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1 es exhibited a clinical score of 1 (pinpoint erythema).
2 nt is often initiated by symptoms or visible erythema.
3 , consisting mainly of infusion syndrome and erythema.
4 ced more TTs than UVA1 for the same level of erythema.
5 eukocyte markers, while quantifying clinical erythema.
6 pital because of cough, sputum, pruritus and erythema.
7 on site pain and tenderness, induration, and erythema.
8 delivery, but are sometimes associated with erythema.
9 cant reductions in gingival inflammation and erythema.
10 four patients experienced only minimal skin erythema.
11 SpeB- strains appeared pale with surrounding erythema.
12 ility and reproducibility for both scale and erythema.
13 rized by epidermal hyperplasia, scaling, and erythema.
14 ndoscopy showing areas of nonspecific patchy erythema.
16 and as tenderness (33 [66%] of 50 [51-79]), erythema (20 [40%] of 50 [26-55]), and pruritus (41 [82%
17 local adverse events with imiquimod included erythema (27%), scabbing or crusting (21%), flaking (9%)
18 mon adverse events were again injection-site erythema (33 [8%] of 416 with 50 mg sirukumab every 4 we
20 eported injection site pain (52% vs 17%) and erythema (73% vs 25%) more frequently than placebo recip
21 acterized by less transient and nontransient erythema, a more lateral distribution of erythema and te
22 ter an immediate allergic reaction including erythema, abdominal pain, vomiting, and anaphylactic sho
23 , 28-53 years of age), the mean reduction in erythema across six doses of UVR (300-800 mJ/cm(2) in 10
24 a decrease in ulceration (alpha = 0.068) and erythema (alpha = 0.005) at the mid-point with continued
26 inant, disorder characterized by generalized erythema and cutaneous blistering at birth followed by h
30 patients (50.2%), followed by injection site erythema and fatigue in 227 (25.6%) and 212 patients (23
32 in the 1 g idarucizumab group (infusion site erythema and hot flushes), one in the 5 g plus 2.5 g ida
33 enotype consisting of early-onset patches of erythema and hyperkeratosis, as well as SCA manifesting
34 ed cutaneous immunity based on the extent of erythema and induration after intradermal VZV antigen in
42 d on the day of treatment, and short-lasting erythema and pain at the site of application were common
45 ed to PCV13 (facial diplegia, injection-site erythema and pyrexia, autoimmune hemolytic anemia, and s
47 for association with skin cGVHD involvement (erythema and sclerosis, skin symptoms), lacrimal dysfunc
49 t radiation dose-response curves for sunburn/erythema and suppression of the contact hypersensitivity
52 ent erythema, a more lateral distribution of erythema and telangiectasia, less neurogenic mast cell a
54 the cessation of spread of infection-related erythema and the absence of fever at 48 to 72 hours.
55 esponse (healing of erosions, improvement in erythema, and alleviation of pain), adverse effects, and
56 dema, lupus erythematosus [occurring twice], erythema, and dermohypodermitis all experienced by one [
59 nces in vaccination take rates, lesion size, erythema, and induration or in serum neutralizing-antibo
70 edema in mice, and reduced susceptibility to erythema arising from narrow-band 311-nm UVR in humans.
72 dpoint was a >/=20% reduction in the area of erythema at 48-72 hours in the intent-to-treat populatio
74 at the mid-point with continued reduction of erythema at the final (alpha = 0.075) time measurement.
76 ion level, which contrasts strongly with the erythema-based sun protection factors (mainly indicative
79 ction characterized by respiratory distress, erythema, decreased body temperature, unresponsiveness,
80 rate cutaneous inflammation characterized by erythema, dermal infiltrates of CD45(+) leukocytes, and
83 ( Fig 1 ), there was generalized periareolar erythema, dimpling, firmness, and fixation involving the
84 irradiated with UVB (three times, 1 minimal erythema dose (80 mJ/cm(2)), weekly) for 10 weeks, and e
85 ts and 11 controls were exposed to 1 minimum erythema dose (MED) of UVR delivered from Waldmann UV-6
86 -UVB) regimens start with 70% of the minimal erythema dose (MED) with 20% increments at each treatmen
87 s that measured time-related UVR in standard erythema dose (SED) and corresponding sun diaries (mean,
88 rotein changes in the skin after one minimal erythema dose of spectrally pure UVA1 (50 J cm(-2)) and
89 in vivo by looking at changes in the minimal erythema dose with subsequent doses of UV radiation.
90 ion, median (IQR) sunburn threshold (minimal erythema dose) was 28 (20-28) and 20 (20-28) mJ/cm(2) in
91 during the study was 39.1 (30.9) as standard erythema dose, comparable to a quarter of the median sum
92 lated radiation, ranging from 0 to 2 minimum erythema dose, on gluteal skin, with or without sunscree
97 exposures ranging from 0.65 to 3.9 standard erythema doses (SEDs), which were equivalent to 15-90 mi
98 n between our subjects' UVB and UVA1 minimal erythema doses implies that UVA1 and UVB erythema occur
100 nically important differences in tendency to erythema during a standard 70/20% NB-UVB twice-weekly re
101 Side effects, including the development of erythema during phototherapy, were similar for the two l
104 narof leads to compound-driven reductions in erythema, epidermal thickening, and tissue cytokine leve
108 common after intradermal administration (31% erythema for full subcutaneous dose and 77% for intrader
109 se events in this period were injection-site erythema (four [1%] with placebo, 22 [8%] with 50 mg sir
110 Pulsed-dye laser was used for pruritus and erythema; fractional CO2 laser was used for stiffness an
111 sis bullosa simplex with migratory circinate erythema (frameshift mutation c1649delG in the V2 domain
113 eria at follow-up visits: fever; increase in erythema (>25%), swelling, or tenderness (days 3-4); no
114 ommon adverse events were diarrhoea, rash or erythema, hepatic adverse events, and neutropenia (not r
116 Area Severity Index (IASI), which integrates erythema (IASI-E) and scaling (IASI-S); transepidermal w
117 ticipants (78%) in the umbrella group showed erythema in 1 or more sites vs 10 of the 40 participants
118 traviolet radiation, measured objectively as erythema in a sample of 12 body sites on 15 Northern Eur
120 nvestigated UV-induced sunburn apoptosis and erythema in mouse skin as a function of circadian time.
121 GE(2), PGF(2alpha), and PGE(3) accompany the erythema in the first 24-48 h, associated with increased
123 h no difference in AUC analysis for measured erythema index after a geometric series of 10 UVR doses.
124 xtensively evaluated in vivo as a measure of erythema induced in human skin and is expressed as Sun P
125 asured by dermal microdialysis and cutaneous erythema), induces an intense epidermal infiltrate of ma
127 of human diseases, including fifth disease (erythema infectiosum) in children and pure red cell apla
130 over 3 mo did not significantly reduce skin erythema, leukocyte infiltration, or eicosanoid response
131 aracterized by transient figurate patches of erythema, localized or generalized scaling, and frequent
133 lar pain (100%), sleep disturbances (75.3%), erythema migrans (59.7%), headache (46.8%), fatigue (44.
134 ri DNA in skin samples from 90 patients with erythema migrans (EM) and in synovial fluid (SF) from 63
135 ic-treated patients, 53% with culture-proven erythema migrans (EM) had IgG responses to recombinant g
136 fection causes an initial skin lesion called erythema migrans (EM) in human Lyme disease and in model
137 elia burgdorferi spreads in the skin to form erythema migrans (EM) lesions and then disseminates to o
138 hese inflammatory responses in patients with erythema migrans (EM) or Lyme arthritis (LA) to elucidat
139 l manifestation of early Lyme disease is the erythema migrans (EM) skin lesion that develops at the t
140 allel, analyzed B. burgdorferi isolates from erythema migrans (EM) skin lesions in 91 patients, and c
141 urgdorferi genotypes have been isolated from erythema migrans (EM) skin lesions in patients with Lyme
142 bjective was to obtain data on patients with erythema migrans (EM) who have symptoms/signs suggesting
143 ltiplex assays in the serum of patients with erythema migrans (EM), joint fluid of patients with Lyme
144 stage LD but is insensitive in patients with erythema migrans (EM), the most common manifestation of
145 n subjects with Lyme disease presenting with erythema migrans alone (n=36), erythema migrans with neu
146 symptoms was compared between patients with erythema migrans and 81 control subjects without a histo
147 confirmed early LD, based on the presence of erythema migrans and documentation of seroconversion or
148 course of oral doxycycline for treatment of erythema migrans and for a 14-day course for treatment o
149 5 consecutive patients with the diagnosis of erythema migrans and reported original data regarding th
150 variability in the clinical presentation of erythema migrans and the need to factor in multiple comp
152 sitive patients with Lyme disease-associated erythema migrans and were evaluated for an association w
153 chetemia explain why untreated patients with erythema migrans are at risk for dissemination of B. bur
156 from 17 patients who received a diagnosis of erythema migrans between 1991 and 2011 and who had 22 pa
157 ive for B. burgdorferi DNA in a patient with erythema migrans early during therapy and in a patient w
158 ent evidence supports treating patients with erythema migrans for no longer than 10 days when doxycyc
160 0-day doxycycline treatment in patients with erythema migrans has been assessed in the United States
161 ents with early Lyme disease associated with erythema migrans have a positive blood culture based on
164 Lyme disease as either a clinician-diagnosed erythema migrans lesion or a positive standard 2-tiered
165 4.2%; P = 0.006) and more often had multiple erythema migrans lesions (41.9% vs. 15.0%; P < 0.001) th
167 the frequency of nonspecific symptoms among erythema migrans patients was similar to that among cont
168 arious MTTT protocols in patients with acute erythema migrans ranged from 36% (95% confidence interva
170 9 (MMP-9) was selectively upregulated in the erythema migrans skin lesions of patients with acute Lym
171 afzelii, or Borrelia garinii recovered from erythema migrans skin lesions of patients with Lyme borr
172 patients with culture-confirmed episodes of erythema migrans to distinguish between relapse and rein
175 one of the 22 paired consecutive episodes of erythema migrans were associated with the same strain of
176 study assessed whether repeated episodes of erythema migrans were due to the same or different strai
177 Similarly, serum samples from patients with erythema migrans who were infected with the RST1 genotyp
179 esenting with erythema migrans alone (n=36), erythema migrans with neurological disease (n=12), and c
180 s with probable LD, based on the presence of erythema migrans without documented seroconversion or of
181 86 antibiotic-treated European patients with erythema migrans, 45 with post-Lyme symptoms and 41 with
183 cells from 27 patients with culture-positive erythema migrans, production of inflammatory cytokines p
186 n 11 (28%) of 39 of subjects presenting with erythema migrans, which increased to 50% at 4 weeks of f
203 City with early Lyme disease associated with erythema migrans; it is the largest number of borrelial
204 determined using serum from 55 patients with erythema migrans; specificity was determined using serum
205 s were well tolerated in the skin, with mild erythema, minimal wheal formation and complete resolutio
206 -independent, cascade of events resulting in erythema, mixed dermal infiltrate, and epidermal hyperpl
210 pothyroidism; grade 3 iridocyclitis, grade 1 erythema multiforme, and grade 3 erythema; and grade 2 i
211 eumonitis, localized myositis, folliculitis, erythema multiforme, or ophthalmological manifestations.
212 on-eczematous contact dermatitis include the erythema multiforme-like, the purpuric, the lichenoid, a
213 een administration routes for injection-site erythema (n=10 [12%] and n=0, respectively) and nausea (
214 ts in the vitespen group were injection-site erythema (n=158) and injection-site induration (n=153).
215 rface area and UVB exposure dose, related to erythema, necessary to achieve a given level of vitamin
216 ed oral ulcers (100%), genital ulcers (62%), erythema nodosum (46%), and papulopustular lesions (54%)
217 ease course (Pcombined = 5.94 x 10(-7)), and erythema nodosum (Pcombined = 2.27 x 10(-6)), respective
218 osition -308 was found to be associated with erythema nodosum in Caucasian sarcoidosis patients (stud
219 a gene adjacent to TNF, was associated with erythema nodosum in female Caucasian sarcoidosis patient
222 e is clinically valuable in the treatment of erythema nodosum leprosum (ENL) and multiple myeloma and
223 nts when evaluated before the onset of acute erythema nodosum leprosum (ENL) and persistently elevate
224 g thalidomide is the treatment of choice for erythema nodosum leprosum (ENL), an inflammatory cutaneo
228 associated with increased susceptibility to erythema nodosum leprosum in an allelic analysis, wherea
230 s "reactional states" (reversal reaction and erythema nodosum leprosum) that result in major clinical
231 lammatory and autoimmune disorders including erythema nodosum leprosum, Behcet's syndrome, discoid lu
232 an investigational agent in the treatment of erythema nodosum leprosum, oral ulcers, graft versus hos
233 wn role in inflammation were associated with erythema nodosum status in 659 sarcoidosis patients and
234 noted: fever, sialadenitis, lymphadenopathy, erythema nodosum, leukocytoclastic vasculitis, transient
238 ed erythema, the number of treatments before erythema occurred did not differ between skin types (P=0
239 e drug eruptions, viral exanthema, and toxic erythema of chemotherapy, Demodex folliculitis is a clin
240 tis was defined as either pus discharge with erythema of the abdominal skin or severe redness (>2 cm
242 ever, bilateral nonexudative conjunctivitis, erythema of the lips and oral mucosa, changes in the ext
244 (aOR 5.17, 95% CI 1.9-14.1), abdominal wall erythema or discolouration at presentation (aOR 2.51, 95
246 n with placebo included injection site pain, erythema, or both (21 [20%] of 107 vs seven [6%] of 110)
247 se (31% prevalence) was associated with skin erythema (P < 0.001); salivary dysfunction (11% prevalen
250 r Scale (VSS), which documents pigmentation, erythema, pliability, and height, and (2) University of
252 rate transient injection-site reactions (eg, erythema, pruritus) were the most frequent IMA901-relate
253 nal treatment, presence of serous discharge, erythema, purulent exudate, separation of the deep tissu
255 an increase in body temperature (fever) and erythema (rash) in comparison with humanized mice inocul
256 l (ie, patch-site) adverse events (including erythema, rash, pruritus, hyperpigmentation, pain, hypop
257 ed skin blood flow (Doppler velocimeter) and erythema (reflectance colorimeter a*) following topical
258 The sensitivity changes broadly followed the erythema response and did not extend beyond the irradiat
261 the dermatological sum score (DSS) assessing erythema, scaling, and plaque elevation on a 4-point sca
263 assessed, showing nonerosive lesions such as erythema, swelling, and lymphoid hyperplasia in 8 patien
264 ry card where any subjective adverse events (erythema, swelling, itching, headache, root hypersensiti
267 ng, or tenderness (days 3-4); no decrease in erythema, swelling, or tenderness (days 8-10); and more
270 Index for Ichthyosis Severity for scale and erythema that provides (1) written descriptions of the f
271 n skin disorder of follicular prominence and erythema that typically affects the proximal extremities
273 UV radiation were pigmentation, scaling, and erythema; the most frequent dermoscopic changes were inc
274 d, although more frequent infusion site pain/erythema/thrombophlebitis was seen with fosaprepitant re
276 ation (18 [44%]), xerosis (8 [20%]), scrotal erythema/ulceration (6 [15%]), and nail splinter hemorrh
284 By contrast, the interrater reliability for erythema was higher during in-person validation compared
285 tential genetic basis for alcohol-associated erythema was investigated as the function of polymorphis
288 eactions occurred in most patients, but only erythema was significantly more common in the VGX-3100 g
292 ptosis, inflammatory cytokine induction, and erythema were maximal following an acute early-morning e
295 tly responsible for the necrolytic migratory erythema, which resolves after amino acid administration
296 was commensurate with development of palatal erythema, which suggests a role for biofilm in the infla
297 ts, one from each group, reported persistent erythema, which was considered to be possibly related to
298 signs are skin lesions (necrolytic migratory erythema), while in subjects with inactivating mutations
299 blood flow without erythema and PG increased erythema with decreased skin blood flow, all as a functi
300 oM) each produced perceptible induration and erythema with moderate cellular infiltration resolving w
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