ライフサイエンスコーパス: epidermal keratinocyte

1 n HSV-1 infection and their interaction with epidermal keratinocytes.

2 re produced by placenta, adrenal glands, and epidermal keratinocytes.

3 ar signals mediating cell differentiation in epidermal keratinocytes.

4 o cellular components of the skin, including epidermal keratinocytes.

5 , and modulating TGF-beta signaling in mouse epidermal keratinocytes.

6 ons is an enhancer element that is active in epidermal keratinocytes.

7 als for UVB irradiation-induced apoptosis in epidermal keratinocytes.

8 ontact or were physically separated from the epidermal keratinocytes.

9 del, correlating with K17 induction in basal epidermal keratinocytes.

10 l regulator that promotes differentiation in epidermal keratinocytes.

11 ortant roles in the survival and motility of epidermal keratinocytes.

12 yaluronan synthase (HAS) activation in human epidermal keratinocytes.

13 ance of proliferation and differentiation in epidermal keratinocytes.

14 adhesion molecule expression and function in epidermal keratinocytes.

15 lammatory skin and in the differentiation of epidermal keratinocytes.

16 EP2 to the plasma membrane of differentiated epidermal keratinocytes.

17 et of the TNF alpha-regulated genes in human epidermal keratinocytes.

18 d the expression of endogenous TRPC in human epidermal keratinocytes.

19 cutaneous IL-33 reduces REG3A expression in epidermal keratinocytes.

20 C/EBPalpha is abundantly expressed in mouse epidermal keratinocytes.

21 cells responded more quickly to therapy than epidermal keratinocytes.

22 a) is an important regulator of apoptosis in epidermal keratinocytes.

23 xygen species and extensive apoptosis of the epidermal keratinocytes.

24 results were obtained by using normal human epidermal keratinocytes.

25 ory neurons but are highly expressed in skin epidermal keratinocytes.

26 kappa B (NF-kappaB) pathways in normal human epidermal keratinocytes.

27 omplex regulation of Col7a1 transcription in epidermal keratinocytes.

28 s the predominant species expressed in basal epidermal keratinocytes.

29 imus block activation of calcineurin/NFAT in epidermal keratinocytes.

30 T1 was predominantly nuclear in normal basal epidermal keratinocytes.

31 differentiation in primary cultures of mouse epidermal keratinocytes.

32 ssed late during terminal differentiation in epidermal keratinocytes.

33 s cytotoxic to dermal fibroblasts as well as epidermal keratinocytes.

34 nse that is specific for proliferating human epidermal keratinocytes.

35 eased activation of calcineurin in psoriatic epidermal keratinocytes.

36 ytokines that can influence the phenotype of epidermal keratinocytes.

37 gulates involucrin (hINV) gene expression in epidermal keratinocytes.

38 gene cluster and is selectively expressed in epidermal keratinocytes.

39 n as well as isolated dermal fibroblasts and epidermal keratinocytes.

40 ate iron transport and accumulation in human epidermal keratinocytes.

41 and squamous differentiation in normal human epidermal keratinocytes.

42 follow UV-caused gene expression changes in epidermal keratinocytes.

43 s involucrin (hINV) gene expression in human epidermal keratinocytes.

44 g interferon-gamma treatment of normal human epidermal keratinocytes.

45 , plasminogen activator inhibitor type 2, in epidermal keratinocytes.

46 ed covalent adduct formation in normal human epidermal keratinocytes.

47 dermal junction and accompanying necrosis of epidermal keratinocytes.

48 is a water and glycerol channel expressed in epidermal keratinocytes.

49 intermediate filament (IF) network of basal epidermal keratinocytes.

50 of intercellular adhesions between mammalian epidermal keratinocytes.

51 nt directional migration in undifferentiated epidermal keratinocytes.

52 al differentiation and hyperproliferation of epidermal keratinocytes.

53 mice lacking p53-mediated POMC induction in epidermal keratinocytes.

54 d that p63 promotes glycolytic metabolism in epidermal keratinocytes.

55 dy was to determine the role of mitoStat3 in epidermal keratinocytes.

56 tes the proliferation and differentiation of epidermal keratinocytes.

57 kin tissue environment has limited effect on epidermal keratinocytes.

58 ostasis, we conditionally knocked out SRF in epidermal keratinocytes.

59 rganotypic three-dimensional cultures of rat epidermal keratinocytes.

60 normal human fibroblasts and neonatal human epidermal keratinocytes.

61 ependent fashion with predominant effects on epidermal keratinocytes.

62 ted the effect of Mg(2+) on TRPV3 in primary epidermal keratinocytes.

63 ages, and myelomonocytic cells as well as in epidermal keratinocytes.

64 In epidermal keratinocytes a syndecan-TRPC4 complex control

65 rough specific toll-like receptors (TLRs) in epidermal keratinocytes, a DFE-induced murine atopic der

66 In response to UVB damage, epidermal keratinocytes activate a specific repair pathw

67 , the major autoantigen in PV, cause loss of epidermal keratinocyte adhesion, resulting in blisters a

68 le to attenuate DeltaNp63alpha expression in epidermal keratinocytes after such stress.

69 date the role of IL-6 in skin wound healing, epidermal keratinocyte and dermal fibroblast cells were

70 L2 and of IFNgamma-induced CCL8 and CXL10 by epidermal keratinocytes and (3) thereby limited the recr

71 9 null) mice, normal proliferation occurs in epidermal keratinocytes and corneal basal cells.

72 ins in skin was due to both synthesis within epidermal keratinocytes and deposition from granulocycte

73 t STRA6 is constitutively expressed in human epidermal keratinocytes and dermal fibroblasts and is re

74 hich inactivates cortisol), was expressed in epidermal keratinocytes and dermal fibroblasts in human

75 ted Smad1/5/8 are expressed in human primary epidermal keratinocytes and dermal fibroblasts.

76 lysis using RNA isolated from cultured human epidermal keratinocytes and dermal fibroblasts.

77 xpression of 12 435 genes were determined in epidermal keratinocytes and dermal fibroblasts.

78 x steroids by primary cultures of human skin epidermal keratinocytes and dermal fibroblasts.

79 mobility group box 1 (HMGB1) from UV-damaged epidermal keratinocytes and driven by Toll-like receptor

80 The present study demonstrates that epidermal keratinocytes and fibroblast cells express sig

81 (ERK) activation in both normal human adult epidermal keratinocytes and five of seven SCCHN cell lin

82 ed the role of several coactivators in human epidermal keratinocytes and found that, although these c

83 It is mainly found in epidermal keratinocytes and has been hypothesized to be

84 proapoptotic and proinflammatory actions on epidermal keratinocytes and has been implicated in the p

85 3 channel (TRPV3) is abundantly expressed in epidermal keratinocytes and has important roles in senso

86 ng cyclin-dependent kinase 4 (Cdk4) in human epidermal keratinocytes and human mammary epithelial cel

87 a transporter-directed RNAi screen in human epidermal keratinocytes and identified SLC22A20 (OAT6) a

88 elective regulation of TSLP transcription in epidermal keratinocytes and IEC.

89 leading to the accumulation of mutations in epidermal keratinocytes and immunosuppression, which con

90 TRPV4 ion channels function in epidermal keratinocytes and in innervating sensory neuro

91 d that C/EBPalpha is induced in normal human epidermal keratinocytes and in the epidermis of human su

92 cation is the final differentiative step for epidermal keratinocytes and involves dramatic cell conde

93 ncrease in H2O2 was observed in normal human epidermal keratinocytes and its production was inhibited

94 lammation-induced activation of Axl in human epidermal keratinocytes and LCs.

95 ied indeed a functioning GFRP/GTPCHI axis in epidermal keratinocytes and melanocytes in the cytosol,

96 rolled by skin-specific factors derived from epidermal keratinocytes and not by resident dendritic ce

97 nal diacylglycerol/phorbol ester receptor in epidermal keratinocytes and suggest that activation of t

98 hat HVEM and LTbetaR were expressed on human epidermal keratinocytes and that LIGHT could directly pr

99 that AQP3 has a pro-differentiative role in epidermal keratinocytes and that PLD2 activity is necess

100 sly shown that RasGRP1 is expressed in mouse epidermal keratinocytes and that transgenic mice overexp

101 itin sulfate proteoglycan 4) was detected in epidermal keratinocytes and the cardiac conduction syste

102 Fas ligand (FasL) causes apoptosis of epidermal keratinocytes and triggers the appearance of s

103 pholipase-A2 type X (sPLA2-X) is released by epidermal keratinocytes and we have shown that lysophosp

104 of Agouti transcripts and protein in primary epidermal keratinocytes, and BMP signaling positively re

105 --a stimulus of angiogenesis--is produced by epidermal keratinocytes, and elevated levels have been f

106 of wound repair, predominantly expressed in epidermal keratinocytes, and peaked in the subsequent pr

107 transport of melanosomes from melanocytes to epidermal keratinocytes, and then were treated with kine

108 these cells fail to transdifferentiate into epidermal keratinocytes, and there was no improvement in

109 potent chemotactic and mitogenic factor for epidermal keratinocytes, and these properties are centra

110 ucing directly the synthesis of keratin 6 in epidermal keratinocytes, and thus changing the compositi

111 em cells in Terc(-/-) mice, and normal human epidermal keratinocytes are also ALT-positive.

112 Epidermal keratinocytes are complex cells that create a

113 a basis for our hypothesis that normal human epidermal keratinocytes are involved in the initiation a

114 Epidermal keratinocytes are particularly suitable candid

115 higher levels of endogenous bcl-2 protein in epidermal keratinocytes as assessed by immunoblotting an

116 alterations in growth and differentiation of epidermal keratinocytes, as well as a marked increase in

117 city accompanied by MIF release in mouse ear epidermal keratinocytes, as well as in human keratinocyt

118 We confirmed AIRE expression in HaCaT epidermal keratinocytes, as well as its interaction with

119 nal in TPA-induced shedding of TNFalpha from epidermal keratinocytes; (b) PKCepsilon-mediated signals

120 Notch1-deficient epidermal keratinocytes become progressively hyperplasti

121 the transcriptional activity of NFkappaB in epidermal keratinocytes, both under basal treatment and

122 physiology of the cornified cell envelope in epidermal keratinocytes but may reside in the challenges

123 inally thought of as a disorder primarily of epidermal keratinocytes, but is now recognised as one of

124 , the results indicate that HSV enters human epidermal keratinocytes, but not neurons, by a low-pH, e

125 We show how the epidermal keratinocyte can direct hMSC differentiation s

126 intrinsic sensory transduction mechanisms in epidermal keratinocytes can directly elicit AP firing in

127 Smad7 induction in epidermal keratinocytes caused an increase in keratinocy

128 ltaNp63alpha overexpression in primary human epidermal keratinocytes causes decreased cell proliferat

129 During differentiation, epidermal keratinocytes cease to express integrins, but

130 A treatment was observed in the immortalized epidermal keratinocyte cell line NHEK-HPV, whereas PMA d

131 NT5E was also located on epidermal keratinocytes, cells of the dermis, and on noc

132 isplayed increased DMBA-induced apoptosis in epidermal keratinocytes compared to wild-type mice.

133 Unirradiated normal human epidermal keratinocytes contained 1.49 (+/- 0.11) 8-oxo-

134 Conversely, epidermal keratinocytes contained less stromal Igfbp4 an

135 ting bacterial invasion of canine progenitor epidermal keratinocytes (CPEK).

136 Consistent with in vivo results, epidermal keratinocytes cultured from (Epid)CaR(-/-) mic

137 ction of ODC activity did not rescue primary epidermal keratinocyte cultures isolated from ODCER2 mic

138 the culmination of signals exchanged between epidermal keratinocytes, dermal fibroblasts and leukocyt

139 Although epidermal keratinocyte development and differentiation p

140 N-acetyltransferase 1 alone as normal human epidermal keratinocytes did not express mRNA for N-acety

141 mber, functions as an important regulator of epidermal keratinocyte differentiation and survival.

142 order chromatin remodeling in the context of epidermal keratinocyte differentiation in the skin.

143 The expression of epidermal keratinocyte differentiation markers was affec

144 Calcium induces epidermal keratinocyte differentiation, but the mechanis

145 Proper epidermal keratinocyte differentiation, which is necessa

146 kinase-independent activity is required for epidermal keratinocyte differentiation.

147 idermal equivalent model that mimics in vivo epidermal keratinocyte differentiation.

148 In cultures of normal human epidermal keratinocytes dioxin accelerates cell differen

149 ion of protein kinase C-alpha (PKC alpha) to epidermal keratinocytes display a 10-fold increase in PK

150 In human skin, epidermal keratinocytes do not express IGF-1, and hence

151 As epidermal keratinocytes do not express integrin alpha(v)

152 h targeted overexpression of human PlGF-2 in epidermal keratinocytes, driven by a keratin 14 promoter

153 domain (TRADD)-dependent TNFR1 signaling in epidermal keratinocytes drives skin inflammation in Shar

154 cluding bone and cartilage, but not in basal epidermal keratinocytes, exhibit normal epidermal differ

155 Mice overexpressing soluble VEGFR3 in epidermal keratinocytes exhibited hypoplastic cutaneous

156 We found that human epidermal keratinocytes exposed to a vesicating dose (30

157 holipid classes was measured in normal human epidermal keratinocytes exposed to cumene hydroperoxide

158 It is therefore likely that normal human epidermal keratinocytes exposed to similar oxidative str

159 Terminally differentiating epidermal keratinocytes express a large number of struct

160 Epidermal keratinocytes express at least six CYP4F enzym

161 ere was a paracellular barrier defect in rat epidermal keratinocytes expressing a Ppp2r2a siRNA.

162 g, the dermal fibroblast has advantages over epidermal keratinocytes for delivering C7 to DEB patient

163 Epidermal keratinocytes form a structural and immune bar

164 The initial events that specify epidermal keratinocytes from ectodermal progenitor cells

165 POMC was secreted by both human epidermal keratinocytes (from 5 healthy donors) and matc

166 that microtubule disruption in normal human epidermal keratinocytes grown in low calcium media condi

167 phenotype through the transgenic delivery of epidermal keratinocyte growth factors or inflammatory me

168 induced skin cancer using immortalized human epidermal keratinocyte (HaCaT) cells through repetitive

169 212 murine cell line keratinocytes and human epidermal keratinocytes (HEK), express cell-surface GITR

170 sion was evaluated in cytokine-treated human epidermal keratinocytes (HEK)s, murine PAM 212 cell line

171 vels of miR-205 and miR-184 in primary human epidermal keratinocytes (HEKs) and corneal epithelial ke

172 Elevating FIH-1 levels in primary human epidermal keratinocytes (HEKs) and human corneal epithel

173 variants that are expressed in primary human epidermal keratinocytes (HEKs) and the biochemical activ

174 ions of select target genes in primary human epidermal keratinocytes (HEKs) using chromatin immunopre

175 Human epidermal keratinocytes (HEKs) were used to assess if QD

176 e growth arrest and differentiation of human epidermal keratinocytes (HEKs).

177 nking activities in cultured human and mouse epidermal keratinocytes in a modified in situ assay.

178 crease in integrin alpha9beta1 expression in epidermal keratinocytes in cutaneous and corneal wounds.

179 -time imaging and the number and diameter of epidermal keratinocytes in each epidermal cell layer as

180 for effects on human dermal fibroblasts and epidermal keratinocytes in monolayer culture, and for ef

181 py to be expressed at the plasma membrane of epidermal keratinocytes in mouse skin.

182 Here we report that epidermal keratinocytes in psoriatic lesions are charact

183 eta(-/-) mice exhibit increased apoptosis in epidermal keratinocytes in response to carcinogen treatm

184 mice produce hyperproliferative psoriasiform epidermal keratinocytes in response to stimulation.

185 ar function of Merlin in AJ establishment in epidermal keratinocytes in vitro and confirm that a role

186 nds, we compared VEGF production by oral and epidermal keratinocytes in vitro.

187 e antiproliferative effects on human primary epidermal keratinocytes in vitro.

188 alpha9beta1 is seen in basal and suprabasal epidermal keratinocytes in wounds.

189 in a variety of cell lines and primary human epidermal keratinocytes, indicating that the protein was

190 ith loss of Connexin 26 and Connexin 30 from epidermal keratinocyte intercellular junctions and accum

191 Here we show that attenuating PTEN in epidermal keratinocytes is a predisposing factor for UVB

192 he sonic hedgehog (shh) signaling pathway in epidermal keratinocytes is a primary event leading to th

193 otype whereby intercellular adhesion between epidermal keratinocytes is disrupted, adversely affectin

194 How p63 regulates metabolism in epidermal keratinocytes is incompletely understood, and

195 esults not only reveal how UV irradiation of epidermal keratinocytes is sensed by the innate immune s

196 utaneous wounds, the number of proliferating epidermal keratinocytes is significantly reduced in K14-

197 sults indicate that the inhibition of JNK in epidermal keratinocytes is sufficient to initiate their

198 tingly, the prolonged expression of Snail in epidermal keratinocytes is sufficient to recapitulate ea

199 (PKC) isoforms have been associated with the epidermal keratinocyte (KC) granular layer differentiati

200 ripheral blood mononuclear cells (PBMCs) and epidermal keratinocytes (KC) from patients with psoriasi

201 Primary epidermal keratinocytes (KC) from TG mice exhibit signif

202 Epidermal keratinocytes (KCs) and cannabinoid (CB) recep

203 During malignant transformation in skin, epidermal keratinocytes (KCs) frequently acquire the cap

204 und that IFN-beta was abundantly produced by epidermal keratinocytes (KCs) in psoriasis and during wo

205 The nucleus of epidermal keratinocytes (KCs) is a complex and highly co

206 ) that are in close contact with surrounding epidermal keratinocytes (KCs).

207 odermal progenitor cell specification to the epidermal keratinocyte lineage.

208 Inhibition of this pathway in epidermal keratinocytes may account, in part, for the th

209 sufficient retinoic acid synthesis by keloid epidermal keratinocytes may contribute to the pathogenes

210 Epidermal keratinocytes migrate through the epidermis up

211 Normal epidermal keratinocyte morphology is also retained.

212 of cSCC cell lines (n = 8) and normal human epidermal keratinocytes (n = 11) with real-time quantita

213 tutively expressed in cultured normal canine epidermal keratinocytes (NCEKs), and its expression leve

214 easible to discriminate between normal human epidermal keratinocytes (NHEK) and dermal fibroblasts (N

215 n peroxide (H(2)O(2)) levels in normal human epidermal keratinocytes (NHEK) and melanocytes (mel) wer

216 ifferentiated or differentiated normal human epidermal keratinocytes (NHEK) in culture, but is dramat

217 yme was detected in cultures of normal human epidermal keratinocytes (NHEK) in response to Ca(2+)-ind

218 owed that EGCG treatment of the normal human epidermal keratinocytes (NHEK) inhibits ultraviolet (UV)

219 ation of DNA ligase in cultured normal human epidermal keratinocytes (NHEK) on exposure to the DNA-da

220 how that a single low-dose exposure of human epidermal keratinocytes (NHEK) to an FS20 light source i

221 Rapidly proliferating normal human epidermal keratinocytes (NHEK) were infected with retrov

222 TLR4 mRNA expression in primary normal human epidermal keratinocytes (NHEK).

223 When microtubules of normal human epidermal keratinocytes (NHEKs) grown in low calcium med

224 Here, we used normal human epidermal keratinocytes (NHEKs) to determine the effects

225 We used normal human epidermal keratinocytes (NHEKs) to model N-BP effects on

226 ration and apoptosis studies in normal human epidermal keratinocytes (NHEKs) were conducted by MTT (3

227 ing sites using ChIP-on-chip in normal human epidermal keratinocytes (NHEKs).

228 al vein endothelial cells (HUVECs) and human epidermal keratinocytes (NHEKs).

229 ytes during wound repair and in normal human epidermal keratinocytes (NHEKs).

230 , in a dose-dependent manner in normal human epidermal keratinocytes (NHEKs).

231 eek for 8 weeks induced p53 mutations in the epidermal keratinocytes of 100% of the mice.

232 was significantly elevated (17-fold) in the epidermal keratinocytes of 7,12-dimethylbenz[a]anthracen

233 are intrinsic to cutaneous neurons and that epidermal keratinocytes only modulate this transduction.

234 of interferon-gamma and, unlike normal human epidermal keratinocytes, p27 association with cdk2 did n

235 In human HaCaT and primary epidermal keratinocytes, photodynamic induction of apopt

236 Hair regenerated from epidermal keratinocyte progenitor cells isolated from mu

237 carcinogen-treated transgenic skin, whereas epidermal keratinocyte proliferation in vitro was not af

238 No significant differences in TPA-induced epidermal keratinocyte proliferation were observed in C/

239 and displayed no detectable abnormalities in epidermal keratinocyte proliferation, differentiation, o

240 ion of COX-2 with either inhibitor decreased epidermal keratinocyte proliferation.

241 differentiation in monolayer cultures of rat epidermal keratinocytes (REKs), but high PpIX signals we

242 cycle exit and differentiation of suprabasal epidermal keratinocytes require nuclear IkappaB kinase a

243 homeostasis and terminal differentiation of epidermal keratinocytes required for formation of the sk

244 differentiation-mitosis checkpoint in human epidermal keratinocytes, resulting in impaired cell divi

245 in the maturation and activity of ADAM17 in epidermal keratinocytes, resulting in significantly upre

246 an 15-LOX expression vectors or normal human epidermal keratinocytes results in the production and ex

247 X receptor (RXR)-alpha and RXR-beta in mouse epidermal keratinocytes (RXR-alphabeta(ep-/-)) or a topi

248 factor-2 in skin of mice lacking RXRalpha in epidermal keratinocytes (RXRalpha(ep-/-) mice), which in

249 xpressed only by thymic epithelial cells and epidermal keratinocytes, Skint-1 drives specifically the

250 T pathway as new molecular targets of CsA in epidermal keratinocytes, suggesting a previously unknown

251 ression of CTIP2 was ablated specifically in epidermal keratinocytes, suggests that CTIP2 functions i

252 Fra-1 and Fra-2) that are key controllers of epidermal keratinocyte survival and differentiation, and

253 integration site 1) as a regulator of human epidermal keratinocyte survival.

254 Following UV exposure, epidermal keratinocytes synthesize proopiomelanocortin (

255 ls of VEGF protein and mRNA were observed in epidermal keratinocytes than in oral keratinocytes after

256 study, we have demonstrated in normal human epidermal keratinocytes that disruption of actin structu

257 tive correlations included the morphology of epidermal keratinocytes, the appearance of nests of nevu

258 In cultured epidermal keratinocytes, the induction of keratin 6 is t

259 ling after a calcium switch in primary human epidermal keratinocytes, the loss of this receptor did n

260 During terminal differentiation of the epidermal keratinocytes, the nucleus undergoes a program

261 During terminal differentiation of the epidermal keratinocytes, the nucleus undergoes programme

262 in-specific factor(s) derived primarily from epidermal keratinocytes, thereby providing a mechanism f

263 alpha-2-glycoprotein is normally produced by epidermal keratinocytes, these studies raise the possibi

264 ifferentiation and inhibits proliferation in epidermal keratinocytes through interaction with the vit

265 ore, we propose that loss of cell contact in epidermal keratinocytes through reactive oxygen species-

266 expression of KLK5 and KLK7 in normal human epidermal keratinocytes to better understand how these m

267 fic IQGAP requirement in vivo, we engineered epidermal keratinocytes to express individual IQGAP prot

268 eir surfaces were seeded with cultured human epidermal keratinocytes to form a skin equivalent.

269 epidermal growth factor (EGF) stimulation of epidermal keratinocytes to initiate the cell migration c

270 ession of multiple host-defense molecules in epidermal keratinocytes to promote healing.

271 se characterized by reduced adherence of the epidermal keratinocytes to the underlying dermis, and is

272 Furthermore, epidermal keratinocytes treated with p38 MAPK inhibitors

273 itochondrial apoptotic signaling pathways in epidermal keratinocytes, triggering apoptosis and afford

274 In transgenic mice overexpressing Bmx in epidermal keratinocytes, tumors induced by a two-stage c

275 ed transgenic mice that overexpress TRPV3 in epidermal keratinocytes under the control of the keratin

276 Thus, normal human epidermal keratinocytes undergo profound lipid oxidation

277 Surface epithelial cells, such as the epidermal keratinocyte, undergo a process of terminal ce

278 The major cell type of the epidermis, the epidermal keratinocyte, undergoes a carefully choreograp

279 We demonstrate here that in epidermal keratinocytes, unliganded heterodimers of vita

280 Epidermal keratinocytes, unlike other cells, express man

281 tional responses to ultraviolet radiation in epidermal keratinocytes using microarray chips containin

282 geted the overexpression of RasGRP1 to basal epidermal keratinocytes using the keratin 5 promoter.

283 ion in ultraviolet-B-irradiated normal human epidermal keratinocytes was also assessed and correlated

284 rehensively the JNK-regulated genes in human epidermal keratinocytes, we compared the transcriptional

285 16) E6 and E7 gene-immortalized normal human epidermal keratinocytes, we demonstrated intracellular a

286 In epidermal keratinocytes, we observed higher proportions

287 ma on human skin cells, primary normal human epidermal keratinocytes were exposed to ultraviolet-B ra

288 Immortalized HaCaT and adult human epidermal keratinocytes were used as a model and the eff

289 cells, showing several similarities to basal epidermal keratinocytes, were found to respond to extern

290 recently shown expression of RasGRP1 in the epidermal keratinocytes where it can mediate Ras activat

291 integrin alpha3beta1 is highly expressed in epidermal keratinocytes, where it regulates both cell-au

292 Differentiation of cultured primary epidermal keratinocytes with 0.12 mm Ca(2+) or 12-O-tetr

293 demonstrated that treatment of normal human epidermal keratinocytes with cis-UCA resulted in increas

294 We show that stimulation of epidermal keratinocytes with EGF, but not with other gro

295 To address this issue, we treated human epidermal keratinocytes with five EFNAs individually and

296 day revealed a functional circadian clock in epidermal keratinocytes with hundreds of transcripts reg

297 as altered differentiation and activation of epidermal keratinocytes with increased expression of hai

298 fibroblasts led to increased invasiveness of epidermal keratinocytes with pre-malignant features.

299 e presence of functional opioid receptors on epidermal keratinocytes, with attendant regulation of ke

300 tured human skin, or isolated cultured human epidermal keratinocytes, with triiodothyronine (100 pmol