal enterotoxin B resulted in a multisystem autoimmune infla

111 n-1-null mice challenged with staphylococcal enterotoxin B showed enhanced T cell accumulation in tis

112 Staphylococcal enterotoxin B stimulated similar cytokine patterns in me

113 on-induced proliferation (via staphylococcal enterotoxin B stimulation) but inhibited homeostatic pro

114 In response to staphylococcal enterotoxin B stimulation, beta-cat(Tg) mice exhibited a

115 ction by cells in response to staphylococcal enterotoxin B stimulation.

116 imulation of the TCR with the staphylococcal enterotoxin B superantigen.

117 allenge with the staphylococcal superantigen enterotoxin B were comparable between WT and DKO mice.

118 T plus a polyclonal stimulus (staphylococcal enterotoxin B) or specific bacterial Ags, and effects on

119 demonstrated by superantigen (staphylococcal enterotoxin B)-induced deletion of Vbeta8(+) T cells.

120 phenotype was reproduced with staphylococcal enterotoxin B, a heterologous SAg that also targets Vbet

121 jected intraperitoneally with staphylococcal enterotoxin B, a pyrogenic superantigen, and their infla

122 ogical warfare agents, ricin, staphylococcal enterotoxin B, and epsilon toxin, in complex human biofl

123 or the toxins (cholera toxin, staphylococcal enterotoxin B, and ricin) were 1.6, 0.064, and 1.6 ng/mL

124 wild-type mice injected with staphylococcal enterotoxin B, and the administration of heparan sulfate

125 ted with anti-CD3 antibody or staphylococcal enterotoxin B, we found that chloramphenicol induces the

126 fferences in levels of Staphylococcus aureus enterotoxin B-induced cytokines between the two groups,

127 en human AD skin and allergen/staphylococcal enterotoxin B-induced mouse skin lesions, particularly i

128 ing a robust immune response (staphylococcal enterotoxin B-induced T cell activation).

129 eta8(+) T cells isolated from staphylococcal enterotoxin B-injected mice did not exhibit Ag-independe

130 ed pulmonary HIV-specific and staphylococcal enterotoxin B-reactive CD4(+) memory responses, includin

131 memory CD4+ T lymphocytes and staphylococcal enterotoxin B-stimulated cytokine production by total CD

132 Staphylococcal enterotoxin B-stimulated IL-2-producing cells were more

133 ther anti-CD3/28 antibody- or staphylococcal enterotoxin B-stimulated single-positive CD4(+) and CD8(

134 inhibitor U-73122 sensitizes staphylococcal enterotoxin B-treated mice to dexamethasone in vivo.

135 r cells (PBMCs) stimulated by staphylococcal enterotoxin B.

136 escribed for the detection of staphylococcal enterotoxin B.

137 spores or Pep(263) but not by staphylococcal enterotoxin B.

138 atopic dermatitis-associated staphylococcal enterotoxin B.

139 mmunized with a superantigen, staphylococcal enterotoxin B.

140 superantigens (toxic shock syndrome toxin 1, enterotoxins B and C, and enterotoxin-like X) and cytoly

141 e two MRSA isolates, produced staphylococcal enterotoxins B, C, D, and E on overnight culture.

142 On Staphylococcus enterotoxin-B stimulation, which stimulates T cells thro

143 CPA), beta-toxin (CPB), epsilon-toxin (ETX), enterotoxin, beta2-toxin (CPB2), and perfringolysin O.

144 lococcal enterotoxin B (SEB), staphylococcal enterotoxin C (SEC), and toxic shock syndrome toxin-1 (T

145 n Valentine leukocidin (PVL), staphylococcal enterotoxin C-1 (SEC-1), and phenol-soluble modulin alph

146 island encodes and expresses staphylococcal enterotoxin C3 (SEC3) and staphylococcal enterotoxin-lik

147 We also demonstrated that staphylococcal enterotoxins can cross a CaCo-2 epithelial monolayer in

148 Rotavirus NSP4 is a viral enterotoxin capable of causing diarrhea in neonatal mice

149 Staphylococcus aureus enterotoxins cause debilitating systemic inflammatory re

150 Clostridium perfringens enterotoxin causes the gastrointestinal (GI) symptoms of

151 Domain I of Clostridium perfringens enterotoxin (cCPE) binds to the second extracellular loo

152 terminal fragment of Clostridium perfringens enterotoxin (cCPE) is a natural ligand for claudin-4.

153 Clostridium perfringens enterotoxin (CPE) action starts when the toxin binds to

154 gens type A strains producing C. perfringens enterotoxin (CPE) cause human food poisoning and antibio

155 tridium perfringens type A strains producing enterotoxin (CPE) cause one of the most common bacterial

156 Clostridium perfringens enterotoxin (CPE) causes food poisoning and antibiotic-a

157 Clostridium perfringens enterotoxin (CPE) causes the gastrointestinal symptoms o

158 Clostridium perfringens enterotoxin (CPE) causes the symptoms of a very common f

159 ed by type A isolates carrying a chromosomal enterotoxin (cpe) gene (C-cpe), while C. perfringens-ass

160 oisoning (FP) strains carrying a chromosomal enterotoxin (CPE) gene or the genetically related type C

161 belong to type C, carry beta-toxin (cpb) and enterotoxin (cpe) genes on large plasmids, and express b

162 Clostridium perfringens enterotoxin (CPE) is a major cause of food poisoning and

163 Clostridium perfringens enterotoxin (CPE) is a pore-forming toxin with a unique,

164 Clostridium perfringens enterotoxin (CPE) is responsible for causing the gastroi

165 Clostridium perfringens enterotoxin (CPE) is the etiological agent of the third

166 al of food-associated stresses, and (ii) the enterotoxin (CPE) responsible for the symptoms of this f

167 ulate in the intestinal tract and produce an enterotoxin (CPE) that is responsible for the symptoms o

168 many EN strains also express C. perfringens enterotoxin (CPE), suggesting that CPE could be another

169 -4) as receptors for Clostridium perfringens enterotoxin (CPE).

170 as the receptor for Clostridium perfringens enterotoxin (Cpe).

171 x [DI] = 0.924); generally positive only for enterotoxin D (74.5%); and resistant to clindamycin (98.

172 late and a high prevalence of staphylococcal enterotoxin D and the enterotoxin gene cluster.

173 anticity by non-toxic derivatives of type II enterotoxins dependent upon GD1a/TLR2 cooperative activi

174 the CT-related Escherichia coli heat-labile enterotoxin designated LT(R192G), or CpG oligodeoxynucle

175 bility of the astrovirus capsid to act as an enterotoxin, disrupting the gut epithelial barrier.

176 e-mutant of the Escherichia coli heat-labile enterotoxin (dmLT) adjuvant using microneedles.

177 d variant of the superantigen staphylococcal enterotoxin E (SEA/E-120) linked to fragment antigen bin

178 GFP fusion) were activated by Staphylococcus enterotoxin E-coated Raji cells, NDE1 and dynein failed

179 tive type C isolates appeared to carry their enterotoxin-encoding cpe gene on a cpb plasmid.

180 elta act mutant (a T2SS-associated cytotoxic enterotoxin-encoding gene) and a Delta act Delta vasH mu

181 similarity between the iap/ibp plasmids and enterotoxin-encoding plasmids of type A isolates.

182 s have confirmed older observations that the enterotoxins enhance enteric bacterial colonization and

183 ieved to be the key factor in staphylococcal enterotoxin enteropathy.

184 Staphylococcal food poisoning is caused by enterotoxins excreted into foods by strains of staphyloc

185 ults suggest that the host may also modulate enterotoxin expression because cells intoxicated with he

186 vel i.d. adjuvant of the type II heat-labile enterotoxin family, elicited strong systemic PspA-specif

187 pha1 and alpha2 is an important component of enterotoxin function and rotavirus pathogenesis, further

188 Enterotoxins G and I and enterotoxin-like superantigens

189 The enterotoxin gene cluster (egc) and the collagen adhesin

190 Genes of the enterotoxin gene cluster, seg, sei, sem, sen, and seo, w

191 ence of staphylococcal enterotoxin D and the enterotoxin gene cluster.

192 ar forms containing iap/ibp genes and silent enterotoxin gene sequences, with or without an IS1151-li

193 ious studies based on a design that involved enterotoxin genes cloned into a nontoxigenic fimbriated

194 ual clonal groups maintained the same set of enterotoxin genes even though they were isolated over ex

195 ults in increased transcription of the major enterotoxin genes nhe, hbl, and cytK and the virulence r

196 d six distinct clones, which contained fewer enterotoxin genes than strains without lukF-PV.

197 taining open reading frames with homology to enterotoxin genes, restriction-modification systems, tra

198 ng heat-stable (estA) and heat-labile (eltA) enterotoxin genes.

199 IgE antibody concentrations in serum against enterotoxins, grass pollen (GP), and house dust mite all

200 on the mechanisms of action of the rotavirus enterotoxin highlight this pleiotropic protein as a good

201 Two families of bacterial heat-labile enterotoxins (HLTs) have been described: the type I HLTs

202 ere significantly increased in patients with enterotoxin IgE and nonatopic asthma.

203 Staphylococcal enterotoxin IgE antibodies, but not IgE against inhalant

204 We hypothesize that the presence of enterotoxin IgE in serum indicates the involvement of st

205 Enterotoxin IgE positivity was significantly greater in

206 a higher FEV(1) percent predicted value, and enterotoxin IgE was associated with a lower FEV(1) perce

207 percent of patients with severe asthma with enterotoxin IgE were considered nonatopic.

208 asthma (OR, 11.09; 95% CI, 4.1-29.6) versus enterotoxin IgE-negative subjects.

209 monstrated significantly increased risks for enterotoxin IgE-positive subjects to have any asthma (OR

210 at primary cultured IMFs bind staphylococcal enterotoxins in a MHC class II-dependent fashion in vitr

211 Secretory diarrheas caused by bacterial enterotoxins, including cholera and traveler's diarrhea,

212 Adhesins and enterotoxins, including heat-labile (LT) and heat-stable

213 or colonization factor antigens (CFAs), and enterotoxins, including heat-labile enterotoxins (LT) an

214 as they are the final, rate-limiting step in enterotoxin-induced fluid secretion in the intestine.

215 Clostridium perfringens enterotoxin is a common cause of food-borne and antibiot

216 n of the small intestine may determine which enterotoxin is maximally expressed.

217 CPE (Clostridium perfringens enterotoxin) is the major virulence determinant for C. p

218 structures of the group V SAG staphylococcal enterotoxin K (SEK) alone and in complex with the TCR hV

219 Among them, NSP4 is the enterotoxin, known to disrupt cellular Ca(2+) homeostasi

220 own virulence genes which included those for enterotoxins, leukocidins, hemolysins, and surface prote

221 Staphylococcal enterotoxin-like K (SEl-K) is a potent mitogen that elic

222 xin-like superantigens M and N declined, but enterotoxin-like superantigens K, L, and Q increased.

223 Enterotoxins G and I and enterotoxin-like superantigens M and N declined, but ent

224 cal enterotoxin C3 (SEC3) and staphylococcal enterotoxin-like toxin L (SElL), as confirmed by quantit

225 syndrome toxin 1, enterotoxins B and C, and enterotoxin-like X) and cytolysins (alpha-, beta-, and g

226 also had a high prevalence of staphylococcal enterotoxin-like X.

227 conditions for the optimal expression of one enterotoxin limit the expression of the other.

228 released by this pathogen is the heat-labile enterotoxin LT, which upsets the balance of electrolytes

229 (CFs) such as CFA/I fimbriae and heat-labile enterotoxin (LT) are important virulence factors and pro

230 wo highly homologous substrates: heat-labile enterotoxin (LT) from enterotoxigenic Escherichia coli (

231 appears that toxins such as the heat-labile enterotoxin (LT) from Escherichia coli can help overcome

232 sibility of a vaccine containing heat-labile enterotoxin (LT) from ETEC delivered to the skin by patc

233 ent evidence suggesting that the heat-labile enterotoxin (LT) provides a colonization advantage for e

234 at express K88 (F4)(+) fimbriae, heat-labile enterotoxin (LT), heat-stable enterotoxin b (STb), and e

235 r effector molecule of ETEC, the heat-labile enterotoxin (LT), may enhance these interactions by stim

236 virulence factors, ETEC produces heat-labile enterotoxin (LT).

237 ibosyltransferase toxin known as heat-labile enterotoxin (LT).

238 meric B subunit of type IIb Escherichia coli enterotoxin (LT-IIb-B(5)), a doughnut-shaped oligomeric

239 binding subunit of type IIb Escherichia coli enterotoxin (LT-IIb-B(5)).

240 toxic mutant of Escherichia coli heat-labile enterotoxin (LT-K63) and CpG1826 as model adjuvants coul

241 a 24 h period) in which either or both ETEC enterotoxins (LT and heat-stable toxin [ST]) were detect

242 As), and enterotoxins, including heat-labile enterotoxins (LT) and heat-stable enterotoxins (ST), are

243 olera toxin (CT) and the type II heat-labile enterotoxins (LT-IIa and LT-IIb) are potent immunologica

244 ytes and their interaction with bacteria and enterotoxins may account for the noted increased suscept

245 Our data indicate that specialized, secreted enterotoxins may play a major role in one of these strat

246 rther development as antisecretory drugs for enterotoxin-mediated secretory diarrheas.

247 stigate whether IgE to Staphylococcus aureus enterotoxins might be relevant to disease severity in ad

248 ce of an Escherichia coli mutant heat-labile enterotoxin [mLT(R192G)] or cholera toxin subunit B as a

249 e toxin B with no cross-reactions with other enterotoxins, nontoxigenic C. difficile, or other Clostr

250 this study reports on the mechanism by which enterotoxin NSP4 exerts cytotoxicity and the mechanism b

251 cAMP agonists, cholera toxin, or heat-stable enterotoxin of E. coli (STa toxin), with IC50 down to ap

252 The type IIb heat-labile enterotoxin of Escherichia coli (LT-IIb) and its nontoxi

253 The heat-stable enterotoxin of Escherichia coli (STa) is a potent stimul

254 hway and the effect of Staphylococcus aureus enterotoxins on the regulation of the pro-inflammatory n

255 Two types of enterotoxins, one heat labile and the other heat stable,

256 ucing adjuvants, such as type II heat-labile enterotoxin or cholera toxin, resulted in increased morb

257 taglandin E(2), Escherichia coli heat-stable enterotoxin, orexins, and carbonated beverages stimulate

258 strain carrying the gene for staphylococcal enterotoxin P (sep).

259 ne production associated with staphylococcal enterotoxin pathogenesis.

260 ds guanylin, uroguanylin, or the heat-stable enterotoxin peptide (ST).

261 acute lung response to Staphylococcus aureus enterotoxin, peripheral injection of poly(I:C) manifeste

262 However, some enterotoxin-positive type C isolates appeared to carry t

263 s cholera toxin or the heat-labile or stable enterotoxins produced by Escherichia coli) that invade c

264 o C coli (around two times), and heat-stable enterotoxin-producing E coli ([ST-ETEC] around 1.5 times

265 (OR: 1.46; 95% CI: 1.11, 1.91), heat-labile enterotoxin-producing E. coli (OR: 1.55; 95% CI: 1.04, 2

266 % CI, 24.4%-26.7%]), followed by heat-stable enterotoxin-producing Escherichia coli (AF, 18.4% [95% C

267 ulence factors such as ystA, responsible for enterotoxin production, ail, attachment invasion locus g

268 SM101 reduced the levels of sporulation and enterotoxin production, supporting the involvement of Ab

269 cterized by distinct colonization factor and enterotoxin profiles.

270 f asthma to determine whether staphylococcal enterotoxins promote TH2 differentiation of allergen-spe

271 archaic plasmid that encoded staphylococcal enterotoxins R, J, and P.

272 s in various cell systems, and the bacterial enterotoxin receptor guanylyl cyclase C (GCC), the princ

273 tory diarrheas caused by bacterial and viral enterotoxins remain a significant cause of morbidity and

274 enterotoxin and Escherichia coli heat-labile enterotoxin reversed passive latex agglutination (VET-RP

275 IgE antibodies against Staphylococcus aureus enterotoxins (SAEs).

276 Labeled staphylococcal enterotoxin (SE) A, SED, and SEE were used to isolate si

277 e signaling that results from staphylococcal enterotoxin (SE) exposure.

278 Specific IgE to Staphylococcus aureus enterotoxins (SE-IgE) has been associated with asthma.

279 However, unlike the classical enterotoxins SEB and toxic shock syndrome toxin 1 (TSST-

280 eus) carriage and sensitization to S. aureus enterotoxins (SEs) have been associated with allergic di

281 es from involved skin express staphylococcal enterotoxins (SEs) that induce crosstalk between maligna

282 istance 26 A), cholera toxin and heat-labile enterotoxin (shortest distance 31 A), anti-HIV antibody

283 diarrheagenic bacterial heat-stable peptide enterotoxin ST.

284 es as the receptor for bacterial heat-stable enterotoxin (ST) peptides and the guanylin family of gas

285 Oral delivery of the heat-stable enterotoxin (ST), an exogenous GUCY2C ligand, opposed RI

286 eat-labile enterotoxins (LT) and heat-stable enterotoxins (ST), are the key virulence factors in ETEC

287 Originally identified as a target of E. coli enterotoxin STa, GC-C is an important regulator of physi

288 enterotoxigenic Escherichia coli heat-stable enterotoxin STa, which deregulates this pathway and caus

289 exit duct to drive efflux of antibiotics and enterotoxin STII out of the bacterial cell.

290 ux of macrolide antibiotics and secretion of enterotoxin STII.

291 es worldwide, often by producing heat-stable enterotoxins (STs), which are peptides structurally homo

292 Some bacteria produce secretory enterotoxins (such as cholera toxin or the heat-labile o

293 f inflammation and the presence of S. aureus enterotoxin (superantigen)-specific IgE in the nasal pol

294 ellular receptor, is a multifunctional viral enterotoxin that induces diarrhea in murine pups.

295 i use OMVs to deliver bundles of heat labile enterotoxin to host cells.

296 HRV infection and RV enterotoxin treatment of HIEs caused physiological lumen

297 clfB, cna, map/eap; P < .0001 for all) and 5 enterotoxins (tst, sea, sed, see, and sei; P </= .005 fo

298 E. coli-like colonies were screened for ETEC enterotoxins using a GM1 enzyme-linked immunosorbent ass

299 The distribution of ETEC enterotoxins varied between the symptomatic children (44

300 PE-induced cytotoxicity by preincubating the enterotoxin with soluble full-length recombinant claudin