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1 binding mode similar to the natural product epoxomicin.
2 ST motif, or via proteasomal inhibition with epoxomicin.
3 pe generation is enhanced in the presence of epoxomicin.
4 teasome-specific inhibitors, lactacystin and epoxomicin.
5 he inhibitors MG115, MG132, lactacystin, and epoxomicin.
11 K inhibitor (U0126) or proteasome inhibitor (epoxomicin) also up-regulated Bim accumulation and rende
12 alues for the specific proteasome inhibitors epoxomicin and bortezomib for each of the catalytic site
14 temically to mice, the proteasome inhibitors epoxomicin and proteasome inhibitor-1 increased bone vol
17 cells to proteasome inhibitors (bortezomib, epoxomicin, and MG132), but not to proteotoxic or ER str
20 The proteasome inhibitors, lactacystin and epoxomicin, attenuated MIP-1beta induced CCR5 down-modul
21 wed that the proteasome inhibitors MG132 and epoxomicin blocked a postentry step in vaccinia virus (V
22 roteasome inhibitors MG132, lactacystin, and epoxomicin blocked PICT1 degradation, whereas the inhibi
23 lthough addition of the proteasome inhibitor epoxomicin blocked the presentation of two epitopes, pre
26 fic inhibitors, lactacystin-beta-lactone and epoxomicin, could not stabilize HNE-modified proteins in
27 in as a molecular probe, we demonstrate that epoxomicin covalently binds to the LMP7, X, MECL1, and Z
29 t to peptide aldehyde proteasome inhibitors, epoxomicin does not inhibit nonproteasomal proteases suc
30 requirement for proteasomal processing of an epoxomicin-enhanced epitope, an in vitro proteasome dige
31 nfirm that both the epoxomicin-inhibited and epoxomicin-enhanced epitopes are processed cytosolically
34 ystin and the specific proteasomal inhibitor epoxomicin increased soluble protein levels of the poly(
35 G1 is stabilized by the inhibitors MG132 and epoxomicin, indicating that it is degraded by the 26S pr
41 y injected six doses of naturally occurring (epoxomicin) or synthetic (Z-lle-Glu(OtBu)-Ala-Leu-al [PS
42 ed bovine erythrocyte proteasome reveal that epoxomicin potently inhibits primarily the chymotrypsin-
44 bitors PSI (Z-Ileu-Glu(OtBu)-Ala-Leu-CHO) or epoxomicin recapitulated many of the degenerative change
45 e therefore evaluated a novel, irreversible, epoxomicin-related proteasome inhibitor, carfilzomib.
49 ect was reversed by the proteasome inhibitor epoxomicin, suggesting a Sir2alpha-mediated ubiquitin/pr
50 PC12 cell model, aggregation was reduced by epoxomicin, suggesting that some other protein(s) induce
54 nce it is prevented by either lactacystin or epoxomicin, two inhibitors of proteasomal degradation.
56 replication was inhibited by both MG132 and epoxomicin, which would account for the effect on interm
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