ライフサイエンスコーパス: escape of @2 from

1 therapy may also be required to prevent the escape of cancers from a given state of oncogene addicti

2 unching also is the driving force behind the escape of RNA polymerase from a regulatory pause of the

3 quired to ensure preparedness for a possible escape of RPV from a laboratory or its deliberate releas

4 rotein kinase C (PKC) cascade which promotes escape of the bacterium from a macrophage-like cell phag

5 Cytotoxic activity follows Hsc70-mediated escape of RTA from an otherwise destructive pathway faci

6 Escape of tumor cells from apoptotic-mediated stimuli re

7 rived niche that facilitates the therapeutic escape of melanoma cells from BRAF inhibition.

8 phase (data not shown), consistent with the escape of p185neu* from cell cycle-dependent regulation.

9 IL8 pathway in mediating macroH2A1-dependent escape of HCC cells from chemotherapy-induced senescence

10 d for in SHIV strain 89.6P to facilitate the escape of that virus from CTL recognition of the dominan

11 formation in the hTR precursor improves the escape of immature RNP from degradation, but subsequentl

12 r after other stressors that may promote the escape of metastases from dormancy, an issue that is of

13 We show that the escape of hydrogen from early Earth's atmosphere likely

14 isms with either tlyC or pld resulted in the escape of transformants from endosomal vacuoles into the

15 ilst some pH responsive peptides promote the escape of labelled siRNA from endosomes, others may prom

16 ver protein cargo into cells, facilitate the escape of protein from endosomes in response to the redu

17 ng at the ER by a degron in Pca1 leads to an escape of Pca1 from ERAD.

18 , low-mass galaxies probably facilitated the escape of ionizing radiation from galaxies when the Univ

19 Mutational escape of HIV-1 from HIV-1-specific CD8(+) T lymphocytes

20 block escape pathways.IMPORTANCE Mutational escape of HIV-1 from HIV-1-specific CD8(+) T lymphocytes

21 lution of viruses with cellular life, or the escape of genetic material from host genomes.

22 These results indicate that the escape of HIV-1 from host humoral immunity may play a di

23 le of serine protease inhibitor (SPI) in the escape of MSCs from host immunosurveillance through the

24 unity, but the perturbations associated with escape of the fungus from host defenses remain ill-defin

25 ot support a role for FasL expression in the escape of melanoma cells from immune destruction.

26 may present an evolutionary strategy for the escape of pathogens from immune destruction.

27 elopment of regulatory T cells may allow the escape of tumor cells from immune surveillance by the CT

28 tter to function efficiently facilitates the escape of virus from immune control and the collapse of

29 ent can contribute to immune suppression and escape of tumours from immunological detection and clear

30 The same mechanism could explain the escape of CPPs from intracellular endosomes that are enr

31 plicate sequences diverge-until an eventual "escape" of the sequences from its influence.

32 ic replication, but may also account for the escape of vIL6 from K13-induced transcriptional suppress

33 ntended genome editing occurring through the escape of strains from laboratories, coupled with the pr

34 Cyclin T1 induction is involved in the escape of HIV-1 from latency.

35 main boundaries at C(r) would facilitate the escape of molecules from membranes was further supported

36 We demonstrate that mutational escape of HIV-1 from Nef-specific CTL in vitro leads to

37 The mechanisms that regulate the escape of Pol II from pausing and the relationship to ch

38 thogen, and adenosine synthesis also enabled escape of Bacillus anthracis from phagocytic clearance.

39 O) is a major virulence factor implicated in escape of Listeria monocytogenes from phagocytic vacuole

40 rast to other cells, neutrophils prevent the escape of Shigella from phagocytic vacuoles in which the

41 completely eradicate leukaemic cells and the escape of these cells from previous control has led to a

42 ial subpopulation and, in vivo, promoted the escape of the latter from primary implantation sites and

43 velopment of memory CD8 T cells requires the escape of CTLs from programmed cell death.

44 opment of memory CD8(+) T lymphocytes is the escape of progenitors from programmed cell death, but ho

45 es regulate their expression by limiting the escape of RNA polymerase from promoter-proximal pause si

46 eld fluorescence microscopy, and tracked the escape of dye from single vesicles by watching the incre

47 of individuals with CF that facilitates the escape of the microorganism from SP-A-mediated phagocyti

48 c approaches, thus contributing to avoid the escape of malignant cells from stress-elicited immune re

49 In this report, we document that escape of AAV from the endocytic pathway in NIH 3T3 cell

50 We present data indicating that escape of AAV from the endosome and trafficking of viral

51 suggest that both crowding and binding limit escape of AMPARs from the synapse.

52 e severe inflammatory response and increased escape of bacteria from the colon into the lymphatic sys

53 intestinal lesions provide the mechanism for escape of C. albicans from the gastrointestinal tract.

54 y assigned, as in the bacterial case, to the escape of counterions from the surface of the particles.

55 e first substrate binding event prevents the escape of diatomic ligands from the distal heme binding

56 ential as small as 20 mV strongly biased the escape of DNA from the pore.

57 Escape of F. tularensis from the phagosome into the cyto

58 vial fluid hyaluronan, on the trans-synovial escape of fluid from the joint cavity in the steady stat

59 ies, the presence of products resulting from escape of free radicals from the solvent cage can be sup

60 oring cells but that LLO is required for the escape of L. monocytogenes from the double-membrane vacu

61 shown that PI-PLC plays a role in efficient escape of L. monocytogenes from the primary phagosome.

62 reted pore-forming protein essential for the escape of L. monocytogenes from the vacuole formed upon

63 Escape of microtubules from the surface was prevented by

64 ids) to play a central role in mediating the escape of motile bacteria from the LC interface.

65 complex, a finding consistent with the slow escape of PAP from the nonreactive E.PAP.E(2) complex.

66 Large crystallites sublimated by escape of particles from the perimeter.

67 sociation, thereby permitting rapid promoter escape of Pol II from the preinitiation complex.

68 anti-OmpB monoclonal antibody inhibited the escape of R. conorii from the phagosome, resulting in in

69 hesized that the molecular events leading to escape of S. aureus from the endosome involved the Agr v

70 This aneuploidy may facilitate the continued escape of such cells from the normal checkpoint mechanis

71 2, 3-diphosphoglycerate partially inhibited escape of the bacteria from the primary phagocytic vacuo

72 Escape of the bacterium from the phagosome to the cytoso

73 he cell by clathrin-mediated endocytosis and escape of the virus from the endosome.

74 l-specific phospholipase C (PI-PLC), mediate escape of this pathogen from the phagocytic vacuole of m

75 potential to combat cancer and overcome the escape of tumor cells from the cell death machinery.

76 ls (DCs), is one potential mechanism for the escape of tumors from the host immune system.

77 The escape of water from the sixth position upon the formati

78 provides the ability to directly measure the escape of electrons from their Coulombically bound radic

79 The rate of escape of (3)He from this fullerene was monitored by (3)

80 Escape of polymerase from this paused state has been pro

81 dation, while Ln-332 and HSC-CM promoted the escape of FAK from ubiquitination, probably inducing a p

82 olecular mechanisms are responsible for the "escape" of these BMDCs from VEGFR1 inhibition would faci

83 e coats prey-capture threads and hampers the escape of prey from webs, thereby increasing the foragin