ライフサイエンスコーパス: epithelial-mesenchymal transition

1 , including differentiation, development and epithelial mesenchymal transition.

2 led SLUG), implicating LGR4 in regulation of epithelial-mesenchymal transition.

3 Fringe, is specifically required for cardiac epithelial-mesenchymal transition.

4 tumors by increasing cell proliferation and epithelial-mesenchymal transition.

5 orming growth factor beta (TGF-beta)-induced epithelial-mesenchymal transition.

6 dependent cell invasion, MMP1 activation and epithelial-mesenchymal transition.

7 e front edge, a phenotype reminiscent of the Epithelial-Mesenchymal Transition.

8 loss of NOTCH activity favors the process of epithelial-mesenchymal transition.

9 , Bmi-1, and other genes associated with the epithelial-mesenchymal transition.

10 ulate transcription of Fascin along with the epithelial-mesenchymal transition.

11 transcription of many of these genes and the epithelial-mesenchymal transition.

12 cross-talk emerged as an integrated part of epithelial-mesenchymal transition.

13 s been shown to accelerate tumorigenesis and epithelial-mesenchymal transition.

14 tively correlated with genes associated with epithelial-mesenchymal transition.

15 Hippo signaling activity, nor does it induce epithelial-mesenchymal transition.

16 192 promoter methylation and expression with epithelial-mesenchymal transition.

17 involving resident stem/progenitor cells and epithelial-mesenchymal transitions.

18 OXC2 is induced and necessary for successful epithelial-mesenchymal transition, a developmental progr

19 Following epithelial-mesenchymal transition, acquisition of avian

20 pha, a major isoform of the p63 gene, led to epithelial-mesenchymal transition and acquisition of ste

21 and stabilize SNAIL1, a key factor promoting epithelial-mesenchymal transition and breast cancer meta

22 or invasiveness and metastasis by increasing epithelial-mesenchymal transition and by enriching a can

23 istance can occur following the emergence of epithelial-mesenchymal transition and by reactivation of

24 leading to subsequent downregulation of the epithelial-mesenchymal transition and cancer stem-like c

25 e counterbalanced by its ability to suppress epithelial-mesenchymal transition and cell invasion.

26 ve formation, Dll4-Notch1 signaling leads to epithelial-mesenchymal transition and cushion formation.

27 o the control of miRNAs that are involved in epithelial-mesenchymal transition and cystic kidney dise

28 romotes TGF-beta-induced gene expression and epithelial-mesenchymal transition and enhances cell migr

29 tion of beta-catenin suppressed EGF-mediated epithelial-mesenchymal transition and facilitated epithe

30 tory cytokines and chemokines and markers of epithelial-mesenchymal transition and fibrosis/remodelin

31 Notch activation in Pten-null mice promoted epithelial-mesenchymal transition and FOXC2-dependent tu

32 Moreover, ILK inhibition blocked KRAS-driven epithelial-mesenchymal transition and growth factor-stim

33 p53-responsive miRNAs, which may facilitate epithelial-mesenchymal transition and growth of liver tu

34 ing in promoting inflammation and hepatocyte epithelial-mesenchymal transition and migration, we used

35 n of HOXB7 in breast cancer cells induces an epithelial-mesenchymal transition and promotes tumor pro

36 Resistin was found to promote epithelial-mesenchymal transition and stemness in breast

37 ssociated with stemness, chemoresistance and epithelial-mesenchymal transition and suppressed the pro

38 ially directed mesoendoderm differentiation, epithelial-mesenchymal transition and the ensuing cell m

39 complete an epigenetic program that favored epithelial-mesenchymal transition and the generation of

40 can relieve HIP1 suppression of Akt-mediated epithelial-mesenchymal transition and thereby lead to de

41 ays in tumor cells, followed by induction of epithelial-mesenchymal transition and upregulation of E3

42 ization upon kidney injury and contribute to epithelial-mesenchymal transition and, potentially, to m

43 tercalation, and cells can delaminate during epithelial-mesenchymal transitions and metastasis.

44 strated that loss of NMI expression promotes epithelial-mesenchymal-transition and results in enhance

45 vasion, migration, proliferation, apoptosis, epithelial-mesenchymal transition, and anchorage-indepen

46 dherin, induces expression of markers of the epithelial-mesenchymal transition, and enhances the moti

47 ial for primitive cardiac valve formation by epithelial-mesenchymal transition, and NOTCH1 mutations

48 We report the epithelial to hybrid epithelial/mesenchymal transition as a result of decreas

49 Gefitinib-mediated ROS correlated with epithelial-mesenchymal transition, as well as striking p

50 In addition, we showed that some epithelial-mesenchymal transition-associated genes are u

51 dial cells were highly adipogenic through an epithelial-mesenchymal transition both in vitro and in v

52 Cs) from primary human keratinocytes through epithelial-mesenchymal transition by modulating the epit

53 cells in vitro and in vivo and regulated the epithelial-mesenchymal transition by repressing AKT/glyc

54 hat by analogy to the cadherin switch during epithelial-mesenchymal transition, cancer cells also und

55 ession of TAZ/YAP target genes implicated in epithelial-mesenchymal transition, cell migration, and i

56 te cancer progression phenomena, such as the epithelial-mesenchymal transition, cell motility, invasi

57 sis via altered gene expression, stimulating epithelial-mesenchymal transition, cellular migration/in

58 rcinomas become invasive after undergoing an epithelial-mesenchymal transition characterized by the a

59 of mesenchymal cadherins, a hallmark of the epithelial-mesenchymal transition, contributes to migrat

60 enhanced expression of epithelial genes and epithelial-mesenchymal transition downregulation signatu

61 ETS-transcription factor ETV1 is involved in epithelial-mesenchymal transition during pancreatic deve

62 ate the changes of glucose metabolism during epithelial mesenchymal transition (EMT) and the role of

63 pathy (PVR) is mediated by proliferation and epithelial mesenchymal transition (EMT) of retinal pigme

64 ization of marker genes and proteins for the Epithelial Mesenchymal Transition (EMT) pathway only in

65 The epithelial mesenchymal transition (EMT) promotes tumor m

66 primary human hepatocytes (PHH) induces the epithelial mesenchymal transition (EMT) state and extend

67 /Twist has been implicated in the process of epithelial mesenchymal transition (EMT), an intermediate

68 (NPCs) led RGs to undergo changes resembling epithelial-mesenchymal transition (EMT) along with exube

69 EB1) is a transcription factor that promotes epithelial-mesenchymal transition (EMT) and acts as an o

70 MAOA induces epithelial-mesenchymal transition (EMT) and augments hyp

71 Twist-mediated oncogenic functions including epithelial-mesenchymal transition (EMT) and cancer stem

72 Hedgehog (Hh) pathway is involved in epithelial-mesenchymal transition (EMT) and cancer stem

73 udin expression and suppresses Slug-mediated epithelial-mesenchymal transition (EMT) and cell invasiv

74 Furthermore, depletion of DLG5 induced epithelial-mesenchymal transition (EMT) and disrupted ep

75 mounts of IL-1beta and TNF which facilitated epithelial-mesenchymal transition (EMT) and epithelial c

76 ts extend the role of ZEB2 beyond regulating epithelial-mesenchymal transition (EMT) and establish ZE

77 TAT3 induces TWIST1 to drive a p27-dependent epithelial-mesenchymal transition (EMT) and further acti

78 m cell cycle regulation and cell survival to epithelial-mesenchymal transition (EMT) and gastrulation

79 enchymal transformation during developmental epithelial-mesenchymal transition (EMT) and has been ass

80 uced expression of Slug, ovarian cancer cell epithelial-mesenchymal transition (EMT) and invasion.

81 y studied types of phenotypic plasticity are epithelial-mesenchymal transition (EMT) and mesenchymal-

82 us studies coupled beta3 integrin (ITGB3) to epithelial-mesenchymal transition (EMT) and metastasis,

83 hway plays critical roles during cancer cell epithelial-mesenchymal transition (EMT) and metastasis.

84 ion in the prostate epithelium promotes both epithelial-mesenchymal transition (EMT) and metastatic c

85 e (RTK) AXL has been intrinsically linked to epithelial-mesenchymal transition (EMT) and promoting ce

86 erent mechanisms: TGF-beta1 via induction of epithelial-mesenchymal transition (EMT) and SKP2 via dow

87 eq analysis reveals that REST is involved in epithelial-mesenchymal transition (EMT) and stemness acq

88 , PIPKIgamma, expression is upregulated upon epithelial-mesenchymal transition (EMT) and together wit

89 Collective cell migration (CCM) and epithelial-mesenchymal transition (EMT) are common to ca

90 e, we demonstrate that Notch1 activation and epithelial-mesenchymal transition (EMT) are coupled to p

91 Convergent extension (CE) movements and epithelial-mesenchymal transition (EMT) are important fo

92 Glioma stem cells (GSC) and epithelial-mesenchymal transition (EMT) are strongly ass

93 Albumin also stimulated an epithelial-mesenchymal transition (EMT) as indicated by

94 h in vitro and in vivo also induced HCC cell epithelial-mesenchymal transition (EMT) as well.

95 The epithelial-mesenchymal transition (EMT) bestows cancer c

96 then found to result from TGFbeta1-triggered epithelial-mesenchymal transition (EMT) by beta-cells, r

97 CAF-CM induced the epithelial-mesenchymal transition (EMT) by regulating ex

98 In cancer cells, the epithelial-mesenchymal transition (EMT) confers the abil

99 Finally, mir-200 was able to antagonize the epithelial-mesenchymal transition (EMT) driven by mutant

100 is a bHLH transcription factor that promotes epithelial-mesenchymal transition (EMT) during developme

101 During epithelial-mesenchymal transition (EMT) epithelial cells

102 Epithelial-mesenchymal transition (EMT) has been associa

103 Epithelial-mesenchymal transition (EMT) has been conside

104 Although epithelial-mesenchymal transition (EMT) has been inverse

105 Epithelial-mesenchymal transition (EMT) has been linked

106 Epithelial-mesenchymal transition (EMT) has been recogni

107 and TGFbeta signaling pathways and a loss of epithelial-mesenchymal transition (EMT) in BVE(Cyp24a1-n

108 Epithelial-mesenchymal transition (EMT) in cells is a de

109 The transcription factor AP4 mediates epithelial-mesenchymal transition (EMT) in colorectal ca

110 Functionally, RGC32 facilitated epithelial-mesenchymal transition (EMT) in CRC via the S

111 gated the mechanisms by which SPRY regulates epithelial-mesenchymal transition (EMT) in CRC.

112 Conversely, the introduction of inducers of epithelial-mesenchymal transition (EMT) in cystine-indep

113 e report that mammary tumor cells undergoing epithelial-mesenchymal transition (EMT) in response to t

114 to CD44(High)-CD24(-/Low) (CD44H) cells via epithelial-mesenchymal transition (EMT) in response to t

115 moresistance, and expressed genes related to epithelial-mesenchymal transition (EMT) in solid cancers

116 We found that, during epithelial-mesenchymal transition (EMT) induced by trans

117 related to cancer stem cells (CSCs) markers, epithelial-mesenchymal transition (EMT) inducers and bas

118 The epithelial-mesenchymal transition (EMT) is a complex cel

119 Epithelial-mesenchymal transition (EMT) is a complex pro

120 Epithelial-mesenchymal transition (EMT) is a fundamental

121 Epithelial-mesenchymal transition (EMT) is a fundamental

122 Epithelial-mesenchymal transition (EMT) is a highly cons

123 Epithelial-mesenchymal transition (EMT) is a key event t

124 Epithelial-mesenchymal transition (EMT) is a key reversi

125 Epithelial-mesenchymal transition (EMT) is a normal cell

126 The epithelial-mesenchymal transition (EMT) is a process tha

127 Epithelial-mesenchymal transition (EMT) is an essential

128 The epithelial-mesenchymal transition (EMT) is an important

129 Epithelial-mesenchymal transition (EMT) is an important

130 Epithelial-mesenchymal transition (EMT) is associated wi

131 An epithelial-mesenchymal transition (EMT) is believed to b

132 reveal that HCC progression via induction of epithelial-mesenchymal transition (EMT) is closely assoc

133 Epithelial-mesenchymal transition (EMT) is controlled by

134 The epithelial-mesenchymal transition (EMT) is crucial to ca

135 Epithelial-mesenchymal transition (EMT) is implicated in

136 Epithelial-mesenchymal transition (EMT) is induced by tr

137 Epithelial-mesenchymal transition (EMT) is prominent in

138 Epithelial-mesenchymal transition (EMT) is regarded as a

139 The epithelial-mesenchymal transition (EMT) is regulated by

140 Epithelial-mesenchymal transition (EMT) is required for

141 The epithelial-mesenchymal transition (EMT) is the pivotal m

142 ealing correlated with altered expression of epithelial-mesenchymal transition (EMT) markers and redu

143 sociated proteins with altered expression of epithelial-mesenchymal transition (EMT) markers.

144 AT1 was shown to be required for maintaining epithelial-mesenchymal transition (EMT) mediated cell mi

145 -cadherin expression in association with the epithelial-mesenchymal transition (EMT) occurs frequentl

146 An initial step of the pathogenesis is an epithelial-mesenchymal transition (EMT) of retinal pigme

147 cancer cells, implicating acquisition of an epithelial-mesenchymal transition (EMT) phenotype.

148 demonstrated that VHL knock down induces an epithelial-mesenchymal transition (EMT) phenotype.

149 s, has been linked to the acquisition of the epithelial-mesenchymal transition (EMT) program and self

150 osed as critical regulatory molecules in the epithelial-mesenchymal transition (EMT) program.

151 MaSCs and MaTICs both use epithelial-mesenchymal transition (EMT) programs to acqu

152 Epithelial-mesenchymal transition (EMT) promotes cancer

153 ted with sunitinib and assayed for AXL, MET, epithelial-mesenchymal transition (EMT) protein expressi

154 ific mechanism for HNSCC local invasion: the epithelial-mesenchymal transition (EMT) regulator Twist1

155 at hepatitis C virus (HCV) infection induces epithelial-mesenchymal transition (EMT) state and cancer

156 The induction of pathogenic epithelial-mesenchymal transition (EMT) through microRNA

157 are highly migratory cells, which undergo an epithelial-mesenchymal transition (EMT) to leave the neu

158 s with cabazitaxel and enzalutamide reversed epithelial-mesenchymal transition (EMT) to mesenchymal-e

159 ist1 has a well-established role in inducing Epithelial-Mesenchymal Transition (EMT) to promote tumor

160 Collagen was increased, and cancer cell epithelial-mesenchymal transition (EMT) was inhibited, a

161 Metastatic cancer cells undergo an epithelial-mesenchymal transition (EMT) where they lose

162 in gliomas, are associated with a process of epithelial-mesenchymal transition (EMT), a cellular alte

163 transcription factor known for participating epithelial-mesenchymal transition (EMT), a critical cell

164 in human carcinomas drives the phenomenon of epithelial-mesenchymal transition (EMT), a phenotypic mo

165 c ductal adenocarcinoma (PDAC) cells undergo epithelial-mesenchymal transition (EMT), a process combi

166 members of the miR-200 family that regulate epithelial-mesenchymal transition (EMT), a reversible ph

167 played enhanced proliferation, migration and epithelial-mesenchymal transition (EMT), and acquired a

168 tors (CPs) and promotes their specification, epithelial-mesenchymal transition (EMT), and cardiovascu

169 tumor edge, ceased to proliferate, underwent epithelial-mesenchymal transition (EMT), and expressed m

170 osed mechanism by which CSCs arise is via an epithelial-mesenchymal transition (EMT), and reversal of

171 r localization of Twist and Slug, markers of epithelial-mesenchymal transition (EMT), but also consti

172 ified several MMS genes as essential for the epithelial-mesenchymal transition (EMT), but not for cel

173 of a latent embryonic program, known as the epithelial-mesenchymal transition (EMT), can endow cance

174 show that PRMT1 is an important regulator of epithelial-mesenchymal transition (EMT), cancer cell mig

175 The epithelial-mesenchymal transition (EMT), considered esse

176 The epithelial-mesenchymal transition (EMT), defined as tran

177 We show that TGF-beta induces an epithelial-mesenchymal transition (EMT), generally consi

178 relates with poor prognosis and induction of epithelial-mesenchymal transition (EMT), hence we hypoth

179 sensitivity to various drugs by attenuating epithelial-mesenchymal transition (EMT), Hif1-alpha sign

180 The process of epithelial-mesenchymal transition (EMT), in addition to

181 t example in human cancers of the concept of epithelial-mesenchymal transition (EMT), in which the tw

182 a/BMP-SMAD4 signaling pathway, which induces epithelial-mesenchymal transition (EMT), is known to con

183 Snail1, a key transcription factor of epithelial-mesenchymal transition (EMT), is subjected to

184 fibulin-3 levels inhibited TGF-beta-induced epithelial-mesenchymal transition (EMT), migration, inva

185 TCF transcription induces epithelial-mesenchymal transition (EMT), promoting a tum

186 ed the activation of two major initiators of epithelial-mesenchymal transition (EMT), TGF-beta1 signa

187 luding the inhibition of cell proliferation, epithelial-mesenchymal transition (EMT), the expression

188 miR-200 family and p21 in inhibition of the epithelial-mesenchymal transition (EMT), we observed EMT

189 Because TWIST1 mediates epithelial-mesenchymal transition (EMT), we studied the

190 embryonic development program referred to as epithelial-mesenchymal transition (EMT), whereby epithel

191 of a latent embryonic program, known as the epithelial-mesenchymal transition (EMT), which bestows s

192 Twist is a key transcription factor for Epithelial-mesenchymal transition (EMT), which is a cell

193 FN is an established marker for epithelial-mesenchymal transition (EMT), which occurs du

194 Moreover, hnRNPF regulates an epithelial-mesenchymal transition (EMT)-associated CD44

195 Her2(+) eDCCs activated a Wnt-dependent epithelial-mesenchymal transition (EMT)-like disseminati

196 d ShP51 cells that stably express PXR showed epithelial-mesenchymal transition (EMT)-like morphologic

197 ke value (SUVmax) are associated with a more epithelial-mesenchymal transition (EMT)-like phenotype.

198 Many epithelial-mesenchymal transition (EMT)-promoting transc

199 Additionally, epithelial-mesenchymal transition (EMT)-related genes we

200 iferation, anchor-free colony formation, and epithelial-mesenchymal transition (EMT).

201 ells, consistent with a role in promoting an epithelial-mesenchymal transition (EMT).

202 a gene expression signature associated with epithelial-mesenchymal transition (EMT).

203 herin, Vimentin, and/or Snail1) critical for epithelial-mesenchymal transition (EMT).

204 lved in the control of cellular stemness and epithelial-mesenchymal transition (EMT).

205 s resistance, characteristics endowed by the epithelial-mesenchymal transition (EMT).

206 y demonstrates that nuclear villin regulates epithelial-mesenchymal transition (EMT).

207 oliferation and promoted cell to undergo the epithelial-mesenchymal transition (EMT).

208 that has been mechanistically linked to the epithelial-mesenchymal transition (EMT).

209 stem cell (CSC) states that are activated by epithelial-mesenchymal transition (EMT).

210 hanced DNA repair capacity and activation of epithelial-mesenchymal transition (EMT).

211 FPR2 knock-down, implying the involvement of epithelial-mesenchymal transition (EMT).

212 t, derive from the caudal somites through an epithelial-mesenchymal transition (EMT).

213 ins unclear whether copper has any effect on epithelial-mesenchymal transition (EMT).

214 erentiation state through a process known as epithelial-mesenchymal transition (EMT).

215 h of alternative splicing that occurs during epithelial-mesenchymal transition (EMT).

216 embryonic development program referred to as epithelial-mesenchymal transition (EMT).

217 their expression correlates with markers of epithelial-mesenchymal transition (EMT).

218 resses E-cadherin transcription and enhances epithelial-mesenchymal transition (EMT).

219 cells to adopt an invasive phenotype through epithelial-mesenchymal transition (EMT).

220 ironment for tumor cell growth and promoting epithelial-mesenchymal transition (EMT).

221 the endometrial adenocarcinoma (EAC) through epithelial-mesenchymal transition (EMT).

222 diated epigenetic reprogramming known as the epithelial-mesenchymal transition (EMT).

223 Tumor growth, angiogenesis, and epithelial-mesenchymal transition (EMT)/extracellular ma

224 ecific 84-1 monoclonal antibody that detects epithelial-mesenchymal transitioned (EMT) CTCs.

225 Understanding epithelial-mesenchymal transitions (EMT) during cancer m

226 ivation of peritoneal fibroblasts, mitigated epithelial-mesenchymal-transition (EMT), as well as enha

227 Metastasis is initiated after epithelial-mesenchymal-transition (EMT).

228 Epithelial-mesenchymal transitions (EMTs) are often gove

229 for disassembly of adherens junctions during epithelial-mesenchymal transitions (EMTs), loss of adher

230 ulation was highly attributed to significant epithelial-mesenchymal transition, enhanced Hh activity

231 ear beta-catenin accumulation, and increased epithelial-mesenchymal transition factors and colon cell

232 ment of the cell condensation process during epithelial-mesenchymal transition, forcing cells at the

233 more, Snail expression is associated with an epithelial-mesenchymal transition gene signature charact

234 During this conversion, pluripotency and epithelial-mesenchymal transition genes become markedly

235 expression and with several TGF-beta-related epithelial-mesenchymal transition genes in human HCC.

236 nes correlated with normalized expression of epithelial-mesenchymal transition genes.

237 pression of cancer stem cell-related and key epithelial-mesenchymal transition genes.

238 ethylation, histone modification, as well as epithelial-mesenchymal transition, highlighting potentia

239 portant source of myofibroblasts through the epithelial-mesenchymal transition; however, confirmatory

240 ated transforming growth factor-beta-induced epithelial-mesenchymal transition in A549 cells and HCC8

241 ignatures of receptor proteins associated to epithelial-mesenchymal transition in bladder and breast

242 ceptor expression signatures associated with epithelial-mesenchymal transition in bladder and breast

243 iR-138) as a novel regulator of invasion and epithelial-mesenchymal transition in breast cancer cells

244 High folate also promoted epithelial-mesenchymal transition in cancer cells and ex

245 erized by high expression of ZEB1, including epithelial-mesenchymal transition in epithelial-derived

246 miR-194 stimulated migration, invasion, and epithelial-mesenchymal transition in human prostate canc

247 This study demonstrated that HLJ1 inhibits epithelial-mesenchymal transition in vitro and reduces l

248 duced cell invasion, clonogenic ability, and epithelial-mesenchymal transition in vitro; reduced tumo

249 iveness of established tumors as well as the epithelial-mesenchymal transition, increasing numbers of

250 induced mammary tumorigenesis and suppresses epithelial-mesenchymal transition, intravasation, and me

251 as a key promoter of CC cell proliferation, epithelial-mesenchymal transition, invasion and cancer s

252 pression of E-cadherin, thereby facilitating epithelial-mesenchymal transition, invasion, and metasta

253 of proliferation, cell cycle, self-renewal, epithelial-mesenchymal transition, invasion, and resista

254 much less prone than BM-MSCs to promote the epithelial-mesenchymal transition, invasion, stemness, a

255 Epithelial-mesenchymal transition is one of the critical

256 ion of cellular proliferation, stemness, and epithelial-mesenchymal transition, is up-regulated in co

257 nd reduced E-cadherin expression, indicating epithelial-mesenchymal transition-like abnormalities.

258 rties through promotion of the expression of epithelial-mesenchymal transition-like cellular markers

259 Progenitors emerged by an epithelial-mesenchymal transition-like mechanism that re

260 ion of CD cells with loss of aquaporin-2 and epithelial-mesenchymal transition-like phenotypes.

261 a decreased cellular invasion and migration, epithelial-mesenchymal transition marker levels, lysyl o

262 esponse results in a marked induction of the epithelial-mesenchymal transition marker MMP-1.

263 Many neoplastic cells expressed epithelial-mesenchymal transition markers and coexpresse

264 rties concomitant with the downregulation of epithelial-mesenchymal transition markers Snail2 (SNAI2)

265 Galpha12QL induced ZEB1 and other epithelial-mesenchymal transition markers with fibroblas

266 In primary cultures, epithelial-mesenchymal transition markers, mesenchymal m

267 However, no effects on epithelial-mesenchymal transition markers, proliferation

268 keratinocytes, grown on fibronectin, led to epithelial-mesenchymal transition mediated by a cadherin

269 found that overexpression of MMP19 promotes epithelial-mesenchymal transition, migration, and invasi

270 eered Bcl-xL targeted to the nucleus promote epithelial-mesenchymal transition, migration, invasion a

271 RHAMM by the RB/E2F pathway was critical for epithelial-mesenchymal transition, motility, and invasio

272 rogramming, extracellular matrix remodeling, epithelial-mesenchymal transition, motility, invasion, m

273 ed in regulating branching morphogenesis and epithelial-mesenchymal transition of the mammary gland b

274 Drastic changes of organization, such as epithelial-mesenchymal transitions or the formation of s

275 n translation impact on the configuration of epithelial mesenchymal transition pathways in tumor cell

276 r epitranscriptome, regulating oncogenic and epithelial-mesenchymal transition pathways.

277 Epithelial-mesenchymal transitions play key roles in dev

278 ons; (ii) CRIS-B: TGF-beta pathway activity, epithelial-mesenchymal transition, poor prognosis; (iii)

279 a self-renewing phenotype independent of an epithelial-mesenchymal transition programme or regulatio

280 Although the epithelial-mesenchymal transition regulator Snail (Snai1

281 -beta transcriptional, tumor-suppressing and epithelial-mesenchymal transition responses.

282 rmore, this gain of ERbeta function enhances epithelial-mesenchymal transition signaling, thereby inc

283 EMT (epithelial-mesenchymal-transition)-signaling regulates T

284 with sustained activation of prosurvival and epithelial-mesenchymal transition-signaling pathways.

285 n previously that HCV infection generates an epithelial-mesenchymal transition state and tumor-initia

286 verged to identify subsets with differential epithelial-mesenchymal transition status, carcinoma in s

287 s and for mRNAs encoding proteins related to epithelial-mesenchymal transition, stemness, and Wnt sig

288 )-DD, which promotes cellular proliferation, epithelial-mesenchymal transition, stromal reaction, and

289 omain they generate migratory progenitors by epithelial-mesenchymal transition that mature into neuro

290 ces signaling networks comprising ERK1/2 and epithelial-mesenchymal transition that subsequently conf

291 d differentiation as well, but also promotes epithelial-mesenchymal transitions that lead to cataract

292 , two major transcription factors triggering epithelial-mesenchymal transition, the repression of the

293 ut of the periventricular zones, possibly by epithelial-mesenchymal transition, to colonize and maint

294 SLUG, beyond its known function as an epithelial-mesenchymal transition transcription factor,

295 roliferation, invasion, as well as switch in epithelial-mesenchymal-transition transcription factor e

296 ag1 to Notch1 signaling is required for post-epithelial-mesenchymal transition valvulogenesis.

297 Epithelial-mesenchymal transition was lower in BEV + RES

298 roids are more sensitive to TGF/beta-induced epithelial-mesenchymal transition with highly tumorigeni

299 2C in luminal breast carcinoma cells induced epithelial-mesenchymal transition with morphological and

300 all molecules that inhibit TGF-beta1-induced epithelial-mesenchymal transition without immediate TGF-