ライフサイエンスコーパス: epididymal fat

1 sion of Egr1 while decreasing ATGL levels in epididymal fat.

2 showed significantly reduced body weight and epididymal fat.

3 dexamethasone prevented ATM accumulation in epididymal fat.

4 ependent and PPARalpha-independent manner in epididymal fat.

5 neal fat, but was without effect on GLUT4 in epididymal fat.

6 a, and an enhanced proinflammatory status of epididymal fat.

7 receptor type in islets, liver, kidney, and epididymal fat.

8 ce also exhibit adipocyte hypertrophy in the epididymal fat.

9 ositol-3'-kinase (PI-3-kinase) expression in epididymal fat (26% decrease; P < 0.1).

10 <0.01) less retroperitoneal, mesenteric, and epididymal fat accumulation, compared to their ob/ob cou

11 trophy and interlelukin-6 gene expression in epididymal fat, along with the splenic proinflammatory p

12 white adipose tissues including inguinal and epididymal fats and also in brown adipose tissue but not

13 ethylation of its promoter in progenitors of epididymal fat compared to Con offspring, which was corr

14 el was lower in human visceral fat and mouse epididymal fat compared with their subcutaneous fat.

15 at 6 mo) and had significantly more (P<0.01) epididymal fat content.

16 leasable and extractable LPL activity in the epididymal fat decreased by 75-80% in the diabetic group

17 lation of Foxa3 have a selective decrease in epididymal fat depot and a cell-autonomous defect to ind

18 rger cells (120-160 microm; P < 0.05) in the epididymal fat depot.

19 tissue (VAT) of lean mice, especially in the epididymal fat depot.

20 that regulates the selective enlargement of epididymal fat depots and suppresses energy expenditure

21 depot could affect vascular disease in mice, epididymal fat depots were transplanted into atheroscler

22 se in the diet, whereas [(3)H]NE turnover in epididymal fat did not respond to either monosaccharide.

23 oliferator-activated receptor (PPAR)gamma in epididymal fat; enzymes of fatty acid oxidation and thei

24 in GLUT4 (54 +/- 5% of high-carbohydrate) in epididymal fat from rats on the high-fat diet for 3 week

25 During HFD feeding, epididymal fat initiates adipogenesis after 4 weeks, whe

26 -induced glucose uptake in soleus muscle and epididymal fat; insulin inhibition of lipolysis was also

27 Thus, the rapid increase in epididymal fat mass with the cessation of voluntary whee

28 Absolute and relative epididymal fat mass, mean cell volume, and amount of lip

29 NA methylation in the zfp423 promoter in the epididymal fat of OB/Obe offspring, which was correlated

30 in confined well-vascularized sites like the epididymal fat pad (EFP) improved graft outcomes, but on

31 Micro) in the confined and well-vascularized epididymal fat pad (EFP) site, a model of the human omen

32 ouse islets engrafted on the intra-abdominal epididymal fat pad ameliorated streptozotocin-induced hy

33 ia occurred in all mice after removal of the epididymal fat pad bearing the islet graft.

34 This study indicates that the epididymal fat pad maybe a useful islet transplant site

35 eeded scaffolds were then implanted onto the epididymal fat pad of syngeneic mice with streptozotocin

36 The epididymal fat pad was evaluated as a site of islet tran

37 t affected until week 28 (decreased by 14%); epididymal fat pad weight also decreased (25%) at this t

38 yperleptinemia and reduction in food intake, epididymal fat pad weight declined 55% in wild-type but

39 air-fed group, a significant increase in the epididymal fat pad weight, BAT weight, and plasma leptin

40 A significant increase in the epididymal fat pad weight, interscapular brown adipose t

41 ell as lower hepatic triglyceride levels and epididymal fat pad weights than in SHR harboring mutant

42 3) Does short-term elimination of T cells in epididymal fat pad without disturbing the systemic T cel

43 o the devices, which were implanted into the epididymal fat pad(s) of streptozocin diabetic mice.

44 er visceral adipose tissue mass estimated by epididymal fat pad, associated with iron accumulation in

45 w as 50 islets, either intraportal or in the epididymal fat pad, displayed similar glucose tolerance

46 ic mass of syngeneic islets implanted in the epididymal fat pad, followed by a subrenal capsular impl

47 that for M-CPT I in RNA isolated from whole epididymal fat pad, this was reversed in purified adipoc

48 mia and increased homocysteine levels in the epididymal fat pad, which was associated with decreased

49 quantified by magnetic resonance imaging and epididymal fat-pad weights.

50 cally, adiponectin KO mice possessed smaller epididymal fat pads and showed reduced body weight compa

51 To determine the mechanism, epididymal fat pads from normal wild-type (+/+) and obes

52 Acute depletion of T cells from epididymal fat pads improved insulin action in young DIO

53 have greater deposits of s.c. fat and larger epididymal fat pads in comparison with wild-type mice.

54 PPARgamma-regulated genes were higher in the epididymal fat pads of Adipoq-LPL mice than control mice

55 .4-microm pore membranes when implanted into epididymal fat pads of rats.

56 adipocytes from SPARC-null versus wild-type epididymal fat pads were 252 +/- 61 and 161 +/- 33 micro

57 stablish feasibility of fat transplantation, epididymal fat pads were harvested from wild-type C57BL/

58 Incubation of epididymal fat pads with leptin or its i.v. injection in

59 ties, including low fertility, an absence of epididymal fat pads, and a tendency to develop blepharit

60 nd in primary adipocytes isolated from mouse epididymal fat pads, in response to acute activation of

61 ere found to have lower body weight, smaller epididymal fat pads, lower blood levels of nonesterified

62 n messenger RNA (mRNA) and protein levels in epididymal fat pads.

63 that GSK-3 activity increased twofold in the epididymal fat tissue and remained unchanged in muscle a

64 ntrast, GSK-3 activity did not change in the epididymal fat tissue of A/J mice, regardless of the typ

65 th reduced levels of the pP65 protein in the epididymal fat tissue, suggesting less activation of the

66 1 protein levels were significantly lower in epididymal fat tissues from db/db and high fat diet-indu

67 is associated with a prolonged overshoot in epididymal fat triacylglycerol synthesis.

68 The rate of triacylglycerol synthesis in epididymal fat was 4.2-fold greater in SED5 than in WL5,

69 C/EBPalpha protein levels in epididymal fat were 30% greater in SED5 than in WL5.

70 H2O content and wt of epididymal fat were increased by RSG and correlated to i

71 t and GSK3 phosphorylation and activities in epididymal fat were opposite to those of brain after str