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1 -3 ovarian carcinoma, A375 melanoma, or A431 epidermoid carcinoma.
2 receptor expression is upregulated in human epidermoid carcinoma.
4 e show that dense cultures of human salivary epidermoid carcinoma A253 cells exhibited elevated expre
5 Madin-Darby canine kidney (MDCK), and human epidermoid carcinoma (A253) cells contain the T-cell fac
6 filipin also inhibited CT activity in human epidermoid carcinoma A431 and Jurkat T lymphoma cells th
7 st two-hybrid interaction library from human epidermoid carcinoma A431 cells and identified four cell
8 ed/activated in response to insulin in human epidermoid carcinoma A431 cells as well as in mouse 3T3-
10 We show that ultraviolet-B exposure of human epidermoid carcinoma A431 cells results in G1 cell cycle
14 ) prevents completely the formation of human epidermoid carcinoma A431 xenografts in athymic mice.
16 on of Bcl-2 in PDT apoptosis-sensitive human epidermoid carcinoma (A431) cells resulted in enhanced a
19 n to have excellent oral activity in a human epidermoid carcinoma (A431) xenograft model in nude mice
20 radiation- and chemotherapy-resistant human epidermoid carcinoma and a hormone-refractory prostate a
21 or regressions at 10 mg/kg in the A431 human epidermoid carcinoma and at 40 mg/kg for the SF767 human
22 ocotrienol would suppress the growth of A431 epidermoid carcinoma and B16-F10 melanoma in vitro and i
25 cumulated in KB-V-1, a vinblastine-resistant epidermoid carcinoma but not in KB-3-1, drug-sensitive w
26 n accordance with its false identity as oral epidermoid carcinoma, but only 57 articles that provided
27 e experimental treatment of established A431 epidermoid carcinoma, C33a cervix carcinoma, and LS174T
28 g a soluble Mr 42,000 sialidase into a human epidermoid carcinoma cell line (A431) provided an approa
30 of IL-6 following UV irradiation of a human epidermoid carcinoma cell line (KB), and of normal human
31 rplastic parathyroid glands and in the human epidermoid carcinoma cell line A431, which mimics hyperp
32 cell lines (KB100 and KB300) from the human epidermoid carcinoma cell line KB by exposure to CPT.
34 n in human hepatoma cell lines and the human epidermoid carcinoma cell line, A431 which express TGFal
37 rface expressed EGFRs in A431 cells, a human epidermoid carcinoma cell line, is composed of two subpo
46 its degradation rate in infected A431 human epidermoid carcinoma cells and resulting in the alterati
47 f all tumor cells tested, only A431 (A431-V) epidermoid carcinoma cells developed partial resistance
49 tional regulator of VEGF expression, in A431 epidermoid carcinoma cells under both normoxic and hypox
50 ding of IRDye 800CW EGF to intact A431 human epidermoid carcinoma cells was quantified in a microplat
54 CKI-responsive cell line (A431 human vulvar epidermoid carcinoma cells with functional Rb) and one C
55 we re-expressed a series of CD151 mutants in epidermoid carcinoma cells with near total, RNAi-mediate
56 s, including WI-38 lung diploid cells, A-431 epidermoid carcinoma cells, and HeLa cervix epitheloid c
57 tyrosine in response to HGF-SF in A431 human epidermoid carcinoma cells, expressing the HGF/SF recept
58 is of loss-of-function mutants of A431 human epidermoid carcinoma cells, lacking protein kinase A, pr
66 rine P388 leukemia, as well as against human epidermoid carcinoma KB/8.5 implanted sc in athymic mice
70 a panel of tumor cell lines including human epidermoid carcinoma of the nasopharynx (KB) and its eto
71 east cancer (MCF-7), lung carcinoma (A-549), epidermoid carcinoma of the nasopharynx (KB), renal canc
73 perior in vivo antitumor activity in a human epidermoid carcinoma xenograft model with no overt toxic
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