ライフサイエンスコーパス: esophageal mucosa

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1 normal, inflamed, metaplastic, and malignant esophageal mucosa.

2 le 1, 29 patients (81%) had no cancer in the esophageal mucosa.

3 esponse to food antigens in contact with the esophageal mucosa.

4 tly increased ERK2 phosphorylation levels in esophageal mucosa.

5 without acid is not sufficient to damage the esophageal mucosa.

6 extensive expansion involving up to 17 cm of esophageal mucosa.

7 sin B activity across normal and tumor human esophageal mucosa.

8 ach, or the infrequent methylation of normal esophageal mucosa accompanied by methylation in all norm

9 ) was found in the basal layer of the normal esophageal mucosa and in IEUs and cytomegalovirus ulcera

10 system, we show comprehensive microscopy of esophageal mucosa and of coronary arteries in vivo.

11 A, MGMT, and TIMP3) of methylation in normal esophageal mucosa and stomach, or the infrequent methyla

12 led to esophagitis, marked thickening of the esophageal mucosa, and enhanced expression of COX-2.

13 Esophageal mucosa from 3 patients whose ulcers healed or

14 We used electron microscopy on esophageal mucosa from an affected family member carryin

15 issues and in 96% (96/100) of distant normal esophageal mucosa from cancer specimens.

16 e level of TNF-alpha messenger RNA (mRNA) in esophageal mucosa from patients with cytomegalovirus-ass

17 Electron microscopy of squamous esophageal mucosa harboring the S631G variant revealed d

18 gus (BE) and malignant transformation of the esophageal mucosa in mice.

19 Long-term systematic follow-up of the esophageal mucosa including multistaged biopsies is requ

20 ether an esophageal prick test, in which the esophageal mucosa is challenged by local injection of al

21 he biofilm microorganisms suggested that the esophageal mucosa is more permissive to invasion than th

22 not differ significantly between the distal esophageal mucosa of controls (median, 25.5 cell layers

23 ucosa of patients with NERD, from the distal esophageal mucosa of patients with ERD, and the distal-m

24 tions as dilated intercellular spaces in the esophageal mucosa of patients with GERD compared to cont

25 Proximal and distal esophageal mucosa of patients with NERD have more superf

26 s were obtained from the proximal and distal esophageal mucosa of patients with NERD, from the distal

27 pithelium lining the upper two-thirds of the esophageal mucosa possess a non-neuronal cholinergic sys

28 ith extensive alterations of the lingual and esophageal mucosa that were absent in mice given the hwp

29 ial and the sensitivity or resistance of the esophageal mucosa to the reflux material are important f

30 The epithelial turnover time of the healthy esophageal mucosa was approximately 11 days (twice that

31 lammatory mediators S100a8 and S100a9 in the esophageal mucosa with accompanying esophageal epithelia

32 Contact of esophageal mucosa with acid, before inducing heartburn,

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