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1 transcription polymerase chain reaction, and enzyme-linked immunoassay.
2 and tetanus toxoid were measured by indirect enzyme-linked immunoassay.
3 ne attack complex C5b-9 concentrations using enzyme-linked immunoassay.
4 e changes validated by mass spectrometry and enzyme-linked immunoassay.
5 d by real-time polymerase chain reaction and enzyme-linked immunoassay.
6 LA2 was measured in plasma aliquots using an enzyme-linked immunoassay.
7 drolysis of cyclic (c)GMP levels with a cGMP enzyme-linked immunoassay.
8 by PDE inhibitors were quantitated by a cGMP enzyme-linked immunoassay.
9 protein C (pepC10) were evaluated by kinetic enzyme-linked immunoassay.
10 eceptors were performed on plasma samples by enzyme-linked immunoassay.
11 rs by end-point dilution by use of an HTLV-I enzyme-linked immunoassay.
12 or estradiol and progesterone metabolites by enzyme-linked immunoassay.
13 ntigens was determined by flow cytometry and enzyme-linked immunoassay.
14 Plasma c-erbB-2 levels were quantified by enzyme-linked immunoassay.
15 and 15 normal donors was determined using an enzyme-linked immunoassay.
16 d IL-6 concentrations were measured using an enzyme-linked immunoassay.
17 d PACAP and its receptor (PAC1) levels using enzyme-linked immunoassay.
18 matic activity in a solution compatible with enzyme-linked immunoassays.
19 often a necessary step in the development of enzyme-linked immunoassays.
20 analyzed using cell-proliferation assays and enzyme-linked immunoassays.
21 erferon (IFN-gamma), as measured by cytokine enzyme-linked immunoassays.
22 and NS4a and E2-HVR-1 peptides were used in enzyme-linked immunoassays.
26 apoB-100] were measured by chemiluminescent enzyme-linked immunoassay and commercial assays, respect
27 avidity assay by modifying the Ortho 3.0 HCV enzyme-linked immunoassay and tested 997 serum or plasma
28 The presence of OxPL was confirmed using enzyme-linked immunoassays and immunohistochemistry of c
30 ar MUC5AC concentration was quantified using enzyme-linked immunoassay, and conjunctival goblet cell
31 unoblots, glycosidase digestion, intact cell enzyme-linked immunoassay, and extracellular calcium-sti
34 of receptor phosphorylation determined in an enzyme-linked immunoassay, as well as by Western blottin
37 st homologous virus-like particles (VLPs) by enzyme-linked immunoassay but did not react with denatur
38 etion was shown, with immunofluorescence and enzyme-linked immunoassay, by loss of binding of PF sera
39 d seven respiratory viruses through culture, enzyme-linked immunoassay (EIA) and polymerase chain rea
41 he conventional radioimmunoassay (RIA) to an enzyme-linked immunoassay (EIA) for the measurement of H
42 positive results in the Platelia Aspergillus enzyme-linked immunoassay (EIA), due to contamination wi
45 to HCV (anti-HCV) using a second-generation enzyme-linked immunoassay (EIA-2), followed by recombina
47 s with chronic HCV infection were assayed in enzyme-linked immunoassays (EIAs) for antibody binding t
51 n tears, feces and serum were measured using enzyme-linked immunoassays (ELISA) after topical and sys
56 on of cell surface proteins, and intact cell enzyme-linked immunoassay indicated that disruption of a
59 ted generation of cyclic AMP, as measured by enzyme-linked immunoassay (p = .05 polynomial regression
60 uorescent microbead-based immunoassay and/or enzyme-linked immunoassay platform to characterize the a
63 on occurs is presently unknown, quantitative enzyme-linked immunoassay showed the presence of the equ
64 sence of antibodies to HCV was determined by enzyme-linked immunoassay, supplementary recombinant imm
66 ha and interleukin-6 levels were analyzed by enzymes-linked immunoassay using randomly selected plasm
70 ith reactive rapid test results were offered enzyme-linked immunoassay, Western blot, and plasma HIV-
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