ライフサイエンスコーパス: epitopic

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1 The epitopic and extraepitopic compensatory mutations introd

2 +) T cells were expanded by using autologous epitopic and variant peptides.

3 Both proteins were recognized by a mono-epitopic antibody raised against a peptide of GMEB-2.

4 ency virus replication, the relevance of the epitopic breadth of those CTL responses remains unexplor

5 Although no epitopic changes were detected under the conditions of t

6 o the NH(2)-terminal region can modulate the epitopic conformation and troponin I and tropomyosin bin

7 Epitopic cross-reactivity does not explain the increased

8 of full length proteins (containing multiple epitopic determinants) for presentation to T cells.

9 ent expansion of the magnitude, breadth, and epitopic diversity of Env-specific binding antibody resp

10 is achieved through convergence on a common epitopic focus by utilizing various complementarity-dete

11 se cardiomyocyte phosphoprotein with limited epitopic homology to p53.

12 Z12 elicited an integrated and multi-epitopic immune response with persistent effector T cell

13 We identified epitopic-like regions in 206 parasite proteins and prese

14 d of the selected sites were associated with epitopic mutational escapes from CTLs.

15 ween TRBV7 and non-TRBV7 clonotypes and this epitopic peptide.

16 Epitopic peptides are identified from a direct compariso

17 Moreover, the HPV-6 epitopic peptides recognized by WIL differed to some ext

18 n to afford reproducible spectra that enable epitopic peptides to be identified in complex mixtures a

19 ascribed to both the optimized design of its epitopic region and the superior surface interacting pro

20 to bind to each of 2 sites within the Lym-1 epitopic region, have been linked to generate small (<2

21 The nine epitopic regions analyzed were also preserved so that, w

22 udy subjects, with a median of 14 individual epitopic regions targeted per person (range, 2 to 42), a

23 In these subjects, the median number of epitopic regions targeted was 13 (range, 2 to 39) and th

24 However, the number of epitopic regions targeted, the protein subunits recogniz

25 of significant similarity, inclusive of the epitopic regions, between allergens and helminth protein

26 in alcoholic hepatitis focused on individual epitopic regions.

27 he A(k)/p46-61 complex, which is hindered by epitopic residue(s) within p46-61.

28 suggests that T cell receptor (TCR) contact (epitopic) residue(s) flanking the minimal 51-59 determin

29 The identification of the epitopic sequence of a monoclonal antibody raised agains

30 ition to the nucleotide changes defining the epitopic sequence of He, a single C-to-G nucleotide tran

31 These modifications created an epitopic site that can be readily distinguished from the

32 ency, suggesting potential reversions of CTL epitopic sites recognized by the immune system of the tr

33 Nevertheless, the NKTcrs recognised distinct epitopic sites within these antigens, including alpha-ga

34 ssible significance of these antibodies, the epitopic specificity of the anti-rhodopsin antibodies wa

35 antibodies examined exhibited the identical epitopic specificity, it is likely that a common mechani

36 e of the time course of the response and the epitopic specificity, using peptides derived from the cy

37 ose of D10/B7 was combined with an IgG1 anti-epitopic tag monoclonal antibody, possibly because decor

38 tion occurs because the Ab binds to a common epitopic tag present at two sites on each of the two VHH

39 e produced and characterized a collection of epitopic tagged, heavy chain-only antibody VH domains (V

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