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1 d growth arrest in the central region of the epiphyses.
2 gatively regulates secondary ossification of epiphyses.
3 h Blount disease and slipped capital femoral epiphyses.
4 d the degree of fusion between diaphyses and epiphyses.
5 nal features, such as short stature and cone epiphyses, also suggest an underlying defect in endochon
8 scans were obtained by pQCT from the distal epiphyses and from the diaphyses of the tibia in each le
10 tyspondyly, generalized abnormalities of the epiphyses and metaphyses, and a distinctive lacy appeara
11 hort stature, hip abnormalities, cone-shaped epiphyses and premature closure of growth plates reflect
13 SCs and putative HSC niches in the calvaria, epiphyses, and diaphyses, at steady state or after HSC t
17 of Phd2 on endochondral ossification of the epiphyses by conditionally deleting the Phd2 gene in ost
21 and subsequently in the chondrocytes of the epiphyses in a graded distribution that decreased toward
23 e (normalized to tissue volume) in long bone epiphyses, indicating that Phd2 expressed in chondrocyte
24 omography to measure trabecular bone of limb epiphyses (long bone articular ends) in modern humans an
27 eriosteum, tendons, and endosteum within the epiphyses of the long bones adjacent to articular joints
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