ライフサイエンスコーパス: estradiol-17beta

1 received a subcutaneous Silastic implant of estradiol-17beta.

2 The estradiol-17beta abolition of inducible MHC class II ant

3 of nulliparous rats with pregnancy levels of estradiol 17beta and progesterone has high efficacy in p

4 and a secondary 46-kDa protein recognized by estradiol-17beta and by a monoclonal antibody directed t

5 ieved by full-term pregnancy or low doses of estradiol-17beta and progesterone administered for 21 da

6 able to affect sertolin expression, whereas estradiol-17beta and progesterone induced a significant

7 radiol (2-ME) is an endogenous metabolite of estradiol-17beta and the oral contraceptive agent 17-eth

8 Under the same experimental conditions, estradiol-17beta and two of its other metabolites, estri

9 In pulp cell cultures, androstenedione, estradiol-17beta, and HGF each stimulated AR mRNA accumu

10 2 (Mrp2) in mediating cholestasis induced by estradiol-17beta(beta-D-glucuronide) (E(2)17G).

11 istant cells display increased resistance to estradiol-17beta(beta-D-glucuronide) (E217G).

12 gnificant portion of specific, high-affinity estradiol-17beta binding-sites are also enriched in plas

13 Estradiol-17beta caused a 2.5-fold increase in NOS enzym

14 beta-Estradiol 17beta-D-glucuronide (E(2)17G), an endogenous

15 ediated bile acid transport was inhibited by estradiol 17beta-d-glucuronide and fumitremorgin C.

16 te, digoxin, and prostaglandin E2 but not of estradiol 17beta-d-glucuronide or p-aminohippurate.

17 nergized transport of cyclic nucleotides and estradiol 17beta-D-glucuronide.

18 of the OATP-C substrates estrone sulfate and estradiol 17beta-d-glucuronide.

19 using a rapid filtration technique and [(3)H]estradiol-17beta-d-glucuronide ([(3)H]E(2)17betaG) as mo

20 Estradiol-17beta-D-glucuronide (E(2)17G), an endogenous

21 -dependent transport of leukotriene C(4) and estradiol-17beta-d-glucuronide into vesicles derived fro

22 ial display technique, we show, herein, that estradiol-17beta (E(2)) and its catechol metabolite 4-hy

23 nic responses of MCF-7 cell proliferation to estradiol-17beta (E(2)) and zeranol (Z, a nonsteroidal g

24 Estradiol-17beta (E(2)) causes cell proliferation in the

25 17beta-stearate (E(2)-17beta-S) with that of estradiol-17beta (E(2)) in stimulating the growth of mam

26 In the epithelial compartment of the uterus, estradiol-17beta (E(2)) induces cell proliferation while

27 ons are the direct targets for the action of estradiol-17beta (E(2)), which acts via the estrogen rec

28 Estradiol-17beta (E(2), 10(-8) m) and acetylcholine (10(

29 -delayed L:D cycle (pdL:D) were treated with Estradiol-17beta (E) or E and Progesterone (E+P).

30 Estradiol-17beta (E) plays an important role in ovarian

31 nd 1 week later half of the animals received estradiol-17beta (E).

32 nd after incubation with 10(-9) mol/L 17beta-estradiol (17beta-E(2)) for 16 to 18 hours.

33 To investigate the involvement of 17beta-estradiol (17beta-E(2)) in protection against oxidative

34 Estradiol 17beta (E2) rapidly induced morphological chan

35 Estradiol-17beta (E2) acts through the estrogen receptor

36 Estradiol-17beta (E2) and G-1, a GPER-selective agonist,

37 and differentiation under the regulation of estradiol-17beta (E2) and progesterone (P4).

38 and those treated with testosterone (T) and estradiol-17beta (E2) for 16 weeks.

39 i.d., 3.18 mm o.d., 1.5 cm long) containing estradiol-17beta (E2) in peanut oil (150 microg/ml) at 0

40 ed 155 genes whose expression was altered by estradiol-17beta (E2) in three immortalized normal human

41 rated that chronic exposure to low levels of estradiol-17beta (E2) increases mean arterial pressure (

42 Estradiol-17beta (E2) upregulates PF formation in develo

43 Previously, we found that estradiol-17beta (E2) with immunosuppressant cyclosporin

44 A primary estrogen, estradiol-17beta (E2), did not induce this LF response.

45 early in G1 as a physiological inhibitor of estradiol-17beta (E2)-induced epithelial cell proliferat

46 Estradiol-17beta (E2)-induced gene expression of pS2 and

47 capsules (control), one capsule filled with estradiol-17beta (E2; low dose), or three capsules of E2

48 ion of two natural steroid estrogens [17beta-estradiol (17beta-E2) and estrone (E1)] and two syntheti

49 We demonstrate that 17beta-estradiol (17beta-E2) increases cell survival against H2

50 primary neurons, we demonstrate that 17beta-estradiol (17beta-E2) stimulates formation of vesicles c

51 and found that low concentrations of 17beta-estradiol (17beta-E2), 17alpha-E2, 17beta-E2-BSA, and IC

52 ones, 17alpha-estradiol (17alpha-E2), 17beta-estradiol (17beta-E2), and estrone (E1), are routinely d

53 e and after overnight incubation with 17beta-estradiol (17beta-E2, 10(-9) mol/L).

54 Estradiol-17beta (E2beta), a potent vasodilator, has its

55 Exposure to estradiol-17beta (E2beta, 10(-)10 to 10(-)6 M) caused a

56 f three natural estrogenic hormones (17alpha-estradiol, 17beta-estradiol, and estrone) in aqueous sol

57 %, and 77% of the initial amounts of 17alpha-estradiol, 17beta-estradiol, and estrone, respectively.

58 r the first time, that a naturally occurring estradiol-17beta-fatty acid ester has a differential, st

59 ptor (ER)-positive MCF-7 cells abolished the estradiol-17beta growth stimulatory effect that was obse

60 The administration of estradiol-17beta induces pituitary hyperplasia followed

61 In addition to its role in reproduction, estradiol-17beta is critical to the regulation of energy

62 Although estradiol-17beta is known to increase spine density and

63 Although ovarian estrogen, estradiol-17beta, is a key modulator of normal reproduct

64 ERalpha signaling mediates major effects of estradiol-17beta on energy balance, raising the possibil

65 ontrolled trial of estrogen therapy (1 mg of estradiol-17beta per day) in 664 postmenopausal women (m

66 Lhcgr, Cyp11a1, Hsd17b1, and Pappa mRNAs and estradiol-17beta production.

67 this hypothesis, we compared the activity of estradiol-17beta-stearate (E(2)-17beta-S) with that of e

68 es for estrogen may mediate rapid effects of estradiol-17beta that contribute to proliferation of hum

69 lts in changes opposite to those reported in estradiol-17beta-treated mice and suggests that ERalpha

70 has been detected in different tissues, and estradiol-17beta treatment protects against experimental

71 ted interleukin-6 levels in incubations with estradiol-17beta were increased 2500-fold by IL-1beta (P

72 ta and ERgamma fusion proteins can each bind estradiol-17beta with high affinity.