ライフサイエンスコーパス: erythroleukemia

1 ythroid progenitors and eventually promoting erythroleukemia.

2 t to the development of Friend virus-induced erythroleukemia.

3 e for the resistance to Friend virus-induced erythroleukemia.

4 r the constitutively activated mutant caused erythroleukemia.

5 with GATA1 function, thereby contributing to erythroleukemia.

6 irus in check and developed fatal FV-induced erythroleukemia.

7 dysregulation of PU.1 expression can lead to erythroleukemia.

8 o directly address the role of p53 in Friend erythroleukemia.

9 y also have possible therapeutic utility for erythroleukemia.

10 ration of erythroid progenitors and inducing erythroleukemia.

11 also be a useful marker for the diagnosis of erythroleukemia.

12 ensued, most commonly lymphoid leukemia and erythroleukemia.

13 of the Moloney virus from T-cell leukemia to erythroleukemia.

14 d to the erythroid lineage, and induction of erythroleukemia.

15 xplanation of v-ErbA activity in AEV-induced erythroleukemia.

16 trongly associated with Friend virus-induced erythroleukemia.

17 ring the progression of Friend-virus-induced erythroleukemia.

18 forming virus (SFFV) results in a multistage erythroleukemia.

19 F1IP and MLF1 deregulation in the genesis of erythroleukemias.

20 globin but not beta-globin; transgenic mouse erythroleukemia 585 cells express predominantly human be

21 Fv-2 resistance to Friend virus-induced erythroleukemia acts through nonimmunological mechanisms

22 actor PU.1, an oncogene implicated in murine erythroleukemia, acts to functionally cross-antagonize o

23 those from mice susceptible to SFFV-induced erythroleukemia also express a short form of the recepto

24 ll mice exhibited accelerated progression to erythroleukemia and accelerated death following diagnosi

25 elated factor 2 (Nrf2) pathway in K562 human erythroleukemia and other cell types after treatment wit

26 ith AGI-6780 induced differentiation of TF-1 erythroleukemia and primary human acute myelogenous leuk

27 t in a broad range of malignancies including erythroleukemia and solid tumors.

28 lase 1 (HDAC1), and histone deacetylase 2 in erythroleukemia and T cell leukemia cells.

29 e exhibit decreased susceptibility to Friend erythroleukemia and that expansion of erythroid progenit

30 utes to both early and late stages of Friend erythroleukemia and that persistence of F-gp55 expressio

31 etion in Gab2 are less susceptible to Friend erythroleukemia and the expansion of erythroid progenito

32 s are >/= 20%, the disorder is classified as erythroleukemia, and when BM blasts are < 20%, as myelod

33 Further, the data demonstrate that erythroleukemias arising in Friend virus-infected p53 nu

34 ne leukemia virus (F-MuLV) in F-MuLV induced erythroleukemia, as well as that of the 10A1 and Graffi

35 ression may be required not only to initiate erythroleukemia but to also maintain erythroleukemia fol

36 ce does not independently prevent FV-induced erythroleukemia but works in concert with the immune sys

37 severe Friend virus-induced splenomegaly and erythroleukemia by 6 to 8 weeks postinfection.

38 Here, we use a mouse erythroleukemia cell (MEL) system for studying how repet

39 ressed differentially before and after mouse erythroleukemia cell (MELC) induction.

40 oter during dimethyl sulfoxide-induced mouse erythroleukemia cell differentiation.

41 ty (3.1 nM) and specificity similar to mouse erythroleukemia cell HS2NF5.

42 ctivation function, we cotransfected a human erythroleukemia cell line (K562) with a locus control re

43 In non-B cell lines, like the murine erythroleukemia cell line (MEL), the most distal IgH con

44 amma-globin promoter complexes in K562 human erythroleukemia cell line and primary human fetal and ad

45 Using a human erythroleukemia cell line and primary murine BM cells, w

46 This segment can be excised from an avian erythroleukemia cell line by restriction enzyme digestio

47 These segments were excised from an avian erythroleukemia cell line by restriction enzyme digestio

48 When transfected into the erythroleukemia cell line Dami, promoter-luciferase cons

49 In the erythroleukemia cell line Dami, transfected promoter-luc

50 ylation of sf-Stk can also be detected in an erythroleukemia cell line derived from an SFFV-infected

51 e factors in the proliferation of the murine erythroleukemia cell line HCD57.

52 f JunB in the erythropoietin (EPO)-dependent erythroleukemia cell line HCD57.

53 livery of this transcription factor into the erythroleukemia cell line K562 resulted in an increase o

54 ated proapoptotic activity against the human erythroleukemia cell line K562.

55 romatin interactions for TFII-I in the human erythroleukemia cell line K562.

56 on to a 2-D gel separation of the same human erythroleukemia cell line lysate, the IEF-NP RP HPLC pro

57 inal erythroid differentiation of the murine erythroleukemia cell line MEL, whereas its overexpressio

58 screening, and lack of functional effects in erythroleukemia cell line TF-1 and CD34(+) progenitor ce

59 ony-stimulating factor (GM-CSF) in the human erythroleukemia cell line TF-1.

60 btraction cDNA library approach from a mouse erythroleukemia cell line that had been induced to polyp

61 detected in cells of the K562 line, a human erythroleukemia cell line, and in CD34+ primary human he

62 However, a mouse erythroleukemia cell line, CB3, does not express hnRNP A

63 In TF-1 cells, an erythroleukemia cell line, granulocyte-macrophage colony

64 er stable plasmid transfection of the murine erythroleukemia cell line, MEL585.

65 In an erythroleukemia cell line, TF-1, high levels of p105/p50

66 vely expressed in the factor-dependent human erythroleukemia cell line, TF1.

67 In contrast, studies with the K562 human erythroleukemia cell line, which is often used for compa

68 s well as EPO protein in K562 cells, a human erythroleukemia cell line.

69 In contrast, the growth of erythroleukemia cell lines derived from Friend murine le

70 e JNK inhibitor blocked the proliferation of erythroleukemia cell lines derived from these mice.

71 ulation of PU.1 transcription in established erythroleukemia cell lines differed depending upon the l

72 ocytes from Friend SFFV-infected mice and in erythroleukemia cell lines from Friend MuLV-infected mic

73 ed mice but did not alter AP1 DNA binding in erythroleukemia cell lines from Friend SFFV-infected mic

74 Studies of cultured mouse erythroleukemia cell lines indicate that one aspect of P

75 Two human erythroleukemia cell lines, HEL and K562, and a human pr

76 We have established an erythroleukemia cell model to study how Notch regulates

77 n erythrocyte membranes and from K562 (human erythroleukemia) cell membranes, has robust peptidylprol

78 fR IgG3-Av) inhibits the proliferation of an erythroleukemia-cell line.

79 in undifferentiated and differentiated human erythroleukemia cells (HEL) using SEEL using the sialylt

80 We have previously shown that erythroleukemia cells (K562) transfected with vascular a

81 surface coreceptor for entry, we used human erythroleukemia cells (K562), which allow parvovirus B19

82 cell-mediated chromosome transfer into human erythroleukemia cells (K562).

83 lyzed the responses of HeLa cells and murine erythroleukemia cells (MELC) to hexamethylene bisacetami

84 tation assays and minigene-transfected mouse erythroleukemia cells (MELCs).

85 We have purified an mSin3A complex from K562 erythroleukemia cells and identified three new mSin3A-as

86 g directly and specifically to HS2 in living erythroleukemia cells and in mouse fetal liver.

87 s, into a defined chromosomal site in murine erythroleukemia cells and monitored the stability of the

88 using DNA-protein binding studies and human erythroleukemia cells and promoter activity using lucife

89 close to 100 % of negatively selected mouse erythroleukemia cells and ranges from 10 to 50 % in embr

90 of CR2 were expressed on the surface of K562 erythroleukemia cells and their binding ability assessed

91 CReP was also required for exocytosis from erythroleukemia cells and thus appears to play a broader

92 Attachment of B. burgdorferi N40 to human erythroleukemia cells and to human saphenous vein endoth

93 ogenous enzyme or cocultured with human K562 erythroleukemia cells as an exogenous source of TPI.

94 With the use of DS19 erythroleukemia cells as model particles with frequency-

95 Induced overexpression of PU.1 in K562 erythroleukemia cells blocks hemin-induced erythroid dif

96 n of megakaryocytic differentiation of human erythroleukemia cells by 12-O-tetradecanoylphorbol-13-ac

97 er transfer of gamma globin genes into mouse erythroleukemia cells can be used for the analysis of re

98 To address this issue, we used hybrid murine erythroleukemia cells containing a single copy of human

99 In differentiated ES cells and erythroleukemia cells containing the LCR-deleted chromos

100 d (65)Zn uptake activity in transfected K562 erythroleukemia cells expressing hZip2 from the CMV prom

101 ude that two independent pathways operate in erythroleukemia cells for nitric oxide-mediated protecti

102 Recombinant OPN and BSP can protect murine erythroleukemia cells from attack by human complement as

103 In our preliminary study, we found that K562 erythroleukemia cells have an extremely low level of end

104 astoma cell line was cultured with the human erythroleukemia cells IA lacking PrP(C).

105 hSK1(-)(9b) is especially abundant in human erythroleukemia cells in culture.

106 onstructs transfected into NIH 3T3 and mouse erythroleukemia cells indicated that the housekeeping pr

107 cetylation increased significantly in murine erythroleukemia cells induced to differentiate in cultur

108 ADNP or ADNP2 in zebrafish embryos or mouse erythroleukemia cells inhibited erythroid maturation, wi

109 -globin gene expression is evident in murine erythroleukemia cells lacking the p45 subunit of NF-E2.

110 and FTE/S3a from lysates of Rauscher murine erythroleukemia cells overexpressing both proteins.

111 , we examined (65)Zn uptake activity in K562 erythroleukemia cells overexpressing hZIP1.

112 e expression of the -90 beta-ZF-DBD in mouse erythroleukemia cells reduced the binding of KLF1 with t

113 d chemosensitization was observed on K562/R7 erythroleukemia cells resistant to doxorubicin, especial

114 rgeted introduction of this allele into K562 erythroleukemia cells results in a proliferation defect

115 K562 erythroleukemia cells stably transfected with constructs

116 x transcript is expressed at lower levels in erythroleukemia cells than reticulocytes.

117 A large panel of mouse erythroleukemia cells that bear a single copy of integra

118 TNF-alpha expression in the proliferation of erythroleukemia cells that is distinct from the effect o

119 nous Sias on transfected cells, and by using erythroleukemia cells to allow experimental manipulation

120 talin synthesis enhances sensitivity of K562 erythroleukemia cells to CDC, whereas overexpression of

121 study, we used nuclear extracts from murine erythroleukemia cells to purify a protein complex that b

122 hese compounds also induced HL-60 and murine erythroleukemia cells to undergo partial differentiation

123 have now analyzed laminin binding to murine erythroleukemia cells transfected with various human B-C

124 K562 erythroleukemia cells treated with the HbF inducers hemi

125 Mouse erythroleukemia cells treated with the phosphorothioate

126 ntegration into three defined loci in murine erythroleukemia cells using recombinase-mediated cassett

127 st extracellular loop of the P2Y(2)R to K562 erythroleukemia cells was inhibited by antibodies agains

128 We also show that AEV-transformed erythroleukemia cells were resistant to TGF-beta-induced

129 Finally, transfection of murine erythroleukemia cells with human B-CAM cDNA induces bind

130 megakaryocytes (and megakaryocyte-like human erythroleukemia cells), a regulatory role in cellular de

131 w levels of p53 (HeLa cells) or no p53 (K562 erythroleukemia cells).

132 The adhesion of K562 erythroleukemia cells, a cell line expressing a single f

133 activated K+ channels of human erythrocytes, erythroleukemia cells, and ferret vascular smooth muscle

134 gulatory protein 2, in iron-deficient murine erythroleukemia cells, and in human patients with ISCU m

135 tial for erythroid differentiation of murine erythroleukemia cells, and serine/threonine phosphorylat

136 e mRNA was unchanged in iron-depleted murine erythroleukemia cells, and the stability of mature ferro

137 3 accelerates differentiation of both murine erythroleukemia cells, as well as fetal liver cells, whe

138 ene in both normal erythroid progenitors and erythroleukemia cells, as well as in macrophages.

139 thyl sulfoxide treatment of wild-type murine erythroleukemia cells, but not a mutant clone of dimethy

140 to the LCR occurs in undifferentiated murine erythroleukemia cells, but phosphorylation of LCR-associ

141 syngeneic fully autochthonous system (FBL-3 erythroleukemia cells, C57BL/6 mice, and highly purified

142 cytosine methyltransferase isoform in mouse erythroleukemia cells, Dnmt1, exhibits potent dead-end i

143 e assessed in stable transfectants of murine erythroleukemia cells, in which the activities of cyclin

144 on and reduced with RUNX1 knockdown in human erythroleukemia cells, indicating that PCTP is regulated

145 is required for beta-globin transcription in erythroleukemia cells, induces histone H3 hyperacetylati

146 In K562 human erythroleukemia cells, knockdown of LPA2 enhanced erythr

147 also p53 mutants, such as p53 M133K in human erythroleukemia cells, leading to pathologic gene expres

148 In luciferase reporter studies in human erythroleukemia cells, mutation of each site decreased a

149 During differentiation of mouse erythroleukemia cells, protein levels of KCCs paralleled

150 3-integrin gene transcription in human K-562 erythroleukemia cells, Raf activation in NIH 3T3 cells l

151 in vitro erythroid differentiation in murine erythroleukemia cells, resulting in phenotypical cross-c

152 In murine erythroleukemia cells, the endogenous Lbd1 and LMO2 prot

153 In murine erythroleukemia cells, UNC0638 and Ehmt2 CRISPR/Cas9-med

154 In K562 erythroleukemia cells, we have identified four alternati

155 ) cDNA transfected into HeLa cells and mouse erythroleukemia cells, where it was expressed in the con

156 Unlike SFFV-derived erythroleukemia cells, which depend on PU.1 expression f

157 formulate a metabolic model relevant to K562 erythroleukemia cells.

158 nit-erythroid-derived erythroblasts and TF-1 erythroleukemia cells.

159 of occupancy during differentiation of mouse erythroleukemia cells.

160 y complementation analysis in NF-E2-null CB3 erythroleukemia cells.

161 lacking hnRNP A1 was purified from CB3 mouse erythroleukemia cells.

162 to induce differentiation of cultured murine erythroleukemia cells.

163 a-globin promoter in undifferentiated murine erythroleukemia cells.

164 ared acetylation of the locus in MEL and CB3 erythroleukemia cells.

165 NA) is up-regulated after induction of mouse erythroleukemia cells.

166 th p50(cdc37) were detected in cultured K562 erythroleukemia cells.

167 inducing differentiation of cultured murine erythroleukemia cells.

168 rexpression enhances hemoglobin synthesis in erythroleukemia cells.

169 ) targeting this site (+60 ZF-DBD) in murine erythroleukemia cells.

170 substantial effect on toxicity toward human erythroleukemia cells.

171 ar extracts derived from hematopoietic human erythroleukemia cells.

172 ubregions of the LCR in human K562 and mouse erythroleukemia cells.

173 -geo reporter at three genomic sites in K562 erythroleukemia cells.

174 randomly chosen locus in the genome of mouse erythroleukemia cells.

175 uitment to the beta-globin promoter in mouse erythroleukemia cells.

176 pression was also demonstrated in K562 human erythroleukemia cells.

177 s an Epo upregulated gene in Rauscher murine erythroleukemia cells.

178 nce involved in inducible AQP1 regulation in erythroleukemia cells.

179 en characterized in hemin-treated human K562 erythroleukemia cells.

180 ted for the native MTase derived from murine erythroleukemia cells.

181 NK cell-susceptible targets, including K562 erythroleukemia cells.

182 and exon 1B to the proximal 3' ss in murine erythroleukemia cells.

183 hibitor JQ1 induced differentiation of mouse erythroleukemia cells.

184 etic cell lines, including 32DWT18 and human erythroleukemia cells.

185 ation of the virus-transformed cells, murine erythroleukemia cells.

186 ineered ribonuclease, is also toxic to human erythroleukemia cells.

187 after site-directed integration into murine erythroleukemia cells.

188 Runx1-regulated repression element in murine erythroleukemia cells.

189 in cell proliferation and/or the survival of erythroleukemia cells.

190 o search for Tal1/SCL target regions in K562 erythroleukemia cells.

191 ospho-STAT5; (iii) induce apoptosis in human erythroleukemia cells; and (iv) suppress pathologic cell

192 ith 10% to < 20% BM blasts from TNCs fulfill erythroleukemia criteria; however, by considering blasts

193 single-cell clones derived from HFV-infected erythroleukemia-derived cells (H92), there were up to 20

194 s a Stat3 target gene in the early stages of erythroleukemia development.

195 nitiate erythroleukemia but to also maintain erythroleukemia following Friend virus infection.

196 ally significant differences between NBM and erythroleukemia gene expression.

197 in vivo progression of Friend virus-induced erythroleukemia has been suggested but not clearly defin

198 nes, the mechanism by which gp55-A initiates erythroleukemia has remained a mystery.

199 re found with the JAK2(V617F)-positive human erythroleukemia HEL cell line.

200 Human erythroleukemia HEL cells showed AQP1 transcript express

201 -myristate 13-acetate (PMA)-stimulated human erythroleukemia (HEL) and CHRF 288-11 cells, which have

202 ed regulation of the serglycin gene in human erythroleukemia (HEL) and CHRF 288-11 cells, which have

203 iferation of the Jak2-V617F expressing human erythroleukemia (HEL) cell line by promoting marked cell

204 When the human erythroleukemia (HEL) cell line is induced to differenti

205 he cellular action of NO, we inhibited human erythroleukemia (HEL) cell-surface PDI expression using

206 Platelets and human erythroleukemia (HEL) cells also contained ER beta and A

207 ivation of endogenous alphaIIbbeta3 in human erythroleukemia (HEL) cells and beta1 integrin activatio

208 We demonstrate in both human erythroleukemia (HEL) cells and primary human CD34(+) he

209 d in bipotential human cells (K562 and human erythroleukemia (HEL) cells), proerythroblastic mouse (M

210 Here, we used human platelets and human erythroleukemia (HEL) cells, which express integrin alph

211 rinostat-treated, JAK2V617F-expressing human erythroleukemia (HEL) cells.

212 f two independent cell lines, K562 and human erythroleukemia (HEL).

213 s of the cultured JAK2V617F-expressing human erythroleukemia HEL92.1.7 and Ba/F3-JAK2V617F cells.

214 lobin locus in a human fetal liver and mouse erythroleukemia hybrid cell (A181gamma cell) that contai

215 s of the spleen focus-forming virus initiate erythroleukemia in adult mice.

216 e and -resistant mice but is unable to cause erythroleukemia in Fv-2-resistant mice.

217 leen focus-forming virus (SFFV) causes rapid erythroleukemia in mice due to expression of its unique

218 Friend virus induces the development of erythroleukemia in mice through the interaction of a vir

219 ng the initial stage of Friend virus-induced erythroleukemia in mice, interaction of the viral protei

220 Friend virus induces an erythroleukemia in susceptible mice that is initiated by

221 cute erythroid hyperplasia and eventually to erythroleukemia in susceptible strains of mice.

222 disease is essential for the progression of erythroleukemia in the presence of differentiation signa

223 n is a requirement for progression of Friend erythroleukemia in vivo.

224 ssion of PU.1 in erythroid precursors causes erythroleukemias in mice.

225 by proviral insertion or transgenesis causes erythroleukemias in mice.

226 The erythroleukemia induced by Friend virus is a multistage

227 Erythroleukemia induced by the anemia strain of Friend v

228 we replaced the U3 region of the LTR of the erythroleukemia-inducing Friend murine leukemia virus (F

229 The erythroleukemia-inducing Friend spleen focus-forming vir

230 The erythroleukemia-inducing Friend spleen focus-forming vir

231 The erythroleukemia-inducing Friend spleen focus-forming vir

232 rget cells, in vivo, cooperate to accelerate erythroleukemia induction.

233 ted mice, but caused a fatal, transplantable erythroleukemia instead.

234 Susceptibility to SFFV-induced erythroleukemia is conferred by the Fv-2 gene, which enc

235 positive) from shotgun analyses of the human erythroleukemia K562 cell line.

236 )-induced cytotoxicity in a subline of human erythroleukemia K562 cells (K/VP.5) and in K/VP.5 cells

237 We showed previously that human erythroleukemia K562 cells are resistant to antineoplast

238 Erythroid differentiation of human erythroleukemia K562 cells by hemin simultaneously incre

239 oduction was induced by glutamate, and human erythroleukemia K562 cells in which H(2)O(2) production

240 rmore, we detected the 2.8-kb PDGF-B mRNA in erythroleukemia K562 cells upon 12-O-tetradecanoylphorbo

241 arabine (Ara C)-dependent differentiation of erythroleukemia K562 cells, we observed effects that ind

242 Incubation of the RNA with erythroleukemia K562 cytosolic extract results in deaden

243 , ZFP161) and measure reporter expression in erythroleukemia (K562) and liver carcinoma (HepG2) cell

244 of LEF to induce differentiation of a human erythroleukemia (K562) cell line and show that LEF induc

245 In human erythroleukemia (K562) cells, the highly related protein

246 n in both the DHL-4 B cell line and the K562 erythroleukemia line.

247 outs via CRISPR/Cas9 using the immortal JK-1 erythroleukemia line.

248 TfR2-alpha mRNA were significantly higher in erythroleukemia (M6) marrow samples than in nonmalignant

249 leukemia, suggesting that a key mechanism in erythroleukemia may be the collaboration of lesions dist

250 (SF2/ASF) expression in differentiated mouse erythroleukemia mediates a differentiation stage-specifi

251 sor mSin3A is associated with TAL1 in murine erythroleukemia (MEL) and human T-ALL cells.

252 d for production of stably transfected mouse erythroleukemia (MEL) cell clones and pools.

253 Herein we have derived stable Friend mouse erythroleukemia (MEL) cell clones expressing either Mfrn

254 cus before and after the induction of murine erythroleukemia (MEL) cell differentiation.

255 physically interacts with Mfrn1 during mouse erythroleukemia (MEL) cell differentiation.

256 erentiation block and can be grown as murine erythroleukemia (MEL) cell lines.

257 NF-related complex (PYR complex) from murine erythroleukemia (MEL) cell nuclear extract that binds py

258 ide (HMBA)-induced differentiation of murine erythroleukemia (MEL) cells and blocked differentiation;

259 y pathway is recapitulated in cultured mouse erythroleukemia (MEL) cells and targets nonsense-free mR

260 Murine erythroleukemia (MEL) cells are a model system to study

261 Friend murine erythroleukemia (MEL) cells are transformed erythroid pr

262 ide (DMSO)-induced differentiation of murine erythroleukemia (MEL) cells as a model, transcription of

263 uring erythrocytic differentiation of murine erythroleukemia (MEL) cells induced by dimethylsulfoxide

264 nditional expression of C/EBPalpha in murine erythroleukemia (MEL) cells induced myeloid-specific gen

265 recipitate with Tal1 in extracts from murine erythroleukemia (MEL) cells induced to differentiate wit

266 The in vitro differentiation of murine erythroleukemia (MEL) cells is a dramatic example of tum

267 Mouse erythroleukemia (MEL) cells represent an important cell

268 ion of terminal differentiation in the mouse erythroleukemia (MEL) cells requires a decline in the le

269 ell lines, whereas A-kinase-deficient murine erythroleukemia (MEL) cells show impaired hemoglobin pro

270 in dynamics to the differentiation of murine erythroleukemia (MEL) cells, a model system for erythroi

271 ary conservation was established using mouse erythroleukemia (MEL) cells, a well studied erythropoies

272 ce adult erythroid differentiation in murine erythroleukemia (MEL) cells, but only SCFAs concurrently

273 er enhancement than the intact core in mouse erythroleukemia (MEL) cells, indicating the presence of

274 We have cloned from murine erythroleukemia (MEL) cells, thymus, and stomach the cDN

275 tions from untreated and DMSO-treated Murine ErythroLeukemia (MEL) cells.

276 riptional unit in plasmid-transfected murine erythroleukemia (MEL) cells.

277 sulfoxide-induced differentiation of murine erythroleukemia (MEL) cells.

278 n normal and alpha-spectrin-deficient murine erythroleukemia (MEL) cells.

279 t (DeltaLCR) heterozygous mice and in murine erythroleukemia (MEL) cells.

280 s and the block to differentiation in murine erythroleukemia (MEL) cells.

281 ene expression from these mutations in mouse erythroleukemia (MEL) cells.

282 uring SAHA-induced differentiation of murine erythroleukemia (MEL) cells.

283 d probed for SAHA-binding proteins in murine erythroleukemia (MEL) cells.

284 rentiated, but not in differentiated, murine erythroleukemia (MEL) cells.

285 F) increase during differentiation of murine erythroleukemia (MEL) cells.

286 phosphorylation is not detectable in murine erythroleukemia (MEL) or other hematopoietic cells.

287 n and contributes to the formation of murine erythroleukemias (MEL).

288 The Friend murine retroviral erythroleukemia model involves mitogenic activation of t

289 d mechanistic studies in the TF-1 IDH2 R140Q erythroleukemia model system and found that IDH2 mutant

290 which spontaneously recover from FV-induced erythroleukemia, neutralization of gamma interferon (IFN

291 n insertion in SB-induced JAK2V617F-positive erythroleukemias, present in 87.5% and 65%, respectively

292 r miR-92a, results in B-cell hyperplasia and erythroleukemia, respectively.

293 ious data demonstrating a role for MLF1IP in erythroleukemias, suggest a possible function for this p

294 In Friend virus-induced erythroleukemia, the spleen focus-forming virus integrat

295 Friend erythroleukemia virus has long served as a paradigm for

296 fected cells that occurs early during Friend erythroleukemia virus infection.

297 elta(12)-PGJ(3) to mice infected with Friend erythroleukemia virus or those expressing the chronic my

298 ivation in another cell line-Rauscher murine erythroleukemia- which expresses the EPO receptor endoge

299 his disease had the overall appearance of an erythroleukemia, with an accumulation of immature erythr

300 jected into adult mice, SFFV induces a rapid erythroleukemia, with susceptibility being determined by