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2 [TfR]), we developed a series of human K562 erythroleukemic cell clones retrovirally transduced with
3 n applied successfully to analysis of murine erythroleukemic cell cultures and this, when coupled wit
9 O7e cells, a growth factor-dependent CD34(+) erythroleukemic cell line, and in cord blood-derived hum
10 pressed in the reticulocytes and in the K562 erythroleukemic cell line, but not in a lymphocytic cell
12 in mRNA levels in single K562 cells, a human erythroleukemic cell line, with and without 30 microM he
14 age of the abundant mRNAs from a single K562 erythroleukemic cell or a single dorsal root ganglion ne
16 eased expression of the gamma-globin gene in erythroleukemic cells as well as in primary erythroblast
17 ucleosomal chromatin derived from K562 human erythroleukemic cells by micrococcal nuclease digestion.
19 athways stopped the proliferation of primary erythroleukemic cells from Friend SFFV-infected mice, wi
22 cate that the lineage plasticity observed in erythroleukemic cells reflects an intrinsic property of
24 n-globin gene in the adult stage and in K562 erythroleukemic cells to analyze the inactive state of t
25 of 4T1 breast adenocarcinoma cells and K562 erythroleukemic cells with IFN-gamma triggers the cellul
26 nt occurs early in differentiation of murine erythroleukemic cells, this suggests that ribosomal cont
34 econd stage is characterized by emergence of erythroleukemic clones (MEL cells) which typically bear
36 ally characterized from cDNA clones of human erythroleukemic (HEL) cell mRNA, a cell line exhibiting
38 e nitric oxide synthase (NOS-2) in the human erythroleukemic K562 cell line results in constitutive p
45 f activated Erg was validated by reproducing erythroleukemic pathology in mice transplanted with feta
46 oson-driven disease and strong selection for erythroleukemic pathology with transformation of bipoten
49 reduced AP1 DNA-binding activity in primary erythroleukemic splenocytes from Friend SFFV-infected mi
50 n agonistic DR3 monoclonal antibody in human erythroleukemic TF-1 cells, which express DR3 endogenous
53 involved in lysis of K562 cells because the erythroleukemic tumor target cells expressed no class II
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