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1 mens, human esophageal cell lines, and mouse esophagi.
2 ning in ZD esophagi compared with that in ZS esophagi.
3 were detected in the suprabasal layers of ZD esophagi.
4 enes in the PEITC + NMBA versus NMBA-treated esophagi.
5 ragastric zinc compared with zinc-sufficient esophagi.
6 -deficient esophagus and in zinc-replenished esophagi after treatment with intragastric zinc compared
7 expressed in the NMBA-treated versus control esophagi and 1,936 genes in the PEITC + NMBA versus NMBA
8 y that many laparoscopic patients with short esophagi are unrecognized and perhaps treated inappropri
9 tly, tumors were consistently observed in ZD esophagi at very early time points.
10 hosphorylated forms of Rb was detected in ZD esophagi by immunoblotting.
11 ng cell nuclear antigen was characterized in esophagi by immunohistochemistry at 0, 24, and 48 h, and
12 6ink4a showed reduced nuclear staining in ZD esophagi compared with that in ZS esophagi.
13 cial and transient infections of tongues and esophagi (detected by histology) at 1 to 2 weeks after o
14 ptosis accelerator, was markedly stronger in esophagi from Zn-/DFMO+ animals that showed increased ap
15         Fifty one RNAs, comprising 24 normal esophagi (NE), 18 BEs, and nine EACs were hybridized to
16 ell nuclear antigen (PCNA) expression in the esophagi of NMBA treated cyclin D1 mice.
17 y identified 2,261 dysregulated genes in the esophagi of rats that received a 1-week exposure to the
18 n, morphometric changes were observed in the esophagi of rats treated with N-nitrosomethylbenzylamine
19 udy evaluating the morphological features of esophagi resected for endstage achalasia showed marked d
20 was 88% in ZD rats with highly proliferative esophagi versus 0% in ZS rats.
21 h keratinized and nonkeratinized surfaces of esophagi were colonized to a considerably greater extent
22 re sacrificed 24 h after the last treatment; esophagi were excised and processed for histologic gradi
23 S) as well as the expression of c-Jun in the esophagi, were evaluated to investigate the mechanism(s)

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