ライフサイエンスコーパス: estradiol

1 0 (no activity) to 1 (bioactivity of 17beta-estradiol).

2 of PCOS phenotype induced by testosterone or estradiol.

3 rol mice but was unchanged by time of day or estradiol.

4 in Met carriers but only in the presence of estradiol.

5 gulated during the reproductive cycle and by estradiol.

6 pients, including elevated levels of AMH and estradiol.

7 campal function are selectively modulated by estradiol.

8 alpha-AF2 in the metabolic actions of 17beta-estradiol.

9 primary and metastatic tumors in response to estradiol.

10 g mechanisms from ERalpha, ERbeta, or 17beta-estradiol.

11 ted mechanisms or via local aromatization to estradiol.

12 endent CTSD mRNA upregulation in response to estradiol.

13 catalyzes the conversion of testosterone to estradiol.

14 depressive symptoms during open-label use of estradiol.

15 gh levels of testosterone and its byproduct, estradiol.

16 inhibit its function, an effect mediated by estradiol.

17 mm, K20), in nociceptors incubated with beta-estradiol.

18 e nucleus when MCF7 cells are incubated with estradiol.

19 e were implanted with trenbolone acetate and estradiol.

20 RH neurons to be activated by kisspeptin and estradiol.

21 35-mer aptamer for a small molecule, 17beta-estradiol.

22 domly assigned to receive either oral 17beta-estradiol (1 mg per day, plus progesterone [45 mg] vagin

23 The animals received beta-estradiol 17-valerate once a week and were treated daily

24 he animals were treated with 100 mug/kg beta-estradiol 17-valerate or vehicle (control) over 7 and 28

25 Interestingly, the endobiotic estradiol-17-glucuronide and the xenobiotic indomethacin

26 rated that chronic exposure to low levels of estradiol-17beta (E2) increases mean arterial pressure (

27 Estradiol-17beta (E2) upregulates PF formation in develo

28 Lhcgr, Cyp11a1, Hsd17b1, and Pappa mRNAs and estradiol-17beta production.

29 16alpha-(18)F-fluoro-17beta-estradiol ((18)F-FES) is a PET tracer for ER with relati

30 vels (-4.9%; 95% CI: -8.7, -0.8%); PFOS with estradiol (-4.0%; 95% CI: -7.7, -0.1%), testosterone (-5

31 In the absence of exogenous estradiol, a TD by ACE interaction was observed on BOLD

32 diol production via the aromatase enzyme and estradiol action via the alpha isoform of the estrogen r

33 Estradiol acts via estrogen receptor alpha (ERalpha)-exp

34 ound that the protection conferred by 17beta-estradiol against obesity and insulin resistance require

35 T binding, indicating a protective effect of estradiol against SERT loss.

36 r prodrug of the main human estrogen, 17beta-estradiol, alleviates hot flushes in rat models of therm

37 Two different ERalpha ligands, estradiol and 27HC, work together to promote EMH during

38 xidizes the hydroxyl group at position 17 of estradiol and 5-androstenediol using NAD(+) as cofactor.

39 17beta-HSD14 is a SDR enzyme able to oxidize estradiol and 5-androstenediol using NAD(+).

40 te evidence for association to the change in estradiol and allopregnanolone over the course of pregna

41 Endogenous estradiol and estrone are linked causally to increased r

42 or some low polarity analytes including both estradiol and estrone.

43 and 64% (P = 0.0009) when administered with estradiol and genistein, respectively.

44 These data indicate that 17beta-estradiol and GPER independently regulate hippocampal me

45 Lower estradiol and LH levels were found in urine and saliva s

46 mass indices and lower levels of endogenous estradiol and luteinizing hormone.

47 Notably, the interactions between estradiol and PFASs were significant for PFOS (p = 0.026

48 Modeling intra-individual fluctuations in estradiol and progesterone may provide unique insight in

49 investigated the influence of testosterone, estradiol and progesterone on initiation and maintenance

50 In addition, the differential effects of estradiol and progesterone on these uterine leiomyoma su

51 Long-term administration of both estradiol and testosterone in mice can result in prostat

52 To study the associations of endogenous estradiol and testosterone with carotid plaque compositi

53 To test the hypotheses that estradiol and time of day signals alter GnRH neuron resp

54 while their affinities for estrogen agonist (estradiol) and antagonist (4-hydroxytamoxifen) are reduc

55 re we show that physiologic estrogen (17beta-estradiol) and progesterone reciprocally regulate melani

56 ether removal of natural estrogens (estrone, estradiol, and estriol) and overall SSF performance for

57 In WTP influents, estrogens (estrone, 17beta-estradiol, and estriol), androgens (androstenedione, and

58 Complex phenol donors tyrosine, estradiol, and griseofulvin follow the predictive model.

59 ha protein levels, reduced responsiveness to estradiol, and reduced growth rates.

60 od to estimate probability distributions for estradiol- and time of day-dependent parameters was used

61 agonist, leuprolide acetate; leuprolide plus estradiol; and leuprolide plus progesterone.

62 Using [2, 4, 6, 7 - (3)H]estradiol as an example compound, this protocol uses sol

63 by AR activation, and potently stimulated by estradiol as well as estrogenic metabolites of 5alpha-di

64 ed), including the hormones testosterone and estradiol as well as steroidal drugs.

65 orfenicol, pyrimethamine, estrone and 17beta-estradiol at levels from 0.095 to 2.7 mug/kg.

66 erved with placebo treatment, treatment with estradiol attenuated the effects of ACE and TD such that

67 Estradiol-based contraceptives and hormonal replacement

68 osterone propionate (Testos; n = 10), 0.5 mg estradiol benzoate (E2; n = 10), or vehicle (control gro

69 r ovariectomy, and administration of 17-beta-estradiol benzoate (EB) restored this escalated anxiety-

70 of research and monitoring have been 17beta-estradiol (beta-E2) and 17alpha-ethinylestradiol, due bo

71 a4beta2-selective positive modulators 17beta-estradiol (betaEST) and desformylflustrabromine (dFBr),

72 Ralpha (231 G521R ER), which is defective in estradiol binding.

73 In contrast, estradiol but not G1 increased Akt phosphorylation level

74 Aqueous micropollutants such as estradiol can have a large environmental impact-even at

75 Across women, lower 17beta-estradiol concentrations were related to more pronounced

76 ts regarding the mechanisms by which loss of estradiol contributes to this vulnerability is lacking.

77 17beta-estradiol-D5 was used as internal standard.

78 Estradiol deficiency disrupted cortical microarchitectur

79 suggesting effects of both testosterone and estradiol deficiency.

80 Neutrophils treated with 17beta-estradiol demonstrated an enhanced oxidative burst but d

81 en together, these findings point to a novel estradiol-dependent pathway that modulates cocaine-induc

82 at lesions of the mPOA or microinjections of estradiol directly into the mPOA increased cocaine-induc

83 Estradiol disrupts the Cxcl1 gradient and favors neutrop

84 cutive function that are unmasked by loss of estradiol during menopause.

85 es indicates that the gonadal hormone 17beta-estradiol (E(2)) impacts the structure and function of t

86 als were compared with OVX animals receiving estradiol (E) alone or E with progesterone (P).

87 at ovariectomized (Ovx) monkeys treated with estradiol (E) for 28 days supplemented with placebo or p

88 r a subcutaneous sham implant (OVX), 17-beta estradiol (E) implant (OVX+E) or E implant plus cyclic o

89 TOT classically antagonizes estradiol (E2) -dependent breast cancer cell growth, but

90 RalphaKO mice exhibited elevated circulating estradiol (E2) acting on E2-responsive tissue/cells such

91 Estradiol (E2) acutely potentiates glutamatergic synapti

92 We found recently that 17beta-estradiol (E2) acutely suppresses GABAergic inhibition i

93 17beta-estradiol (E2) also influences hypothalamic programming

94 ations and systemic administration of 17beta-estradiol (E2) alter spine density in the dorsal hippoca

95 found in treated and natural waters, 17beta-estradiol (E2) and 17alpha-ethynylestradiol (EE).

96 We investigated the potential of 17beta-estradiol (E2) and estrogen receptor (ER) signaling for

97 pared to male rats, and that ovarian-derived estradiol (E2) and progesterone (P4) are essential for t

98 he same consultations blood plasma levels of estradiol (E2) and thyroid hormones (TSH, T3t, T4t) were

99 transcriptional effects of crosstalk between estradiol (E2) and TNFalpha, identifying a large set of

100 enzthiazoline-6-sulfonate) (ABTS) and 17beta-estradiol (E2) as the probing substrates.

101 Estradiol (E2) can act in the brain in a relatively fast

102 examined whether the steroid hormone, 17beta-estradiol (E2) can exert long-lasting beneficial effects

103 ponses in female mice while pioglitazone and estradiol (E2) co-treatment ameliorated TFH cells and GC

104 17-beta-estradiol (E2) enhanced responsiveness of BMSCs of wild-

105 The sex-steroid hormone estradiol (E2) enhances the psychoactive effects of coca

106 ted by an immunospecific detection of 17beta-Estradiol (E2) following the competitive inhibition form

107 Paradoxically, agonists at ERalpha such as estradiol (E2) have demonstrated clinical efficacy in pa

108 e measured serum androstenedione (A4), T and estradiol (E2) in both sexes and social classes, during

109 s have focused on the role of female sex and estradiol (E2) in pulmonary arterial hypertension (PAH),

110 d that KA-induced SE stimulates synthesis of estradiol (E2) in the hippocampus, a brain region common

111 adipocyte lineage was suppressed by 17-beta-estradiol (E2) in WT female mice but not in NOER or MOER

112 Although systemically injected 17beta-estradiol (E2) increases CA1 dendritic spine density, th

113 ChIP assay demonstrated estradiol (E2) induced ESR1 binding to Pck-1, G6Pase, Fa

114 We found 17beta-estradiol (E2) inhibited hepatic gluconeogenic genes suc

115 Estradiol (E2) is a steroid hormone that negatively affe

116 eading to pLTF are well studied in males and estradiol (E2) is known to be required, it has seldom be

117 during estrous cycle stages in which 17beta-estradiol (E2) is naturally high (e.g., proestrus vs est

118 The inaccuracy of 17-beta estradiol (E2) measurements affects its use as a biomark

119 Stereoisomers of estradiol (E2) or trenbolone (TB) can occur together in

120 Estradiol (E2) perfusion rapidly increases the strength

121 Here we have assessed the role that estradiol (E2) plays in regulating the dynamics of GABAA

122 at it may have contributed to a reduction in estradiol (E2) production.

123 It is known that 17-beta estradiol (E2) regulates adipose tissue function and VEG

124 positive feedback effects of ovarian 17beta-estradiol (E2) regulating release of gonadotropin releas

125 Exposure to 0.1 pM, 10 pM, and 1 nM 17 beta-estradiol (E2) resulted in monotonic inhibition of mamma

126 We previously found that 17beta-estradiol (E2) stimulates apolipoprotein A-IV (apoA-IV)

127 The ability of 17beta-estradiol (E2) to enhance hippocampal object recognition

128 We have previously reported that 17beta-estradiol (E2) treatment activates Notch signaling in EC

129 Here we show that 17beta-estradiol (E2) up-regulates total CaM level in endotheli

130 based chemotherapy, had postmenopausal serum estradiol (E2), and had received tamoxifen for >/= 1 yea

131 the enzyme converting testosterone to 17beta-estradiol (E2), contributes to the regulation of this pl

132 25% of patients had persistent elevation in estradiol (E2), defined as E2 greater than 10 pg/mL (to

133 Estrogen, 17beta-estradiol (E2), is a powerful therapeutic agent that pla

134 ses the conversion of testosterone to 17beta-estradiol (E2), thereby reducing E2-dependent vitellogen

135 ization reagent for the measurement of serum estradiol (E2), with simultaneous analysis of underivati

136 istrome that substantially overlaps with the estradiol (E2)-dependent ERalpha cistrome.

137 ndent synaptic plasticity and memory, 17beta-estradiol (E2).

138 em by interacting with the endogenous ligand estradiol (E2).

139 tase, which produces the neurosteroid 17beta-estradiol (E2).

140 estrus levels of the primary estrogen 17beta-estradiol (E2; 10 mug/kg, i.p., 1-h pretreatment) nor a

141 ter exposure to two concentrations of 17beta-estradiol (E2beta; 2 ng/L and 50 ng/L) during four disti

142 (BPA) and the pharmaceutical 17alpha-ethinyl estradiol (EE) are synthetic chemicals with estrogen-lik

143 e bioavailability and bioactivity of ethinyl estradiol (EE2) sorbed onto SWCNTs in a fish gastrointes

144 In avian systems, rapid estradiol effects can be mediated via local alterations

145 However, whether estradiol enhances the neurochemical response to cocaine

146 Prediagnostic concentrations of estradiol, estrone, and 13 metabolites were measured in

147 Four steroidal estrogens (17beta-estradiol, estrone, estriol, and 17alpha-estradiol) were

148 Estradiol, estrone, testosterone, luteinizing hormone (L

149 We studied how estradiol feedback regulates GnRH excitability, a key de

150 Estradiol feedback regulates gonadotropin-releasing horm

151 tropic glutamate receptors are important for estradiol feedback, but it is not known where they fit i

152 Initial open-label treatment with estradiol followed by randomized, double-blind, placebo-

153 Release of estradiol from the drug-polyketal conjugate microparticl

154 acebo group versus 0.0044 mm per year in the estradiol group (P=0.008).

155 nificantly between the placebo group and the estradiol group in either postmenopause stratum.

156 rates of CIMT progression in the placebo and estradiol groups were similar (0.0088 and 0.0100 mm per

157 Estradiol had no significant effect on cardiac CT measur

158 Although 17beta-estradiol has long been known to regulate memory functio

159 release was stimulated with optimal doses of estradiol, IL-1, and TNF-alpha (10 ng/mL) from 15 minute

160 her with the facts that they antagonize beta-estradiol in a functional assay in MCF-7 human breast ca

161 cant association was identified for PFNA and estradiol in control group.

162 lated bacteria, and those changes induced by estradiol in its signalling at the single cell level.

163 R-beta), and antagonize the activity of beta-estradiol in MCF-7 human breast cancer cells.

164 BPS also has potencies similar to that of estradiol in membrane-mediated pathways, which are impor

165 gesterone in controls only, and decreased by estradiol in PMDD LCLs.

166 sets for annual changes in gonadotropins and estradiol in rainbow trout.

167 uct a voltammperometric biosensor for 17beta-estradiol in the 0.9-11 pM range.

168 This study revealed that 17beta-estradiol in the brain mediated the physiological action

169 eizures by promoting the synthesis of 17beta-estradiol in the brain.

170 the expression of P450arom, but also 17beta-estradiol in the cerebral cortex.

171 support the notion of an important role for estradiol in the etiology of male breast cancers, simila

172 r data imply both serotonergic signaling and estradiol in the mechanisms by which sex-steroid hormone

173 d calcium transients in the presence of beta-estradiol, in an IP3 receptor-dependent manner.

174 ha-positive breast tumors and treatment with estradiol increased SUSD3 expression in ERalpha-positive

175 Treatment with estradiol increased the trabecular and cortical bone mas

176 w that treating mice with estradiol to model estradiol increases during pregnancy induced HSC prolife

177 hasone inducible vector pOpOff2 and into the estradiol induced vector pER8.

178 Some estradiol-induced changes observed in previous studies w

179 hether the suppressive effects of HCD on the estradiol-induced GnRH/LH surge were overcome by neuron-

180 ary vascularization over time in response to estradiol-induced prostatic enlargement.

181 Overexpression of FUS3 driven by an estradiol-inducible promoter increased oil contents in A

182 hreshold for synaptic plasticity when 17beta-estradiol is elevated.

183 The steroid 17beta-estradiol is known to acutely potentiate glutamatergic s

184 17beta-estradiol led to a significant upregulation in pentose p

185 were percent change from baseline in 17beta-estradiol levels (E2) and tricuspid annular plane systol

186 positively associated with net decreases in estradiol levels (p = .02) from baseline within the GnRH

187 Estradiol levels above 10 pg/ml and testosterone levels

188 450arom inhibitor, letrozole, reduced 17beta-estradiol levels and completely suppressed the elongatio

189 atics, PFASs were positively associated with estradiol levels and negatively associated with testoste

190 the pubertal rise and premenopausal fall of estradiol levels and results in the obstetrically most a

191 ce, and suggest approaches to restore 17beta-estradiol levels as a novel treatment option for SERT de

192 he Grady Trauma Project and found that serum estradiol levels associates with DNA methylation across

193 Increases in estradiol levels correlated negatively with decreases in

194 meostasis in adult men, and testosterone and estradiol levels must decline substantially to impact th

195 e largely unrelated to risk, but circulating estradiol levels showed a significant association.

196 Serum estradiol levels were inversely associated with fat atte

197 a1 suppression, decreased circulating 17beta-estradiol levels, abnormal fat accumulation, and glucose

198 1) expression and reduced circulating 17beta-estradiol levels.

199 nd chemical formulas similar to estrone- and estradiol-like compounds.

200 Thus, the anti-oxidative effects of 17beta-estradiol may be involved in the prevention of seizures

201 unmodified breast cancer cell lines blocked estradiol-mediated CTSD induction, inhibited growth in s

202 gions contained genes known to interact with estradiol metabolism and cancer.

203 Finally, estradiol microinjections followed by microdialysis were

204 ulture of isolated uterine ILC2s with 17beta-estradiol modified expression of a number of genes.

205 he molecular mechanisms through which 17beta-estradiol modulates hippocampal memory.

206 A switch from estradiol negative to positive feedback initiates the Gn

207 Estradiol-negative feedback decreased glutamatergic tran

208 Moreover, during the ovulatory phase (high estradiol), neutrophil numbers decrease in the vaginal l

209 The acute vasodilatory effects of 17beta-estradiol (non-specific estrogen receptor (ER) agonist),

210 = 0.68) when administered in the presence of estradiol (nonselective) and genistein (ERbeta-selective

211 ed with vehicle or various concentrations of estradiol (nonspecific ER agonist) or genistein (ERbeta-

212 aromatase inhibitors that lower circulating estradiol occurs in up to 50% of patients, generally lea

213 erize the effects of DHEA, prolactin, 17beta-estradiol on insulin-growth factor-1 and -2 (IGF-1, -2)

214 There was an interaction between Patch and estradiol on NA.

215 The effect of estradiol on transcriptional activity was dependent on t

216 nd Ki values was observed in the presence of estradiol or genistein.

217 4+ neurons, cultured in the presence of beta-estradiol or PPT.

218 irecting group furnishes 2-arylated estrone, estradiol, or estriol depending on the method used.

219 Dysregulation of the PGE2-estradiol pathway during the second week by treatment wi

220 Microparticles were prepared from the estradiol-polyketal conjugate, where estradiol was incor

221 its use to synthesize high molecular weight estradiol-polyketal conjugates by addition polymerizatio

222 ific agonist, and to a lesser extent 17alpha-estradiol, possibly acting through ER-X, prevented this

223 hat neonatal testosterone treatment, but not estradiol, produces histological changes in female rat l

224 findings suggest that the decline in ovarian estradiol production during menopause plays a significan

225 glandin production and activity but also for estradiol production via the aromatase enzyme and estrad

226 ptomatic) during the first month of combined estradiol/progesterone compared with the last month of l

227 The findings demonstrate that the change in estradiol/progesterone levels from low to high, and not

228 trual Tension scores in the second and third estradiol/progesterone months did not significantly diff

229 bo month, and the second and third months of estradiol/progesterone.

230 ebo and then 3 months of continuous combined estradiol/progesterone.

231 placebo month, or second and third months of estradiol/progesterone.

232 Estradiol provision via neural aromatization decreases n

233 Estradiol rapidly (within 30 minutes) influences a varie

234 xy for individuals' prenatal testosterone-to-estradiol ratio, second-to-fourth digit-ratio (2D:4D rat

235 wer) progesterone levels and progesterone to estradiol ratios were associated with reducing smoking u

236 Ovarian estradiol regulates the pattern of GnRH (negative feedba

237 ased GnRH excitability and was essential for estradiol regulation of excitability.

238 intravaginal testosterone cream (IVT) or an estradiol-releasing vaginal ring (7.5 mug/d) in patients

239 17beta-estradiol replacement in SERT (-/-) mice reversed the ob

240 d ERbeta and demonstrate that the window for estradiol's beneficial effects on memory and hippocampal

241 Characterizing estradiol's role in stress circuitry in vivo in humans m

242 tion between estrus cycle-related changes in estradiol secretion and BDNF Val(66)Met genotype on meas

243 The predicted estradiol serum concentrations (convoluted from in vitro

244 rs), including serum levels of testosterone, estradiol, sex hormone binding globulin, and androstened

245 odel, we review our current understanding of estradiol signaling in the regulation of sexual receptiv

246 VI1-silenced cells, suggesting that EVI1 and estradiol signaling merge in MAPK activation.

247 f these neurons and neuroendocrine output by estradiol.SIGNIFICANCE STATEMENT The brain regulates fer

248 However, estradiol significantly decreased NF-kappaB transcriptio

249 In contrast, 17beta-estradiol significantly increased the abundance of trans

250 riectomized females supplemented with 17beta-estradiol succumbed to P. aeruginosa challenge earlier t

251 le mice, while the effect was abrogated with estradiol supplementation, suggesting that the sex-diffe

252 We questioned the effect of E alone or E+P (estradiol supplemented with progesterone) on gene expres

253 Above 10% inhibition of estradiol synthesis by aromatase inhibitors, noticeable

254 izures, focusing on the regulation of 17beta-estradiol synthesis in the brain.

255 age without or with adult testosterone plus estradiol (T+E) to promote carcinogenesis.

256 Although implantation of testosterone and estradiol (T+E2) pellets for 2 months in wild-type mice

257 At baseline, total estradiol (TE) and total testosterone (TT) were measured

258 sociated with the risk of T2D, whereas total estradiol (TE) was associated with increased risk of T2D

259 arent from the absence of interferences from estradiol, testosterone and progesterone.

260 e and removed significantly more estrone and estradiol than nonaugmented filters.

261 ly, the threshold levels of testosterone and estradiol that initiate bone loss are uncertain.

262 rence may be regulated by estrogens, such as estradiol, that are synthesized in the spinal cord and b

263 Like 17beta-estradiol, the non-steroid Br-PBTC only requires one alp

264 Women with past PMD who continued estradiol therapy and all women in the control group rem

265 the development of a brain-selective 17beta-estradiol therapy to relieve hot flushes without undesir

266 Oral estradiol therapy was associated with less progression o

267 ffects that were similar to those induced by estradiol, there were some notable differences, includin

268 Serum estradiol, thyroid hormones, and urinary free cortisol l

269 Here we show that treating mice with estradiol to model estradiol increases during pregnancy

270 VMH PI3K activity blocked effects of 17beta-estradiol to stimulate energy expenditure, but did not a

271 ignificant while adjusting for sex hormones (estradiol-to-progesterone ratio in women and testosteron

272 consumed, the geometric mean total and free estradiol, total and free testosterone, and luteinizing

273 the relationship between levels of PFAS and estradiol, total testosterone, and IGF-1 in 2,292 childr

274 tion (IVIVC) for drug-in-adhesive (DIA) type estradiol transdermal drug delivery systems (TDDS).

275 s occurs on a daily basis in ovariectomized, estradiol-treated (OVX+E) mice; GnRH neurons are suppres

276 Estradiol-treated MCF7 cells stably expressing PR-B exhi

277 17beta-Estradiol-treated ovariectomized female mice demonstrate

278 Cyclic estradiol treatment in aged ovariectomized animals resto

279 led, parallel-design evaluation of continued estradiol treatment was evaluated at an outpatient resea

280 ormal cortical morphology, whereas in males, estradiol treatment, or IL-6 deletion, recapitulates the

281 As Wnt16 expression is enhanced by estradiol treatment, we hypothesized that the bone-spari

282 % loss of MSBs that was restored with cyclic estradiol treatment.

283 is trial was to evaluate safety of IVT or an estradiol vaginal ring in patients with early-stage BC r

284 ido were randomized to 12 weeks of IVT or an estradiol vaginal ring.

285 reater when aromatization of testosterone to estradiol was also suppressed, suggesting effects of bot

286 With TNP, higher-than-usual estradiol was associated with greater decreases in NA.

287 However with PBO, lower-than-usual estradiol was associated with greater decreases in NA.

288 and exemestane, a sensitive assay for serum estradiol was checked and returned at 16 pg/mL (61 pmol/

289 17beta-estradiol was detected below its quantitation limit in a

290 rom the estradiol-polyketal conjugate, where estradiol was incorporated into the polymer backbone.

291 Estradiol was measured at baseline and weeks 4 and 12 us

292 ull estrogen antagonist, while the effect of estradiol was not.

293 Dipeptide conjugates with coprostanol and estradiol were synthesized by this method for potential

294 eta-estradiol, estrone, estriol, and 17alpha-estradiol) were tested in these assays.

295 TAF in MED12-LM proliferated in response to estradiol, whereas progesterone had no effect.

296 o guest molecules, dextromethorphan and beta-estradiol, which are widely found as pollutants in groun

297 ronger among children with higher than lower estradiol, with odds ratios (OR) for asthma ranging from

298 After a median of 5 years, the effect of estradiol, with or without progesterone, on CIMT progres

299 f bisphenol A, triclosan and 17alpha-ethinyl estradiol without generating obviously toxic byproducts.

300 en were hypogonadal, and that treatment with estradiol would attenuate this effect.