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1 t a useful predictor of pathologic nocturnal esophageal reflux.
2 after a meal can reduce acidic postprandial esophageal reflux.
3 ophagus, an otherwise benign complication of esophageal reflux, affects approximately three million A
4 phageal motility abnormalities, and proximal esophageal reflux among patients with end-stage lung dis
5 ngeal reflux at least once a day and/or high esophageal reflux at least 5 times a day, subsequently u
6 sed as a pathophysiological factor in gastro-esophageal reflux disease (GERD) and as a target for GER
7 ty-four-hour pH monitoring documented Gastro-Esophageal Reflux Disease (GERD) in 53.4% of patients.
13 f comorbidities is similar except for gastro-esophageal reflux disease and dyslipidemia that appear t
14 able after both procedures except for gastro-esophageal reflux disease and dyslipidemia, which were m
17 sensitive cough reflex (P = .03) but similar esophageal reflux exposure and erosive disease, together
19 We have previously shown that gastroduodenal-esophageal reflux (GDER) together with N-methyl-N-benzyl
21 ility that the high prevalence of pathologic esophageal reflux might simply have resulted from calcul
22 ccurring 1 or more times per day and/or high-esophageal reflux occurring 5 or more times per day.
24 flux episodes and the percentage of proximal esophageal reflux off-PPI did not change significantly a
25 to detect laryngopharyngeal reflux and high-esophageal reflux (reflux 2 cm distal to the upper esoph
26 fects, severe hypotonia, pathological gastro-esophageal reflux, retinal disease, and sinus-node dysfu
28 may falsely attribute exertional dyspnea and esophageal reflux to asthma, leading to excess rescue me
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