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1 on characterized by a prolonged and painless erection.
2 rone, however, reduced the latency period to erection.
3 ller proportion of males displayed reflexive erection.
4 eurons may have different roles in mediating erection.
5 us females, and (after RF lesions) reflexive erection.
6 penile erection, and NOS inhibitors prevent erection.
7 tes erectile physiology, including sustained erection.
8 anatomic basis of the nerves responsible for erection.
9 elevated adenosine-induced prolonged penile erection.
10 suggesting that EETs are required for normal erection.
11 shed model of electrically stimulated penile erection.
12 pherally, notably through its role in penile erection.
13 g magnetic resonance imaging with artificial erection.
14 e NO-cGMP pathway is important during penile erection.
15 c and spinal cord regions involved in penile erection.
16 fficacy in anesthetized dog model for penile erection.
17 The two drugs also reduce erection.
18 leads to sustained NO production and maximal erection.
19 that PI3-kinase/Akt physiologically mediates erection.
20 orpus cavernosum tissue that is required for erection.
21 ry behavior and facilitated vocalization and erection.
22 e ignored as a potential regulator of penile erection.
23 s cavernosum pressure to culminate in penile erection.
24 lated erections, and PS phases exhibiting an erection.
25 ts into the central mechanisms which control erections.
26 immobility, 22-kHz vocalization, and penile erections.
28 surgery) and included: 1) ability to have an erection, 2) maintain an erection, 3) attain orgasm, 4)
29 ability to have an erection, 2) maintain an erection, 3) attain orgasm, 4) dry orgasm, and 5) whethe
30 tions prior to treatment reported functional erections; 531 (53% [95% CI, 50%-56%]) of patients witho
31 x of Erectile Function on ability to achieve erection, ability to maintain erection, ejaculation freq
32 f PDE5 inhibitor and the generation of early erections after radical prostatectomy as a strategy to i
33 denosine was induced during sustained penile erection and contributes to PI3K/AKT activation and subs
35 opamine receptor agonist, facilitates penile erection and is effective in patients suffering from ere
36 s featured with prolonged and painful penile erection and is prevalent among males with sickle cell d
37 forms of nNOS are major mediators of penile erection and so may be targets for therapeutic intervent
39 with low sexual desire and reduced nocturnal erections and ejaculate volume, all of which improve wit
43 Although the neural mechanisms of PS-related erections are unknown, the forebrain likely plays a crit
44 ncluding micturition, defecation, and penile erection, as well as to brain networks controlling nutri
46 ed by race (38.4% of black men reported firm erections at > or =18 months vs 25.9% of Hispanic and 21
47 the ability to get an erection, maintain an erection, attain orgasm, and being sexually active in co
49 e the chances of a man to recover functional erections but also return him to his preoperative erecti
50 neuronal NO synthase (nNOS) initiates penile erection, but has not been thought to participate in the
52 , a specific PDE5 inhibitor, promotes penile erection by blocking the activity of PDE5, which causes
53 ts in the sildenafil group reported improved erections compared with 13 (10%) of 127 patients in the
55 e beta splice variant of nNOS elicits normal erection despite a decrease in stimulus-response charact
56 management model that includes daily vacuum erection device therapy, dietary supplementation and PDE
58 use of PDE5 inhibitor and the generation of erections early after radical prostatectomy are of some
59 ity to achieve erection, ability to maintain erection, ejaculation frequency, orgasm frequency, and s
63 sphorylation, and subsequent impaired penile erection featured with the reduction of ratio of maximal
64 ements in ability to achieve and maintain an erection, frequency of ejaculation, and orgasm frequency
65 es both initiation and maintenance of penile erection, implying unique approaches for treating erecti
69 nt and efficacious in vivo, eliciting penile erections in rats at a dose of 0.03 micromol/kg, with a
70 Rho-kinase antagonism stimulates rat penile erection independently of nitric oxide, introduces a pot
72 ponsible for tip elongation, whereas papilla erection is a hydraulic process driven by blood flow.
75 A key role for nitric oxide (NO) in penile erection is well established, but the relative roles of
76 ic oxide (NO), a mediator involved in penile erection, is synthesized by the nitric oxide synthase (N
77 n the training set, symptoms of poor morning erection, low sexual desire, erectile dysfunction, inabi
78 d an adverse effect on the ability to get an erection, maintain an erection, attain orgasm, and being
80 bsequent evaluation of 59a in the rat penile erection model revealed in vivo activity, comparable in
81 rats were tested for copulation, noncontact erection (NCE) evoked by remote cues from estrous female
82 in sexual arousal, inferred from noncontact erection (NCE) evoked in male rats by remote cues from e
84 d to the working hypothesis that Dd sorocarp erection occurs by two superimposed processes: one perio
85 he distal ends of the Rad50 coiled coils and erection of a 1000 A scaffold to productively bridge DNA
86 tween infected and naive animals through the erection of a steel mesh was effective at stopping virus
87 hological differentiation contributes to the erection of aerial hyphae by decreasing the surface tens
88 in family of fungal proteins involved in the erection of aerial hyphae in the filamentous fungus Schi
89 howed that the mycelial architecture and the erection of aerial hyphae were affected by the expressio
92 istic analyses of these data have led to the erection of phylogenetic hypotheses that appear to be at
93 r than 50% of patients understood the terms "erection" or "impotent." Only 5% of patients understood
95 d in a significant decrease in the number of erections per hour of PS, number of PS-related erections
98 [95% CI, 45%-52%]) of those with functional erections prior to treatment reported functional erectio
104 eatment reversed abnormalities in key penile erection signaling molecules, including phosphodiesteras
105 inability to obtain and/or maintain a penile erection sufficient for adequate sexual relations, is al
107 aseline function, 31 (89%, 95% CI 73-97) had erections sufficient for penetration 12 months after foc
110 is implicated in normal and abnormal penile erection, the exact role and the underlying mechanism fo
111 le provides an overview of the physiology of erection, the pathophysiology of ED, and modern patient
112 The ability of ABT-724 to facilitate penile erection together with the favorable side-effect profile
113 xide (NO) pathway is known to mediate penile erection under normal conditions, we hypothesized that t
117 mean score for the question about achieving erections was 100 percent higher after treatment than at
120 ABT-724 dose-dependently facilitates penile erection when given s.c. to conscious rats, an effect th
121 A lesioning selectively disrupted PS-related erections while leaving waking-state erections intact.
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