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1 sms other than the action of tamoxifen as an estrogen antagonist.
2 3G), and could be inhibited by tamoxifen, an estrogen antagonist.
3 iated by estrogen, estrogen deprivation, and estrogen antagonists.
4 reporter gene, and was inhibited by specific estrogen antagonists.
5 r in the presence of estrogen metabolites or estrogen antagonists.
6 ity, the antiestrogens then function as pure estrogen antagonists.
7               Evidence is presented that the estrogen antagonist 4-hydroxytamoxifen (HT) can occupy n
8 sphorylation was also induced by the partial estrogen antagonist 4-hydroxytamoxifen, but not by the c
9 hanges were also observed in the presence of estrogen antagonist and were corroborated in the estroge
10 Brg1/Brm node is a major cellular target for estrogen antagonists, and thereby also implicate prohibi
11                                              Estrogen antagonists are universally employed in the bre
12 l group in 4 affords analogues with dominant estrogen antagonist effects in MCF-7 cells.
13 tin structural changes could be repressed by estrogen antagonist ICI 182 780 in MCF-7 cells yet were
14                        Inclusion of the pure estrogen antagonist ICI 182,780 blocked the increase in
15                                Moreover, the estrogen antagonist ICI 182,780 does not inhibit the ant
16 s highly enhanced by estrogen and blocked by estrogen antagonist ICI 182,780.
17  activation and reversed the activity of the estrogen antagonists ICI182780 (ICI) and tamoxifen (TAM)
18  characteristic feature of AF2ER is that the estrogen antagonists ICI182780 and tamoxifen (TAM) act a
19 ved in U2OS cells, SP500263 acts as a strong estrogen antagonist in an MCF-7 breast cancer proliferat
20                   Tamoxifen acts as a strong estrogen antagonist in human breast but as an estrogen a
21 n receptor modulator (SERM) that is a potent estrogen antagonist in mammary and uterine tissue while
22                           While acting as an estrogen antagonist in the breast, tamoxifen can have es
23                     The compound is a potent estrogen antagonist in the rat uterus.
24 with male slices or exposed to estrogen, and estrogen antagonists inhibited song circuit development
25 hromatin modifiers Brg1/Brm are required for estrogen antagonist-mediated growth suppression through
26                        Pretreatment with the estrogen antagonist Nafoxidine blocked this induction.
27 ulted in a substantial (10-fold) increase in estrogen antagonist potency relative to raloxifene in an
28 ues and serum lipids while displaying potent estrogen antagonist properties in the breast and uterus.
29  triarylethylenes to improve ER affinity and estrogen antagonist properties, were prepared with the u
30 naling was prevented and even potentiated by estrogen antagonists such as tamoxifen, suggesting that
31 naling was prevented and even potentiated by estrogen antagonists such as tamoxifen, suggesting that
32               Simultaneous administration of estrogen antagonist tamoxifen (8 mg/kg per day) with E2
33 d controls, whereas females administered the estrogen antagonist tamoxifen prior to stress were not i
34 he absence of estrogen or in the presence of estrogen antagonists tamoxifen or ICI 182780; second, am
35 ependent cellular inhibitory responses to an estrogen antagonist, tamoxifen (TAM), via at least in pa
36 gulatory gene NAP1L1, an adhesin MAGE-D2, an estrogen-antagonist, the metastasis marker MTA1, the apo
37                        In rat uteri they are estrogen antagonists/weak agonists and decrease the conc
38 dditive impact on cell growth, whereas known estrogen antagonists were found to neutralize agonist ef
39 ct of BPA was blocked by Fulvestrant, a full estrogen antagonist, while the effect of estradiol was n
40 ls, whereas resveratrol functioned as a pure estrogen antagonist with T47D and LY2 cells.

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