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1 modified quenching constant, followed by (-)-epigallocatechin-3-gallate.
2 nterventions provided similar amounts of (-)-epigallocatechin-3-gallate.
3 e we show that the tea-derived flavanol, (-)-epigallocatechin 3-gallate [(2R,3R)-5,7-dihydroxy-2-(3,4
4 d reactivities of the green tea extract, (-)-epigallocatechin-3-gallate [(2R,3R)-5,7-dihydroxy-2-(3,4
5 inistration of green tea extracts containing epigallocatechin 3-gallate, a potent DYRK1A inhibitor, t
8 showed that 3-d supplementation of combined epigallocatechin-3-gallate and resveratrol (EGCG+RES) in
9 ea polyphenol preparation containing 65% (-)-epigallocatechin-3-gallate] and caffeine on 4-(methylnit
10 h tea polyphenols or the major tea component epigallocatechin-3-gallate blocked expression of the hyp
11 phanyl-3-butenyl isothiocyanates; G) and (-)-epigallocatechin-3-gallate (E) were investigated in colo
12 echin (EGC), epicatechin 3-gallate (ECG) and epigallocatechin 3-gallate (EGCG) and caffeine in 29 com
13 sly, we showed that the green tea polyphenol epigallocatechin 3-gallate (EGCG) inhibits growth of NF6
15 ted that the major green tea polyphenol, (-)-epigallocatechin 3-gallate (EGCG), exerts potent neuropr
16 abilities of apigenin, baicalein, curcumin, epigallocatechin 3-gallate (EGCG), genistein, quercetin,
18 sly, we showed that the green tea polyphenol epigallocatechin-3 gallate (EGCG) inhibits growth and tr
19 ole of green tea polyphenols, the effects of epigallocatechin-3 gallate (EGCG) on Her-2/neu-overexpre
20 ype, the effects of the green tea polyphenol epigallocatechin-3 gallate (EGCG) on mammary tumor cells
21 ecursor N-acetyl-L-cysteine (NAC), curcumin, epigallocatechin-3 gallate (EGCG), and p38 inhibitor SB2
23 sms of the protection conferred by sugars to epigallocatechin-3-gallate (EGCG) against deterioration.
24 catechin, (-)-epigallocatechin (EGC) and (-)-epigallocatechin-3-gallate (EGCG) and butylated hydroxyt
25 s of two main constituents of green tea, (-)-epigallocatechin-3-gallate (EGCG) and caffeine, on intes
26 s known to modulate its aggregation, such as epigallocatechin-3-gallate (EGCG) and dopamine (DA).
29 Recent reports suggest green tea-derived (-)-epigallocatechin-3-gallate (EGCG) can attenuate neuronal
31 we have shown that green tea polyphenol (-)-epigallocatechin-3-gallate (EGCG) exerts a beneficial ro
36 (4-hydroxyphenyl) retinamide (4-HPR) and (-)-epigallocatechin-3-gallate (EGCG) in altering expression
38 y was undertaken to evaluate the efficacy of epigallocatechin-3-gallate (EGCG) in down-regulating Mcl
39 of epigenetic bioactive botanicals including epigallocatechin-3-gallate (EGCG) in green tea polypheno
41 owed that the bioactive green tea polyphenol epigallocatechin-3-gallate (EGCG) inhibits growth in sof
44 with anti-amyloid agents like the polyphenol epigallocatechin-3-gallate (EGCG) is emerging as an expe
49 fect of vanillin, resveratrol, curcumin, and epigallocatechin-3-gallate (EGCG) on the aggregation of
51 to scavenge H2O2 (400muM) and influence (-)-epigallocatechin-3-gallate (EGCG) oxidation (400muM) in
52 ract and its main polyphenol constituent (-)-epigallocatechin-3-gallate (EGCG) possess potent neuropr
54 Polyphenon E with a standardized dose of epigallocatechin-3-gallate (EGCG) was administered using
56 major effective component in green tea, (-)-epigallocatechin-3-gallate (EGCG)'s potential benefits t
57 major green tea polyphenols (catechins), (-)-epigallocatechin-3-gallate (EGCG), (-)-epigallocatechin,
58 resent study, we investigated the effects of epigallocatechin-3-gallate (EGCG), a bioactive green tea
61 d antioxidants included three phenolics: (-)-epigallocatechin-3-gallate (EGCG), a flavanoid polypheno
62 the present study, we examined the effect of epigallocatechin-3-gallate (EGCG), a green tea polypheno
64 on CLL B cells, and we studied the impact of epigallocatechin-3-gallate (EGCG), a known receptor tyro
70 nefits of green tea are mainly attributed to epigallocatechin-3-gallate (EGCG), a polyphenolic compou
72 as conducted to determine to what extent (-)-epigallocatechin-3-gallate (EGCG), a widely used dietary
74 rions, Sindbis virus, and the polyphenol (-)-epigallocatechin-3-gallate (EGCG), and has been implicat
75 ation (1-500 muM) of a model polyphenol, (-)-epigallocatechin-3-gallate (EGCG), and matrix pH (2-7) o
76 muscle for the major green tea catechin, (-)-epigallocatechin-3-gallate (EGCG), but effects were obse
77 effect of two small molecules, FK506 and (-)-epigallocatechin-3-gallate (EGCG), known to inhibit alph
78 ea extract (GTE) and its major catechin, (-)-epigallocatechin-3-gallate (EGCG), on collagen homeostas
79 larly its major polyphenolic constituent (-)-epigallocatechin-3-gallate (EGCG), possesses remarkable
80 bond-containing tea polyphenols, such as (-)-epigallocatechin-3-gallate (EGCG), potently and specific
81 yphenols (GTPP) and their active ingredient, epigallocatechin-3-gallate (EGCG), protects cells from s
82 eritoneal (i.p.) injection (20 mg/kg) of (-)-epigallocatechin-3-gallate (EGCG), the main polyphenolic
87 n previously that topical application of (-)-epigallocatechin-3-gallate (EGCG), the major polyphenol
88 e and animal model studies indicate that (-)-epigallocatechin-3-gallate (EGCG), the major polyphenol
96 ion by two dietary polyphenols, curcumin and epigallocatechin-3-gallate (EGCG), with that by the endo
99 nts, such as the vitamin E analog trolox and epigallocatechin-3-gallate, enhanced hypoxia-induced cas
100 ) at pH 7.5 and 25 degrees C: catechins [(-)-epigallocatechin-3-gallate, (-)-epigallocatechin, (-)-ep
101 ergies for (-)-epicatechin-3-gallate and (-)-epigallocatechin-3-gallate for the binding site on BSA n
104 e candidate compounds: rifampicin, curcumin, epigallocatechin-3-gallate, myricetin, and scyllo-inosit
105 biologically active green tea catechin, (-)-epigallocatechin-3-gallate or (-)-EGCG, has been shown t
106 In a limited phytonutrient screen, EGCG ((-)-epigallocatechin 3-gallate), the major phytochemical in
107 Studies from our laboratory have shown that epigallocatechin-3-gallate, the major polyphenol present
108 By using this system, the natural product epigallocatechin 3-gallate was found to block Wnt signal
109 oxaldehyde, ferulic acid, vanillic acid, and epigallocatechin-3-gallate, were effective inhibitors of
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