ライフサイエンスコーパス: epipodophyllotoxin

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1 anthracyclines, taxanes, camptothecins, and epipodophyllotoxins.

2 ng plan support the relative safety of using epipodophyllotoxins according to the therapeutic plans o

3 Cytochrome P450 (CYP) 3A metabolizes epipodophyllotoxins and other chemotherapeutic agents.

4 Epipodophyllotoxins are associated with leukemias charac

5 ive doses of alkylators, anthracyclines, and epipodophyllotoxins are below thresholds usually associa

6 Although epipodophyllotoxins are commonly used in contemporary tr

7 oup clinical trials were identified that use epipodophyllotoxins at low (<1.5 g/m2 etoposide), modera

8 Tumor cell resistance to anthracyclines and epipodophyllotoxins can be due to reduced drug accumulat

9 cer mortality (radiation, alkylating agents, epipodophyllotoxins), cardiac mortality (chest irradiati

10 CYP3A metabolism generates epipodophyllotoxin catechol and quinone metabolites, whi

11 The variant may decrease production of the epipodophyllotoxin catechol metabolite, which is the pre

12 pipodophyllotoxin is identical to etoposide, epipodophyllotoxin contains a 4'-methoxyl group on the E

13 toxin treatment and the relationship between epipodophyllotoxin cumulative dose and risk are not well

14 Within the context of the epipodophyllotoxin cumulative dose range and schedules o

15 ubicin, and other agents, factors other than epipodophyllotoxin cumulative dose seem to be of primary

16 4beta-[(4' '-benzamido)-amino]-4'-O-demethyl-epipodophyllotoxin derivatives (11-23) were designed to

17 s shall aid in future design of novel potent epipodophyllotoxin derivatives.

18 Topoisomerase poisons such as the epipodophyllotoxin etoposide are widely used effective c

19 as anthracyclines (e.g., doxorubicin) or the epipodophyllotoxin etoposide.

20 s with MLL gene translocations that followed epipodophyllotoxin exposure were compared with the same

21 s and could be categorized into five groups: epipodophyllotoxins (group 1), inducers of DNA damage (g

22 Except for the sugar, 4'-demethyl epipodophyllotoxin is identical to etoposide, epipodophy

23 f DNA topoisomerase II covalent complexes by epipodophyllotoxins may play a role in the genesis of le

24 risk for treatment-related leukemia and that epipodophyllotoxin metabolism by CYP3A4 may contribute t

25 ng agents (RR, 2.2; 95% CI, 1.6 to 3.0), and epipodophyllotoxins (RR, 2.3; 95% CI, 1.2 to 4.5) increa

26 od to develop predictive QSAR models for 157 epipodophyllotoxins synthesized previously in our ongoin

27 We found that the group 1 epipodophyllotoxins (teniposide and etoposide) consisten

28 approximately 140-fold more resistant to the epipodophyllotoxin, teniposide (VM-26), than the parenta

29 The incidence of secondary leukemia after epipodophyllotoxin treatment and the relationship betwee

30 ates of the risk of secondary leukemia after epipodophyllotoxin treatment.

31 ion results in resistance to anthracyclines, epipodophyllotoxins, vinca alkaloids, and some alkylatin

32 amptothecin-(para)-4beta-amino-4'-O-demethyl Epipodophyllotoxin (W1), and camptothecin-(ortho)-4beta-

33 mptothecin-(ortho)-4beta-amino-4'-O-demethyl Epipodophyllotoxin (W2)] on cleavable complex formation

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