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1 d patients have undergone implantation of an epiretinal 4 x 4 electrode array designed to directly st
2 d human subjects underwent implantation with epiretinal 4 x 4 electrode arrays designed to directly s
3 laucomatous eyes, pars plana vitrectomy with epiretinal and internal limiting membrane peel was ineff
6 lantation and electrical stimulation with an epiretinal array did not result in damage that could be
8 age X-linked RP underwent implantation of an epiretinal array over the macula in the right eye and su
11 es on optical coherence tomography, we found epiretinal cells and vitreous collagen fibrils on the in
12 lasmin treatment, presence and topography of epiretinal cells and vitreous collagen remnants on the i
15 gularities or irregularity of each category (epiretinal, intraretinal, or RPE/choroidal irregularity)
16 gularities evaluated in the study (including epiretinal irregularities, abnormal retinal thickness, i
17 pectral-domain optical coherence tomography, epiretinal material of homogenous reflectivity without c
19 ommon complications were cataract (0.31/EY), epiretinal membrane (0.16/EY), and recurrent macular ede
20 roliferative diabetic retinopathy (PDR) (5), epiretinal membrane (4), vitreomacular traction syndrome
21 %), and all other baseline parameters except epiretinal membrane (84.3%), which was detected at a sig
22 (aHR, 1.43) and >/=2+ (aHR, 1.59) vs. none; epiretinal membrane (aHR, 1.25); peripheral anterior syn
23 e had a higher growth rate than eyes without epiretinal membrane (difference, 0.16; 95% CI, 0.03-0.30
25 00 (Carl Zeiss Meditech, Jena, Germany) with epiretinal membrane (ERM) and internal retinal membrane
27 selected among 369 patients examined for an epiretinal membrane (ERM) by Cirrus spectral-domain OCT
28 , or vehicle, and at appropriate time points epiretinal membrane (ERM) formation and retinal detachme
30 , the intraretinal glial response results in epiretinal membrane (ERM) formation, a proliferative and
32 e and clinical associations of patients with epiretinal membrane (ERM) who develop central-peripheral
33 (PPV): 6 for vitreous hemorrhage (VH), 1 for epiretinal membrane (ERM), and an additional 8 for tract
34 criteria included FTMH >400 mum, presence of epiretinal membrane (ERM), and aphakia in the study eye.
35 ndergoing pars plana vitrectomy to remove an epiretinal membrane (ERM), and test subjects (n = 7) wit
41 fluid (n = 6), subretinal exudation (n = 6), epiretinal membrane (n = 3), retinal hemorrhage (n = 2),
42 cyclitic membrane (n = 18, 69%), neoplastic epiretinal membrane (n = 6, 23%), and persistent hyaloid
44 al [CI], 1.57-4.34), a previous diagnosis of epiretinal membrane (RR, 5.60; 95% CI, 3.45-9.07), uveit
45 , and fibroblasts (the cell types crucial in epiretinal membrane [ERM] formation) under identical mic
47 inner nuclear layer in eyes with concomitant epiretinal membrane and glaucomatous optic neuropathy.
48 l membrane, 338 +/- 23 mum; and eyes with an epiretinal membrane and surface wrinkling, 405 +/- 22 mu
50 R) GUIDELINES: New evidence-based Idiopathic Epiretinal Membrane and Vitreomacular Traction Preferred
55 cluding postoperative cystoid macular edema, epiretinal membrane formation, macular folds, and, ultim
57 native internal limiting membrane in 2 eyes, epiretinal membrane in 1 eye, and remnant cortical vitre
58 8 eyes (29%) and 6 eyes (38%) (P = .34), and epiretinal membrane in 5 eyes (20.8%) and 4 eyes (25%) (
62 naive wet AMD group (group 3, n = 10) and an epiretinal membrane or macular hole group (group 4, n =
64 patients in the single-peeling group had an epiretinal membrane remaining in the central fovea posto
65 ted internal limiting membrane specimens and epiretinal membrane specimens removed from 25 eyes of 25
68 fects of "single peeling," in which only the epiretinal membrane was peeled, and "double peeling," in
71 l subfield thicknesses were: eyes without an epiretinal membrane, 338 +/- 23 mum; and eyes with an ep
72 , cataract, vitritis, cystoid macular edema, epiretinal membrane, and disc edema may occur in patient
73 lar edema, posterior capsular opacification, epiretinal membrane, and intraocular lens subluxation.
75 n younger patients (<65 years), eyes without epiretinal membrane, eyes with FTMH, phakic eyes, and ey
76 pathy, retinal vein occlusion, macular hole, epiretinal membrane, macular degeneration, retinal detac
77 e diabetic retinopathy, and the other 13 for epiretinal membrane, macular hole, vitreous opacities, o
78 3 months (20/94 vs 20/35 for eyes without an epiretinal membrane, P = .002) and at 6 months follow-up
80 retinoschisis, myopic traction maculopathy, epiretinal membrane, vitreoretinal traction, optic or sc
81 etinal Defect Associated With High Myopia or Epiretinal Membrane," published online January 22, 2015,
89 ntraocular pressure (IOP); (7) macula pucker/epiretinal membrane; (8) cataract; and (9) quality of li
90 er were diagnosed in 52 out of 264 eyes with epiretinal membranes (19.7%), of which 28 (55.0%) had co
91 ectopic inner foveal layers associated with epiretinal membranes (ERMs) and to present a new optical
92 The development of symptoms in patients with epiretinal membranes (ERMs) often corresponds with the a
95 ile of cells growing out of human idiopathic epiretinal membranes (iERMs) and testing their prolifera
100 oid layer and was associated with tractional epiretinal membranes and/or vitreomacular traction.
103 n of HGFR in human donor eyes and in several epiretinal membranes associated with proliferative vitre
104 n of HGFR in human donor eyes and in several epiretinal membranes associated with proliferative vitre
105 eadily detected in the cellular component of epiretinal membranes associated with PVR, whereas little
107 ces (ECM) is important in the development of epiretinal membranes found in proliferative vitreoretino
111 he RPE monolayer in human donor eyes, and in epiretinal membranes obtained from patients with PVR.
113 eptors (PDGFRs) are present and activated in epiretinal membranes of patient donors, and they are ess
116 epiretinal proliferation, whereas tractional epiretinal membranes presenting contractive properties w
117 ative vitreoretinopathy (PVR) and idiopathic epiretinal membranes was analyzed by immunohistochemistr
121 Eyes without epiretinal membranes and with epiretinal membranes without surface wrinkling were not
122 opathy, ophthalmological lesions (cataracts, epiretinal membranes, and retinal hamartomas), and cutan
124 n of the retinal inner layers (DRIL), cysts, epiretinal membranes, microaneurysms, subretinal fluid,
125 These conditions include macular holes, epiretinal membranes, retinal detachments, and retinopat
126 er nuclear layer in patients with idiopathic epiretinal membranes, with and without glaucomatous opti
135 s) with and without lamellar hole-associated epiretinal proliferation (LHEP) and standard epiretinal
136 It was often associated with nontractional epiretinal proliferation and a retinal "bump." Moreover,
138 ructure showed that lamellar hole-associated epiretinal proliferation of lamellar macular holes mainl
142 was categorized as lamellar hole-associated epiretinal proliferation, whereas tractional epiretinal
143 ologic and ultrastructural analysis of these epiretinal proliferations peeled at the time of silicone
144 man subjects were implanted chronically with epiretinal prostheses consisting of two-dimensional elec
145 result of retinitis pigmentosa, who received epiretinal prostheses implanted monocularly as part of a
146 an effort to restore functional form vision, epiretinal prostheses that elicit percepts by directly s
147 light perception, and an implanted Argus II epiretinal prostheses used a touchscreen to trace white
149 The Argus I implant is the first-generation epiretinal prosthesis approved for an investigational cl
150 lanted blind human subjects with a prototype epiretinal prosthesis consisting of a 4 x 4 array of 16
151 with severe RP (implanted with a 16-channel epiretinal prosthesis in 2004) on nine individual electr
153 ee categories for intermodality comparisons: epiretinal, retinal/subretinal, and RPE/choroidal irregu
154 Implantation of an electrode array on the epiretinal side (i.e., side closest to the ganglion cell
158 the KA/AMPA antagonist NBQX, suggesting that epiretinal stimulation evoked glutamate release from bip
163 ation of dense, contractile membranes on the epiretinal surface and prevented development of traction
166 the contraction of fibrotic membranes on the epiretinal surface of the neurosensory retina, resulting
170 ntified Muller cells are present in diabetic epiretinal tissues and appear to undergo the same progre
172 18 and was more strongly associated with the epiretinal vessels than with inner retinal vessels.
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