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1 in over 75% of phagocytic cups during amebic erythrophagocytosis.
2 moved at an accelerated rate in the liver by erythrophagocytosis.
3 ase, EhC2PK is involved in the initiation of erythrophagocytosis.
4 ophages died of exposure to heme released on erythrophagocytosis.
5 gnificantly reduced the efflux of 59Fe after erythrophagocytosis.
6 ity and protects red blood cells (RBCs) from erythrophagocytosis.
7 erythrocytes, nor did it enhance the rate of erythrophagocytosis.
8 roglial HO-1 and reduced injury by enhancing erythrophagocytosis.
9 -British (FAB) M4/5 morphology and prominent erythrophagocytosis.
10 CR3) on activated monocytes, thus leading to erythrophagocytosis.
12 sociated with host parasitemia; mediators of erythrophagocytosis and cellular stress were notable com
13 at 9 and 14 days after inoculation revealed erythrophagocytosis and deposition of an iron-negative p
14 ion of a PH domain in trophozoites inhibited erythrophagocytosis and enhanced motility, providing gen
16 ld-type animals exhibit comparable levels of erythrophagocytosis and platelet clearance in response t
17 pared with HM-1:IMSS, Rahman has a defect in erythrophagocytosis and the ability to cause amoebic col
18 d infiltrates, Kupffer cell hyperplasia with erythrophagocytosis, and an inconstant presence of eosin
19 formed granulomas, Kupffer cell hyperplasia, erythrophagocytosis, and microvesicular fatty metamorpho
20 establish phosphatidylserine involvement in erythrophagocytosis by amebae and suggest the existence
24 ation of maternal blood sinuses, the massive erythrophagocytosis by trophoblasts, the alteration of t
27 t FPN1 functions in the export of iron after erythrophagocytosis, FPN1 was stably expressed in J774 m
30 escribe a novel pathophysiologic pathway for erythrophagocytosis in the context of tissue macrophage
31 complement via complement receptor-mediated erythrophagocytosis in the spleen; and (3) when opsonize
32 In rat peritoneal macrophages, engaged in erythrophagocytosis in vitro, endotoxin stimulated heme
33 thy, fever, liver failure, pancytopenia, and erythrophagocytosis indicative of a hemophagocytic syndr
37 ad mutant of EhC2PK displayed a reduction in erythrophagocytosis, it appears that kinase activity is
39 m, we examined the effect of iron status and erythrophagocytosis on FPN1 expression in J774 macrophag
40 orthern analyses of iron-related genes after erythrophagocytosis revealed a 16-fold increase in FPN1
41 ressed FPN1 mRNA and protein induction after erythrophagocytosis, suggesting that FPN1 induction resu
42 nd strong induction of FPN1 expression after erythrophagocytosis suggests that FPN1 plays a role in i
43 expressing FPN1 released 70% more 59Fe after erythrophagocytosis than control cells, consistent with
48 ntial YM1(+) tissue macrophage accumulation, erythrophagocytosis within the liver, spleen, and bone m
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