ライフサイエンスコーパス: enterovirus

1 us B, 1 metapneumovirus, 1 adenovirus, and 1 enterovirus).

2 inovirus, 1 influenza C, 1 adenovirus, and 1 enterovirus).

3 itis or encephalitis who tested positive for enterovirus.

4 lity following infection with a common human enterovirus.

5 11 children were positive for rhinovirus or enterovirus.

6 rain of coxsackievirus, a positive-sense RNA enterovirus.

7 ditional non-EV-A71, non-CV-A16 serotypes of enterovirus.

8 dynamics caused by the multiple serotypes of enterovirus.

9 FMD) is a common childhood illness caused by enteroviruses.

10 a reemerging illness caused by a variety of enteroviruses.

11 ease is a common childhood illness caused by enteroviruses.

12 ractions known from previous work with human enteroviruses.

13 relatively minor abundance and occurrence of Enteroviruses.

14 cements occurred by recombination with other enteroviruses.

15 embling the uncoating intermediates of other enteroviruses.

16 requently the site of receptor attachment in enteroviruses.

17 virus 19 (SV19), baboon enterovirus (BaEV), enterovirus 112 (EV112), and EV115, have been solely ass

18 rystal structures of the picornavirus bovine enterovirus 2 (BEV2) and the cytoplasmic polyhedrosis vi

19 s infection and that this is mediated by the enterovirus 2A protease (2A(pro)).

20 The results demonstrate that the enterovirus 2A protease directly cleaves the nuclear por

21 Unlike other enterovirus 3A proteins, HRV 3A failed to bind GBF1.

22 parainfluenza virus (PIV) types 1-4 (7.5%); enterovirus (5.9%); respiratory syncytial virus (RSV; 5.

23 Human enterovirus 68 (EV-D68) is a member of the EV-D species,

24 Human enterovirus 68 (EV68) is a member of the EV-D species, w

25 o differentially mediates the replication of enterovirus 68 and rhinovirus 16.

26 ion is also important for the replication of enterovirus 68 but disadvantageous to human rhinovirus 1

27 Incubation of coxsackievirus A24 and enterovirus 70 with 2.5 mM NVC-422 for 1 hour reduced th

28 human adenovirus (HAdV), coxsackievirus A24, enterovirus 70, and herpes simplex-virus-1 (HSV-1) was t

29 immunodeficiency virus 1, hepatitis C virus, enterovirus 70, and variant Creutzfeldt-Jakob disease.

30 Infection of enterovirus 71 (EV71) and associated hand, foot, and mou

31 Human enterovirus 71 (EV71) and coxsackievirus A16 (CA16) are

32 Enterovirus 71 (EV71) and coxsackievirus A16 (CVA16) are

33 Enterovirus 71 (EV71) can cause hand-foot-and-mouth dise

34 Enterovirus 71 (EV71) causes a spectrum of neurological

35 Enterovirus 71 (EV71) causes hand, foot, and mouth disea

36 ered a top priority for the control of human enterovirus 71 (EV71) infection outbreaks.

37 no antivirals or vaccines available to treat Enterovirus 71 (EV71) infections.

38 Enterovirus 71 (EV71) is a human pathogen that causes ha

39 Enterovirus 71 (EV71) is a major public health threat th

40 Enterovirus 71 (EV71) is a picornavirus that causes outb

41 Enterovirus 71 (EV71) is an emerging pathogen causing ha

42 Enterovirus 71 (EV71) is an important emerging human pat

43 Enterovirus 71 (EV71) is one of the primary causative ag

44 Enterovirus 71 (EV71) is responsible for most of the sev

45 Enterovirus 71 (EV71) is responsible for seasonal outbre

46 Like other enteroviruses, enterovirus 71 (EV71) relies on phosphatidylinositol 4-k

47 Enterovirus 71 (EV71) remains a leading pathogen for acu

48 NCE We report here that a stable full-length enterovirus 71 (EV71) reporter construct was used to vis

49 eous nuclear ribonucleoprotein A1 stimulates enterovirus 71 (EV71) translation in part through specif

50 on and rapid quantification method for human enterovirus 71 (EV71) using a portable surface plasmon r

51 mice with empty, immature particles of human enterovirus 71 (EV71), a picornavirus that causes severe

52 tion on three Type 1 IRESs: poliovirus (PV), enterovirus 71 (EV71), and bovine enterovirus (BEV).

53 ovirus 11 (E11), coxsackievirus B (CVB), and enterovirus 71 (EV71), and that contrary to an immortali

54 coxsackievirus B (CVB), poliovirus (PV), and enterovirus 71 (EV71), co-opt the host cell secretory pa

55 The main causative agents are enterovirus 71 (EV71), coxsackievirus A16 (CVA16), and,

56 es titers against coxsackievirus A16 (CA16), enterovirus 71 (EV71), PV I-III and HIV-1 were performed

57 ver, little is known about its connection to enterovirus 71 (EV71).

58 Enterovirus 71 also binds to LIMP-2 (also known as SCARB

59 h as poliovirus, coxsackievirus, rhinovirus, enterovirus 71 and foot-and-mouth disease virus.

60 ver, the LIMP-2/SCARB2 binding sequences for enterovirus 71 and GCase are not similar, indicating tha

61 Here, we report the crystal structure of enterovirus 71 and show that, unlike in other enteroviru

62 gue virus, and the recent deadly outbreak of enterovirus 71 in Cambodian children is discussed.

63 e possible mechanisms and treatment of fatal enterovirus 71 infections to prevent the abrupt progress

64 Among enterovirus 71 infections, brainstem encephalitis progre

65 e to reduce the 1-week mortality of stage 3B enterovirus 71 infections.

66 As of now, no cure is available for enterovirus 71 infections.

67 Human enterovirus 71 is a picornavirus causing hand, foot, and

68 Enterovirus 71 may cause serious neurological disease in

69 It is most typically caused by enterovirus 71 or coxsackievirus A16 and results in asym

70 at, upon silencing, increased poliovirus and enterovirus 71 production by from 10-fold to >50-fold in

71 We show that the 3A protein of enterovirus 71 recruits an enzyme, phosphatidylinositol

72 rt a 3.2-A-resolution X-ray structure of the enterovirus 71 virion complexed with the capsid-binding

73 ermore, we show that the compound stabilizes enterovirus 71 virions and limits its infectivity, proba

74 rus replication also enhanced replication of enterovirus 71, a clinically relevant virus to which vac

75 g HIV, hepatitis C virus, hepatitis B virus, enterovirus 71, influenza virus, respiratory syncytial v

76 I4KB to the RNA replication sites.IMPORTANCE Enterovirus 71, like other human enteroviruses, replicat

77 Enterovirus 71-induced brainstem encephalitis with pulmo

78 ignificantly reduced the 1-week mortality of enterovirus 71-induced pulmonary edema and/or neurogenic

79 describes four vsRNAs that were detected in enterovirus 71-infected cells using next-generation sequ

80 confirmed deaths (93%) were associated with enterovirus 71.

81 ates replication of RNA from Hepatitis C and Enterovirus 71.

82 es: influenza A (104/556, 18.7%), rhinovirus/enterovirus (82/556, 14.7%), coronavirus and human metap

83 childhood illness caused by serotypes of the Enterovirus A species in the genus Enterovirus of the Pi

84 uman enterovirus A71 (EV-A71) belongs to the Enterovirus A species in the Picornaviridae family.

85 etween 2008 and 2013, with the two serotypes Enterovirus A71 (EV-A71) and Coxsackievirus A16 (CV-A16)

86 Human enterovirus A71 (EV-A71) belongs to the Enterovirus A sp

87 Enterovirus A71 (EV-A71) is a major cause of hand, foot,

88 Enterovirus A71 (EV-A71) is the major cause of severe ha

89 r 27,692 (6.9%) cases; EV71, CA16, and other enteroviruses accounted for 59.5%, 14.1%, 26.4%, respect

90 Enterovirus, adenovirus A, Salmonella spp., Campylobacte

91 ic Escherichia coli) and viruses (rotavirus, enterovirus, adenovirus) in hand rinses, stored water, a

92 sing age and infection with RSV, rhinovirus, enterovirus, adenovirus, and bocavirus.

93 nant ToV-PLP protein derived from this novel enterovirus also showed strong deubiquitination and deIS

94 human enteric viruses (norovirus GI and II, enterovirus and group A rotavirus) and supplementary hyd

95 Enterovirus and human herpesvirus type 6 had agreements

96 buvirus is intermediate between those of the enteroviruses and cardioviruses, with a shallow, narrow

97 the ability of 3C(pro) proteins from diverse enteroviruses and coxsackieviruses to interfere with typ

98 Twenty-five additional enteroviruses and eight parechoviruses could not be type

99 tions of the structure-function relations of enteroviruses and enterovirus-related virus-like particl

100 onsensus criteria for species assignments in enteroviruses and other picornavirus genera.

101 In conclusion, enteroviruses and parechoviruses may be transmitted betw

102 an commercially available assays that detect enteroviruses and rhinoviruses without distinguishing be

103 of receptor engagement among ICAM-1-binding enteroviruses and rhinoviruses.

104 , and surveillance (acute flaccid paralysis, enterovirus, and environmental) needs further strengthen

105 ce when considering the replication of human enteroviruses, and we believe that these data are unatta

106 Enteroviruses are among the most common viral infectious

107 Enteroviruses are among the suspected environmental trig

108 Enteroviruses are dependent upon host nuclear RNA bindin

109 Enteroviruses are recognized as important pathogens in p

110 Recently, we discovered not only that enteroviruses are transmitted via vesicles as population

111 ing three species (A, B, and C) of the genus Enterovirus, are responsible for the majority of upper r

112 ed: 25 with acute flaccid myelitis, two with enterovirus-associated encephalitis, five with enterovir

113 roviruses of the same species as CAV9, Human enterovirus B (HEV-B), identified examples from 5 types

114 se serotypes, simian virus 19 (SV19), baboon enterovirus (BaEV), enterovirus 112 (EV112), and EV115,

115 irus (PV), enterovirus 71 (EV71), and bovine enterovirus (BEV).

116 ted Enterovirus species F, previously bovine enterovirus (BEVs), viruses that are globally prevalent

117 is described frequently among non-rhinovirus enteroviruses but appears to be sporadic in rhinoviruses

118 f EV71 and suggested EV71 as the most common enterovirus causing HFMD in Shawo.

119 oV-PLP from a recombination event.IMPORTANCE Enteroviruses comprise a highly diversified group of vir

120 This study of the pancreatropic enterovirus Coxsackievirus B4 (CVB4) shows that although

121 and (c) influence on the replication of the enterovirus CVB3.

122 y novel predictive markers for the course of enterovirus (CVB3) cardiomyopathy by screening for nonco

123 simplex viruses 1 and 2, Epstein-Barr virus, enterovirus, cytomegalovirus, and chikungunya virus).

124 Coxsackievirus A16 (CV-A16), CV-A6, and enterovirus D68 (EV-D68) belong to the Picornaviridae fa

125 Enterovirus D68 (EV-D68) caused a widespread outbreak of

126 Enterovirus D68 (EV-D68) has been infrequently reported

127 CR (RT-PCR) assay for the detection of human enterovirus D68 (EV-D68) in clinical specimens.

128 isease in pediatric patients associated with enterovirus D68 (EV-D68) in the fall of 2014.

129 f severe respiratory illness associated with enterovirus D68 (EV-D68) infection was reported in mid-A

130 States experienced a nationwide outbreak of Enterovirus D68 (EV-D68) infection.

131 tes experienced an unprecedented outbreak of enterovirus D68 (EV-D68) infections.

132 Human enterovirus D68 (EV-D68) is a causative agent of childho

133 Enterovirus D68 (EV-D68) is a member of Picornaviridae a

134 Enterovirus D68 (EV-D68) is a member of the Picornavirid

135 Enterovirus D68 (EV-D68) is an emerging pathogen that ca

136 In August 2014, an outbreak of enterovirus D68 (EV-D68) occurred in North America, caus

137 During the enterovirus D68 (EV-D68) outbreak of 2014, the BioFire F

138 Enterovirus D68 (EV-D68)-associated acute flaccid myelit

139 ates, coincident with a national outbreak of enterovirus D68 (EV-D68)-associated severe respiratory i

140 strengthen the putative association between enterovirus D68 and acute flaccid myelitis and the conte

141 st the possibility of an association between enterovirus D68 and neurological disease in children.

142 plausibility support an association between enterovirus D68 and the recent increase in acute flaccid

143 aimed to investigate the association between enterovirus D68 infection and acute flaccid myelitis dur

144 a rare yet severe clinical manifestation of enterovirus D68 infection in susceptible hosts.

145 s was significantly higher during a national enterovirus D68 outbreak occurring from August 2014 thro

146 ation sequencing, viral genome recovery, and enterovirus D68 phylogenetic analysis.

147 Of six coding polymorphisms in the clade B1 enterovirus D68 polyprotein, five were present in neurop

148 n and acute flaccid myelitis during the 2014 enterovirus D68 respiratory outbreak in the USA.

149 Phylogenetic analysis revealed that all enterovirus D68 sequences associated with acute flaccid

150 tory illness were both infected by identical enterovirus D68 strains.

151 with acute flaccid myelitis but negative for enterovirus D68 using the two-tailed Fisher's exact test

152 Enterovirus D68 viraemia was identified in a child exper

153 Enterovirus D68 was detected in respiratory secretions f

154 Enterovirus D68 was implicated in a widespread outbreak

155 acute flaccid myelitis who were positive for enterovirus D68 with those with acute flaccid myelitis b

156 from five (45%) of 11 children were typed as enterovirus D68.

157 idence of an alternative infectious cause to enterovirus D68.

158 If enterovirus-D68 infections continue to happen in an ende

159 nd temporally associated with an outbreak of enterovirus-D68 respiratory disease.

160 n in children associated with an outbreak of enterovirus-D68 respiratory illness.

161 terovirus-associated encephalitis, five with enterovirus-D68-associated upper respiratory illness, an

162 ere present in neuropathogenic poliovirus or enterovirus D70, or both.

163 he specificity of encapsidation of C-cluster enteroviruses depends on an interaction between capsid p

164 proteins, were synthesized and conjugated to enteroviruses echovirus 1 and coxsackievirus B3.

165 In this study, a novel enterovirus (enterovirus species G [EVG]) (EVG 08/NC_USA

166 Like other enteroviruses, enterovirus 71 (EV71) relies on phosphati

167 Extensive research has identified enterovirus (EV) infections as key environmental trigger

168 Acute enterovirus (EV) meningitis is a major cause of hospital

169 oster virus (VZV), cytomegalovirus (CMV), or enterovirus (EV) NAAT with CSF samples between 2008 and

170 Twenty distinct human enterovirus (EV) serotypes, two bovine EV types, six hum

171 f rhinovirus C (RV-C), a recently identified Enterovirus (EV) species, are the causative agents of se

172 Enteroviruses (EVs) are a genetically and antigenically

173 Enteroviruses (EVs) are among the most frequent infectio

174 Enteroviruses (EVs) are implicated in a wide range of di

175 Enteroviruses (EVs) are important human pathogens associ

176 Enteroviruses (EVs) causing persisting infection are cha

177 Enteroviruses (EVs) have been connected to type 1 diabet

178 ify the entire capsid coding region of human enteroviruses (EVs) including PV.

179 the frequency of infection and diversity of enteroviruses (EVs) infecting monkeys (primarily mandril

180 Rhinoviruses (RVs), respiratory enteroviruses (EVs), influenza virus, respiratory syncyt

181 ctions in infants similar to those caused by enteroviruses (EVs).

182 This was investigated for a diverse range of enteroviruses (EVs; species A to D), human rhinoviruses

183 To better understand how enteroviruses exert these effects on nuclear transport,

184 the detected virus had greatest homology to Enterovirus F type 1 (indicating that the virus should b

185 Positive rhinovirus/enterovirus FA targets revealed patterns loosely associa

186 Many members of the genus Enterovirus (family Picornaviridae) cause HFMD.

187 al case of cross-order recombination between enterovirus G (order Picornavirales) and torovirus (orde

188 LP functions as an innate immune antagonist; enterovirus G may therefore gain fitness through the acq

189 Several enterovirus genotypes and CRESS DNA genomes were associa

190 an rhinovirus (HRV) and other members of the enterovirus genus bind small-molecule antiviral compound

191 ; genus Hepatovirus) and some members of the Enterovirus genus, are released from cells nonlytically

192 different frequencies within species of the Enterovirus genus.

193 Two recent studies reveal that enteroviruses harness the PI4P-cholestrol exchange cycle

194 onaviruses, human metapneumovirus (hMPV) and enteroviruses have also impacted humans globally.

195 Because bovine enteroviruses have not been previously reported in U.S.

196 , Streptococcus agalactiae, cytomegalovirus, enterovirus, herpes simplex virus 1 and 2, human herpesv

197 %), HAdV (5.3%), Norwalk virus (6.6%), Human enterovirus (HEV) (9.2%), Human parechovirus (1.3%), Sap

198 The cutaneous manifestations of human enterovirus (HEV) infection are usually limited, such as

199 has potential impact on understanding other enterovirus-host cell interactions.

200 This report provides novel insights into enterovirus-host interactions and describes an approach

201 routine clinical tests, including rhinovirus/enterovirus, human metapneumovirus, and coronavirus.

202 results provide evidence for the presence of enterovirus in pancreatic islets of type 1 diabetic pati

203 piratory panel was used to detect rhinovirus/enterovirus in respiratory specimens; suspected EV-D68-p

204 miological features of HFMD and infection by enteroviruses in China.

205 y indicate the presence and density of these enteroviruses in the population and prolonged virus circ

206 Mumps virus, enteroviruses (including human parechovirus), HHV-6A, HP

207 ids were susceptible to infection by diverse enteroviruses, including echovirus 11 (E11), coxsackievi

208 Our results suggest that enterovirus-induced miR-146a facilitates viral pathogene

209 ocrine cells are permissive, suggesting that enteroviruses infect specific cell populations in the hu

210 ons in permeability and nuclear transport in enterovirus-infected cells and how NPCs remain functiona

211 ible for alterations in nuclear transport in enterovirus-infected cells that lead to the cytoplasmic

212 s rapidly targeted for degradation following enterovirus infection and that this is mediated by the e

213 diabetic patients have prolonged/persistent enterovirus infection associated with an inflammation pr

214 future studies involving various degrees of enterovirus infection in mice, not just severe infection

215 tive for adenovirus, human herpesvirus 6, or enterovirus infection or with acute bacterial infection

216 reening as a gene whose depletion restricted enterovirus infection.

217 cine administered during the high season for enterovirus infections (April-September) was significant

218 Enterovirus infections are associated with a number of s

219 Enterovirus infections can vary from asymptomatic infect

220 Enterovirus infections have been linked to type 1 diabet

221 However, the events associated with enterovirus infections of the human gastrointestinal tra

222 together, our studies provide insights into enterovirus infections of the human intestine, which cou

223 teroids from human small intestines to study enterovirus infections of the intestinal epithelium.

224 ng preventive and therapeutic strategies for enterovirus infections.

225 ications that occur in a small percentage of enterovirus infections.

226 observed between human rhinovirus and other enterovirus infections.

227 Adenovirus, bocavirus-1, coronavirus, enteroviruses, influenza virus, metapneumovirus, parainf

228 Although EV71, as well as other enteroviruses, initiates a remodeling of intracellular m

229 Enteroviruses invade their hosts by crossing the intesti

230 We are targeting unfettered enterovirus IRES activity in cancer with PVSRIPO, the ty

231 Viral infection with enteroviruses is a suspected trigger for T1D, but a caus

232 hows that while recombination with non-Sabin enteroviruses is associated with cVDPV, the recombinatio

233 er coxsackieviruses, which are prototypes of enteroviruses, is dependent on an interaction of capsid

234 is described frequently among non-rhinovirus enteroviruses, it seems to occur more rarely in rhinovir

235 Enterovirus, kobuvirus, parechovirus, parvovirus, and ro

236 Adenovirus and enterovirus levels in the wells were associated with pre

237 Further, recombination with species C enteroviruses may indicate the presence and density of t

238 Enterovirus morphogenesis, which involves the encapsidat

239 previous in vitro studies suggest that some enteroviruses not only evade these protective effects bu

240 models, we assessed the effects of nonpolio enteroviruses (NPEVs) and diarrhea on the odds of seroco

241 is study represents an overview of non-polio enteroviruses (NPEVs) isolated from acute flaccid paraly

242 es of the Enterovirus A species in the genus Enterovirus of the Picornaviridae family.

243 Analysis of several enteroviruses of the same species as CAV9, Human enterov

244 ssays cannot differentiate EV-D68 from other enteroviruses or HRV, and improved rapid diagnostic tool

245 The assay did not detect any other enteroviruses or rhinoviruses tested and did detect dive

246 screened using real-time PCR for the genera Enterovirus, Parechovirus, and Sapelovirus, and positive

247 ed using real-time PCR assays for the genera Enterovirus, Parechovirus, and Sapelovirus; PCR-positive

248 i virus, Anellovirus, Astrovirus, Bocavirus, Enterovirus, Parechovirus, Picobirnavirus, and Rotavirus

249 Enterovirus PCR of cerebrospinal fluid, blood, and recta

250 nationwide, who were rhinovirus-positive or enterovirus-positive in hospital testing, were submitted

251 Enteroviruses proteolyze nuclear pore complex (NPC) prot

252 ride or peptidoglycan of bacterial origin to enterovirus provides thermal protection through stabiliz

253 lar PS lipids are co-factors to the relevant enterovirus receptors in mediating subsequent infectivit

254 irus/rhinovirus or rhinovirus/non-rhinovirus enterovirus recombinants are fully viable.

255 te-Sabin isolate and Sabin isolate-non-Sabin enterovirus recombination after accounting for the time

256 Network) inpatient study of 61 children with enterovirus-related neurological disease during a 2013 o

257 ture-function relations of enteroviruses and enterovirus-related virus-like particles, including thei

258 Many RNA viruses, including enteroviruses, remodel host membranes to generate organe

259 .IMPORTANCE Enterovirus 71, like other human enteroviruses, replicates its genome within host cells,

260 of BPIFB6 expression dramatically suppresses enterovirus replication in a pan-viral manner.

261 These results reveal a mechanism of enterovirus replication that involves a selective strate

262 de new insight into the molecular network of enterovirus replication.

263 et whose function could be targeted to alter enterovirus replication.

264 6 (BPIFB6), whose expression is required for enterovirus replication.

265 ns as a positive regulator of CVB, and other enterovirus, replication by controlling secretory pathwa

266 rrected] for C. jejuni, Salmonella spp., and enteroviruses, respectively.

267 ive sequence analysis of 170 closely related enteroviruses revealed that the SLII region lacks conser

268 Enterovirus RNA was analyzed from isolated pancreatic is

269 ts contained VP1(+) cells, and the amount of enterovirus RNA was low.

270 Enterovirus RNA was more commonly identified in feces (4

271 ratory syncytial virus (RSV), parainfluenza, enterovirus, rotavirus, norovirus, Campylobacter, and Sa

272 Most frequently detected were rhinoviruses/enteroviruses (RV/EV) (21%) and influenza viruses (21%);

273 everity, laboratory confirmation status, and enterovirus serotype.

274 ed by month, age, sex, disease severity, and enterovirus serotype.

275 ple size or evaluation of detection for more enterovirus serotypes are not well investigated in Chong

276 ical community size (CCS) of HFMD associated enterovirus serotypes CV-A16 and EV-A71 and we explore w

277 Our objective was to identify enterovirus serotypes that could be involved in the init

278 noglobulin-like receptors used by most other enteroviruses share a conserved mechanism for priming vi

279 ing human populations, comprising members of enterovirus species B, J, and H and a large novel group

280 ence was most similar to recently designated Enterovirus species F, previously bovine enterovirus (BE

281 In this study, a novel enterovirus (enterovirus species G [EVG]) (EVG 08/NC_USA/2015) was is

282 rs of characteristic species groups, such as enterovirus species groups A to H or rhinovirus species

283 Enterovirus-specific RNA sequences were detected in four

284 Enteroviruses such as poliovirus and coxsackievirus recr

285 d bacterial pathogens but was antagonized by enteroviruses, such as rhinovirus and poliovirus, which

286 rotease and prevented C3 cleavage, rendering enteroviruses susceptible to intracellular complement se

287 Coxsackievirus B (CVB) is a common enterovirus that can cause various systemic inflammatory

288 oxsackievirus B3 (CV-B3) is a cardiovirulent enterovirus that utilizes a 5' untranslated region (5'UT

289 nterovirus 71 and show that, unlike in other enteroviruses, the "pocket factor," a small molecule tha

290 interval [CI], 81.3 to 89.7%) for rhinovirus/enteroviruses to 98.6% (95% CI, 96.5 to 99.6%) for PIV 2

291 was done for C. jejuni, Salmonella spp., and enteroviruses to estimate risk of infection and illness.

292 n-mediated endocytosis (CME) is harnessed by enteroviruses to traffic cholesterol from the plasma mem

293 ine (m(7)G) "cap"-independent translation at enterovirus type 1 internal ribosomal entry sites (IRESs

294 neutralizing antibodies against 41 different enterovirus types in a unique longitudinal sample series

295 erscores the need for robust surveillance of enterovirus types, enabling improved understanding of vi

296 Enteroviruses use a type I Internal Ribosome Entry Site

297 ined for the presence of human rhinovirus or enterovirus using the FilmArray Respiratory Panel (RP) a

298 Strikingly, no known simian enteroviruses were detected among these NHP populations.

299 Enteroviruses were the most frequently detected pathogen

300 exhibits antiviral activity against several enteroviruses, whereas truncated G3BP1 that cannot form

301 inovirus, 3 adenovirus, 2 influenza B, and 1 enterovirus), which was also significantly higher than t