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1 onsider the Susceptible-Infectious-Recovered epidemic model.
2 , analogous to the reproduction number of an epidemic model.
3 ial distancing measures in an age structured epidemic model.
4 quilibrium is equivalent to that of standard epidemic models.
5 nce of including changing mixing patterns in epidemic models.
6 tochastic models were developed: patient and epidemic models.
7 ted for the unforced and forced SIR and SEIR epidemic models.
8 question has been studied extensively using epidemic models.
9 ns differ strongly from the ones provided by epidemic models.
10 is similar to standard results for diffusive epidemic models.
11 series susceptible-infected-recovered (TSIR) epidemic models.
12 ate in a worldwide-structured metapopulation epidemic model a timescale-separation technique for eval
15 ation for the susceptible-infected-recovered epidemic model applicable to arbitrary dynamic networks.
18 udy the spread of social phenomena relies on epidemic models by establishing analogies between the tr
19 re to include differential susceptibility to epidemic models can lead to a systematic over/under esti
21 fferences between these 2 fields and how the epidemic modeling community is rising to the challenges
23 is stochastic model is based on an influenza epidemic model, expressed in terms of a system of ordina
26 s in natural populations, thus supporting an epidemic model for the evolution of selfish genes, where
28 g diseases have led to the widespread use of epidemic models for evaluating public health strategies.
31 ssible mechanisms or interactions assumed by epidemic models has been limited: either independent of
33 phone commuting network is considered in the epidemic model, however preserving to a high degree the
36 d final size of an epidemic for a variety of epidemic models in homogeneous and heterogeneous populat
37 as simulated using stochastic age-structured epidemic models in settings conducive to high or low mea
38 wer questions that are often addressed using epidemic models, in particular: on the basis of potentia
43 early on in well-mixed populations mean that epidemic models may be linearised and we can calculate o
47 data from satellites, and weather stations, epidemic models rely on human interactions, multiple dat
48 y on epidemiological dynamics, we propose an epidemic model structured according to immunity level th
50 EIR (susceptible-exposed-infectious-removed) epidemic model that includes a smooth transition in the
53 stochastic simulations with a compartmental epidemic model to quantify the impact of genetic diversi
55 se detailed geographic surveillance data and epidemic models to estimate the critical community size
61 model coupled with the individual-based SIS epidemic model where susceptible individuals adopt a pre
63 uced as a means of optimally melding dynamic epidemic models with epidemiological observations and da
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