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1  the development of anti-EHV-1 agents in the equine.
2                               We selected an equine and an avian H3 influenza virus for further devel
3 ine influenza viruses (CIV) H3N8 and H3N2 of equine and avian origins, respectively.
4  CIV subtypes, the H3N8 and H3N2 viruses, of equine and avian origins, respectively.
5 view and biological characterization of H3N8 equine and canine influenza viruses using various experi
6                                     Although equine and canine viruses hardly replicated in the respi
7 ere numerous genetic differences between the equine and canine viruses, this variation did not result
8 e amount of test material for both rhPDGF-BB/equine and rhPDGF-BB/beta-TCP groups was comparable, but
9                               Both rhPDGF-BB/equine and rhPDGF-BB/beta-TCP have the potential to supp
10 firmed an electron transferase activity with equine and yeast cytochrome c, suggesting a conservation
11 deria mallei causes rapidly fatal illness in equines and humans when contracted by inhalation and als
12 the highly contagious disease "strangles" in equines and zoonotic meningitis in human.
13                        We selected 11 swine, equine, and avian H3 influenza viruses and evaluated the
14 inked sialic acid motif (SA2,3Gal) in avian, equine, and canine species; the alpha2,6-linked sialic a
15 e we characterize the replication of canine, equine, and human IAVs in the trachea of the dog, a spec
16  degeneration, absent fluid around the cauda equine, and hypertrophy of the ligamentum flavum.
17                         We produced purified equine antisera from horses hyperimmunized with EBOV vir
18 ALB/c mice were given up to 2 mg of purified equine antisera per animal, at 30 minutes, 1 or 2 days p
19 rease the possibility of serum sickness, the equine antisera was digested with pepsin to generate F(a
20 tential target of IgE-mediated reactivity to equine antivenom and a possible cause of the high incide
21 ty reactions during the first application of equine antivenom.
22 tope are associated with hypersensitivity to equine antivenoms.
23                                              Equine arteritis virus (EAV) has a global impact on the
24 ro susceptibility of CD3(+) T lymphocytes to equine arteritis virus (EAV) infection and establishment
25 olecule and possible cell entry receptor for equine arteritis virus (EAV).
26 site, we determined the crystal structure of equine arteritis virus PLP2 in complex with ubiquitin (1
27 findings for processing of glycoprotein 3 of equine arteritis virus.
28  examined the potential of H3N8 from canine, equine, avian, and seal origin to productively infect pi
29 he gel coating reduced the immunogenicity of equine BChE, unlocking the possibility to use non-human
30                                Understanding equine behaviour and cognition can inform horse welfare
31 ection of H. capsulatum var. farciminosum in equine blood and at high frequency among horses exhibiti
32 hat is associated with the administration of equine blood products; its etiologic agent has remained
33 iry manure slurry and a dry stack mixture of equine, bovine, and ovine manure.
34 to provide the first detailed description of equine breed diversity.
35 th four H3N8 viruses from different origins (equine, canine, avian, and seal) were performed.
36  WT virus infection, IFN-beta mRNA levels in equine cells infected with PLP2 mutants were increased b
37          Following induction of ovulation by equine chorionic gonadotropin (eCG)/human CG (hCG) treat
38 umulations of trophoblast cells that produce equine chorionic gonadotropin between days 40 and 120 of
39     In horses, the CXCL16 gene is located on equine chromosome 11 (ECA11) and encodes a glycosylated,
40 rRNA operon was validated for application to equine clinical samples.
41  this study, we evaluated the ability of the equine clinical treatments N-acetylcysteine, EDTA, and h
42 s immunocompromised; both patients had known equine contact.
43 on cold), human enteric coronavirus (HECoV), equine coronavirus (ECoV), and equine torovirus Berne (B
44 us studies in our laboratory have identified equine CXCL16 (EqCXCL16) to be a candidate molecule and
45 ic killer of equine neutrophils and identify equine-CXCRA and CXCR2 as its target receptors.
46 iation/association kinetics for yeast versus equine cytochrome c indicate that formation of mammalian
47 ides generated by enzymatic digestion of the equine cytochrome c with trypsin.
48 per day, intravenously, on days -4 to -2, or equine derived 30 mg/kg per day, intravenously, on days
49                                              Equine-derived heptavalent botulinum antitoxin (HBAT), t
50                                 Despeciated, equine-derived, heptavalent botulinum antitoxin (HBAT) i
51 e for association studies of immune-mediated equine diseases and for evolutionary analysis of genetic
52                                  The eastern equine encephalitis (EEE) and Venezuelan equine encephal
53 ern equine encephalitis (EEE) and Venezuelan equine encephalitis (VEE) viruses are pathogens that inf
54                                      Eastern equine encephalitis diagnostic serum antibody can appear
55 is, western equine encephalitis, and eastern equine encephalitis expressing the surface proteins of a
56 oly I:C) and a potential vaccine [Venezuelan equine encephalitis replicon particles expressing MERS-C
57 , or an unrelated, intramuscular, Venezuelan equine encephalitis replicon vaccine expressing EBOV GP.
58 EBOV glycoprotein (GP)-expressing Venezuelan equine encephalitis viral replicon particle vaccine prot
59                                      Eastern equine encephalitis virus (EEEV) is a representative mem
60 ghly pathogenic phenotype.IMPORTANCE Eastern equine encephalitis virus (EEEV) is one of the most path
61 equine encephalitis virus (WEEV), or eastern equine encephalitis virus (EEEV) when given individually
62 an equine encephalitis virus (VEEV), eastern equine encephalitis virus (EEEV), and western equine enc
63 equine encephalitis virus (WEEV) and eastern equine encephalitis virus (EEEV), two New World alphavir
64 equine encephalitis virus (VEEV) and Eastern equine encephalitis virus (EEEV), which have demonstrate
65 quine encephalitis virus (WEEV), and eastern equine encephalitis virus (EEEV).
66 ycoprotein of natural North American eastern equine encephalitis virus (NA-EEEV) isolates and demonst
67                                   Venezuelan equine encephalitis virus (VEE) empty replicon particles
68    The genome from the alphavirus Venezuelan equine encephalitis virus (VEE) was modified to express
69 are the encephalitic alphaviruses Venezuelan equine encephalitis virus (VEEV) and Eastern equine ence
70 her related alphaviruses, such as Venezuelan equine encephalitis virus (VEEV) and Semliki Forest viru
71 usly, a replicon vaccine based on Venezuelan equine encephalitis virus (VEEV) demonstrated protective
72        New attenuated variants of Venezuelan equine encephalitis virus (VEEV) designed in this study
73 teins of Sindbis virus (SINV) and Venezuelan equine encephalitis virus (VEEV) form cytoplasmic comple
74                                   Venezuelan equine encephalitis virus (VEEV) is a mosquito-borne RNA
75                                   Venezuelan equine encephalitis virus (VEEV) is a pathogenic alphavi
76                                   Venezuelan equine encephalitis virus (VEEV) is a previously weaponi
77                                   Venezuelan equine encephalitis virus (VEEV) is a reemerging virus t
78                                   Venezuelan equine encephalitis virus (VEEV) is a select agent that
79                                   Venezuelan equine encephalitis virus (VEEV) is an alphavirus that i
80                                   Venezuelan equine encephalitis virus (VEEV) is an alphavirus with a
81                                   Venezuelan equine encephalitis virus (VEEV) is an emerging pathogen
82                                   Venezuelan equine encephalitis virus (VEEV) is an important human a
83                                   Venezuelan equine encephalitis virus (VEEV) is an important human a
84                                   Venezuelan equine encephalitis virus (VEEV) is one of the most path
85 we have developed a noncytopathic Venezuelan equine encephalitis virus (VEEV) mutant that can persist
86   The infectious titer of WNV and Venezuelan equine encephalitis virus (VEEV) TC83 in the brains of A
87           The structures of human Venezuelan equine encephalitis virus (VEEV), an alphavirus, in comp
88 ins drive the assembly of vRCs of Venezuelan equine encephalitis virus (VEEV), and G3BPs were shown t
89 , Rift Valley fever virus (RVFV), Venezuelan equine encephalitis virus (VEEV), and herpes simplex vir
90                  The alphaviruses Venezuelan equine encephalitis virus (VEEV), eastern equine encepha
91 ocess of one of the alphaviruses, Venezuelan equine encephalitis virus (VEEV), to understand its adap
92 se severe encephalitis in humans: Venezuelan equine encephalitis virus (VEEV), western equine encepha
93  potential vaccine candidates for Venezuelan equine encephalitis virus (VEEV), western equine encepha
94 ics are currently unavailable for Venezuelan equine encephalitis virus (VEEV), which elicits flu-like
95 vel of heterologous proteins from Venezuelan equine encephalitis virus (VEEV)-based replicons.
96 emonstrate that Tc bovine-derived Venezuelan equine encephalitis virus (VEEV)-specific TcPAbs are hig
97                                      Western equine encephalitis virus (WEEV) and eastern equine ence
98 rotropic alphaviruses, which include western equine encephalitis virus (WEEV) and Fort Morgan virus,
99 quine encephalitis virus (EEEV), and western equine encephalitis virus (WEEV) are arthropod-borne pos
100                                      Western equine encephalitis virus (WEEV) has caused several epid
101                                      Western equine encephalitis virus (WEEV) is an arbovirus from th
102 an equine encephalitis virus (VEEV), western equine encephalitis virus (WEEV), and eastern equine enc
103 an equine encephalitis virus (VEEV), western equine encephalitis virus (WEEV), or eastern equine ence
104 ily using the neurotropic alphavirus western equine encephalitis virus (WEEV).
105 o pathogenic mosquito-borne viruses (Eastern equine encephalitis virus [EEEV], Western equine encepha
106 equine encephalitis virus [WEEV], Venezuelan equine encephalitis virus [VEEV], and Chikungunya virus
107 rn equine encephalitis virus [EEEV], Western equine encephalitis virus [WEEV], Venezuelan equine ence
108 r atomic resolution structures of Venezuelan equine encephalitis virus and dengue virus revealed tran
109  inhibitors of the replication of Venezuelan equine encephalitis virus and other alphaviruses.
110 er encephalitic arboviruses, such as eastern equine encephalitis virus and West Nile virus, underscor
111  very amino-terminal subdomain of Venezuelan equine encephalitis virus capsid protein, SD1, plays a c
112 ance survival of mice given a lethal western equine encephalitis virus challenge.
113 IXV) is an enzootic member of the Venezuelan Equine Encephalitis Virus complex and belongs to the New
114 of the mosquito-borne North American eastern equine encephalitis virus in myeloid-lineage cells by bi
115 ase manifestations characteristic of eastern equine encephalitis virus infection in humans.
116 ase (PARP) family in clearance of Venezuelan equine encephalitis virus mutants from infected cells.
117 a virus-vectored vaccine (Kp47/47-Venezuelan equine encephalitis virus replicon particle) for safety,
118 opagating vaccine vector based on Venezuelan equine encephalitis virus replicon particles (VRP) expre
119      Using synthetic genomics and Venezuelan equine encephalitis virus replicons (VRPs) expressing sp
120 s of >100 in cell-based assays using western equine encephalitis virus replicons.
121 pagating, truncated derivative of Venezuelan equine encephalitis virus that targets dendritic cells (
122 n coronavirus 229E), Togaviridae (Venezuelan equine encephalitis virus), and Hepeviridae (HEV), indic
123 nly one other NT human arbovirus (Venezuelan equine encephalitis virus), which is also poorly underst
124 ing primary mosquito infection by Venezuelan equine encephalitis virus, an arbovirus causing neurolog
125 arations of bacteriophage lambda, Venezuelan equine encephalitis virus, and Staphylococcus aureus dur
126  lethal alphavirus infection with Venezuelan equine encephalitis virus, and this was associated with
127 he alphaviruses Sindbis virus and Venezuelan equine encephalitis virus, as well as La Crosse bunyavir
128 ellow fever virus, Sindbis virus, Venezuelan equine encephalitis virus, measles virus, influenza A vi
129                       Venezuelan and western equine encephalitis viruses (VEEV and WEEV; Alphavirus;
130 bis, Chikungunya, and eastern and Venezuelan equine encephalitis viruses and demonstrate that a small
131             Venezuelan, western, and eastern equine encephalitis viruses are New World alphaviruses t
132 halitic alphaviruses (eastern and Venezuelan equine encephalitis viruses) based upon either fusion of
133 , including western, eastern, and Venezuelan equine encephalitis viruses, cause serious and potential
134  for Venezuelan equine encephalitis, western equine encephalitis, and eastern equine encephalitis exp
135 ause Venezuelan equine encephalitis, western equine encephalitis, and eastern equine encephalitis.
136  seen with Japanese encephalitis, Venezuelan equine encephalitis, and Rift Valley fever viruses; and
137  developed a combined vaccine for Venezuelan equine encephalitis, western equine encephalitis, and ea
138 lenge with the viruses that cause Venezuelan equine encephalitis, western equine encephalitis, and ea
139 is, western equine encephalitis, and eastern equine encephalitis.
140                                              Equine encephalosis virus (EEV) distribution was thought
141      To characterize the role of biofilms in equine endometritis, six mares were inoculated with lux-
142  present during clinical cases of infectious equine endometritis.
143 n content and organisation, in young and old equine energy storing and positional tendons.
144 gnant endometrium at levels similar to other equine epithelia.
145  prior hysterectomy received oral conjugated equine estrogen (0.625 mg/day) or placebo.
146 signment to hormone therapy (HT) (conjugated equine estrogen (CEE) alone or CEE plus medroxyprogester
147  with a uterus were randomized to conjugated equine estrogens (0.625 mg/d [estrogen]) plus medroxypro
148 ostmenopausal women to placebo or conjugated equine estrogens (0.625 mg/d) plus medroxyprogesterone a
149 ociations between the use of oral conjugated equine estrogens (CEE) (0.625 mg/day) plus medroxyproges
150                                   Conjugated equine estrogens (CEE, 0.625 mg/d) plus medroxyprogester
151 en with an intact uterus received conjugated equine estrogens (CEE; 0.625 mg/d) plus medroxyprogester
152  to receive 4 y of 0.45 mg/d oral conjugated equine estrogens (o-CEE) plus 200 mg/d micronized proges
153                              Oral conjugated equine estrogens (o-CEE), 0.45 mg/d, or transdermal 17be
154 r hysterectomy were randomized to conjugated equine estrogens alone (0.625 mg/d) or placebo with a me
155 ical significance in the trial of conjugated equine estrogens alone in women with prior hysterectomy
156  a mean of 7.1 years among 10 251 conjugated equine estrogens alone participants.
157 hey had a uterus (N=16 608) or to conjugated equine estrogens only if they had prior hysterectomy (N=
158 one, replication was not observed in primary equine fetal liver cultures or after electroporation of
159                                 Although the equine fossil record represents a textbook example of ev
160 V from different lineages and sublineages, A/equine/Georgia/1/1981 (eq/GA/81) was selected to produce
161 unt of new cementum formed for the rhPDGF-BB/equine group (4.8 +/- 1.3 mm) was significantly higher (
162 y higher (P <0.01) in favor of the rhPDGF-BB/equine group.
163 he complex collagen architecture of juvenile equine growth cartilage.
164                                    Avian and equine H3 viruses elicited broadly cross-reactive antibo
165 s for use in the event of transmission of an equine H3N8 influenza virus to humans.
166 -as well as those from humans, swine and the equine H3N8 lineage--sharing an ancestor with them in th
167  pandemic virus and, independently, the 1963 equine H3N8 panzootic lineage.
168 l genomic segments and demonstrates that the equine H7N7 lineage is a sister clade to strains from bi
169 no acid sequence differences between the two equine HAs located on the virus membrane-distal molecula
170 igosaccharide cellopentaose, and the protein equine heart cytochrome c.
171                                              Equine hepacivirus (EHCV; nonprimate hepacivirus) is a h
172 e many animal and primate hepaciviruses, the equine hepaciviruses remain the closest genetic relative
173 the SRT of IE62 was replaced with the SRT of equine herpesvirus 1 (EHV-1) IEP, its trans-activation a
174 accination remains the best option to combat equine herpesvirus 1 (EHV-1) infection, and several diff
175                                              Equine herpesvirus 1 (EHV-1) is a major pathogen affecti
176 from herpes simplex virus type 1 (HSV-1) and equine herpesvirus type 1 (EHV-1) failed to support DNA
177                   The cytolytic animal virus equine herpesvirus type 1 (EHV-1) was evaluated for its
178                                              Equine herpesvirus type 1 (EHV1), a well-known member of
179 n linked to the neuropathogenic phenotype of equine herpesvirus-1 (EHV-1).
180 multiple introductions of MRSA CC398 in a UK Equine Hospital, identifying an emerging zoonotic pathog
181 -hemolytic Streptococcus dysgalactiae in the equine host is increasingly recognized.
182 ion does not seem by itself to contribute to equine hybrid sterility.
183                                              Equine hybrids, in particular a quartet pedigree compose
184 sessing allelic differences of ZF domains in equine hybrids.
185 rdm9 an interesting candidate to evaluate in equine hybrids.
186                                         Such equine IgG may find utility as a post-exposure prophylac
187    Herein we describe production of purified equine IgG obtained from horses immunized with plasmid D
188                        Administration of the equine IgG over 5 days to cynomolgus macaques infected 2
189                          We aimed to dissect equine immune responses after experimental NPHV infectio
190  Following viral challenge, an activation of equine immune responses was observed.
191 ift from the recommended polyclonal human or equine immunoglobulins to monoclonal antibody therapies.
192 ritis virus (EAV) has a global impact on the equine industry as the causative agent of equine viral a
193  source of significant economic loss for the equine industry from high rates of abortion in pregnant
194 mportant due to major economic losses in the equine industry.
195 genetic analyses of isolates from 2 cases of equine infection with the EEE virus and 1 case of human
196           This study, using CA proteins from equine infectious anemia virus (EIAV) as an example, dem
197                               The lentivirus equine infectious anemia virus (EIAV) encodes the small
198 f N-tropic murine leukemia virus (N-MLV) and equine infectious anemia virus (EIAV) infection, promoti
199 image human immunodeficiency virus (HIV) and equine infectious anemia virus (EIAV) particles.
200 n we studied the full-length CA protein from equine infectious anemia virus (EIAV), a lentivirus shar
201  HIV-1, feline immunodeficiency virus (FIV), equine infectious anemia virus (EIAV), or N-tropic murin
202                                StarGen is an equine infectious anemia virus (EIAV)-based lentiviral v
203 planted into the heterologous Gag protein of equine infectious anemia virus (EIAV).
204 tibody escape data from horses infected with equine infectious anemia virus.
205 humans; however, a recent report showed that equine influenza A viruses (IAVs) can be isolated from p
206 ne influenza virus (CIV) emerged from A/H3N8 equine influenza virus (EIV) around the year 2000 throug
207 H3N8 originated from a direct transfer of an equine influenza virus (EIV) in the early 2000s.
208 V; H3N8) originated after the transfer of an equine influenza virus (EIV) into dogs.
209 tudied the intra- and interhost evolution of equine influenza virus in vaccinated horses.
210 sponses to study the within-host dynamics of equine influenza virus infection in horses.
211 , which originated from the transfer of H3N8 equine influenza virus to dogs; and the H3N2 CIV, which
212 04 an hemagglutinin 3 neuraminidase 8 (H3N8) equine influenza virus was transmitted from horses to do
213                                              Equine influenza viruses (EIV) are responsible for rapid
214 ibution of K186 and E186 among H3N8 CIVs and equine influenza viruses (EIVs), the ancestors of H3N8 C
215                                              Equine influenza viruses have crossed the species barrie
216  it is important to develop vaccines against equine influenza viruses in the event that an EIV evolve
217  and 16S rRNA gene sequence showed that most equine isolates could also be differentiated from S. dys
218  other antimicrobial agents against clinical equine isolates of Corynebacterium pseudotuberculosis.
219 lciforme strains (12/15) were recovered from equine keratitis infections; however, strains of F. kera
220 est the potential anti-rotavirus activity of equine lactadherin and support the feasibility of develo
221                             We observed that equine MaSCs (eMaSCs) maintain their growth potential in
222 s with prominent pigment synthesis mimicking equine melanoma represent a rare variant of biologically
223 ing (SAXS) was used to study the behavior of equine metmyoglobin (Mb) and bovine pancreatic trypsin i
224 .e., bovine pancreatic trypsin inhibitor and equine metmyoglobin, on the conformational ensemble of a
225                                   An in vivo equine model of infectious endometritis was also develop
226 contribution of different Mphi subsets in an equine model of naturally occurring tendon injury.
227 nt of spontaneous osteochondral repair in an equine model.
228 EAV entry receptor in EAV-susceptible cells, equine monocytes.
229 ing to other sequence types particularly the equine MRSA ST8.
230 hat LukPQ is a potent and specific killer of equine neutrophils and identify equine-CXCRA and CXCR2 a
231            The closest homolog of HCV is the equine nonprimate hepacivirus (NPHV), which shares simil
232  the symptoms of EEV can be similar to other equine notifiable diseases this is a significant finding
233 group and centre, to receive oral conjugated equine oestrogen (0.625 mg per day; n=5310) or matched p
234 hPDGF-BB) combined with either a particulate equine or a beta-tricalcium phosphate (beta-TCP) matrix.
235 r with rabies immunoglobulin (RIG) of either equine or human origin.
236 f hydroxyapatite and collagen bone blocks of equine origin (eHAC), infused with recombinant human pla
237 ne influenza virus (CIV) H3N2 (CIV-H3N2) and equine-origin CIV H3N8 (CIV-H3N8), are enzootic in the c
238 respiratory disease in dogs, and include the equine-origin H3N8 and the avian-origin H3N2 viruses.
239 tion, utilizing bioluminescence imaging with equine P. aeruginosa isolates from this study.
240   Experience with formulating guidelines for equine parasite control illustrates that end-users strug
241 ge of the vector ecology of these tick-borne equine pathogens, emphasizing tick transmissibility and
242 nalyses confirmed that the tentatively named equine pegivirus (EPgV) represents a novel species withi
243 h most of these animals were coinfected with equine pegivirus (EPgV), also a flavivirus, EPgV viral l
244                                              Equine piroplasmosis is a disease of Equidae, including
245        The invasive trophoblast cells of the equine placenta migrate into the endometrium to form end
246 hod for the detection of alpha-cobratoxin in equine plasma has now been developed.
247 hod to confirm the presence of alpha-Cbtx in equine plasma.
248                                    DAT is an equine polyclonal antibody that is not commercially avai
249 l (GI) tracts of six species (bovine, ovine, equine, porcine, chicken, and deer) and from two human-d
250  ends decades of speculation, explaining how equine pregnancies survive without measurable circulatin
251 physiological concentrations and to maintain equine pregnancy in the absence of luteal progesterone i
252 justify future efficacy studies for purified equine products in NHPs.
253 oc mobilization from a direct predecessor of equine pVAPA.
254  circular virulence plasmids associated with equine (pVAPA) and porcine (pVAPB variant) R. equi isola
255 lance was implemented from 2011-2016 in a UK Equine Referral Veterinary Hospital and identified 81 me
256 perelastosis cutis, also known as hereditary equine regional dermal asthenia.
257 roxide to disrupt in vitro biofilms and kill equine reproductive pathogens (Escherichia coli, Pseudom
258 ine respiratory mucosal explants and primary equine respiratory epithelial cells (EREC), grown at the
259  alphaherpesvirus family, was used to infect equine respiratory mucosal explants and primary equine r
260 ing specific nucleic acid sequences for four equine respiratory pathogens as representative examples,
261 cryo-electron microscopy structure of native equine rhinitis A virus (ERAV), together with the struct
262 ure of a unique, massively expanded state of equine rhinitis A virus that provides insight into how t
263 her we conducted a retrospective analysis of equine sera, which had been collected for diagnosis of o
264 1 to the inhibitory effect of both human and equine SERINC5.
265                         The binding modes to equine serum albumin (ESA) of two nonsteroidal anti-infl
266 -circulating bioscavenger nanogel by coating equine serum-derived BChE with a zwitterionic polymer ge
267               In the acute postinjury stage, equine SF exhibits poor boundary lubrication properties,
268                           The mu(kinetic) of equine SF from joints with acute injury (0.036) was high
269 APL characteristics intermediate to those of equine SF from joints with acute injury and normal equin
270 on of high molecular weight HA (4,000 kd) to equine SF from joints with acute injury reduced the mu(k
271                                              Equine SF from joints with chronic injury had mu(kinetic
272 njury (0.036) was higher (+39%) than that of equine SF from normal joints (0.026).
273                                              Equine SF from normal joints, joints with acute injury,
274 e basis for this, and adding HA to deficient equine SF restored lubrication function.
275  and molecular weight, on the mu(kinetic) of equine SF samples from normal joints and joints with acu
276                                              Equine SF samples were analyzed for HA, PRG4, and SAPL c
277                           Compared to normal equine SF, SF from joints with acute injury had a lower
278 e mu(kinetic) to a value near that of normal equine SF.
279  SF from joints with acute injury and normal equine SF.
280 ecreased with increasing HA concentration in equine SF.
281 e for gene flow involving three contemporary equine species despite chromosomal numbers varying from
282 vidual, woolly mammoths, polar bears and two equine species, we confirm that DNA methylation survives
283 ring tendons, such as the human Achilles and equine superficial digital flexor tendon (SDFT), are hig
284 n-resolving mediators in naturally occurring equine tendon injury with disease stage and age.
285 es, and to investigate the role of HA in the equine to canine cross-species transfer, we used X-ray c
286 irus (HECoV), equine coronavirus (ECoV), and equine torovirus Berne (BEV) are enzymatically active, r
287 y for the homologous protein of a torovirus, equine torovirus, which is more closely related to coron
288            Twelve defects received rhPDGF-BB/equine treatment, 12 defects received rhPDGF-BB/beta-TCP
289 Pseudomonas aeruginosa strains isolated from equine uterine infections.
290 hat Gram-negative bacteria isolated from the equine uterus are capable of producing a biofilm in vitr
291  for elimination of bacterial biofilm in the equine uterus.
292 gle nonantibiotic treatment commonly used in equine veterinary practice was able to reduce the CFU an
293 he equine industry as the causative agent of equine viral arteritis (EVA), a respiratory, systemic, a
294 In the fusion subdomain of canine and recent equine virus HAs a unique difference is observed by comp
295 s of the HAs from two antigenically distinct equine viruses and from a canine virus.
296 well in the upper respiratory tract, but the equine viruses replicated poorly in the lungs.
297                                The swine and equine viruses replicated well in the upper respiratory
298 antiviral activity of lactadherin sourced by equines, we undertook a proteomic analysis of milk fat g
299 -1 strain KyA is attenuated in the mouse and equine, whereas wild-type strain RacL11 induces severe i
300 irus 1 (EHV-1) is a major pathogen affecting equines worldwide.

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