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1 tment with recombinant human erythropoietin (epoetin).
2 herapy received filgrastim and 6.8% received epoetin.
3 igher in the comparison of peginesatide with epoetin.
4 matologic response before considering use of epoetin.
5 matologic response before considering use of epoetin.
6 ment with either subcutaneous or intravenous epoetin.
7 alysis treatment and intravenous maintenance epoetin.
8 gy (ASCO/ASH) recommendations for the use of epoetin.
9 ered, on average, an additional 3306 U/wk of epoetin.
10 versus 60,600 dollars and 64,311 dollars for epoetin.
11 ge for patients who continued treatment with epoetin (-0.75 g/L, -2.26 to 0.75) (p<0.0001 for both co
12 SP was threefold longer than for intravenous Epoetin (25.3 versus 8.5 h), a difference of 16.8 h (95%
13 ume of distribution was similar for NESP and Epoetin (52.4 +/- 2.0 ml/kg versus 48.7 +/-2.1 ml/kg; me
15 e, administered monthly, was as effective as epoetin, administered one to three times per week, in ma
17 design, the single-dose pharmacokinetics of Epoetin alfa (100 U/kg) and an equivalent peptide mass o
18 eekly epoetin alfa (40K arm) or 120,000 U of epoetin alfa (120K arm) SC every 3 weeks for 18 addition
19 io, to either GM-CSF (0.3-5.0 microg/kg.d) + epoetin alfa (150 IU/kg 3 times/wk) or GM-CSF (0.3-5.0 m
21 sly (SC), followed by either standard weekly epoetin alfa (40K arm) or 120,000 U of epoetin alfa (120
22 be lower at day 29 among patients receiving epoetin alfa (adjusted hazard ratio, 0.79; 95% CI, 0.56
24 ninferiority study to evaluate the impact of epoetin alfa (EPO) on tumor outcomes when used to treat
26 showed a trend in overall survival favoring epoetin alfa (P =.13, log-rank test), and Cox regression
28 tarting chemotherapy, were randomized 2:1 to epoetin alfa 150 to 300 IU/kg (n = 251) or placebo (n =
31 enrollment to receive three weekly doses of epoetin alfa 40,000 U subcutaneously (SC), followed by e
33 ts who had received the Eprex formulation of epoetin alfa and 1 in a patient who had received Neoreco
34 erall tumor response was similar between the epoetin alfa and placebo groups after three chemotherapy
37 ; P=0.67) at 6 months did not differ between epoetin alfa and placebo, but declines in stroke volume
39 s transfused during the 12-week study in the epoetin alfa and the placebo groups were 5.9 and 9.5, re
43 multiple times and pooling preservative-free epoetin alfa caused this outbreak of bloodstream infecti
46 t that in newly diagnosed patients with SCLC epoetin alfa does not affect tumor response to chemother
49 were punctured multiple times, and residual epoetin alfa from multiple vials was pooled and administ
50 the first week (first CMR) was larger in the epoetin alfa group (19.9% LV mass; 95% CI, 14.0-25.7% LV
51 ell units transfused (4.5+/-4.6 units in the epoetin alfa group and 4.3+/-4.8 units in the placebo gr
52 red in 4 of 45 (9%) patients in the GM-CSF + epoetin alfa group compared with 1 of 21 (5%) patients w
53 oncentration at day 29 increased more in the epoetin alfa group than in the placebo group (1.6+/-2.0
54 e interval {CI}, 13.3-18.2% LV mass] for the epoetin alfa group vs 15.0% LV mass [95% CI, 12.6-17.3%
55 red-cell transfusion (relative risk for the epoetin alfa group vs. the placebo group, 0.95; 95% conf
57 ving combination therapy, patients receiving epoetin alfa had significant improvements in HRQL compar
59 ective study evaluating the effectiveness of epoetin alfa in maintaining RBV dose, alleviating anemia
61 inical trials have shown that treatment with epoetin alfa increases hemoglobin levels, reduces fatigu
65 s or reactions were associated with doses of epoetin alfa of more than 4000 U (multivariate odds rati
67 olled clinical trial assessed the effects of epoetin alfa on transfusion requirements, hematopoietic
68 led trial (N93-004) evaluated the effects of epoetin alfa on tumor response to chemotherapy and survi
69 domly assigned to treatment with intravenous epoetin alfa or matching saline placebo administered wit
72 s maintained in the prechemotherapy range in epoetin alfa patients, but decreased substantially in pl
73 ajority of these patients received Eprex, an epoetin alfa product marketed exclusively outside the Un
76 inical trial data showed that treatment with epoetin alfa resulted in clinically meaningful as well a
80 patients were randomized to 40,000 units of epoetin alfa subcutaneously weekly or placebo for an 8-w
81 were randomly assigned to receive a dose of epoetin alfa targeted to achieve a hemoglobin level of 1
82 were associated with higher median doses of epoetin alfa than the 188 other sessions (6500 vs. 4000
83 ted from pooled epoetin alfa, empty vials of epoetin alfa that had been pooled, antibacterial soap, a
85 and the adverse event profile of once-weekly epoetin alfa therapy in community-based practice are sim
86 mmunity-based trial suggest that once-weekly epoetin alfa therapy increases hemoglobin levels, decrea
87 the 2,964 patients assessable for efficacy, epoetin alfa therapy resulted in significant increases i
94 he authors aimed to evaluate the efficacy of epoetin alfa treatment on the outcome of OHCA patients i
95 ized, double-blind clinical trial evaluating epoetin alfa versus placebo in anemic cancer patients.
97 were maintained in 88% of patients receiving epoetin alfa vs. 60% of patients receiving placebo (P <
106 that preservative-free, single-use vials of epoetin alfa were punctured multiple times, and residual
108 or rescue PCI, a single intravenous bolus of epoetin alfa within 4 hours of PCI did not reduce infarc
109 sized that treating anemia with subcutaneous epoetin alfa would be associated with reverse ventricula
111 safety phase and a single dose (60,000 U of epoetin alfa) efficacy phase; the Reduction of Infarct E
112 mulating factor (GM-CSF) and erythropoietin (epoetin alfa) in anemic, neutropenic patients with myelo
114 erapy with recombinant human erythropoietin (epoetin alfa) might reduce the need for red-cell transfu
119 t between groups (observation, 35.42 months; epoetin alfa, 31.47 months; hazard ratio, 1.04; 95% CI,
120 enter, placebo-controlled, clinical trial of epoetin alfa, 40,000 U subcutaneously, once weekly vs. m
121 anemic critically ill patients treated with epoetin alfa, all dosing regimens were well tolerated an
123 S. liquefaciens was isolated from pooled epoetin alfa, empty vials of epoetin alfa that had been
124 ety cohort, of the 125 patients who received epoetin alfa, the composite outcome of death, MI, stroke
133 a mouse model, single-dose CNTO 530 (unlike epoetin-alfa or darbepoietin-alfa) bolstered red cell pr
135 quently can be treated via administration of epoetin-alfa, darbepoietin-alfa, or methoxy-PEG epoetin-
137 the present study to determine if utilizing epoetin alpha (EPO) with or without a higher dose of RVN
139 a patient who was treated with fixed-dosage epoetin alpha in the poorest versus best performing unit
141 owth factor injections (darbepoetin alpha or epoetin alpha) during combination therapy with standard
142 01 continues to support a positive effect of epoetin-alpha therapy on the quality of life of patients
144 ents and trials of longer-acting versions of epoetin-alpha, such as the novel erythropoiesis-stimulat
145 ogists and anemia managers adjusted doses of epoetin and intravenous iron as clinically indicated.
146 s individualized and specific application of epoetin and iron for each patient, and significant cost
147 anel found good evidence to recommend use of epoetin as a treatment option for patients with chemothe
148 anel found good evidence to recommend use of epoetin as a treatment option for patients with chemothe
149 hronic kidney disease patients who developed epoetin-associated PRCA and had 3 months or more follow-
150 Among chronic kidney disease patients with epoetin-associated PRCA, epoetin discontinuation and imm
152 en January 1998 and April 2004, 175 cases of epoetin-associated pure red-cell aplasia were reported f
154 d to intravenous methoxy polyethylene glycol-epoetin beta every 2 weeks (-0.71 g/L, 95% CI -2.20 to 0
155 igned to receive methoxy polyethylene glycol-epoetin beta every 2 weeks, and 224 to receive it every
157 o had received Neorecormon (a formulation of epoetin beta); both are products that are marketed outsi
158 effectiveness of methoxy polyethylene glycol-epoetin beta, given intravenously at 2-week or 4-week in
161 controlled trial investigated the effect of epoetin-beta to normalize hemoglobin values (13.0-15.0 g
165 s likely to confer a clinical advantage over Epoetin by allowing less frequent dosing in patients tre
166 hemodialysis, subcutaneous administration of epoetin can maintain the hematocrit in a desired target
167 Iron overload before the availability of epoetin constituted a serious problem; our review of the
168 sease patients with epoetin-associated PRCA, epoetin discontinuation and immunosuppressive therapy or
169 erritin 500 to 1200 ng/ml, TSAT <or=25%, and epoetin dosage >or=225 IU/kg per wk or >or=22,500 IU/wk.
171 anemic dialysis patients receiving adequate epoetin dosages and have a ferritin 500 to 1200 ng/ml an
173 ession models were used to estimate the mean epoetin dose and dose adjustment by profit, chain, and a
174 27 +/- 18,021 IU/wk, P = 0.003), whereas the epoetin dose essentially did not change for patients in
178 facilities, for-profit facilities increased epoetin doses 3-fold for patients with hematocrit levels
179 ialysis patients who were receiving adequate epoetin doses and who had ferritin levels between 500 an
180 conate maintains hemoglobin and allows lower epoetin doses in anemic hemodialysis patients with low T
182 ich a patient receives dialysis might affect epoetin dosing patterns and has implications for future
184 bcutaneous route, the average weekly dose of epoetin during the maintenance phase was 32 percent less
185 ess erythropoietin-stimulating agent (median epoetin-equivalent units per week: 5306 units/wk ferric
186 randomized controlled trial supports use of epoetin for patients with anemia associated with low-ris
190 n in 1988, recombinant human erythropoietin (epoetin) has been standard treatment for patients with a
191 m EMERALD 2, peginesatide was noninferior to epoetin in maintaining hemoglobin levels (mean between-g
193 levels in renal anemia that is treated with epoetins is often incomplete and subject to much variati
194 ed that if recombinant human erythropoietin (epoetin) is administered subcutaneously rather than intr
195 iency, and recombinant human erythropoietin (epoetin) is prescribed to correct the anemia partially.
196 ts from the Food and Drug Administration and epoetin manufacturers were reviewed for information on c
197 esatide once monthly or continued to receive epoetin one to three times a week, with the doses adjust
199 imilar patient case-mix, the average dose of epoetin ranged from 17,832 U/wk at chain 5 (nonprofit fa
201 ibution of iron deficiency toward anemia and epoetin resistance among end-stage renal disease (ESRD)
202 times more likely to receive filgrastim and epoetin, respectively, after controlling for other facto
206 ronic kidney disease developed antibodies to epoetin, resulting in pure red cell aplasia (PRCA).
208 t, and 615 were assigned to receive doses of epoetin sufficient to maintain a hematocrit of 30 percen
209 gluconate group required significantly less epoetin than their DRIVE dose (mean change of -7527 +/-
210 target range, with an average weekly dose of epoetin that is lower than with intravenous administrati
215 ho were receiving long-term hemodialysis and epoetin therapy to treatment with either subcutaneous or
217 ical trials supports the use of subcutaneous epoetin thrice weekly (150 U/kg tiw) for a minimum of 4
218 ical trials supports the use of subcutaneous epoetin thrice weekly (150 U/kg) for a minimum of 4 week
219 were assigned to receive increasing doses of epoetin to achieve and maintain a hematocrit of 42 perce
221 re receiving hemodialysis, administration of epoetin to raise their hematocrit to 42 percent is not r
225 incidence of RBC transfusion for placebo and epoetin treatment arms was 39.6% and 25.3% (P = .005), r
226 venously at 2-week or 4-week intervals, with epoetin treatment one to three times per week for haemog
227 stimulating agent is as safe as conventional epoetin treatment, and can maintain anaemia management i
229 ts of supplementation with ergocalciferol on epoetin utilization and other secondary outcomes in pati
230 ficiency or deficiency, but had no effect on epoetin utilization or secondary biochemical and clinica
233 zed, controlled clinical trial comparing two epoetins were examined by the techniques of functional d
234 nalogue of recombinant human erythropoietin (Epoetin) which has an increased terminal half-life in an
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