ライフサイエンスコーパス: estrogen deficiency

1 l as the increase in B cell number caused by estrogen deficiency.

2 is also required for the bone loss caused by estrogen deficiency.

3 atment option for osteoporosis to ameliorate estrogen deficiency.

4 tions of aldosterone as a consequence of the estrogen deficiency.

5 se of infection that are most susceptible to estrogen deficiency.

6 e acquisition was achieved or as a result of estrogen deficiency.

7 k for cardiovascular diseases because of the estrogen deficiency.

8 s and strength in mice with bone loss due to estrogen deficiency.

9 as RA or periodontitis and in conditions of estrogen deficiency.

10 loss caused by inflammation, infection, and estrogen deficiency.

11 are protected from bone loss despite severe estrogen deficiency.

12 otected from the bone loss characteristic of estrogen deficiency.

13 nant of increased bone resorption induced by estrogen deficiency.

14 n are critical to the bone wasting effect of estrogen deficiency.

15 the altered bone turnover characteristic of estrogen deficiency.

16 e in osteoclastic bone resorption induced by estrogen deficiency.

17 ytes is required for the bone loss caused by estrogen deficiency.

18 , trochanter, and total body, despite severe estrogen deficiency.

19 the pathophysiologic changes that occur with estrogen deficiency.

20 BMD in young women with GnRH analog-induced estrogen deficiency.

21 nts that lead to cancellous bone loss during estrogen deficiency.

22 ssion of periodontal disease associated with estrogen deficiency.

23 e in the developed world, is associated with estrogen deficiency.

24 ng behaviors, and osteopenia associated with estrogen deficiency.

25 However, in long-term estrogen deficiency (34 weeks), there was no significant

26 healthy bone cells after short durations of estrogen deficiency (5 weeks), and exceeded the osteogen

27 event the development of glomerulosclerosis, estrogen deficiency accelerated the progression of glome

28 city to adapt to dyslipidemia, hypertension, estrogen deficiency and diabetes mellitus that appear to

29 The impact of estrogen deficiency and osteopenia/osteoporosis on perio

30 Estrogen deficiency and pulmonary diseases are associate

31 clast and osteocyte apoptosis in response to estrogen deficiency and replacement.

32 ouse models of high bone turnover, including estrogen deficiency and sustained exposure to parathyroi

33 Ca absorption and vitamin D action in aging, estrogen deficiency, and adaptation to a low Ca diet.

34 serum cholesterol, and adipose tissues after estrogen deficiency, and show that the benzothiophene ra

35 asts resorb could also be enhanced following estrogen deficiency, and since sex steroids regulate apo

36 and increased marrow adiposity during aging, estrogen deficiency, and skeletal unloading.

37 kappaB and a cytokine strongly implicated in estrogen-deficiency bone loss, was suppressed in osteocl

38 ators of increased osteoclastogenesis during estrogen deficiency, but increases in RANKL, the final e

39 tion contributes to the bone loss induced by estrogen deficiency, but it is presently unknown whether

40 is instrumental for the bone loss induced by estrogen deficiency, but the responsible mechanism is un

41 This study examined the effects of estrogen deficiency by ovariectomy (OVX) and 17beta-estr

42 These observations strongly suggest that estrogen deficiency causes bone loss by lowering thiol a

43 ss has been made in our understanding of how estrogen deficiency causes bone loss, the mechanisms inv

44 Estrogen deficiency decreased glomerular estrogen recept

45 Caffeine and/or estrogen deficiency decreased TBA in the unligated teeth

46 Estrogen deficiency expands hemopoietic stem and progeni

47 r caffeine administration for 65 days and/or estrogen deficiency for 51 days.

48 f their study support previous findings that estrogen deficiency has a dramatic effect on bone homeos

49 r 1 y potently attenuated BMD loss caused by estrogen deficiency, improved bone turnover, promoted a

50 iscerning the specific roles of androgen and estrogen deficiency in bone loss.

51 however, the specific roles of androgen and estrogen deficiency in hypogonadal bone loss are unclear

52 Estrogen deficiency in menopause is a major cause of ost

53 ase in bone resorption observed in states of estrogen deficiency in mice is mainly caused by lack of

54 Female gender in aged mice and estrogen deficiency in middle-aged mice appears to incre

55 Duration of estrogen deficiency in postmenopausal state confers fibr

56 Whether duration of estrogen deficiency in postmenopausal state dictates an

57 We hypothesize that estrogen deficiency in the brains of women with AD may b

58 the assay is presented in which the role of estrogen deficiency in the development and progression o

59 ial mediators of the bone-wasting effects of estrogen deficiency in vivo.

60 The fact that IGF-1 may compensate for estrogen deficiency in wound repair, and potentially oth

61 Female age-related estrogen deficiency increases the risk of osteoporosis,

62 unity to examine the influence of endogenous estrogen deficiency, indicated by age at menopause, on r

63 Estrogen deficiency induced bone loss is associated with

64 various cytokines contribute majorly to the estrogen deficiency-induced bone loss.

65 e formation and bone resorption in mediating estrogen deficiency-induced cancellous bone loss was inv

66 Estrogen deficiency induces bone loss by upregulating os

67 ow T cell number is a key mechanism by which estrogen deficiency induces bone loss in vivo.

68 ells to be central to the mechanism by which estrogen deficiency induces bone loss, but the mechanism

69 Under conditions of estrogen deficiency, infection, and inflammation, the ne

70 re disproportionally susceptible, suggesting estrogen deficiency is a significant risk factor for chr

71 Estrogen deficiency is known to play a critical role in

72 Estrogen deficiency is the dominant pathogenic factor fo

73 The bone wasting induced by estrogen deficiency is, in part, a consequence of increa

74 Age-related estrogen deficiency leads to accelerated bone resorption

75 rly menopause appear at risk for worse ARMD, estrogen deficiency may also contribute to the onset or

76 Estrogen deficiency may contribute to the development an

77 Estrogen deficiency may increase susceptibility to forma

78 Elevated iron stores, oxidative stress, and estrogen deficiency may place postmenopausal women at gr

79 sed in postmenopausal women, suggesting that estrogen deficiency may play a role in the accumulation

80 Estrogen deficiency may predispose to dysregulated synth

81 our current understanding of the process of estrogen deficiency-mediated bone destruction and explor

82 the underlying cause of glucocorticoid- and estrogen deficiency-mediated osteoporosis, and at least

83 are valid models for study of the effects of estrogen deficiency on bone metabolism.

84 s to evaluate the effects of caffeine and/or estrogen deficiency on ligature-induced bone loss (BL),

85 logical function in RPE through ERs and that estrogen deficiency or excess may cause dysregulation of

86 ses in lean mass, muscle size, and strength; estrogen deficiency primarily accounted for increases in

87 Thus, prolonged estrogen deficiency promotes GS and renders MCs insensit

88 ion-unfavorable condition created by gonadal estrogen deficiency reduced the level of differential ex

89 The Ovx rat is a useful model of estrogen deficiency, replicating many aspects of post-me

90 a novel Egr-1/Sp-1 complex in stromal cells, estrogen deficiency results in enhanced levels of free S

91 Because estrogen deficiency results in significantly elevated al

92 Estrogens seem to retard whereas estrogen deficiency seems to accelerate progressive glom

93 Changes in alveolar BMD precede estrogen deficiency, suggesting that early signs of redu

94 ss around the time of menopause is driven by estrogen deficiency, the roles of estrogens and androgen

95 thogenesis of osteoporosis is the ability of estrogen deficiency to increase osteoclast formation by

96 bone matrix during osteoclastic resorption, estrogen deficiency unleashes signals that can stimulate

97 to simulate metabolic bone diseases such as estrogen deficiency, vitamin D deficiency, senescence an

98 The finding that estrogen deficiency was associated with increased produc