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1 th a tumor suppressor function in cancers of epithelial origin.
2 ation, but it is also expressed in tumors of epithelial origin.
3 cumented in a variety of invasive cancers of epithelial origin.
4 s receptor is largely restricted to cells of epithelial origin.
5 apture live CTCs and only apply to tumors of epithelial origin.
6 lonies in culture independent of the site of epithelial origin.
7 poptotic signaling in proliferating cells of epithelial origin.
8 sed to the environment and in many tumors of epithelial origin.
9 human keratinocytes and other cell lines of epithelial origin.
10 at occurs during metastasis of cancers of an epithelial origin.
11 evere development defects and lack organs of epithelial origin.
12 progression of cancers, especially those of epithelial origin.
13 ereas adult tumors are usually carcinomas of epithelial origin.
14 tumor previously demonstrated to be of lens epithelial origin.
15 activity was reported in various cancers of epithelial origin.
16 lication in keratinocytes and other cells of epithelial origin.
17 The majority of cancer is of surface/cyst epithelial origin.
18 y observed in neoplasia,notably in tumors of epithelial origin.
19 used in clinical trials targeting cancers of epithelial origin.
20 rrence and development of various cancers of epithelial origin.
21 men, >80% of malignant ovarian tumors are of epithelial origin.
22 in the malignant transformation of cells of epithelial origin.
23 at q31.1 is common in a variety of tumors of epithelial origin.
24 ession of Dab2 in the tumor cells, which are epithelial origin.
25 a marker of rapidly proliferating tumors of epithelial origin.
26 to over 300 tumour samples of germ cell and epithelial origin.
27 and embryonic tissues, primarily in those of epithelial origin.
28 FN-lambda receptor is restricted to cells of epithelial origin.
29 frequently associated with human cancers of epithelial origin.
30 ally distinct tumors of both mesenchymal and epithelial origin.
31 riety of solid tumors, particularly those of epithelial origin.
32 factor (EGF), a potent mitogen for cells of epithelial origin.
33 of monocyte, T lymphocyte, B lymphocyte, and epithelial origins.
34 ransfection in nontumorigenic human cells of epithelial origin (293HEK, MCF-10A adenoma, and NHDF-Ad
35 +) tumors of lymphoid (B, T, and NK cell) or epithelial origin and five Burkitt's lymphoma cell lines
36 he predominant isoform expressed in cells of epithelial origin and frequently overexpressed in cancer
37 ase that is overexpressed in cancer cells of epithelial origin and in normal tissues during events in
38 in cytokine release by cells of respiratory epithelial origin and suggest that further work in the a
39 erexpressed in tumor cells of mesenchymal or epithelial origin and these molecules are required for t
40 d positive for cytokeratin, confirming their epithelial origin, and also expressed alpha-SMA and fibr
41 in several carcinomas, which are cancers of epithelial origin, and is crucial to metastatic tumor ce
42 y an inflammatory cytokine in a cell line of epithelial origin, and the results suggest a potential m
43 n receptors that are characteristic of their epithelial origin, and upregulate the expression of adhe
44 a previously unreported late complication of epithelial origin, and we suggest a potential link betwe
45 her EBV-positive malignancies of lymphoid or epithelial origin arise in individuals with seemingly in
46 uamous cell carcinoma and related cancers of epithelial origin, as well as to test and develop novel
47 that is predominantly expressed in cells of epithelial origin but can also be induced in other cell
48 xpression has been reported in cell lines of epithelial origin, but its function in these cells remai
49 sive breast cancers, and other cancers of an epithelial origin (carcinomas) but not in cancers of a m
50 pCAM) antigen, a common marker for tumors of epithelial origin, employing bionanoconjugates as signal
51 r, we have evidence that for solid tumors of epithelial origin, extreme levels of genomic instability
52 pressed in most malignant adenocarcinomas of epithelial origin (for example, colon, breast and ovaria
53 ch signaling in cancers of hematopoietic and epithelial origins has been established, its role in tum
54 roteins produced by neutrophils and cells of epithelial origin have chemotactic and activating effect
55 ated in prostate cancer and other cancers of epithelial origin, highlighting its potential as a targe
56 tein expressed in the majority of cancers of epithelial origin in humans, were induced by vaccination
61 as well as in six human tumour cell lines of epithelial origin, including two colon carcinoma lines.
62 expression or function in various tumors of epithelial origin is associated with a more invasive phe
65 re, the HR HPV type, rather than the site of epithelial origin of the cells, determines the efficacy
70 of antiviral ISGs preferentially in cells of epithelial origin, recent studies have defined additiona
71 pidermis and human and murine skin tumors of epithelial origin revealed that only differentiated, non
72 specifically binds to human cancer cells of epithelial origin such as pancreatic cancer cells (PANC-
73 nd is frequently overexpressed in cancers of epithelial origin such as pancreatic cancer, switching p
74 lymphoid-derived cells, but not in cells of epithelial origin, suggesting that PDT-induced rapid and
75 s indicated that they are endocrine cells of epithelial origin that do not express the same transcrip
76 ad and neck (SCCHN) is a group of cancers of epithelial origin that may provide an ideal model for th
77 ed into human cell lines of fibroblastic and epithelial origins that differed in their expression of
78 certain malignancies, particularly those of epithelial origin, they also provide a rationale for dev
79 hat are preferentially expressed on cells of epithelial origin; thus, we henceforth refer to them as
81 e ras gene are most often found in tumors of epithelial origin, we explored the signaling pathways ut
82 hat VEGFRs are not present on tumor cells of epithelial origin, we found that VEGFR-1 is present and
84 or selected human cell lines of neuronal and epithelial origin, whereas HVEM or nectins could be used
86 nt of the broad spectrum of human cancers of epithelial origin with mutant RAS that have undergone EM
87 ar how Ras activation affects human cells of epithelial origin with p53 mutation and/or telomerase ac
88 hly metastatic tumors of neuroendocrine, not epithelial, origin within the urogenital tract to give p
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