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1 almost exclusively in strains cultured from equids.
2 l nervous system (CNS) disease in humans and equids.
3 ent serotypes, causes devastating disease in equids.
4 s causing neurological disease in humans and equids.
5 , occasionally causing disease in humans and equids.
6 tion and disease in experimentally immunized equids.
7 a browser, unlike the grazing diet of modern equids.
8 atory, systemic, and reproductive disease of equids.
10 replication during acute disease in infected equids and serve as resilient reservoirs of virus infect
11 e species of middle to late Pleistocene NWSL equid, and demonstrate that it falls outside of crown gr
13 complete reproductive isolation, and promote equids as a fundamental model for understanding the inte
14 resolution record of continental climate and equid body size change shows a directional size decrease
15 aracterized the Prdm9 gene in all species of equids by analyzing sequence variation of the ZF domains
17 clinical symptoms in experimentally infected equids coincided with rapid widespread seeding of viral
18 e extinct 'New World stilt-legged', or NWSL, equids constitute a perplexing group of Pleistocene hors
19 cant amounts of C(4) grasses were present in equid diets beginning at 9.9 Ma and in rhinocerotid diet
20 n and replication in experimentally infected equids during acute disease episodes and during asymptom
22 notably the suid Notochoerus, the hipparion equid Eurygnathohippus, the giraffid Sivatherium, and th
24 nthropogenic forces can dramatically reshape equid gastrointestinal microbiomes, which has broader im
25 that a single-nucleotide polymorphism in the equid herpesvirus type 1 DNA polymerase gene is associat
26 , we report that the nonneurovirulent strain equid herpesvirus type 1 strain NY03 caused lethal neuro
30 ecline and demise of two Alaskan Pleistocene equids, I selected a large number of fossils from the la
31 re balanced view of the virus as a threat to equids in a diverse range of settings, thus leading to a
33 he identification and precise demarcation of equid/Perissodactyl-specific features that for the first
37 te from these data that rhinos diverged from equids prior to the occurrence of the mutations causing
43 (MHC) class I genes isolated from a range of equid species and more distantly related members of the
44 ontained genes and alleles that are found in equid species and one group specific to the rhinoceros.
46 ve figured centrally in that debate, because equid species dominated North American late Pleistocene
47 pha1, alpha2 and theta globin genes from six equid species have been determined to investigate relati
49 horoughbreds and 42 samples from three other equid species that the T-allele was ancestral and there
50 the sequence and number of ZF domains among equid species, ranging from five domains in the Tibetan
53 ckness virus (AHSV) is a lethal arbovirus of equids that is transmitted between hosts primarily by bi
54 midges and causes African horse sickness in equids, with mortality reaching up to 95% in naive horse
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