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1  almost exclusively in strains cultured from equids.
2 l nervous system (CNS) disease in humans and equids.
3 ent serotypes, causes devastating disease in equids.
4 s causing neurological disease in humans and equids.
5 , occasionally causing disease in humans and equids.
6 tion and disease in experimentally immunized equids.
7 a browser, unlike the grazing diet of modern equids.
8 atory, systemic, and reproductive disease of equids.
9 t causes a severe and often fatal disease in equids and humans.
10 replication during acute disease in infected equids and serve as resilient reservoirs of virus infect
11 e species of middle to late Pleistocene NWSL equid, and demonstrate that it falls outside of crown gr
12 es have been identified only in primates and equids, and are expressed in placenta.
13 complete reproductive isolation, and promote equids as a fundamental model for understanding the inte
14 resolution record of continental climate and equid body size change shows a directional size decrease
15 aracterized the Prdm9 gene in all species of equids by analyzing sequence variation of the ZF domains
16                      Persistent infection of equids by equine infectious anemia virus (EIAV) is typic
17 clinical symptoms in experimentally infected equids coincided with rapid widespread seeding of viral
18 e extinct 'New World stilt-legged', or NWSL, equids constitute a perplexing group of Pleistocene hors
19 cant amounts of C(4) grasses were present in equid diets beginning at 9.9 Ma and in rhinocerotid diet
20 n and replication in experimentally infected equids during acute disease episodes and during asymptom
21 sands of humans and hundreds of thousands of equids during the past century.
22  notably the suid Notochoerus, the hipparion equid Eurygnathohippus, the giraffid Sivatherium, and th
23 ndrial and partial nuclear genomes from NWSL equids from across their geographic range.
24 nthropogenic forces can dramatically reshape equid gastrointestinal microbiomes, which has broader im
25 that a single-nucleotide polymorphism in the equid herpesvirus type 1 DNA polymerase gene is associat
26 , we report that the nonneurovirulent strain equid herpesvirus type 1 strain NY03 caused lethal neuro
27 erent vertebrates, including birds, rodents, equids, humans, and nonhuman primates.
28 ferent vertebrates including birds, rodents, equids, humans, and nonhuman primates.
29                 In placental tissue from the equid hybrids and the horse parent, the allelic expressi
30 ecline and demise of two Alaskan Pleistocene equids, I selected a large number of fossils from the la
31 re balanced view of the virus as a threat to equids in a diverse range of settings, thus leading to a
32 luding non-human primates, canines, felines, equids, ovids, suids, bovins, salmonids and murids.
33 he identification and precise demarcation of equid/Perissodactyl-specific features that for the first
34 ociated with sporadic outbreaks in human and equid populations in the Western Hemisphere.
35 ca through sporadic outbreaks into human and equid populations.
36  as a mechanism to ensure its maintenance in equid populations.
37 te from these data that rhinos diverged from equids prior to the occurrence of the mutations causing
38                          The timing of these equid regional extinctions and accompanying evolutionary
39 y other members of the order Perissodactyla (equid, rhinoceros and tapir species).
40  another group contained the majority of the equid sequences identified.
41                            In analyses of 30 equid sera positive in an ELISA with whole cells, 24 (80
42                             Although dog and equid sera with antibodies to whole-cell B. burgdorferi
43 (MHC) class I genes isolated from a range of equid species and more distantly related members of the
44 ontained genes and alleles that are found in equid species and one group specific to the rhinoceros.
45       Here we match variation in striping of equid species and subspecies to geographic range overlap
46 ve figured centrally in that debate, because equid species dominated North American late Pleistocene
47 pha1, alpha2 and theta globin genes from six equid species have been determined to investigate relati
48                       Although multiple NWSL equid species have been named, our palaeogenomic and mor
49 horoughbreds and 42 samples from three other equid species that the T-allele was ancestral and there
50  the sequence and number of ZF domains among equid species, ranging from five domains in the Tibetan
51                           In contrast, naive equids subjected to the low- or high-dose challenge deve
52                  Serum samples from dogs and equids suspected of having canine or equine borreliosis,
53 ckness virus (AHSV) is a lethal arbovirus of equids that is transmitted between hosts primarily by bi
54  midges and causes African horse sickness in equids, with mortality reaching up to 95% in naive horse

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