ライフサイエンスコーパス: erythrocyte

1 opy number (mean number of CR1 molecules per erythrocyte).

2 disease prevention and treatment beyond the erythrocyte.

3 tes modify their human host cell, the mature erythrocyte.

4 n diffusional loss of the inhibitor from the erythrocyte.

5 of which included both endothelial cells and erythrocytes.

6 an fibroblasts perfused with mouse and human erythrocytes.

7 he surface of Plasmodium falciparum-infected erythrocytes.

8 erentiating primary erythroblasts and mature erythrocytes.

9 ared with other reticulocyte populations and erythrocytes.

10 essential for the parasite's development in erythrocytes.

11 regulating placental CSA binding of infected erythrocytes.

12 with cell membranes in hemin-preconditioned erythrocytes.

13 '-dichlorodihydroflurescein contained inside erythrocytes.

14 kinase is critical for the invasion of host erythrocytes.

15 rvival of Plasmodium falciparum within human erythrocytes.

16 assay suitable for use with small numbers of erythrocytes.

17 physically adhered spheres to the surface of erythrocytes.

18 cies cause malaria by proliferating in human erythrocytes.

19 are established when merozoites invade human erythrocytes.

20 may lead to dehydration of PIEZO1-mutant HX erythrocytes.

21 essential for parasite survival within host erythrocytes.

22 te receptors to facilitate invasion of human erythrocytes.

23 the (18)O-isotope of CO2 and CA activity in erythrocytes.

24 egress, trapping merozoites within infected erythrocytes.

25 gy of the parasite during development inside erythrocytes.

26 d on some extra-arterial skeletal muscle and erythrocytes.

27 e detection by growth and replication within erythrocytes.

28 e MALDI-TOF analysis confirmed only SOD from erythrocytes.

29 apicomplexan parasites that replicate within erythrocytes.

30 aroxysmal nocturnal hemoglobinuria patient's erythrocytes.

31 e elements: sex, timing, origin, nausea, and erythrocytes.

32 of PLA2 but not LPCAT in human and mouse SCD erythrocytes.

33 vasion of Duffy-positive and -negative human erythrocytes.

34 pression of the Duffy blood group antigen on erythrocytes.

35 creased in vivo gamma-globin levels in their erythrocytes.

36 oduction and imbalanced redox homeostasis in erythrocytes.

37 igs (Cavia porcellus) lack CD59, at least on erythrocytes.

38 hat are expressed on the surface of infected erythrocytes.

39 e formation of a pore between merozoites and erythrocytes.

40 rm of malaria, Plasmodium falciparum, invade erythrocytes.

41 histolytica (Eh) trophozoites with ingested erythrocytes.

42 echanism of parasite killing within infected erythrocytes.

43 several in vitro tests and assays involving erythrocytes.

44 ility pathways (NPPs) in Plasmodium-infected erythrocytes.

45 ion was assessed by isotope incorporation in erythrocytes 14 d after the test meals.The lipid emulsio

46 by measuring the isotopic label abundance in erythrocytes 14 days after administration, and serum hep

47 These parasites invade erythrocytes, a complex process involving multiple ligan

48 falciparum, occurring when parasite-infected erythrocytes accumulate in the brain.

49 , mDCs incubated with P. falciparum-infected erythrocytes activated antigen-specific naive CD4(+) T c

50 t study was to investigate E2 levels and SOD erythrocyte activity in patients with age-related catara

51 Adenosine signalling via erythrocyte ADORA2B induces PKA phosphorylation, ubiquit

52 The main adverse effect of primaquine is erythrocyte age and dose-dependent acute haemolytic anae

53 onstrate in this study that histones promote erythrocyte aggregation, sedimentation, and using a nove

54 e of murine RBCs permits B cell responses to erythrocyte Ags and show that Siglec-mediated B cell tol

55 cations in the SPTAN1 gene, encoding the non-erythrocyte alphaII spectrin, have been associated with

56 based "self-associated molecular pattern" on erythrocytes also helps maintain neutrophil quiescence i

57 Erythrocyte and leukocyte zinc concentrations and zinc t

58 at perturb volume homeostasis jeopardize the erythrocyte and may lead to its premature destruction.

59 nd DHA from FFQs and also computed predicted erythrocyte and plasma scores directly from food intake

60 Secondary analyses using predicted erythrocyte and plasma scores of EPA and DHA yielded sli

61 fer, Nr4a1-deficient MPPS contribute more to erythrocyte and platelet populations than do wild-type M

62 die when prevented from escaping their host erythrocytes and because some Syk inhibitors have displa

63 d in iron metabolism and the phagocytosis of erythrocytes and blood-borne pathogens are significantly

64 Prior to infecting erythrocytes and causing malaria symptoms, Plasmodium pa

65 SOD1 levels were measured in erythrocytes and CSF.

66 and MERTK reduced efferocytosis of eryptotic erythrocytes and hematoma clearance, worsened neurologic

67 A block the changes in the deformability of erythrocytes and inhibit merozoite invasion by directly

68 strated by hemagglutination assay with human erythrocytes and intact virus, MERS-CoV Sia-binding acti

69 r 1 (AE1) mediates Cl(-)/HCO3(-) exchange in erythrocytes and kidney intercalated cells where it func

70 ATP is released from erythrocytes and platelets, and purinoceptors and ectonu

71 Sequestered parasitized erythrocytes and reduced uninfected red blood cell defor

72 ges of malarial infection, merozoites invade erythrocytes and replicate within a parasitophorous vacu

73 osemicarbazide reduced neutrophil binding to erythrocytes and restored neutrophil activation.

74 uned to generate functional blood vessels or erythrocytes and salvage ischemic tissue.

75 ditioning typically results in donor-derived erythrocytes and stable mixed chimerism of recipient-der

76 lic and dominant antibodies against infected erythrocytes and suggest that insertion of templated DNA

77 enables efficient removal of H2 antigen from erythrocytes and thereby accomplishes the conversion of

78 on nanoparticles, S1 or S1(A) bound to human erythrocytes and to human mucin in a strictly Sia-depend

79 ese complexes are found both in the infected erythrocyte, and within the parasite-derived compartment

80 ness, Trypanosoma brucei, with that of human erythrocytes, and identified glucose transport and glyce

81 s expressed by its merozoites, binds to host erythrocytes, and interferes with parasite growth.

82 orption in the duodenum, iron recycling from erythrocytes, and iron mobilization from the liver and i

83 ccumulation of protoporphyrins in the blood, erythrocytes, and other tissues.

84 ective erythropoiesis, reduced production of erythrocytes, anemia, and iron overload and PV by erythr

85 n of T-bet(+) B cells and production of anti-erythrocyte antibodies in ex vivo cultures of naive huma

86 n bioactive components but did not raise the erythrocyte antioxidant capacity.

87 esiculation occurring during splenic flow of erythrocytes are addressed via model simulations of RBC

88 Since erythrocytes are enucleated and lack DNA, genetic approa

89 ate that both in vivo and in vitro primitive erythrocytes are generated from hemogenic endothelial ce

90 The plexuses are vascular, carry erythrocytes, are enclosed within a basement membrane, a

91 ies, their hemolytic activity against murine erythrocytes, as well as their cytotoxicity against mamm

92 during parasite development, but fluxed into erythrocytes at the last minute of the parasite lifecycl

93 situ liver perfusion was performed using an erythrocyte-based perfusate.

94 lular localization and ability to bind mouse erythrocytes between the members of the cir family, whic

95 c deformations linked to the function of the Erythrocyte binding antigen family and P. falciparum ret

96 The parasite erythrocyte-binding antigen 175 (EBA175), a protein requ

97 The organism has two erythrocyte-binding protein families: namely, the Duffy-

98 These 2 tubes were tested with a new erythrocyte bulk-lysis protocol allowing acquisition of

99 embranes, preventing the rupture of infected erythrocytes but not parasitophorous vacuoles, and indep

100 essed in all cell types tested except mature erythrocytes, but do not reach the plasma membrane in er

101 ne skeleton maintains the biconcave shape of erythrocytes, but whether spectrins also determine the s

102 m DNA induces autoreactive responses against erythrocytes by activating a population of B cells expre

103 Purified LLMs invaded erythrocytes by an increase of 10-300 fold compared to w

104 substantial changes in the deformability of erythrocytes by binding to glycophorin A and activating

105 The asexual blood stage involves invasion of erythrocytes by merozoites, in which they grow and divid

106 Invasion of erythrocytes by Plasmodial merozoites is a composite pro

107 Invasion of erythrocytes by Plasmodium falciparum merozoites is nece

108 in vitro biochemical reaction controlled by erythrocytes CA and estimation of enzymatic activity of

109 71(neg)/RNA(low) ( approximately 0.55%), and erythrocytes CD71(neg)/RNA(neg) ( approximately 99%).

110 validated experimentally in a novel parasite-erythrocytes co-culture system.

111 els, mayonnaise was the leading predictor of erythrocyte concentrations of cis ALA and one isomer of

112 significantly reduced peritubular capillary erythrocyte congestion and improved histologic scores of

113 The erythrocyte contains a network of pathways that regulate

114 ggest that the P. falciparum- infected human erythrocyte contains numerous high molecular weight prot

115 Sl and McCoy polymorphisms did not influence erythrocyte CR1 clustering, and the effects of the Knops

116 f CD47 on a variety of cell types, including erythrocytes, creates a formidable antigen sink that may

117 175/GPA invasion pathway, we used an ex vivo erythrocyte culture system to decrease expression of GPA

118 ting a phosphorylation cascade that includes erythrocyte cytoskeletal proteins resulting in changes i

119 ow RhopH2 interacts with RhopH1, RhopH3, the erythrocyte cytoskeleton and exported proteins involved

120 egress and a gradual dismantling of the host erythrocyte cytoskeleton over the course of schizont dev

121 pe change due to the sudden breakdown of the erythrocyte cytoskeleton, before permeabilization and ev

122 Our data suggest a non-cell-autonomous erythrocyte defect secondary to the sphingolipid changes

123 All HNF1A(-/-) erythrocyte defects could be simulated by exposure of wi

124 The considerable erythrocyte defects in murine HNF1A deficiency encourage

125 Furthermore, histones impair erythrocyte deformability based on reduced passage of er

126 erogeneous, dominantly inherited disorder of erythrocyte dehydration.

127 yte recruitment to the liver during stressed erythrocyte delivery leads to kidney and liver damage.

128 d lack DNA, genetic approaches to understand erythrocyte determinants of malaria infection have histo

129 e, binding of P. falciparum parasites to the erythrocyte directly activate a signaling pathway throug

130 These erythrocytes display parasite proteins of the PfEMP1 fam

131 Abnormal sickle-shaped erythrocytes disrupt blood flow in small vessels, and th

132 ouse fetuses present misshapen and nucleated erythrocytes, due to impaired actin assembly and cytoske

133 P. falciparum exclusively infects human erythrocytes during clinical illness, and several natura

134 Loss of [(14)C]DADMe-ImmH from erythrocytes during multiple washes is slow and biphasic

135 in A, the most abundant sialoglycoprotein on erythrocytes, engaged neutrophil Siglec-9, a sialic acid

136 Erythrocyte equilibrative nucleoside transporter 1 (eENT

137 CFH had virtually no CA on (self-like) sheep erythrocytes (ES) but retained DAA.

138 Mature erythrocytes express CD47 isoform 2 only, with membrane

139 Because human erythrocytes express high levels of Glut1, take up DHA,

140 We measured erythrocyte FN3K concentrations and enzyme activity in a

141 The parasite makes initial contact with the erythrocyte followed by dramatic deformations linked to

142 lower in Galphai2(-/-) than in Galphai2(+/+) erythrocytes following ex vivo exposure to hyperosmotic

143 or quantitative estimation of CA activity in erythrocytes for clinical testing purposes.

144 y function as an entry pathway into infected erythrocytes for compounds that inhibit parasite egress.

145 r stress model, we show that histones induce erythrocyte fragility and lysis in a concentration-depen

146 density in replicate parallel cultures with erythrocytes from multiple different donors, across mult

147 Silverskin extracts protected erythrocytes from oxidative AAPH- and H2O2-induced hemol

148 ere more likely to interact with LILRB1 than erythrocytes from patients with non-severe malaria, alth

149 bout the effect of extracellular histones on erythrocyte function.

150 llular hypoxia-responsive biolipid promoting erythrocyte glycolysis, O2 delivery and thus new therape

151 compounds in protection against AAPH-induced erythrocyte hemolysis while (-)-epicatechin gallate, (-)

152 onize the protective function of FH in sheep erythrocyte hemolytic assays and increase cell-surface C

153 Secondary disorders of erythrocyte hydration include sickle cell disease, thala

154 ng variant alleles that worsen or ameliorate erythrocyte hydration.

155 exposure to hypoxia in mice and accounts for erythrocyte hypoxic memory and faster acclimatization.

156 ed to adhesion and sequestration of infected erythrocytes (IE) in deep vascular beds, but the endothe

157 tes the binding and accumulation of infected erythrocytes (IE) to blood vessels and tissues.

158 anism against Plasmodium falciparum-infected erythrocytes (IE); however, current phagocytosis assays

159 is caused by Plasmodium falciparum-infected erythrocytes (IEs) that surface-express VAR2CSA and bind

160 m falciparum merozoite antigens and infected erythrocytes (IEs), including functional immunity.

161 nd found to potently inhibit lysis of rabbit erythrocytes in assays of the alternative pathway while

162 Erythrocytes in patients with type-2 diabetes mellitus (

163 emonstrate a previously unsuspected role for erythrocytes in suppressing neutrophils ex vivo and in v

164 Loss of [(14)C]DADMe-ImmH from erythrocytes in the presence of excess unlabeled DADMe-I

165 six P. falciparum isolates growing in human erythrocytes in vitro for 4 years, with 279 clones sampl

166 plasma increased the osmotic fragility of WT erythrocytes in vitro.

167 We then compared erythrocyte incorporation of iron isotopic labels, chang

168 Iron absorption was measured as the erythrocyte incorporation of stable isotopes.

169 clude presumed roles for RhopH2 and CLAG3 in erythrocyte invasion but implicate a RhopH3 contribution

170 ion of gene products critical for growth and erythrocyte invasion of blood stage parasites.

171 anding of the molecular events that underpin erythrocyte invasion remains hampered by the short-perio

172 s identify a dual role for RhopH3 that links erythrocyte invasion to formation of the PSAC/NPP essent

173 Here we show that PMIX is essential for erythrocyte invasion, acting on rhoptry secretory organe

174 aluated 29 antigens likely to be involved in erythrocyte invasion, an essential developmental stage d

175 The series targets both merozoite egress and erythrocyte invasion, but crucially, also blocks transmi

176 g protein homolog 5 (PfRH5) is essential for erythrocyte invasion, is highly conserved among field is

177 After erythrocyte invasion, the intracellular pathogen must in

178 ficant role of the merozoite proteome during erythrocyte invasion, while identifying numerous unknown

179 -interacting protein (PfRipr), essential for erythrocyte invasion.

180 se two determinants in mediating Pfalciparum erythrocyte invasion.

181 ctional iron absorption was estimated by the erythrocyte iron incorporation method.Iron absorption wa

182 The uptake of solutes that use NPPs to enter erythrocytes is also reduced upon RhopH2 knockdown.

183 malaria parasite Plasmodium vivax to invade erythrocytes is dependent on the expression of the Duffy

184 , abundantly expressed aquaporin 1 (AQP1) in erythrocytes is thought not to be part of band 3 complex

185 Furthermore, P. falciparum-infected erythrocytes isolated from patients with severe malaria

186 (FADS) gene cluster have opposite effects on erythrocyte LCPUFAs at 9 mo.To explore whether SNPs in F

187 ells (PBMC) exposed to P. falciprum infected erythrocyte lysates.

188 ressing NK cells, a 12- and 7-fold increased erythrocyte lysis was observed with the IgG1 and IgG3, r

189 In human erythrocytes, malaria parasites perforate their enclosin

190 se findings suggest that histones binding to erythrocytes may contribute to the elevated erythrocyte

191 bound to nor translocated through the intact erythrocyte membrane during parasite development, but fl

192 ortant role for HNF1A in the preservation of erythrocyte membrane integrity, calcium homeostasis, and

193 tering of Complement Receptor 1 (CR1) in the erythrocyte membrane is important for immune-complex tra

194 Plasmodium falciparum erythrocyte membrane protein 1 (PfEMP1) mediates the bin

195 sera was observed, suggesting P. falciparum erythrocyte membrane protein 1 expression.

196 onally efficient coarse-grained model of the erythrocyte membrane reveals that restructuring and cons

197 malaria parasites perforate their enclosing erythrocyte membrane shortly before egress.

198 l lattice structure, resembling the expanded erythrocyte membrane skeleton structure, in the somatode

199 spectrin/actin-binding peptide critical for erythrocyte membrane stability.

200 permeabilization and eventual rupture of the erythrocyte membrane to release the parasites.

201 EM, is distinct from that in the surrounding erythrocyte membrane, with a structure at the apex that

202 We reported an erythrocyte membrane-coated nanogel (RBC-nanogel) system

203 ith Kd = 14 nM; (iii) binding of cdb3-PO4 to erythrocyte membranes is inhibited both by antibodies ag

204 throcytic merozoites and the inner aspect of erythrocyte membranes, preventing the rupture of infecte

205 ss) by sequential rupture of the vacuole and erythrocyte membranes.

206 ymorphisms might influence CR1 clustering on erythrocyte membranes.

207 solated cdb3-PO4 (but not cdb3) to band 3 in erythrocyte membranes; and (v) phosphorylation-induced b

208 but significantly less toxicity toward human erythrocytes (MIC = 1-4 mug/mL and HC50 = 805-1242 mug/m

209 e up DHA, and reduce it to VC, we tested how erythrocytes might impact high dose VC therapies.

210 samples showed a significant improvement in erythrocyte morphology compared to untreated controls.

211 ibed that stabilizes leukocyte viability and erythrocyte morphology in whole blood under ambient stor

212 high concentrations a pro-oxidant effect on erythrocyte morphology was observed.

213 To stabilize erythrocyte morphology, a preservative solution was form

214 hibitors killed trypanosomes without killing erythrocytes, neurons or liver cells.

215 action of this parasite ligand with the host erythrocyte occurs through its two regions present at am

216 Plasmodium spp. target erythrocytes of different ages, but share a common mecha

217 ned, and compared with those of enzymes from erythrocytes of hens and SOD standard.

218 323%) at any given FN3K protein level in the erythrocytes of the negative-GGap group compared with th

219 hy performed with (99m)Tc-labeled autologous erythrocytes or historically with (99m)Tc-sulfur colloid

220 in (Glc-Insulin) and glucose transporters on erythrocytes (or red blood cells, RBCs) membrane.

221 of alpha-globin chains, which contribute to erythrocyte pathology.

222 aughter merozoites, which in turn invade new erythrocytes perpetuating the cycle responsible for mala

223 c signaling is involved in the physiology of erythrocytes, platelets, and leukocytes.

224 exclusively myeloid lineage cells, including erythrocytes, platelets, monocytes, and neutrophils.

225 single cell expression data of T2EC chicken erythrocytes pointed to BATF as a candidate novel regula

226 during clinical illness, and several natural erythrocyte polymorphisms are protective against severe

227 resulting in the physical penetration of the erythrocyte, powered by the parasite's actinomyosin-base

228 four main cell types: duodenal enterocytes, erythrocyte precursors, macrophages, and hepatocytes.

229 uisite multipotent or bipotent megakaryocyte-erythrocyte progenitor stage.

230 ocusing on trajectories toward megakaryocyte-erythrocyte progenitors and lymphoid-primed multipotent

231 underwent baseline laboratory testing, total erythrocyte protoporphyrin (ePPIX) testing, and molecula

232 uble transferrin receptor (sTfR), as well as erythrocyte protoporphyrin.

233 Invasion of immunologically privileged erythrocytes provides a relatively protective niche as w

234 Thus, in human erythrocytes, rebinding of DADMe-ImmH is 50-fold more li

235 parasite growth and the interaction with its erythrocyte receptor basigin is essential for invasion.

236 re, we have identified basigin as the second erythrocyte receptor for PvTRAg38, which is resistant to

237 ntified band 3 as the chymotrypsin-sensitive erythrocyte receptor for the P4 region, but the other re

238 the GPC receptor, supporting a hierarchy of erythrocyte receptor usage in P. falciparum.

239 Each region recognizes its own erythrocyte receptor.

240 In conclusion, PvTRAg38 binds to two erythrocyte receptors basigin and band 3 through P2 and

241 Specific engagement of erythrocyte receptors defines target cell tropism, activ

242 wn as invasion ligands that bind to specific erythrocyte receptors to facilitate invasion of human er

243 nemia-responsive enhancers are essential for erythrocyte regeneration in stress erythropoiesis, a vit

244 cell factor/c-Kit signaling, which promotes erythrocyte regeneration.

245 expansion of a multigene family involved in erythrocyte remodelling, and show that a short region on

246 Here, we use invasion inhibition assays, erythrocyte resealing and high-definition imaging to exp

247 Morphologically, HNF1A(-/-) erythrocytes resembled acanthocytes and displayed increa

248 cine brain, human kidney, lungs, plasma, and erythrocytes resulting in unambiguous identification of

249 te of the spectrin-based cytoskeleton to the erythrocyte's lipid bilayer and thereby contributes crit

250 Here, we report that increased erythrocyte S1P binds to deoxygenated sickle Hb (deoxyHb

251 d trans ALA isomers (0.05% +/- 0.01%) in 395 erythrocyte samples collected in 1989-1990.

252 Participants with IBD-AD showed higher erythrocyte sedimentation rate (ESR), C-reactive protein

253 atory markers (white blood cell [WBC] count, erythrocyte sedimentation rate [ESR], C-reactive protein

254 Laboratory work-up revealed an erythrocyte sedimentation rate of 58 mm/h (reference ran

255 ng hormone level, autoimmune antibody level, erythrocyte sedimentation rate, and C-reactive protein l

256 f the type of uveitis (complete blood count, erythrocyte sedimentation rate, C-reactive protein, tube

257 The second best marker-erythrocyte sedimentation rate-improved the area under t

258 erythrocytes may contribute to the elevated erythrocyte sedimentation rates observed in inflammatory

259 Infected erythrocyte sequestration in the microvasculature plays

260 During Plasmodium falciparum infections, erythrocyte-stage parasites inhibit dendritic cell matur

261 The serological conservation of infected erythrocytes strongly correlated with the expression of

262 This study explores how WZB117 inhibits the erythrocyte sugar transporter glucose transport protein

263 We discovered that erythrocytes suppressed neutrophil activation ex vivo an

264 y imply that the malaria parasite primes the erythrocyte surface through its binding antigens, alteri

265 c modification of sialic acid side chains on erythrocyte surfaces with mild sodium metaperiodate oxid

266 Developing erythrocytes take up exceptionally large amounts of iron

267 The experimental treatments accelerated erythrocyte telomere attrition and increased DNA damage

268 d enhanced phagocytosis of parasite-infected erythrocytes than those in Cd36(-/-) mice in an IFN-gamm

269 te deformability based on reduced passage of erythrocytes through an artificial spleen.

270 ults identify the efferocytosis of eryptotic erythrocytes through AXL/MERTK as a critical mechanism m

271 This allows the erythrocyte to maintain a narrow range of cell hemoglobi

272 usative agents of malaria, modify their host erythrocyte to render them permeable to supplementary nu

273 he binding of Plasmodium falciparum-infected erythrocytes to placental chondroitin sulfate A (CSA) th

274 d be simulated by exposure of wild-type (WT) erythrocytes to sphingosine in vitro and attributed in p

275 protein C receptor (EPCR), allowing infected erythrocytes to synergistically bind both receptors.

276 hat is initiated by cytoadhesion of infected erythrocytes to the brain vasculature, followed by ruptu

277 rate hydroperoxide oxidized Prx2 from intact erythrocytes to the same extent as hydrogen peroxide.

278 We conducted GWAS meta-analyses of six erythrocyte traits in 71,638 individuals from European,

279 ion studies (GWASs) have identified loci for erythrocyte traits in primarily European ancestry popula

280 We identified seven loci for erythrocyte traits including a locus (RBPMS/GTF2E2) asso

281 action as erythrocytic (also associated with erythrocyte traits) or glycemic (associated with other g

282 ional and low sulfite wines on ex vivo human erythrocytes under oxidative stimulus by the cellular an

283 s also suggest pathways for further study of erythrocyte vesiculation that point to the criticality o

284 assette family member, in the maintenance of erythrocyte volume homeostasis.

285 Understanding the pathways regulating erythrocyte water and solute content may reveal innovati

286 copy to define biophysical properties of the erythrocyte, we show that EBA175 binding to GPA leads to

287 n the ability of P. vivax to bind Duffy-null erythrocytes, we analyzed P. vivax parasites obtained fr

288 vitro ATP release from endothelial cells or erythrocytes were assessed by a luciferin-luciferase ass

289 d, MTRAP is essential for gamete egress from erythrocytes, where it is necessary for the disruption o

290 ntial role for signaling pathways within the erythrocyte, which might alter red cell biophysical prop

291 ointly ensured a high yield of valid carrier erythrocytes, which further successfully delivered RNA i

292 Prior studies in erythrocytes-which express particularly high levels of G

293 in complement is remodeled, as epitomized by erythrocytes, whose cytosol is 98% globin.

294 hat recognize I/i carbohydrates expressed by erythrocytes with a specific motif in their framework re

295 constants of 115 to 6 pm Treatment of human erythrocytes with DADMe-Immucillin-H (DADMe-ImmH, 22 pm)

296 ied the mechanism by which the engulfment of erythrocytes with exposed phosphatidylserine directly mo

297 id disturbances in several tissues including erythrocytes with the accumulation of sphingosine as the

298 odies that dominate the response to infected erythrocytes without conferring enhanced protection agai

299 e low G6PD activity in the majority of their erythrocytes, yet are usually reported as G6PD "normal"

300 d AChE solubilized from 8 mL of frozen human erythrocytes, yielding a quantity sufficient for detecti