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1 en oxidation in both free acid and ibuprofen ester form.
2 and most of the Ubc2 was found in the thiol ester form.
3 cited state for the p-coumaroyl and caffeoyl ester forms.
4 itatively converts carboxylic acids to their ester forms.
5 ll as their acetyl, p-coumaroyl and caffeoyl ester forms.
6 sed inhibitor, and the phosphate/phosphonate esters form adducts that mimic intermediates formed in r
11 ss ratios of cholesterol ester to sitosterol ester formed by ACAT1 and ACAT2 were 1.6 and 7.2, respec
12 y unusual hydrolysis pathway from a putative ester, formed by the conjugation of raloxifene diquinone
13 The hydrosilyl-containing alkyl boronate esters formed by this method undergo transformations sel
15 on of alpha,beta-enals to racemic alpha-keto esters, forming gamma-butyrolactones with three contiguo
16 Reductive conversion of the protected amino esters forms highly functionalized cyclopentyl beta-amin
17 rapidly taken up and converted to its inert ester form in peripheral tissues, such as lung, whereas
19 ion of a quinone structure, while the phenyl ester formed in bulk polymerization reactions was not de
20 nitroxide with the B-n-butylcatecholboronate ester formed in situ from urushiol and B-n-butylboronic
21 or confirmation of the identity of TAA fatty esters formed in BEAS-2B cells was obtained via selected
22 ions are reported of the heteroaryl boronate esters formed in situ, demonstrating the usefulness of t
23 ecificity by first analyzing the cholesteryl esters formed in the presence of symmetric PCs labeled a
24 dition, (1)H NMR data suggest that the ethyl esters form inclusion complexes with beta-cyclodextrin.
26 report the synthesis of cyclophanes 18-20 by ester-forming macrocyclization reactions of diols 15 and
30 naline, OAG, PDBu, CPA and the acetoxymethyl ester form of BAPTA (BAPTA-AM) was markedly inhibited by
32 ng intracellular Ca2+ with the acetyl methyl ester form of BAPTA, shortened the action potential and
33 e attributed to the use of the acetoxymethyl ester form of fura-2 to report [Ca2+]i, and that the ste
35 et or cat were loaded with the acetoxymethyl ester form of indo-1 (indo-1 AM) such that approximately
37 creased approximately twofold, and the thiol ester form of Ubc1 increased approximately threefold on
40 the proportions of Ubc3 and -7 in the thiol ester form were significantly higher in the bFGF-treated
41 and glycoside forms and syringic acid in the ester form were the major phenolic acids, and the major
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