ライフサイエンスコーパス: esophageal

1 Lower esophageal acid exposure (percentage time pH < 4) was si

2 Supine esophageal acid exposure before the index operation was

3 significant erosive tooth wear and increased esophageal acid exposure by 24-h multichannel intralumin

4 aseline demographic, clinical, endoscopic or esophageal acid exposure characteristics were significan

5 GERD-Health Related Quality of Life scores, esophageal acid exposure, lower esophageal sphincter pre

6 Barrett's esophagus (BE) and progression to esophageal adenocarcinoma (EA).

7 Bacteria may play a role in esophageal adenocarcinoma (EAC) and esophageal squamous

8 ed receptor5 (TGR5) were highly expressed in esophageal adenocarcinoma (EAC) and precancerous lesions

9 etermine risk of Barrett's esophagus (BE) or esophageal adenocarcinoma (EAC) based on genetic and non

10 igated mechanisms that mediate resistance of esophageal adenocarcinoma (EAC) cells and patient-derive

11 The incidence of esophageal adenocarcinoma (EAC) has increased in many We

12 progression to high-grade dysplasia (HGD) or esophageal adenocarcinoma (EAC) in patients with LGD of

13 Esophageal adenocarcinoma (EAC) is a growing problem wit

14 Esophageal adenocarcinoma (EAC) is a highly lethal cance

15 The incidence of esophageal adenocarcinoma (EAC) is rapidly rising in the

16 Barrett's esophagus (BE) are diagnosed with esophageal adenocarcinoma (EAC) within 1 year of an endo

17 by surgery has become a standard of care for esophageal adenocarcinoma (EAC).

18 their risk of high-grade dysplasia (HGD) or esophageal adenocarcinoma (EAC).

19 rogression to high-grade dysplasia (HGD) and esophageal adenocarcinoma (EAC).

20 significantly enhanced potency against three esophageal adenocarcinoma cell lines compared with the t

21 Esophageal adenocarcinoma is a heterogeneous, chemoresis

22 Esophageal adenocarcinoma is more frequent in non-Hispan

23 In this study, LNM from esophageal adenocarcinoma was objectively detected using

24 e, erosive esophagitis, Barrett's esophagus, esophageal adenocarcinoma, erosive gastritis, gastric ca

25 cell line HUH7, as well as in liver tumors, esophageal adenocarcinoma, glioblastoma multiforme, pros

26 d the risk of gastric cardia adenocarcinoma, esophageal adenocarcinoma, or esophageal squamous cell c

27 In patients with esophageal adenocarcinoma, statin use after diagnosis wa

28 lterations that contribute to development of esophageal adenocarcinoma, we know little about features

29 s representative of the genomic landscape of esophageal adenocarcinoma.

30 sophageal strictures, Barrett esophagus, and esophageal adenocarcinoma.

31 small proportion of patients, development of esophageal adenocarcinoma.

32 GERD) is the strongest known risk factor for esophageal adenocarcinoma.

33 00/CBP and Notch has a synergistic effect in esophageal adenocarcinoma.

34 cell lines representing HER2 overexpressing esophageal adenocarcinomas (EACs) and EGFR overexpressin

35 Esophageal adenocarcinomas have mutations in tumor prote

36 subtypes.We identified 966 incident cases of esophageal adenocarcinomas, 323 cases of esophageal squa

37 mors, 156 glioblastoma multiform samples, 27 esophageal adenocarcinomas, and 269 prostate cancer samp

38 livered at 2 different sites on the anterior esophageal adventitia.

39 5 for a patient subpopulation with increased esophageal allergic inflammation.

40 h L. lactis NCC 2287 significantly decreased esophageal and bronchoalveolar eosinophilia but only whe

41 nd peanut butter consumption and the risk of esophageal and gastric cancers and their different subty

42 azard models to estimate HRs and 95% CIs for esophageal and gastric cancers and their subtypes.We ide

43 s evaluated the effect of nut consumption on esophageal and gastric cancers.The objective was to eval

44 included melanoma, breast, colorectal, lung, esophageal, and hepato-pancreato-biliary/gastric cancer.

45 Esophageal architecture remains unaffected 2 months afte

46 $129764-$173712]), tracheoesophageal fistula/esophageal atresia (WIQR, $39206; median, $105259 [IQR,

47 outcomes in adults with surgically corrected esophageal atresia/tracheaesophageal fistula (EA/TEF).

48 without apparent deleterious effects on the esophageal body.

49 irin users vs non-users after diagnosis with esophageal cancer (48% vs 50% in England and 49% vs 46%

50 Among 3592 patients with esophageal cancer (84.7% adenocarcinoma, 15.2% squamous

51 al responses were observed in a patient with esophageal cancer (duration, 4 months), a patient with u

52 ct on the esophageal toxicity prediction for esophageal cancer (EC) patients administered intensity-m

53 e to neoadjuvant chemoradiotherapy (nCRT) in esophageal cancer (EC) patients is important in a more p

54 he multimodal management of locally advanced esophageal cancer (LAEC), and to assess its independent

55 ancer-specific mortality among patients with esophageal cancer (pooled adjusted HR, 0.98; 95% CI, 0.8

56 cancer-specific mortality after diagnosis of esophageal cancer (pooled adjusted HR, 1.03; 95% CI, 0.8

57 cottish cohorts contained 4654 patients with esophageal cancer and 3833 patients with gastric cancer,

58 idered useful as a new staging parameter for esophageal cancer and could also be of interest for othe

59 promote apoptosis and limit proliferation of esophageal cancer cell lines.

60 analogues were cytotoxic toward gastric and esophageal cancer cells and showed lower IC50 values tha

61 that zinc may inhibit cell proliferation of esophageal cancer cells through Orai1-mediated intracell

62 ulcer (PPU) was analyzed, independent of HV esophageal cancer center status and patient and disease-

63 ethnic disparities in the incidence of total esophageal cancer decreased over time, which was due mai

64 nCRT according to the Chemoradiotherapy for Esophageal Cancer Followed by Surgery Study-regimen can

65 l and ethnic disparities in the incidence of esophageal cancer have not been thoroughly characterized

66 patients diagnosed with potentially curable esophageal cancer impacts overall survival.

67 Data from patients operated on for esophageal cancer in 30 European centers were collected.

68 omy for patients with T1-3N1M0 mid or distal esophageal cancer in the National Cancer Data Base from

69 Esophageal cancer is characterized by early and frequent

70 f an extended lymphadenectomy after nCRT for esophageal cancer is debated.

71 y, early diagnosis and curative treatment of esophageal cancer is possible.

72 D DATA: The optimal treatment for resectable esophageal cancer is unknown.

73 he circumferential resection margin (CRM) in esophageal cancer on survival and recurrence in patients

74 treatment at high-volume centers may improve esophageal cancer outcomes.

75 uggest AC may provide additional benefit for esophageal cancer patients, and merits further investiga

76 adjuvant treatment upon survival for cT3N0M0 esophageal cancer patients, with subgroup analyses by hi

77 the nonoperative treatment of patients with esophageal cancer remains uncertain.

78 All patients with esophageal cancer staged before NAC with PET/CT and afte

79 ited Kingdom's BE gene study and stomach and esophageal cancer study.

80 To study the influence of esophageal cancer surgeon volume upon mortality from upp

81 The complex elective workload of HV esophageal cancer surgeons appears to lower the threshol

82 anastomosis and/or thoracotomy on POM after esophageal cancer surgery in recent years.

83 All consecutive patients who underwent esophageal cancer surgery with reconstruction between 20

84 e relationships led to the centralization of esophageal cancer surgery.

85 Retrospective cohort study of patients with esophageal cancer treated with neoadjuvant chemoradiatio

86 ysplasia (squamous and Barrett's), and early esophageal cancer using resection and ablation technolog

87 Esophagectomies in 1,821 patients with esophageal cancer were conducted by 139 surgeons.

88 elative to placebo in patients with advanced esophageal cancer who had disease progression after chem

89 ears to identify a subgroup of patients with esophageal cancer who may benefit from gefitinib as a se

90 with irregular Z line do not develop HGD or esophageal cancer within 5 years after index endoscopy.

91 hCR) to chemoradiotherapy before surgery for esophageal cancer would enable investigators to study th

92 millimeter +/- 0.15, P < .001) of mice with esophageal cancer xenografts, as well as the smallest re

93 survival for patients with locally advanced esophageal cancer, and to evaluate how pathologic diseas

94 With improved oncologic outcomes in esophageal cancer, there is an increasing focus on funct

95 Patients with esophageal cancer, treated with nCRT plus surgery were i

96 ll carcinoma is a major histological type of esophageal cancer, with distinct incidence and survival

97 osis was associated with a decreased risk of esophageal cancer-specific mortality (HR, 0.61; 95% CI 0

98 d outcomes of patients with locally advanced esophageal cancer.

99 have no impact on survival and recurrence in esophageal cancer.

100 sive surgical techniques in the treatment of esophageal cancer.

101 ificant survival benefit for clinical T3N0M0 esophageal cancer.

102 -positive and, in particular, EGFR-amplified esophageal cancer.

103 en shown to contribute to the progression of esophageal cancer.

104 d short- and long-term oncologic outcomes in esophageal cancer.

105 eferred management approach for locoregional esophageal cancer.

106 ted with chronic gastroesophageal reflux and esophageal cancer.

107 strategy in improving survival of resectable esophageal cancer.

108 assess its prognostic value in patients with esophageal cancer.

109 or predictive biomarkers in the treatment of esophageal cancer.

110 all patients who underwent esophagectomy for esophageal cancer.

111 ive with open esophagectomy in patients with esophageal cancer.

112 Only a minority of esophageal cancers demonstrates a pathologic tumor respo

113 Esophageal cancers develop systems to evade anti-tumor i

114 al xenografts, and nude rats with orthotopic esophageal cancers in four study groups of six animals p

115 s to centers that treated 2.6 (IQR: 1.9-3.3) esophageal cancers per year, and 317 Travel patients who

116 P < .001) of rat orthotopic esophageal cancers.

117 2), cryptococcal infection (P=0.01), oral or esophageal candidiasis (P=0.02), death of unknown cause

118 gnosed with stage pT3-4Nx-0M0 or pT1-4N1-3M0 esophageal carcinoma (squamous cell or adenocarcinoma) f

119 11, patients with a resectable intrathoracic esophageal carcinoma, including the gastroesophageal jun

120 r, in development of Barrett's esophagus and esophageal carcinoma.

121 atients undergoing nonoperative treatment of esophageal carcinoma.

122 focused on a genotoxic aspect of exposure of esophageal cells to acidic bile reflux (BA/A).

123 nistically, our data showed that exposure of esophageal cells to acidic bile salts induces phosphoryl

124 most marked effect of the belt was impaired esophageal clearance of refluxed acid (median values of

125 ulcers showed the occurrence of perforating esophageal complications.

126 ssure (4 vs 9 mm Hg; P < 0.0001), and distal esophageal contraction amplitude (80 vs 90 mm Hg; P = 0.

127 bel use of corticosteroids not optimized for esophageal delivery.

128 esophageal region is an uncommon location of esophageal diverticula, a condition usually diagnosed in

129 Further evaluation demonstrated a mid-esophageal diverticulum at the level of the carina.

130 We found that the mean esophageal dose for ESOwhole, ESOinfield, ESOinfield-tum

131 disease characterized by symptoms related to esophageal dysfunction and an eosinophil-predominant inf

132 is safe and feasible, whereas performance of esophageal endoscopy in the presence of AEF may result i

133 fibrillation ablation and postinterventional esophageal endoscopy were included in the study.

134 atrial fibrillation underwent postprocedural esophageal endoscopy.

135 tment response was independent of effects on esophageal eosinophil maturation or activation.

136 is a potential therapeutic target to reduce esophageal eosinophilia and remodeling.

137 steroid formulation, to reduce symptoms and esophageal eosinophilia in adolescents and adults with E

138 of 11 and 40 years with dysphagia and active esophageal eosinophilia were randomized to receive eithe

139 eceptor-alpha was predominantly expressed on esophageal eosinophils during EoE, in addition to select

140 tion that provides additional perspective on esophageal epithelial biology and the widely prevalent d

141 induced in an undifferentiated, non-dividing esophageal epithelial cell population in patients with a

142 ific Zn(2+) chelator, whereas nontumorigenic esophageal epithelial cells are significantly less sensi

143 s-specific genes were reproduced in vitro in esophageal epithelial cells differentiated in the presen

144 ion was detected in ex vivo-cultured primary esophageal epithelial cells in a subpopulation of cells

145 the nuclei of a subpopulation of basal layer esophageal epithelial cells in patients with active EoE

146 n Atlas in the EoE transcriptome and in EPC2 esophageal epithelial cells.

147 se, involves dysregulated gene expression in esophageal epithelial cells.

148 cells were radioresistant and contributed to esophageal epithelial regeneration following radiation-i

149 in vitro platform that recapitulates normal esophageal epithelial stratification and differentiation

150 CAPN14 induces disruptive effects on the esophageal epithelium by impairing epithelial barrier fu

151 ived progenitor cell population in the mouse esophageal epithelium that is characterized by expressio

152 examination showed complete sloughing of the esophageal epithelium with a striking subepithelial lich

153 he interactions between immune cells and the esophageal epithelium.

154 iferation, thereby leading to atrophy of the esophageal epithelium.

155 eal radiosensitivity can be quantified using esophageal expansion and K-Means clustering to improve t

156 radiation-response and esophageal toxicity, esophageal expansion, as a method to quantify radiosensi

157 The esophageal expansion-response was highly variable betwee

158 Esophageal exposure to gastric refluxate is the primary

159 Herein, we assessed the esophageal expression of IL-33, an epithelium-derived al

160 is associated with the development of atrial-esophageal fistula (AEF) and increased mortality.

161 Pancreatic, esophageal, gastric, pooled colorectal, left-side colon,

162 hypothesis that a defect in tissue-specific esophageal genes is an integral part of EoE pathogenesis

163 Hs-Tyr is localized in the nematode esophageal gland.

164 Significantly increased levels of esophageal GM-CSF expression was detected in the L2-IL5(

165 f the right crus of diaphragm which form the esophageal hiatus are arranged like a "noose" around the

166 We assessed the LES and esophageal hiatus morphology using a block containing th

167 The influence of esophageal high-volume (HV) cancer surgeon status (>/=5

168 Esophageal IgE production was quantified with epsilon ge

169 Eosinophilic esophagitis-like esophageal inflammation was induced in the L2-IL5(OXA) E

170 Eosinophilic esophagitis (EoE) is an esophageal inflammatory disease associated with atopic d

171 pport the importance of immune cell-mediated esophageal injury in esophagitis and confirms the utilit

172 nstrate endoscopically detected asymptomatic esophageal lesions (EDEL) after atrial fibrillation abla

173 The esophageal lumen is lined by a stratified squamous epith

174 ed proximity of their afferent nerves to the esophageal lumen, and therefore greater exposure to noxi

175 Respiratory measurements using esophageal manometry and respiratory inductance plethysm

176 probability score for EoE, p(EoE), based on esophageal mRNA transcript patterns from biopsies of pat

177 Long-term systematic follow-up of the esophageal mucosa including multistaged biopsies is requ

178 ether an esophageal prick test, in which the esophageal mucosa is challenged by local injection of al

179 not differ significantly between the distal esophageal mucosa of controls (median, 25.5 cell layers

180 ucosa of patients with NERD, from the distal esophageal mucosa of patients with ERD, and the distal-m

181 Proximal and distal esophageal mucosa of patients with NERD have more superf

182 s were obtained from the proximal and distal esophageal mucosa of patients with NERD, from the distal

183 esponse to food antigens in contact with the esophageal mucosa.

184 We conclude that esophageal mucosal food allergen injections induce acute

185 rotein in the development and progression of esophageal mucosal metaplasia, dysplasia and carcinoma.

186 ing cohorts of patients newly diagnosed with esophageal or gastric cancer, identified from cancer reg

187 ncreased survival of patients diagnosed with esophageal or gastric cancer.

188 d cancer-specific mortality in patients with esophageal or gastric cancer.

189 thoracic Ivor Lewis esophagectomy (TTIL) for esophageal or gastroesophageal junction (GEJ) cancer.

190 f nodes on prognosis in patients with distal esophageal or gastroesophageal junction adenocarcinoma w

191 Patients with esophageal or GEJ cancer selected for TAMK or TTIL compl

192 sing the zero-heat-flux method compared with esophageal or iliac arterial temperatures measurements.

193 ity of life (HRQOL) domains in patients with esophageal or junctional cancer who received neoadjuvant

194 On the basis of the known esophageal past medical history as well as the absence o

195 Occurrence of esophageal perforating complications during follow-up wa

196 y seems to identify patients at high risk of esophageal perforating complications only occurring in p

197 identified to be a significant predictor for esophageal perforating complications.

198 year) upon 30-day and 90-day mortality from esophageal perforation (EP), paraesophageal hernia causi

199 In 5 of 832 patients (0.6%), an esophageal perforation (n=3) or an esophagopericardial o

200 Esophageal perforation is a dreaded complication of atri

201 Esophageal perforation occurred only in patients with ca

202 ces of stroke-free survival for all types of esophageal perforation.

203 stroesophageal reflux disease by an abnormal esophageal pH study (body mass index <35 kg/m, hiatal he

204 annot reduce PPIs should consider ambulatory esophageal pH/impedance monitoring before committing to

205 can be evaluated with endoscopy and tests of esophageal physiology, to better determine their disease

206 inspiratory effort, and work of breathing by esophageal pressure swings (DeltaPes) and pressure time

207 Esophageal pressure variations decreased from 9.8 (5.8-1

208 ere the indexes of respiratory effort (i.e., esophageal pressure variations, esophageal pressure-time

209 nula on indexes of respiratory effort (i.e., esophageal pressure variations, esophageal pressure-time

210 riate analysis sniff trans-diaphragmatic and esophageal pressure, twitch trans-diaphragmatic pressure

211 Esophageal pressure-time product/min decreased from 165

212 ffort (i.e., esophageal pressure variations, esophageal pressure-time product/min, and work of breath

213 ffort (i.e., esophageal pressure variations, esophageal pressure-time product/min, and work of breath

214 The esophageal prick test deserves further exploration becau

215 We investigated whether an esophageal prick test, in which the esophageal mucosa is

216 cence were used for evaluating expression of esophageal proteins in biopsy specimens from control sub

217 Esophageal radiosensitivity can be quantified using esop

218 -Means clustering to group patients based on esophageal radiosensitivity.

219 Gastro-esophageal reflux disease (GERD) is suggested to be asso

220 f comorbidities is similar except for gastro-esophageal reflux disease and dyslipidemia that appear t

221 able after both procedures except for gastro-esophageal reflux disease and dyslipidemia, which were m

222 causes of heartburn in patients with gastro-esophageal reflux disease.

223 Background acidic esophageal reflux exposure appeared stable over time, wh

224 flux episodes and the percentage of proximal esophageal reflux off-PPI did not change significantly a

225 Mid-esophageal region is an uncommon location of esophageal

226 ce to evaluate surgical performance in major esophageal resection.

227 acial/ethnic disparities in the incidence of esophageal SCC over time in the United States, while dis

228 mor front and predicts for poor prognosis of esophageal SCC, shedding light upon the tumor promoting

229 en extracts, could identify individuals with esophageal sensitization.

230 patients with Barrett's esophagus and human esophageal specimens, we found that BA/A cause significa

231 ic sphincter augmentation (MSA) on the lower esophageal sphincter (LES).

232 veal the anatomical counterpart of the lower esophageal sphincter (LES).

233 extending 5.5 cm proximal to the peak lower esophageal sphincter pressure point was increased by app

234 Life scores, esophageal acid exposure, lower esophageal sphincter pressure, number of beads (size) of

235 Transient lower esophageal sphincter relaxations were not increased by t

236 ion, and electrical stimulation of the lower esophageal sphincter.

237 Human esophageal squamous cancer cells were transduced with lu

238 chemotherapy in a rat model with orthotopic esophageal squamous cancers.

239 e underscored by its pathogenic mutations in esophageal squamous cell cancers (SCCs).

240 ia can enhance the effect of chemotherapy on esophageal squamous cell cancers.

241 tial genome-wide association study (GWAS) on esophageal squamous cell carcinoma (ESCC) in Han Chinese

242 in tumor tissues removed from patients with esophageal squamous cell carcinoma (ESCC) with poor prog

243 role in esophageal adenocarcinoma (EAC) and esophageal squamous cell carcinoma (ESCC), although evid

244 ociated with enhanced malignant potential in esophageal squamous cell carcinoma (ESCC), among the dea

245 susceptible to chronic fungal infection and esophageal squamous cell carcinoma (ESCC).

246 involved in chemoresistance in patients with esophageal squamous cell carcinoma (ESCC).

247 effects on gastric adenocarcinoma (AGS) and esophageal squamous cell carcinoma (KYSE30) cancer cells

248 Esophageal squamous cell carcinoma is a major histologic

249 real data from the 1000 Genomes Project and esophageal squamous cell carcinoma samples show that see

250 ory effects of zinc on cell proliferation in esophageal squamous cell carcinoma through Orai1.

251 In mice bearing esophageal squamous cell carcinoma tumors, to estimate u

252 ore frequent in non-Hispanic whites, whereas esophageal squamous cell carcinoma with risk factors of

253 of esophageal adenocarcinomas, 323 cases of esophageal squamous cell carcinoma, 698 cases of gastric

254 In addition to esophageal squamous cell carcinoma, cancers of the small

255 his effect was not observed in patients with esophageal squamous cell carcinoma.

256 cocutaneous candidiasis, hypothyroidism, and esophageal squamous cell carcinoma.

257 denocarcinoma, esophageal adenocarcinoma, or esophageal squamous cell carcinoma.Among older American

258 enocarcinomas (EACs) and EGFR overexpressing esophageal squamous cell carcinomas (ESCCs).

259 wth factor, and cell cycle pathways, whereas esophageal squamous tumors have a distinct set of mutati

260 Outcomes for esophageal stenting are poor in AEF.

261 and oral cavity; nasopharynx; other pharynx; esophageal; stomach; colon and rectum; liver; gallbladde

262 ce of gastroesophageal reflux disease (26%), esophageal stricture (39%), or both (15%) does not accou

263 s were uncommon: 1 (2%) patient developed an esophageal stricture (grade 2) and 1 (2%) grade 4 esopha

264 tion is a risk factor for the development of esophageal strictures, Barrett esophagus, and esophageal

265 ts have manifestations of fibrosis and gross esophageal strictures.

266 5-year period in 13 high-volume centers for esophageal surgery, we selected a study group of 334 pat

267 s temperature using zero-heat-flux method to esophageal temperature and arterial temperature.

268 ties of atrial fibrillation ablation despite esophageal temperature monitoring.

269 All patients had esophageal temperature probe and a noninvasive cutaneous

270 The esophageal temperature ranged from 33 degrees C to 39.7

271 Targeted esophageal temperature was used in 168 infants.

272 airs of temperature using zero-heat-flux and esophageal temperature were collected and 1,850 triple o

273 erature and between arterial temperature and esophageal temperature were equal to or lower than 1 deg

274 9 degrees C +/- 0.53 degrees C compared with esophageal temperature with an absolute difference of te

275 triple of temperature using zero-heat-flux, esophageal temperature, and arterial temperature.

276 Comparison to the esophageal temperature, considered as the reference in t

277 essed to perforation, and no patient without esophageal thermal ulcers showed the occurrence of perfo

278 red analysis has revealed a profound loss of esophageal tissue differentiation (identity) as an integ

279 events caused by GERD is repeated damage of esophageal tissues by the refluxate.

280 e name AEF, fistulas functionally act 1 way, esophageal to atrial, which accounts for the observed sy

281 The difference in the dose variation, acute esophageal toxicity (AET) and late esophageal toxicity (

282 on, acute esophageal toxicity (AET) and late esophageal toxicity (LET) of four DEs were compared.

283 emonstrates that different DEs influence the esophageal toxicity prediction for EC patients administe

284 definitions of esophagus (DEs) impact on the esophageal toxicity prediction for esophageal cancer (EC

285 imaging biomarker of radiation-response and esophageal toxicity, esophageal expansion, as a method t

286 ials to validate pre-treatment biomarkers of esophageal toxicity.

287 reflux disease by the expression of a unique esophageal transcriptome and the interplay of early life

288 mmune checkpoint modification contributes to esophageal tumor development.

289 Based on current guidelines, clinical T3N0M0 esophageal tumors may or may not receive neoadjuvant tre

290 the endoscopic finding of a longitudinal mid-esophageal ulcer in the presence of proximal stricture m

291 ment, and postablation endoscopy documenting esophageal ulcer may identify patients at higher risk fo

292 Esophageal ulcer seems to precede AEF development, and p

293 Esophageal ulceration and fistula are complications of p

294 One out of 10 postablation esophageal ulcers progressed to perforation, and no pati

295 From 2005 to 2012, patients with esophageal varices (EV) in the National Surgical Quality

296 s with low likelihood of harboring high-risk esophageal varices (EVs) or having clinically significan

297 t, and were case matched to patients without esophageal varices (NEV) based on sex, age, surgery type

298 Most patients (75%) had esophageal varices, 21% were Child-B, and 29% had at lea

299 In 2 patients, full-thickness erosion of the esophageal wall with partial endoluminal penetration of

300 y aneurysm of 9 mm in diameter, abutting the esophageal wall.

301 Cancer cells, mice with subcutaneous cancer esophageal xenografts, and nude rats with orthotopic eso