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1 ex difference in mouse metabolic response to erythropoietin.
2 ed basal expression and hypoxic induction of erythropoietin.
3 tely quantify relative sialylation levels of Erythropoietin.
4 throblasts through a mechanism distinct from erythropoietin.
5 nd an indirect relation that was mediated by erythropoietin.
6 ally significant dimension to the biology of erythropoietin.
7 tion, often caused by impaired production of erythropoietin.
8 er injection, a time course similar to serum erythropoietin.
9 is produced by erythroblasts in response to erythropoietin.
10 erythroid progenitors are hypersensitive to erythropoietin.
11 d with 1 of 392 patients who did not receive erythropoietin.
12 ose-dependent manner in animals treated with erythropoietin.
13 response to hypoxia, including production of erythropoietin.
14 pted in 9 patients due to anemia; 4 received erythropoietin.
15 s to demonstrate the functional existence of erythropoietin.
16 ndomly assigned to receive recombinant human erythropoietin (3000 IU/kg; n=256) or placebo (n=239) in
19 as determined by suppression of circulating erythropoietin, a HIF-2 target and possible pharmacodyna
20 et gene characterized was EPO, which encodes erythropoietin-a glycoprotein hormone that controls eryt
22 on of granulocyte colony-stimulating factor, erythropoietin, aminocaproic acid, and phytonadione was
26 he EPOR dimer in the presence and absence of erythropoietin and a series of agonistic or antagonistic
28 n of this stress response was independent of erythropoietin and BMP4, and was observed in endothelial
29 hropoiesis-stimulating agents (ESAs) such as erythropoietin and darbepoetin in preterm and term infan
33 sion, hemoglobin and hepcidin increased, and erythropoietin and growth differentiation factor-15 decr
36 SnPP administration led to a decrease in erythropoietin and increase in hepcidin serum levels, ch
42 ternal 25(OH)D was inversely associated with erythropoietin at both midgestation (P <0.05) and delive
46 include anaemia due to reduced production of erythropoietin by the kidney; reduced red blood cell sur
50 e reported in 9 of 295 patients who received erythropoietin, compared with 1 of 392 patients who did
52 ood cell units per 8 weeks); pre-study serum erythropoietin concentration (<200 IU/L, 200-500 IU/L, a
54 ly lower than those in the recombinant human erythropoietin control arm in the hemodialysis study, an
56 CT to evaluate whether priming of ECFCs with erythropoietin could enhance their homing to the ischemi
57 genitors delays G1-S progression and arrests erythropoietin-dependent cell growth while favoring term
62 ession in mice constitutively overexpressing erythropoietin (Epo) (Tg6 mice), which leads to excessiv
66 essential mediators of red cell production, erythropoietin (EPO) and its cell surface receptor (EPO
68 dimeric receptor-ligand system, the cytokine Erythropoietin (EPO) and its receptor (EpoR), to dimeriz
71 process relies predominantly on the hormone erythropoietin (EPO) and its transcription factor hypoxi
73 to estimate time trends in hemoglobin (Hgb), erythropoietin (EPO) dose, intravenous (IV) iron dose, f
74 AFPL-Luc was constructed by insertion of the erythropoietin (Epo) enhancer for hypoxic cancer specifi
75 hd2, and Phd3 (Phd(1/2/3)hKO) promoted liver erythropoietin (EPO) expression 1246-fold, whereas renal
76 ascular endothelial growth factor (VEGF) and erythropoietin (EPO) expression were increased during hy
93 asts number, reduces apoptosis, and enhances erythropoietin (Epo) levels in controls, but not in Tfr2
94 ction induced anemia, splenomegaly, elevated erythropoietin (EPO) levels, and extramedullary erythrop
98 nally, all S-HB had increased phosphorylated erythropoietin (EPO) receptor and phosphorylated STAT-5
100 fish, murine, and human models, we show that erythropoietin (EPO) signaling, together with the GATA1
103 sly reported that the topical application of erythropoietin (EPO) to cutaneous wounds in rats and mic
104 when erythropoiesis is acutely stimulated by erythropoietin (EPO) to favor iron supply to maturing er
108 ia-inducible factor 1-alpha (HIF-1alpha) and erythropoietin (EPO) were measured as potential mechanis
111 in erythropoiesis and is the main source of erythropoietin (EPO), an oxygen-sensitive glycoprotein t
112 ond-Blackfan anaemia, are not treatable with erythropoietin (Epo), because the colony-forming unit er
114 ied this approach in vivo to the anemia drug erythropoietin (EPO), to direct its activity to EPO rece
116 reached the kidney and reduced expression of erythropoietin (Epo), which we identified as a MIR122 ta
120 ated that low reticulocytosis and suppressed erythropoietin (Epo)-induced erythropoiesis are features
131 ive of the stress-induced (hemolytic or post-erythropoietin [Epo]) treatment, only native CD11b(lo) s
133 red; this was accompanied by increased renal erythropoietin expression and stabilization of hypoxia-i
134 derlying mechanism is suppression of hepatic erythropoietin expression associated with the downregula
136 G cells, angiotensin II negatively regulated erythropoietin expression in the transformed cells.
138 s of the kidney suppresses renin and induces erythropoietin expression, this study aimed to character
139 um bilirubin, reticulocyte counts, and serum erythropoietin following intestinal HIF-2alpha disruptio
140 lysis study; continuing on recombinant human erythropoietin for the hemodialysis study) for 4 weeks,
141 revention of contrast-induced AKI and use of erythropoietin for the prevention of AKI, two therapeuti
144 g cells of Vhl (-/-(REN)) mice expressed the erythropoietin gene, and they were markedly polycythemic
145 k-in mutation results in upregulation of the erythropoietin gene, erythrocytosis, and augmented hypox
147 34/89 [38.2%; 95% CI, 28.1% to 49.1%]), both erythropoietin groups were futile (first dosing regimen:
149 fewer infants treated with recombinant human erythropoietin had abnormal scores for white matter inju
155 irin dose in 29% of the patients, the use of erythropoietin in 54%, and blood transfusions in 12%.
156 t, we have recently shown that expression of erythropoietin in a GnTI knock-out, FUT8-overexpressing
160 1 induced hypermethylation and repression of erythropoietin in pericytes; these effects were prevente
162 ntiation pathway of humans, and implicate an erythropoietin-independent, macrophage-associated pathwa
163 lts identify EphB4 as a critical mediator of erythropoietin-induced tumor progression and further pro
164 light perception (NLP) despite prescribed IV erythropoietin injections 20,000 units daily for 3 days
165 ignaling, while preventing responsiveness to erythropoietin-instigated signals that promote different
166 omatin and abrogates hepcidin suppression by erythropoietin, iron deficiency, thalassemia, and hemoch
167 fold greater risk of anemia, suggesting that erythropoietin is a sensitive predictor of anemia at del
169 patients with heart failure (HF), high serum erythropoietin is associated with risk of recurrent HF a
170 gulation of the haematopoietic growth factor erythropoietin led to the unexpected discovery of a wide
171 date the mechanisms through which endogenous erythropoietin levels associate with specific outcomes.
172 o-Stat5, most likely because of the elevated erythropoietin levels in response to the HU-induced anem
173 have evaluated the association of endogenous erythropoietin levels with clinical outcomes in the comm
174 pported by evidence that i) mRNA and protein erythropoietin levels within liver and serum increased i
177 nomegaly, and bone pathology, while reducing erythropoietin levels, improving erythrocyte morphology,
181 s unexpected for a JAK1/2 inhibitor, because erythropoietin-mediated JAK2 signaling is essential for
184 Randomized clinical trial of 200 patients (erythropoietin, n = 102; placebo, n = 98) with closed he
185 a nonrandomized subset of 165 infants (n=77 erythropoietin; n=88 placebo), brain abnormalities were
187 dialysis and not receiving recombinant human erythropoietin (nondialysis study) and in patients with
188 d head injury, neither the administration of erythropoietin nor maintaining hemoglobin concentration
190 estigate: 1) the effect of recombinant human erythropoietin on brain edema using diffusion-weighted m
191 oputamen; 2) the effect of recombinant human erythropoietin on brain tissue PO(2) in similar experime
192 local cooling with water and intraperitoneal erythropoietin once a day for five days starting 45 minu
193 e is limited information about the effect of erythropoietin or a high hemoglobin transfusion threshol
197 -stained percent IVNV and AVA; VEGF protein, erythropoietin, phosphorylated extracellular signal-rela
198 sumed cardiac cause, early administration of erythropoietin plus standard therapy did not confer a be
200 stored with ECFCs and almost totally so with erythropoietin priming (control, 72% +/- 2%; ECFCPBS, 90
201 lighted its notable role in ECFC homing with erythropoietin priming (ECFCEPO, 147% +/- 14%, n = 4; EC
205 into fibroblast-like cells resembling native erythropoietin-producing cells located in the tubulointe
207 tudy aims to investigate the significance of erythropoietin-producing hepatocellular (Eph)A2 expressi
208 like kinase suppressor of Ras 1 (KSR1), EPH (erythropoietin-producing hepatocellular carcinoma) recep
211 Beyond the well-defined role of the Eph (erythropoietin-producing hepatocellular) receptor tyrosi
216 nontoxic doses of 5-azacytidine can restore erythropoietin production and ameliorate anemia in the s
217 age-related changes, organ function such as erythropoietin production in the kidney may become subop
219 by erythrocytosis with suppressed endogenous erythropoietin production, bone marrow panmyelosis, and
221 nts with acute-on-chronic liver failure, and erythropoietin promoted hepatic regeneration in animal s
222 te protein/peptide context, such as with the erythropoietin protein, a V3 polypeptide derived from HI
225 failing signal transduction at the homomeric erythropoietin receptor (EpoR) and at the heteromeric in
228 L traptamers) that specifically activate the erythropoietin receptor (EPOR) in mouse cells to confer
231 und and unbound forms, the activation of the erythropoietin receptor (EPOR) was initially proposed to
232 d property of all EMAs, to bind on the human erythropoietin receptor (hEPOR), is therefore exploited.
233 escribe four different rearrangements of the erythropoietin receptor gene EPOR in Philadelphia chromo
235 cal and suggests that studying modulation of erythropoietin receptor pathway may lead to strategies i
236 n receptor subunits, sucrase-isomaltase, the erythropoietin receptor, and two of the subunits of the
237 vates the 3 main myeloid cytokine receptors (erythropoietin receptor, granulocyte colony-stimulating
243 light on the molecular mechanisms underlying erythropoietin repression in kidney myofibroblasts and d
246 for the glycoprofiling of recombinant human erythropoietin, revealing 74 glycoforms in a 60-min run.
250 emoglobin (Hb) response to recombinant human erythropoietin (rhEPO) therapy after hematopoietic cell
252 illegal administration of recombinant human erythropoietin (rHuEPO) among athletes is largely prefer
253 ore complex N-glycans from recombinant human erythropoietin (rHuEPO) expressed in Chinese hamster ova
254 ardioprotective effects of recombinant human erythropoietin (rHuEPO) protein in animal experiments ha
255 pe of juxtaglomerular cells from a renin- to erythropoietin-secreting cell type, presumably in respon
256 errin receptor (sTfR), hepcidin, serum iron, erythropoietin, serum folate, vitamin B-12, C-reactive p
257 These observations suggest that the altered erythropoietin signaling is focal and suggests that stud
258 find-me" signals from apoptotic cells induce erythropoietin signaling within macrophages to prime the
260 lation fingerprinting by comparing different Erythropoietin sources without the need for any sample p
262 6.88 mg/wk active control; P<0.001) and less erythropoietin-stimulating agent (median epoetin-equival
263 sferrin saturation, and intravenous iron and erythropoietin-stimulating agent usage as prespecified s
264 iron stores and reduces intravenous iron and erythropoietin-stimulating agent use while maintaining h
266 Administration boxed warning was issued for erythropoietin-stimulating agents (ESAs) regarding serio
268 gulated EPOR expression, hypersensitivity to erythropoietin stimulation, and heightened JAK-STAT acti
273 ged by ribavirin dosage reduction (71.5%) vs erythropoietin therapy (70.9%), regardless of the timing
276 osporine, abciximab, clopidogrel, tirofiban, erythropoietin, thrombus aspiration, adenosine, glucose-
277 lpha1 type I collagen and PDGFRbeta, produce erythropoietin through HIF2alpha regulation but that pro
278 raumatic administration of recombinant human erythropoietin, thus offering new perspectives in the us
282 EGF, VEGF receptor 1 (VEGFR1), VEGFR2, Tie2, erythropoietin, transforming growth factor beta1, insuli
283 ed in increased heme levels, and hypoxia and erythropoietin treatment increased heme production throu
285 clinical trial of preterm infants, high-dose erythropoietin treatment within 42 hours after birth was
288 cident HF events, and each doubling of serum erythropoietin was associated with a 25% increased risk
289 iesis was shown by Leon Jacobson in 1957 and erythropoietin was eventually purified from the urine of
293 pha, vascular endothelial growth factor, and erythropoietin were higher in term than preterm pups, re
294 CD133(+) progenitor cells stopped producing erythropoietin when they differentiated and acquired an
295 ions after the switch from recombinant human erythropoietin, whereas mean hemoglobin decreased in the
297 describe the total synthesis of homogeneous erythropoietin with consensus carbohydrate domains incor
299 lobin occurred with elevations in endogenous erythropoietin within the range usually observed in the
300 tudy used a factorial design to test whether erythropoietin would fail to improve favorable outcomes
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