ライフサイエンスコーパス: epinephrine

1 (0.5% L-bupivacaine and 1.5% lidocaine with epinephrine).

2 oral antihistamines only, and none received epinephrine.

3 nition of loss of pulse to the first dose of epinephrine.

4 kable rhythm who received at least 1 dose of epinephrine.

5 treatment, and the cardiovascular effects of epinephrine.

6 n plus intrathoracic pressure regulator plus epinephrine.

7 opharynx, with no severe symptoms or uses of epinephrine.

8 e, plasma cortisol, prolactin, oxytocin, and epinephrine.

9 t was not responsive to electrical shocks or epinephrine.

10 stress was modeled by sustained delivery of epinephrine.

11 and 0.23 (95% CI: 0.14 to 0.37) for >5 mg of epinephrine.

12 pic G-protein coupled receptor (GPCR), using epinephrine.

13 be used in anaphylactic shock refractory to epinephrine.

14 onship to the need for prompt treatment with epinephrine.

15 ith ascorbic acid, dopamine, paracetamol and epinephrine.

16 ment with antihistamines, corticosteroids or epinephrine.

17 ed AEs, 59% with antihistamines and 12% with epinephrine.

18 al immunotherapy and some require injectable epinephrine.

19 Thirteen patients (6.7%) required injectable epinephrine.

20 re laryngopharyngeal disorders and no use of epinephrine.

21 der medical supervision and was treated with epinephrine.

22 Advanced life support always included IV epinephrine (0.05 mug/kg).

23 receive either dopamine (5-10 mug/kg/min) or epinephrine (0.1-0.3 mug/kg/min) through a peripheral or

24 nce interval [CI]: 0.27 to 0.84) for 1 mg of epinephrine, 0.30 (95% CI: 0.20 to 0.47) for 2 to 5 mg o

25 Buffering 2% lidocaine with epinephrine 1:100 000 with sodium bicarbonate 8.4% offer

26 und guidance, 20-25 mL of 0.25% bupivacaine (epinephrine 1:400 000) were injected near the triangles-

27 ed either vasopressin (20 IU/CPR cycle) plus epinephrine (1 mg/CPR cycle; cycle duration approximatel

28 (VSE group, n = 130) or saline placebo plus epinephrine (1 mg/CPR cycle; cycle duration approximatel

29 After 4 to 13 weeks, epinephrine (1 mug kg(-1) min(-1)) was infused, and the

30 ,556 eligible patients, 1,134 (73%) received epinephrine; 194 (17%) of these patients had a good outc

31 n therapy (1D-2D), and norepinephrine and/or epinephrine (1D).

32 -enhanced cardiopulmonary resuscitation plus epinephrine (24+/-6 min, 63+/-8 min, and 50+/-9 min, res

33 mine (11)C-hydroxyephedrine was smaller than epinephrine (41 +/- 8 vs. 47% +/- 6% of left ventricle,

34 ive to metabolic degradation, was similar to epinephrine (48 +/- 6 vs. 47% +/- 6%, P = 0.011 vs. perf

35 65-92% in capsaicin-treated animals, as was epinephrine (74%), norepinephrine (33%), and glucagon (4

36 t incubation of normal RBCs and SS-RBCs with epinephrine, a catecholamine that binds to the beta-adre

37 utive patients with cardiac arrest requiring epinephrine according to resuscitation guidelines (from

38 Previous studies showed that epinephrine activates the beta2-adrenergic receptor (B2A

39 ntify hospital variation in rates of delayed epinephrine administration (>5 minutes) and its associat

40 les, compared to without, had lower rates of epinephrine administration (incidence rate per 10,000 st

41 the extent of hospital variation in delayed epinephrine administration and its effect on hospital-le

42 elation between a hospital's rate of delayed epinephrine administration and its risk-standardized rat

43 tion and potential allergic reactions (using epinephrine administration as a surrogate event) after A

44 ributable to nonshockable rhythms, delays in epinephrine administration beyond 5 minutes is associate

45 Delays in epinephrine administration following in-hospital cardiac

46 e effect of peanut-free policies on rates of epinephrine administration for allergic reactions in Mas

47 Hospitals with high rates of delayed epinephrine administration had lower rates of overall su

48 etrospective study, we analyzed (1) rates of epinephrine administration in all Massachusetts public s

49 Prompt epinephrine administration is crucial in managing anaphy

50 Patients with time to epinephrine administration of longer than 5 minutes (233

51 having peanut-free classrooms did not affect epinephrine administration rates.

52 Longer time to epinephrine administration was also associated with decr

53 Longer time to epinephrine administration was associated with lower ris

54 The odds of delay in epinephrine administration were 58% higher at 1 randomly

55 Rates of epinephrine administration were compared for schools wit

56 The odds of C difficile infection and epinephrine administration were significantly higher amo

57 lic access automated external defibrillator, epinephrine administration, and time intervals.

58 er improving hospital performance on time to epinephrine administration, especially at hospitals with

59 al associated with timely defibrillation and epinephrine administration, these findings provide impor

60 Epinephrine (adrenaline) treatment is underused in healt

61 Compared with epinephrine alone, the methylene blue-epinephrine associ

62 Treatment with epinephrine also reduced the systemic accumulation of bo

63 Here we demonstrate that epinephrine alters the neutrophil (polymorphonuclear leu

64 sulted in a 37% increase in plasma levels of epinephrine and a 44% increase in plasma norepinephrine

65 ulinum antitoxin recipients and will require epinephrine and antihistamine treatment and, possibly, i

66 Current treatments for allergies include epinephrine and antihistamines, which treat the symptoms

67 The adrenal stress hormones epinephrine and corticosterone released by emotional aro

68 Thus, the stress-related hormones epinephrine and corticosterone selectively modulate acut

69 hat autonomic neurons are more responsive to epinephrine and corticosterone than are sensory neurons,

70 e neuronal cultures with the stress hormones epinephrine and corticosterone.

71 r hypoglycemia symptom scores and had higher epinephrine and cortisol responses compared with the una

72 the relationship between pre-hospital use of epinephrine and functional survival among patients with

73 impaired glucose sensing, noted by impaired epinephrine and glucagon responses and impaired c-fos ac

74 Therefore, the increased epinephrine and glucagon secretion with declining plasma

75 cogen that markedly elevated the response of epinephrine and glucagon to a given hypoglycemia and inc

76 ds, and more recently with nebulized racemic epinephrine and hypertonic saline.

77 from anaphylactic shock may be refractory to epinephrine and impair cerebral oxygenation and metaboli

78 requiring vasopressors, combined vasopressin-epinephrine and methylprednisolone during CPR and stress

79 vening types had overtly higher 24 h urinary epinephrine and morning plasma ACTH levels, and higher m

80 Epinephrine and norepinephrine are present in the pro-ur

81 This study reveals epinephrine and norepinephrine as novel regulators of ce

82 hesis, which results in dopamine, serotonin, epinephrine and norepinephrine deficiencies.

83 eceived epinephrine during resuscitation and epinephrine and norepinephrine in the early in-hospital

84 Catecholamines such as epinephrine and norepinephrine promote energy expenditur

85 Both circulating epinephrine and norepinephrine released from adrenal med

86 responses to the hormones/neurotransmitters epinephrine and norepinephrine which are found in the ne

87 ources of enteric neurotransmitters, such as epinephrine and norepinephrine, that are known to increa

88 expressed adrenergic receptors (activated by epinephrine) and the glucocorticoid receptor (activated

89 0.30 (95% CI: 0.20 to 0.47) for 2 to 5 mg of epinephrine, and 0.23 (95% CI: 0.14 to 0.37) for >5 mg o

90 mines, 10% called 911, 11% self-administered epinephrine, and 6.4% received no treatment.

91 d epinephrine content and circulating plasma epinephrine, and decreased adrenal CgB.

92 metabolic rate, insulin, glucagon, cortisol, epinephrine, and hunger ratings.

93 r blockers (ARBs), beta-adrenergic blockers, epinephrine, and Kounis syndrome.

94 olamine neurotransmitters, including L-DOPA, epinephrine, and norepinephrine.

95 ic acid, catechol, phenethylamine, tyrosine, epinephrine, and norepinephrine.

96 erall, 13 213 (12.7%) patients had delays to epinephrine, and this rate varied markedly across hospit

97 its treatment is delayed, with little use of epinephrine; and its underlying cause or causes are poor

98 e to LAMA5, and insignificant stimulation by epinephrine as compared to SS-RBCs from untreated patien

99 d with epinephrine alone, the methylene blue-epinephrine association avoided neuronal excitotoxicity

100 n plus epinephrine groups received 0.5 mg of epinephrine at 4.5 and 9 minutes of cardiopulmonary resu

101 neurotransmitters (melatonin, serotonin, and epinephrine) at various concentrations followed by the S

102 riencing anaphylaxis nor being prescribed an epinephrine auto-injector (EAI) contributed to impairmen

103 While 94% of patients took an epinephrine auto-injector (EAI) into risky situations, o

104 en, and to establish the trend of prescribed epinephrine auto-injectors (EAI) among paediatric popula

105 tion is crucial in managing anaphylaxis, but epinephrine auto-injectors (EAIs) are underutilized by p

106 of symptomatic medications that may include epinephrine auto-injectors.

107 ents were also less likely to have filled an epinephrine autoinjector (EAI) prescription or visited a

108 nut allergy relies on allergen avoidance and epinephrine autoinjector for rescue treatment in patient

109 y treated, of which 10% were treated with an epinephrine autoinjector.

110 No epinephrine autoinjectors contain an optimal dose for in

111 prescription, and 60% did not currently have epinephrine available.

112 ibits apoptosis in prostate cancer cells via epinephrine/beta2 adrenergic receptor/PKA/BAD pathway.

113 kable rhythm who received at least 1 dose of epinephrine between 2000 and 2014.

114 The physiologic stimuli, glucagon and epinephrine, both increased hepatic glucose production,

115 in response to adenosine 5'-diphosphate and epinephrine, but variable aggregation defects with other

116 ponse to arachidonic acid, ADP, collagen, or epinephrine by optical aggregometry.

117 cells, increased adrenal norepinephrine and epinephrine content and circulating plasma epinephrine,

118 Refractoriness to epinephrine could be corrected by nitric oxide pathway i

119 ratio = 1.41; 95% CI, 1.01-1.96; p = 0.04), epinephrine cumulate dose less than or equal to 3 mg (ha

120 Stress-related mediators (eg, epinephrine) decrease this threshold, leading to autopha

121 Time to epinephrine, defined as time in minutes from recognition

122 5.4%) at hospitals in the lowest quartile of epinephrine delay, risk-standardized survival was 16% lo

123 an initial nonshockable rhythm who received epinephrine, delay in administration of epinephrine was

124 als in the quartile with the highest rate of epinephrine delays (10.8%; interquartile range, 9.7%-12.

125 circumferential distribution following local epinephrine delivery from a distributed source to the en

126 ction efficiency can be successfully used in epinephrine detection for filtering out signals from asc

127 tion (OR: 0.31; 95% CI: 0.19 to 0.51), lower epinephrine dosage (OR: 0.47; 95% CI: 0.25 to 0.87), and

128 Delay in administration of the first epinephrine dose is associated with decreased survival a

129 lopment of autoinjectors containing a 0.1-mg epinephrine dose suitable for infants, and inclusion of

130 The median time to first epinephrine dose was 1 minute (IQR, 0-4; range, 0-20; me

131 with standard advanced cardiac life support epinephrine dosing (Guideline care).

132 Total epinephrine dosing and defibrillation attempts were not

133 mm with standard American Heart Association epinephrine dosing.

134 mm with standard American Heart Association epinephrine dosing; or 3) Depth 51 mm: target chest comp

135 More nonbradycardia group patients received epinephrine during resuscitation and epinephrine and nor

136 We further hypothesized that the addition of epinephrine during sodium nitroprusside-enhanced cardiop

137 Intracameral phenylephrine or epinephrine, either by direct injection or placement in

138 ines viz., dopamine (DA), levodopa (l-Dopa), epinephrine (EP) and norepinephrine (NE) using cyclic vo

139 cultures revealed that they were composed of epinephrine (EP) and/or norepinephrine (NE) type cells.

140 mination of dopamine (DA) in the presence of epinephrine (EP) at a gold nanoparticles chemically modi

141 ive towards the electrochemical detection of Epinephrine (Ep), in the presence of Serotonine-5-HT (S-

142 /kg) (n = 6); and allergic rats treated with epinephrine (EPI) (10 microg/kg) (n = 6).

143 Both epinephrine (EPI) and norepinephrine (NE) could directly

144 h 5-thioglucose stimulated adrenal medullary epinephrine (Epi) release (3,153%) and feeding (400%), w

145 a or the host, such as autoinducer-3 (AI-3), epinephrine (Epi), and norepinephrine (NE).

146 roxyephedrine, HED), vesicular storage (C-11 epinephrine, EPI), and metabolic degradation (C-11 pheny

147 thylene blue bolus (methylene blue group) or epinephrine (epinephrine group) or both (methylene blue-

148 oups: 1) epinephrine, nebulized with 4 mg of epinephrine every 4 hours starting 1 hour post injury, n

149 ability analysis, and decreased 24-h urinary epinephrine excretion rate by 7%, without a significant

150 those with ACS), the decision to administer epinephrine for anaphylaxis can be difficult, and its be

151 mend standardized interval administration of epinephrine for patients in cardiac arrest.

152 ia point-source release generated transmural epinephrine gradients directly beneath the site of appli

153 ) in the dopamine group and four (7%) in the epinephrine group (p=0.033).

154 bolus (methylene blue group) or epinephrine (epinephrine group) or both (methylene blue-epinephrine g

155 (epinephrine group) or both (methylene blue-epinephrine group).

156 -enhanced cardiopulmonary resuscitation plus epinephrine groups received 0.5 mg of epinephrine at 4.5

157 -enhanced cardiopulmonary resuscitation plus epinephrine groups, respectively; p=0.001).

158 vestigating the effectiveness of adrenaline (epinephrine), H1-antihistamines, systemic glucocorticost

159 Treatment with epinephrine had a significant reduction of the pulmonary

160 antigens and availability of self-injectable epinephrine has been a major focus of research teams, ad

161 after randomization, followed by additional epinephrine if needed.

162 ll decreased with continued cell exposure to epinephrine, implying that activation of ICAM-4-mediated

163 ll 31 patients with a fatal outcome received epinephrine in a titrated manner according to internatio

164 provide significant evidence of the role of epinephrine in BCAM/Lu-laminin and ICAM-4-alpha(v)beta(3

165 The fraction of intramuscular epinephrine in professional emergency treatment increase

166 me was to compare the effects of dopamine or epinephrine in severe sepsis on 28-day mortality; second

167 In the absence of epinephrine in the irrigation bottle, 12.4% of control e

168 propagation with simultaneous imprinting of epinephrine in the polymeric network.

169 We also suggest dobutamine or epinephrine in the presence of cardiogenic shock (2D) an

170 Epinephrine increased rate of matching beats from 35+/-4

171 Epinephrine increases myocardial contractility, decrease

172 can and European Americans for collagen- and epinephrine-induced aggregation, and in European America

173 ime, and reduced survival following collagen/epinephrine-induced pulmonary embolism were also observe

174 rtery and a higher mortality due to collagen/epinephrine-induced pulmonary thromboemboli.

175 o significantly protected from collagen plus epinephrine-induced pulmonary thromboembolism.

176 nt mice were more resistant to collagen- and epinephrine-induced thromboembolism compared with wild-t

177 ls of carotid artery thrombosis and collagen/epinephrine-induced thromboembolism.

178 ury; however, the precise mechanism by which epinephrine influences inflammatory response and wound h

179 acemaker function during circadian rhythm or epinephrine infusion.

180 d 40% O2: (1) during intravenous adrenaline (epinephrine) infusion at 320 ng kg(-1) min(-1) (320 ADR)

181 but there is no absolute contraindication to epinephrine injection in anaphylaxis.

182 ratio to receive (1) an intra-oral lidocaine-epinephrine injection with buffered saline nasal spray b

183 patients who failed endoscopic therapy with epinephrine injection, clip, or thermal therapy.

184 Although epinephrine is essential for successful return of sponta

185 appropriate prescription of self-injectable epinephrine is important.

186 Epinephrine is life-saving in anaphylaxis; second-line m

187 severe IFIS can occur in low-risk eyes when epinephrine is omitted from the irrigation bottle.

188 thway deconvolution revealed that the DMR of epinephrine is originated mostly from the remodeling of

189 Although adrenaline (epinephrine) is a cornerstone of initial anaphylaxis tre

190 Epinephrine levels during hypoglycemia were similar betw

191 the same dose rates, hemodynamic responses, epinephrine levels in the coronary sinus and systemic ci

192 contractility with local application, tissue epinephrine levels were high and variable--only a small

193 Plasma epinephrine levels were normal and increased when the pa

194 stration, correlated strongly with preceding epinephrine levels, and were higher.

195 ee cortisol, 24 h urinary norepinephrine and epinephrine levels, morning plasma ACTH and serum cortis

196 xia resulting in profoundly increased plasma epinephrine levels.

197 that combined glucocorticoids and nebulized epinephrine may be associated with lower hospitalization

198 t additional studies to determine if and how epinephrine may provide long-term functional survival be

199 proinflammatory macrophages is critical for epinephrine-mediated IL-6 production.

200 tagged PMNs in a murine skin wound, chronic, epinephrine-mediated stress was modeled by sustained del

201 e in vascular permeability and collagen- and epinephrine-mediated thromboembolism in mice.

202 The epinephrine-methylene blue association was the most effe

203 Epinephrine must still be considered as the first-line v

204 -enhanced cardiopulmonary resuscitation plus epinephrine (n=10), and active compression-decompression

205 y, sheep were randomized into two groups: 1) epinephrine, nebulized with 4 mg of epinephrine every 4

206 e of competitive concentrations of catechol, epinephrine, norepinephrine, 3,4-dihydroxy-phenylalanine

207 a resulted in significant blunting of plasma epinephrine, norepinephrine, glucagon, cortisol, and gro

208 glucose levels (5.3 +/- 0.1 mmol/L), plasma epinephrine, norepinephrine, glucagon, cortisol, and gro

209 lacebo resulted in significant reductions of epinephrine, norepinephrine, glucagon, growth hormone, c

210 So far, no direct role of epinephrine/norepinephrine in cellular iron homeostasis

211 Here we show that epinephrine/norepinephrine regulates iron homeostasis co

212 al aconitase (ACO2) activity are elevated by epinephrine/norepinephrine that are blocked by the antio

213 To restore the energy balance, epinephrine/norepinephrine-exposed cells may face higher

214 We demonstrate the role of epinephrine/norepinephrine-induced generation of reactiv

215 also observed in liver and muscle tissues of epinephrine/norepinephrine-injected mice.

216 Delayed deteriorations treated with epinephrine occurred in 29 of 315 anaphylaxis cases (9.2

217 (233/1558) compared with those with time to epinephrine of 5 minutes or less (1325/1558) had lower r

218 onectin secretion could not be stimulated by epinephrine or CL in adipocytes isolated from obese/type

219 nduced pulmonary thromboembolism by collagen/epinephrine or long-chain polyphosphate, Klkb1(-/-) mice

220 and selective provocative drug testing with epinephrine or procainamide.

221 Epinephrine or the beta3-adrenergic receptor (AR) agonis

222 to the phenethylamines norepinephrine (NE), epinephrine, or phenylephrine (PE) than are the alpha1B

223 ation responses to serotonin (p = 0.007) and epinephrine (p = 0.004) compared with men.

224 L (3.3 mmol/L) with increases in both plasma epinephrine (P = 0.01) and glucagon (P = 0.01).

225 lar tachycardia was associated with a larger epinephrine/perfusion mismatch (n = 11).

226 wall thickening in myocardial segments with epinephrine/perfusion mismatch (n = 6).

227 s), 52% had never received a self-injectable epinephrine prescription, and 60% did not currently have

228 highly sensitive and selective response for epinephrine, prevalent in aqueous and real samples at ul

229 ression in the adrenal medulla and increased epinephrine production were also observed.

230 that, despite increases in return of pulses, epinephrine reduces long-term survival and functional re

231 ide Y prevents a fasting-induced increase in epinephrine release and results in hypoglycemia in vivo.

232 of the sympathetic nervous system results in epinephrine release and subsequent suppression of the in

233 s the autonomic nervous system that controls epinephrine release from adrenal chromaffin cells and, c

234 catecholamine (CA; dopamine, norepinephrine, epinephrine) release within the social behavior neural n

235 Some epinephrine-resistant cases may play a role in our high

236 sponses in RH rats and restored the impaired epinephrine response to hypoglycemia in STZ-diabetic ani

237 ced an approximately 30% reduction in plasma epinephrine response together with reduced EGP and hypog

238 n the STZ group, consistent with the blunted epinephrine response.

239 abetic rats, and this augmented glucagon and epinephrine responses and hepatic glucose production dur

240 y, SGLT1 knockdown improved the glucagon and epinephrine responses in RH rats and restored the impair

241 Without a rise in glucagon, the epinephrine responses were much larger (DeltaAUC of 204

242 Epinephrine restored partially systemic hemodynamic vari

243 Prolonged systemic exposure of epinephrine resulted in persistent PMN trafficking to th

244 hed a nadir of approximately 2.0 mmol/L, and epinephrine rose to approximately 900 pg/mL.

245 ne in postresuscitation shock, compared with epinephrine/saline placebo, resulted in improved surviva

246 essed, glucagon secretion was recovered, and epinephrine secretion was improved after transplantation

247 thmic contractions caused by norepinephrine, epinephrine, serotonin, and forskolin in atrial trabecul

248 antioxidants, including dopamine, uric acid, epinephrine, serotonin, histamine, and 4-acetaminophen,

249 Nebulized epinephrine should be considered for use in future clini

250 Moreover, we found that epinephrine signaling changes the synergism pattern and

251 The administration of epinephrine significantly increased the contractile ampl

252 est, resuscitation was performed with drugs (epinephrine, sodium bicarbonate, and heparin), ventilati

253 dingly, the mutant alpha2B -AR increases the epinephrine-stimulated calcium signaling.

254 Zyxin-deficient mice exhibit impaired epinephrine-stimulated VWF release, prolonged bleeding t

255 and capillary density, they exhibit impaired epinephrine-stimulated VWF release, reduced levels of hi

256 an important mechanistic link to explain how epinephrine stress exacerbates inflammation via increase

257 B reversal, no patients received atropine or epinephrine, suffered cardiac arrest, or died within 30

258 ation before receipt of a third 1-mg dose of epinephrine), survival rate at hospital discharge among

259 anel of beta(2)-adrenergic receptor ligands (epinephrine, terbutaline, metaproterenol, salmeterol, pr

260 Compared with patients who did not receive epinephrine, the adjusted odds ratio of intact survival

261 oxidizes circulating catecholamines such as epinephrine, there has been no convincing demonstration

262 nerated for arachidonic acid, ADP, collagen, epinephrine, Thrombin receptor activating-peptide, U4661

263 Epinephrine tissue concentration, upregulation of cAMP,

264 pronociceptive mediators, prostaglandin E2, epinephrine, TNFalpha, and interleukin-6, and the neurop

265 Sustained administration of epinephrine to NLB rats mimicked sound stress effect.

266 The addition of epinephrine to sodium nitroprusside-enhanced cardiopulmo

267 C) drug releasing systems were used to apply epinephrine to the anterior surface of the left heart of

268 ere was no difference in the requirement for epinephrine to treat reactions (P = .55).

269 Epinephrine treatment abolished the genotype differences

270 oughout 5 years of follow-up, whereas prompt epinephrine treatment for asystole/pulseless electric ac

271 ital survival with prompt defibrillation and epinephrine treatment in patients with in-hospital cardi

272 In productively infected neuronal cultures, epinephrine treatment significantly increased the levels

273 ity, the rate of 1-year survival with prompt epinephrine treatment was higher than with delayed treat

274 ons (76%) occurred within 4 hours of initial epinephrine treatment.

275 t (</=5 minutes) versus delayed (>5 minutes) epinephrine treatment.

276 iderable systemic effects were observed with epinephrine treatment.

277 oxygenation index were also attenuated with epinephrine treatment.

278 ital discharge with the vasopressin-steroids-epinephrine (VSE) combination.

279 The use of epinephrine was associated with a survival odds ratio of

280 ived epinephrine, delay in administration of epinephrine was associated with decreased chance of surv

281 administration of peripheral or intraosseous epinephrine was associated with increased survival in th

282 Delayed administration of epinephrine was associated with worse outcome.

283 Use of epinephrine was coded as yes/no and by dose (none, 1 mg,

284 ients who achieved ROSC, pre-hospital use of epinephrine was consistently associated with a lower cha

285 The DeltaAUC for epinephrine was greater with Pe than with Po (67 +/- 17

286 Epinephrine was infused systemically or released locally

287 The contractile stimulation by epinephrine was linked to drug tissue levels and commens

288 ired, whereas inhibition of cAMP increase by epinephrine was normal.

289 This adverse association of epinephrine was observed regardless of length of resusci

290 n the SI group, administration of oxygen and epinephrine was significantly lower, whereas minute vent

291 of H1 and H2 blockers, corticosteroids, and epinephrine was similar in the 2 treatment groups.

292 A 120-fold increase in epinephrine was subsequently observed that produced a tr

293 No epinephrine was used during cardiopulmonary resuscitatio

294 Epinephrine was used in 57.9% of cases.

295 able--only a small fraction of the deposited epinephrine was utilized in second messenger signaling a

296 ic allergic reactions or reactions requiring epinephrine were observed.

297 n plus intrathoracic pressure regulator plus epinephrine were significantly increased versus active c

298 the 120 children enrolled (63, dopamine; 57, epinephrine) were similar.

299 in mean arterial pressure >/= 65 mm Hg (1B); epinephrine when an additional agent is needed to mainta

300 with the physiologic neurotransmitter (11)C-epinephrine, which is sensitive to metabolic degradation