ライフサイエンスコーパス: esophageal sphincter

1 sensors located 5 and 20 cm above the lower esophageal sphincter).

2 outcomes (66%) after treatment of the lower esophageal sphincter.

3 f a new magnetic device to augment the lower esophageal sphincter.

4 uction of the abdominal segment of the lower esophageal sphincter.

5 ion, and electrical stimulation of the lower esophageal sphincter.

6 d 3, 6, 9, 12, 15, and 18 cm above the lower esophageal sphincter.

7 incter but an excitatory effect on the upper esophageal sphincter.

8 wed a high prevalence of a hypotensive lower esophageal sphincter (55%) and impaired esophageal peris

9 erized by incomplete relaxation of the lower esophageal sphincter and a loss of normal peristaltic ac

10 of one or more bundles entering at the lower esophageal sphincter and coursing to the forestomach, wh

11 esophagogastric junction contributors (lower esophageal sphincter and crural diaphragm) during deglut

12 geal reflux (reflux 2 cm distal to the upper esophageal sphincter) and ARS.

13 resolution manometry to determine UES, lower esophageal sphincter, and intraesophageal pressure value

14 Lower esophageal sphincter augmentation through injection of m

15 They have an inhibitory effect on the lower esophageal sphincter but an excitatory effect on the upp

16 s was associated with deterioration of lower esophageal sphincter characteristics and increased esoph

17 se were compared between groups (i.e., lower esophageal sphincter characteristics, esophageal acid ex

18 volume into the pharynx 2 cm above the upper esophageal sphincter, directed posteriorly.

19 ker esophagus at 2 and 10 cm above the lower esophageal sphincter during wet swallow.

20 Botulinum toxin injection into the lower esophageal sphincter is an established therapy for the t

21 tulinum toxin (BOTOX) injection of the lower esophageal sphincter largely have replaced cardiomyotomy

22 Separation of the lower esophageal sphincter (LES) and crural diaphragm was quan

23 those who believed in a morphological lower esophageal sphincter (LES) and those who did not.

24 of the esophagus and relaxation of the lower esophageal sphincter (LES) are mediated by nitric oxide

25 In contrast, lower esophageal sphincter (LES) circular muscle maintains spo

26 the physiologic signal transduction of lower esophageal sphincter (LES) circular muscle.

27 The lower esophageal sphincter (LES) in patients with gastroesopha

28 was closer to the upper border of the lower esophageal sphincter (LES) in subjects with a large WC (

29 The motor control of the lower esophageal sphincter (LES) is critical for normal swallo

30 ified adrenergic receptor sites in the lower esophageal sphincter (LES) of animals and humans.

31 esophageal acid exposure and increased lower esophageal sphincter (LES) pressure and length based on

32 Responders had higher preoperative low esophageal sphincter (LES) pressure than nonresponders:

33 rgitation after therapy have low or no lower esophageal sphincter (LES) pressure.

34 Isolated lower esophageal sphincter (LES) relaxation associated with be

35 n the pharynx leads to a long-duration lower esophageal sphincter (LES) relaxation that is an importa

36 proposed that nitrergic nerves mediate lower esophageal sphincter (LES) relaxation with intramuscular

37 On the contrary, during transient lower esophageal sphincter (LES) relaxation, longitudinal musc

38 r discrimination seen during transient lower esophageal sphincter (LES) relaxation-associated reflux

39 s antireflux action is that it reduces lower esophageal sphincter (LES) relaxation.

40 fen reduces the incidence of transient lower esophageal sphincter (LES) relaxations.

41 completeness and propagation velocity, lower esophageal sphincter (LES) resting pressure, LES relaxat

42 In cat spontaneous lower esophageal sphincter (LES), tone is maintained by the ac

43 ic sphincter augmentation (MSA) on the lower esophageal sphincter (LES).

44 and hyoid muscles, esophagus, and the lower esophageal sphincter (LES).

45 veal the anatomical counterpart of the lower esophageal sphincter (LES).

46 onsisting of 24-hour esophageal pH and lower esophageal sphincter manometry.

47 We have previously used the normal lower esophageal sphincter (N-LES) of human organ donors to ex

48 Botulinum toxin injection into the lower esophageal sphincter of patients with achalasia results

49 ossopalatal junction and the timing of upper esophageal sphincter opening relative to glossopalatal j

50 5 cm above the proximal border of the lower esophageal sphincter (PBLES) as appropriate to the locat

51 nce classically manifests as a reduced lower esophageal sphincter pressure (LESP) and loss of distal

52 esistance, 5 cm H2O/L/sec), adjustable lower esophageal sphincter pressure (LESP) and simulated stoma

53 nometry studies revealed a decrease in lower esophageal sphincter pressure (LESP) from 37 +/- 1 mm Hg

54 No significant changes in lower esophageal sphincter pressure and esophageal peristalsis

55 al acid exposure, esophagitis, resting lower esophageal sphincter pressure and medication use.

56 36% decrease in baseline mean resting lower esophageal sphincter pressure in the gastric band group

57 chniques with respect to postoperative lower-esophageal sphincter pressure or postoperative dysphagia

58 extending 5.5 cm proximal to the peak lower esophageal sphincter pressure point was increased by app

59 tion, proton pump inhibitor (PPI) use, lower esophageal sphincter pressure, and patient satisfaction

60 ccording to the size of hiatal hernia, lower esophageal sphincter pressure, Barrett esophagus, and si

61 Life scores, esophageal acid exposure, lower esophageal sphincter pressure, number of beads (size) of

62 The lower esophageal sphincter pressure, the motility of the esoph

63 c pressure, and a reduction in resting lower esophageal sphincter pressure.

64 Lower esophageal sphincter pressures had also returned to norm

65 ere reduced from 7 to 1 (P < .001) and lower esophageal sphincter pressures were reduced from 28 to 9

66 ignificant improvements in postmyotomy lower esophageal sphincter profiles.

67 vulnerability to reflux and transient lower esophageal sphincter relaxation (tLESR) during gastric d

68 y disorder characterized by incomplete lower esophageal sphincter relaxation and aperistalsis resulti

69 lux disease (e.g. vomiting, disordered lower esophageal sphincter relaxation and gastric accommodatio

70 Transient lower esophageal sphincter relaxation is central to the pathog

71 Transient lower esophageal sphincter relaxation is the main mechanism fo

72 The transient lower esophageal sphincter relaxation nadir also typically occ

73 The transient lower esophageal sphincter relaxation onset invariably precede

74 r this patient population is transient lower esophageal sphincter relaxation reducers.

75 assessed the relationship of transient lower esophageal sphincter relaxation to gastroesophageal junc

76 ardinal feature of achalasia, impaired lower esophageal sphincter relaxation, can occur in several di

77 ally for the duration of the transient lower esophageal sphincter relaxation.

78 ergic pathways in modulating transient lower esophageal sphincter relaxation.

79 unction (EGJ) opening during transient lower esophageal sphincter relaxations (tLESRs) using high-res

80 Transient lower esophageal sphincter relaxations and hiatal hernias have

81 sion receptors in triggering transient lower esophageal sphincter relaxations and subsequent reflux.

82 GERD; (2) the mechanism for transient lower esophageal sphincter relaxations and their association w

83 hough there is evidence that transient lower esophageal sphincter relaxations are neurally mediated,

84 dose-dependent inhibition of transient lower esophageal sphincter relaxations in a dog model.

85 Rather, transient lower esophageal sphincter relaxations must occur before the g

86 Termination of transient lower esophageal sphincter relaxations occurred about the time

87 er school of thought is that transient lower esophageal sphincter relaxations result from gastric dis

88 a refute the hypothesis that transient lower esophageal sphincter relaxations result from passive mec

89 Transient lower esophageal sphincter relaxations were not increased by t

90 of compounds that can reduce transient lower esophageal sphincter relaxations.

91 The lower esophageal sphincter shows normalized pressures and rela

92 Normal responses of the upper esophageal sphincter (UES) and esophageal body to liquid

93 eal junction, laryngeal vestibule, and upper esophageal sphincter (UES) and intraluminal pharyngeal d

94 Esophago-upper esophageal sphincter (UES) contractile reflex (EUCR) and

95 the effect of laryngeal stimulation on upper esophageal sphincter (UES) pressure and to determine the

96 tudies of the pressure response of the upper esophageal sphincter (UES) to simulated or spontaneous g

97 detailed manometric assessment of the upper esophageal sphincter (UES).

98 rdings were obtained at 2 cm above the lower esophageal sphincter under 2 study conditions in normal

99 A hypertensive lower esophageal sphincter was diagnosed in three patients, di

100 megaesophagus and achalasia, and their lower esophageal sphincter was resistant to nitric oxide-induc

101 t treatments focused on disrupting the lower esophageal sphincter with pneumatic dilation (70%-90% ef