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1 r phosphorylation and angiotensin-converting enzyme induction.
2 anscription factor responsible for lipogenic enzyme induction.
3 fficiently high level by the slow process of enzyme induction.
4 cell extracts obtained at various stages of enzyme induction.
5 ity or evidence of hepatic biotransformation enzyme induction.
6 nflammatory activity, antiproliferation, and enzyme induction.
7 topoisomerase IIbeta, loss of antioxidative enzyme induction and attenuated protection against ODD b
8 e pools revealed a close correlation between enzyme induction and cytochrome c release, caspase activ
9 wed that a minimal model accounting only for enzyme induction and dilution captures all the substrate
10 ility to adapt to stress through antioxidant enzyme induction and translocation of these proteins to
11 ion of whether bone mass is truly related to enzyme induction, and analogously, of whether reductions
12 intain high potency and efficacy for phase 2 enzyme induction as well as the inhibitory effect on lip
13 on that agr autoactivation, unlike classical enzyme induction, can occur under suboptimal conditions
14 lation exposure, including immune responses, enzyme induction, cellular toxicity, and histopathologic
15 ha and other cytokine production, as well as enzyme induction (cyclooxygenase-2, inducible nitric oxi
17 istance and activities of putative defensive enzymes, induction of defence-related genes and activati
18 ch from other laboratories on the effects of enzyme induction on chemical carcinogenesis as an approa
20 which was additive to the substrate-mediated enzyme induction produced by the ingested erythrocyte he
22 mechanism, whereas the Ah receptor-dependent enzyme induction reduces adducts in liver, probably due
26 erol 7alpha-hydroxylase and initially favors enzyme induction, whereas increased bile acid pool is th
27 activation of the bkd operon by BkdR during enzyme induction which incorporates these results is pre
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