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1 ans rarely confirm the clinical diagnosis of enthesitis.
2 ce, increased risk of axial involvement, and enthesitis.
3 tween bone erosion and new bone formation in enthesitis.
4 of spondyloarthritis, such as synovitis and enthesitis.
5 l bone marrow edema, synovitis, bursitis, or enthesitis.
6 -seven patients with SpA and Achilles tendon enthesitis (20 with early SpA and 17 with chronic SpA) a
8 his is associated with marked improvement of enthesitis and associated osteitis pathology as determin
11 e specific and characteristic development of enthesitis and entheseal new bone formation in the initi
12 of pathological lesions was not specific to enthesitis and might more likely correspond to degenerat
13 g the mechanisms that underlie angiogenesis, enthesitis, and bone resorption in psoriatic arthritis a
14 oint count, evaluation for dactylitis and/or enthesitis, and skin examination) and HLA-B27 typing wer
16 In contrast, older patents tend to manifest enthesitis, axial joint disease, and persistent oligoart
17 , spinal pain, functioning, quality of life, enthesitis, chest expansion, erythrocyte sedimentation r
18 litis, seronegative ankylosing arthritis and enthesitis, conditions stereotypical of human inflammato
19 ht of diagnostic difficulties with detecting enthesitis in clinical examinations and laboratory inves
20 in associated inflammatory features, such as enthesitis in psoriatic arthritis and uveitis in ankylos
21 erosion in association with Achilles tendon enthesitis in SpA is anatomically uncoupled from bone fo
22 drawn attention to the ubiquitous nature of enthesitis in spondyloarthropathies, especially adjacent
23 ts associated with spondyloarthropathy is an enthesitis (inflammation at sites where ligaments, tendo
25 , or joint capsules to bone, which is termed enthesitis, is a characteristic feature of spondyloarthr
26 resonance imaging studies also suggest that enthesitis lesions may be extensive, which could explain
28 tion of HLA-B27 with bone pathology in early enthesitis may have implications for a better understand
29 initive rash (44%), a competing diagnosis of enthesitis-related arthritis (23%), family history of ps
30 tients with SpA and 5 patients with juvenile enthesitis-related arthritis (juvenile ERA); samples wer
31 children and 136 patients with JIA (28 with enthesitis-related arthritis [ERA], 42 with persistent o
33 age was abnormally high in all JIA subtypes (enthesitis-related arthritis was not assessed), most str
35 al assessment of psoriatic nail disease, and enthesitis (using the PsA-modified Maastricht Ankylosing
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