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1 c kidney), Vero (African-green monkey kidney epithelial), 3T12 (mouse fibroblast), and RAW 264.7 (mou
2 pithelium consists of differentiated luminal epithelial and basal myoepithelial cells, as well as und
3 The alpha6beta4 integrin protects adherent epithelial and carcinoma cells from ferroptosis induced
4 EMT that correlate with accessibility of key epithelial and EMT transcription factor binding sites.
5 onitored on genes with a pivotal function in epithelial and hepatic morphogenesis, differentiation an
6 olon, as well as normal human MCF10A mammary epithelial and human peripheral blood mononuclear cells
8 he life of the animal due to the presence of epithelial and mesenchymal stem cells-provides a model f
9 entified a ferrireductase: six-transmembrane epithelial antigen of prostate 4 (STEAP4) was highly ind
10 ffect of miR-143 and miR-145 on the cervical epithelial barrier and to elucidate the mechanisms by wh
12 Loss of claudin-18 was sufficient to impair epithelial barrier function in 16HBE cells and in mouse
13 for the host colonic epithelium and enhance epithelial barrier function through unclear mechanisms.
14 (GI) (patho)physiology; from GI motility and epithelial barrier function to enteric neuroinflammation
16 1 plays a significant role in maintenance of epithelial barrier in the intestine via regulation of ap
21 he secretion of immunoglobulins (Igs) across epithelial barriers, which is achieved via the polymeric
22 of histone modifications in human bronchial epithelial BEAS-2B cells and human nasal RPMI2650 cells
23 din-18.1 mRNA levels were measured in airway epithelial brushings from healthy controls and patients
28 g MP types: endothelial MPs, epithelial MPs (epithelial cell adhesion molecule [EpCAM](+)MPs, E-cadhe
29 Their induced target genes encode diverse epithelial cell adhesion molecules, while mesenchymal ge
30 7 fully blocked CFTR chloride conductance in epithelial cell cultures and intestine after cAMP agonis
37 blind viruses demonstrated the importance of epithelial cell infection for clinical disease, highligh
40 cell lines and primary fibroblasts and human epithelial cell lines or primary human epithelial cells
42 ge occurs through decline in stem/clonogenic epithelial cell loss mediated by microvascular protectio
43 small intestine and translocate through the epithelial cell monolayer by an intracellular pathway to
44 uctance measurement in polymer membranes and epithelial cell monolayers at discrete points in a sampl
45 ic membranes and then demonstrated in living epithelial cell monolayers under physiological condition
47 ofound changes in stem cell differentiation, epithelial cell phenotypes and fibroblast proliferation.
48 an undifferentiated, non-dividing esophageal epithelial cell population in patients with active EoE.
49 intracellular superoxide and inhibited cyst epithelial cell proliferation through extracellular sign
50 of 1,25(OH)2D3 and 24R,25(OH)2D3 on corneal epithelial cell proliferation, migration, and on the vit
52 n of Clusterin leads to divergent effects on epithelial cell regeneration and lung repair during fibr
53 The aim of this study was to evaluate the epithelial cell turnover and expression of proliferation
54 lar signatures such as their non-tumorigenic epithelial cell type, three-dimensional growth, latruncu
55 ions and restrictions of the mesenchymal and epithelial cell types in the developing and mature mouse
57 a human monoclonal antibody specific for the epithelial-cell-derived cytokine thymic stromal lymphopo
58 immune cells (8-12) , other studies detected epithelial cells (13) , and still others detected immune
61 prene SOA on gene expression in human airway epithelial cells (BEAS-2B) through an air-liquid interfa
62 orchestrated trafficking circuits in bladder epithelial cells (BECs) that expels intracellular uropat
64 osal explants and primary equine respiratory epithelial cells (EREC), grown at the air-liquid interfa
66 ontrol and asthmatic primary human bronchial epithelial cells (HBECs) by means of analysis of transep
68 immunohistochemistry study of human corneal epithelial cells (HCECs) and human keratocytes (HKs) cul
70 motes lactational differentiation of mammary epithelial cells (MECs) via its cognate receptor and the
71 tors are recruited to the surface of mammary epithelial cells (MECs), and the vesicle transport syste
74 and immunofluorescent staining of glomerular epithelial cells (podocytes) indicated that VtE ameliora
75 c epithelium and is required to prime thymic epithelial cells (TEC) for effective Treg induction.
78 of CuO NPs induced IL-8 release in the lung epithelial cells already at subtoxic concentrations (1-1
81 nalling pathway that functions in intestinal epithelial cells and may present useful targets in the m
85 ide a unique perspective on and insight into epithelial cells and tissues, whether normal or diseased
87 and BLIMP1 induce lytic EBV reactivation in epithelial cells by synergistically activating the two E
89 hasone to modulate gene expression in airway epithelial cells coincided with its potency to resolve A
92 of genomic architecture in mouse intestinal epithelial cells disclosed that microbiota colonization
93 dative stress, and subsequently apoptosis in epithelial cells during ischemia-reperfusion injury.
98 ates with the actin cytoskeleton in prostate epithelial cells in normal tissues, but not in prostate
101 shed that HIV-1 can infect kidney transplant epithelial cells in the absence of detectable viremia.
102 n molecules, CD44 and annexin II, in tubular epithelial cells in vitro and in vivo, and treatment wit
105 asymmetrically partitioned during mitosis in epithelial cells just before delamination and selection
106 rferon response to viral infection by airway epithelial cells may be a mechanism leading to lung func
107 xpression of IL-36alpha in the renal tubular epithelial cells of a mouse model of unilateral ureteral
109 cus, which is observed in blood and squamous epithelial cells of smokers, but not in lung cancer.
111 lpha defensin HD5 is produced by specialized epithelial cells of the gastrointestinal and genito-urin
112 er, these results suggest beta2ARs on airway epithelial cells promote the asthma phenotype and that t
114 cing of TTP in endometriotic and endometrial epithelial cells revealed differential response to infla
117 We previously reported that HCA2 in A431 epithelial cells transduced Gbetagamma-protein kinase C-
118 ich we specifically ablated Mig-6 in uterine epithelial cells using Sprr2f-cre mice (Sprr2f(cre+)Mig-
119 tion, ROS disassembles adherens junctions in epithelial cells via posttranslational mechanisms, that
121 1 knockout, and wild-type mouse nasal septal epithelial cells were grown at an air-liquid interface (
123 e specificity for islet beta cells and renal epithelial cells were reliably characterized in recipien
125 r-liquid interface cultures of primary nasal epithelial cells were used to measure transepithelial el
126 targeting Stard7 to mitochondria or treating epithelial cells with a mitochondrial-targeted antioxida
127 , NY/108 virus replicated in human bronchial epithelial cells with an increased efficiency compared w
128 es of these cell lines demonstrated that the epithelial cells with surface expression of PD-L1, E-cad
130 hat TGFbeta can induce purified primary lens epithelial cells within the same culture to undergo diff
133 of B cells, dendritic cells, cortical thymic epithelial cells, and medullary thymic epithelial cells.
134 hibition of STAT3 phosphorylation in uterine epithelial cells, and the antitumor effects of P4 are me
135 wn about how LMP1 expression is regulated in epithelial cells, and there are conflicting reports abou
136 myosin accumulation at the apical surface of epithelial cells, as seen in the vertebrate neural tube
139 merous different cell types, such as tubular epithelial cells, endothelial cells, and podocytes, work
140 functioning at the apical plasma membrane of epithelial cells, is required for epithelial polarity.
142 accumulate preferentially in breast luminal epithelial cells, not in basal epithelial or stromal cel
143 t in primary mouse submandibular gland (SMG) epithelial cells, P2X7R activation also induces the asse
144 ing the acute respiratory distress syndrome, epithelial cells, primarily alveolar type (AT) I cells,
146 ined by VEGF that is produced by neighboring epithelial cells, the retinal pigment epithelium (RPE) a
147 lectrically non-excitable cells, for example epithelial cells, this is achieved by primary release of
148 is a major contributor to actin assembly in epithelial cells, where it works with the Ena/VASP famil
149 as a major factor VAT releases to transform epithelial cells-a novel, potential pathway of VAT-enhan
165 tally new roles of TMEM16A in differentiated epithelial cells: TMEM16A provides a mechanism for enhan
166 ronic inflammation, such as products of lung epithelial cellular breakdown and arachidonic acid casca
168 ned in mice lacking a tectorin-based matrix, epithelial cilia or the planar cell polarity genes Vangl
171 rease in the number of blood vessels and sub-epithelial connective tissue matrix components within th
172 adhesion to the scaffold and well-stratified epithelial constructs, with maintenance of a progenitor
174 Primary human airway basal stem cell-derived epithelial cultures and micro-optical coherence tomograp
175 , reducing recurrence presumably by limiting epithelial damage and facilitating microbiome recovery.
176 roorganisms and form EETs at sites of airway epithelial damage to protect the host from infections in
177 recombinant human interferon (IFN)-gamma or epithelial-derived IFN-gamma in constitutively released
179 raphy angiograhy (OCTA) of drusenoid pigment epithelial detachments (PEDs) in a woman affected by Com
182 of proinflammatory cytokines, and subsequent epithelial disorders and mitochondrial dysfunction.
183 ogy can be applied to interrogate intestinal epithelial dynamics and characterize situations in which
184 cover function in an animal model of corneal epithelial dysfunction after surgical transplantation.
186 bility to severe intestinal inflammation and epithelial dysregulation, accompanied by excessive necro
187 4 disrupts the mucosal barrier, resulting in epithelial expression of the IL-6-like cytokine Upd3, le
188 oride channel, resulting in dysregulation of epithelial fluid transport in the lung, pancreas and oth
189 tivates mRNA expression of a broad cohort of epithelial genes, including junctional factor dlg-1 Accu
193 of Jagged1 neither interferes with prostate epithelial homeostasis nor significantly accelerates tum
195 l host defense against pathogen exposure and epithelial injury, yet little is known about how enterop
198 ealing in response to lung injury, restoring epithelial integrity through spreading and migration.
201 an be expanded and differentiated to form an epithelial layer expressing K12 and PAX6, and able to re
203 hod to separately derive lung versus thyroid epithelial lineages, as these two cell types each origin
207 that overexpression of the oncogenic protein epithelial membrane protein-2 (EMP2) correlates with end
208 use lung mesenchyme, the cells recapitulated epithelial-mesenchymal developing lung interactions.
209 re lung, inflammation and injury disrupt the epithelial-mesenchymal interactions required for normal
211 Twist-mediated oncogenic functions including epithelial-mesenchymal transition (EMT) and cancer stem
212 e (RTK) AXL has been intrinsically linked to epithelial-mesenchymal transition (EMT) and promoting ce
214 related to cancer stem cells (CSCs) markers, epithelial-mesenchymal transition (EMT) inducers and bas
216 dial cells were highly adipogenic through an epithelial-mesenchymal transition both in vitro and in v
217 miR-194 stimulated migration, invasion, and epithelial-mesenchymal transition in human prostate canc
218 RHAMM by the RB/E2F pathway was critical for epithelial-mesenchymal transition, motility, and invasio
219 ivation of peritoneal fibroblasts, mitigated epithelial-mesenchymal-transition (EMT), as well as enha
220 Moreover, GRHL2 suppresses the oncogenic epithelial-mesencyhmal transition, thereby acting as a t
225 ize the following MP types: endothelial MPs, epithelial MPs (epithelial cell adhesion molecule [EpCAM
228 tion of p38, prevented TGFbeta from inducing epithelial-myofibroblast transition and cell migration b
229 type 2 airway inflammation in juvenile beta-epithelial Na(+) channel (Scnn1b)-transgenic (Tg) mice.
232 there was a significant increase in corneal epithelial nerve regeneration and substance P-positive n
233 d in M-ILK-deficient mice, and activation of epithelial NF-kappaB and PI3K signaling pathways are res
234 ontribution of innate receptor engagement on epithelial or dendritic cells by HDMs that ultimately me
235 reast luminal epithelial cells, not in basal epithelial or stromal cells, of BRCA1 mutation carriers.
236 e signaling network controls cell migration, epithelial organization, axon patterning and other aspec
241 xins that eliminate glia in mice, intestinal epithelial permeability and proliferation increase; this
246 trinsic tumor-suppressive mechanism, whereby epithelial polarity proteins dictate the cytoarchitectur
249 te: (i) ADMA-mediated NOS uncoupling reduces epithelial production of NO and increases oxygen and nit
251 d large intestines, but caused no defects in epithelial proliferation, barrier integrity, or ultrastr
255 enic signaling produced by retinal pigmented epithelial (RPE) cells under different conditions simula
256 A1 locus with subretinal/sub-retinal pigment epithelial (RPE) hemorrhage related to neovascular AMD (
258 te responses and regional differences in the epithelial secretome participating in RSV lower respirat
259 ative proteomics analysis of the RSV-induced epithelial secretory response in cells representative of
261 mitantly exhibit an expansion of the mammary epithelial stem cell (MaSC) enriched basal/myoepithelial
262 as a central intrinsic regulator of mammary epithelial stem cell quiescence and exhaustion and is ne
266 tion of theory and experiments, we show that epithelial surface cells not only trigger blastoderm exp
269 broblasts may not fully recapitulate that of epithelial tight junction strands, this is the first dir
270 Rab protein distributions during Drosophila epithelial tissue remodeling and show that Rab35 is dyna
274 productive area of study is on single layer epithelial tissues in which the adherence junctions of c
277 a (HCC), a highly metastatic cancer, undergo epithelial to amoeboid transition in physiological envir
278 tion with canonical Wnt signaling during the epithelial to mesenchymal transition (EMT) from NMP to m
279 dherens junctions, dedifferentiation, and an epithelial to mesenchymal transition (EMT) transcription
281 -3 supports the TIC/CSC state and induces an epithelial-to-mesenchymal transition (EMT) by driving ex
284 oor prognosis owing to its role in promoting epithelial-to-mesenchymal transition (EMT), invasiveness
286 henotypes were also affected at the level of epithelial-to-mesenchymal transition and the ERK1/2 sign
287 n the proliferation, survival, migration and epithelial-to-mesenchymal transition of cancer cells.
288 and differentiation, resistance to anoikis, epithelial-to-mesenchymal transition, tumor cell dormanc
289 proteins and with an increased expression of epithelial-to-mesenchymal transition-related genes.
292 nsive analysis indicates that mesenchymal-to-epithelial transition is requisite to initiate SMCs repr
293 uption of cell polarity is a prerequisite in epithelial tumor initiation, the roles of CDC42 in tumor
296 Here we report the generation of alveolar epithelial type 2 cells (AEC2s), the facultative progeni
297 delicate regulation of colonic pH, including epithelial water absorption, nutrient inflow, and lumina
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